acute gastroenteritis is usually caused by bacteria and protozoan
TRANSCRIPT
Acute gastroenteritis is usually caused by bacteria and protozoan. In the Philippines, one of the mostcommon causes of acute gastroenteritis is E. histolytica. The pathologic process starts with ingestion of fecallycontaminated food and water. The organism affects the body through direct invasion and by endotoxin beingreleased by the organism. Through these two processes the bowel mucosal lining is stimulated and destroyed theeventually lead to attempted defecation or tenesmus as the body tries to get rid of the foreign organism in thestomach.
The client with acute gastroenteritis may also report excessive gas formation that may leads toabdominal distention and passing of flatus due to digestive and absorptive malfunction in the system. Feeling offullness and the increase motility of the gastrointestinal tract may progress to nausea and vomiting andincreasing frequency of defecation. Abdominal pain and feeling of fullness maybe relieved only when thepatient is able to pass a flatus.
As the destruction of the bowel continues the mucosal lining erodes due to toxin, direct invasion of theorganism and the action of the hydrochloric acid of the stomach. As the protective coating of the stomach erodesthe digestive capabilities of the acid helps in destroying the stomach lining. Pain or tenderness of the abdomenis then felt by the patient. When the burrows or ulceration reaches the blood vessels in the stomach bleedingwill be induced. Dysentery may be characterized by melena or hematochezia depending on the site and quantityof bleeding that may ensue. Signs of bleeding may be observed also through hematemesis.
As the bowel is stimulated by the organism and its toxin, the intestinal tract secretes water andelectrolytes in the intestinal lumen. The body secretes and therefore lost Chloride and bicarbonate ions in thebowel as the body try to get rid of the organism by increasing peristalsis and number of defecation. Sodium andwater reabsorption in the bowel is inhibited with the lost of the two electrolytes.
Mild diarrhea is characterized by 2-3 stool, borborygmi (hyperactive bowel sound),fluid and electrolyte imbalance and hypernatremia. When the condition continue to progress, protein in the body is excreted to the lumen that further decreases the reabsorption and the body become overwhelmed that leads to intense diarrhea with more than 10 watery stool. Serious fluid volume deficit may lead to hypovolemic shock and eventually death.
Gastroenteritis Overview
If a human adult’s digestive tract were stretched out, it would be 6 to 9 m (20 to 30 ft)
long. In humans, digestion begins in the mouth, where both mechanical and chemical digestion
occur. The mouth quickly converts food into a soft, moist mass. The muscular tongue pushes the
food against the teeth, which cut, chop, and grind the food. Glands in the cheek linings secrete
mucus, which lubricates the food, making it easier to chew and swallow. Three pairs of glands
empty saliva into the mouth through ducts to moisten the food. Saliva contains the enzyme
ptyalin, which begins to hydrolyze (break down) starch—a carbohydrate manufactured by green
plants.Once food has been reduced to a soft mass, it is ready to be swallowed. The
tongue pushes this mass—called a bolus—to the back of the mouth and into the pharynx. This
cavity between the mouth and windpipe serves as a passageway both for food on its way down
the alimentary canal and for air passing into the windpipe. The epiglottis, a flap of cartilage,
covers the trachea (windpipe) when a person swallows. This action of the epiglottis prevents
choking by directing food from the windpipe and toward the stomach.
MouthThe mouth plays a role in digestion, speech, and breathing. Digestion begins
when food enters the mouth. Teeth break down food and the muscular tongue pushes food back
toward the pharynx, or throat. Three salivary glands—the sublingual gland, the submandibular
gland, and the parotid gland—secrete enzymes that partially digest food into a soft, moist, round
lump. Muscles in the pharynx swallow the food, pushing it into the esophagus, a muscular tube
that passes food into the stomach. The epiglottis prevents food from entering the trachea, or
windpipe, during swallowing.
Esophagus
The presence of food in the pharynx stimulates swallowing, which squeezes the food into
the esophagus. The esophagus, a muscular tube about 25 cm (10 in) long, passes behind the
trachea and heart and penetrates the diaphragm (muscular
Gastroenteritis is a condition that causes irritation and inflammation of the stomach and intestines (the gastrointestinal tract). An infection may be caused by bacteria or parasites in spoiled food or unclean water. Some foods may irritate your stomach and cause gastroenteritis. Lactose intolerance to dairy products is one example.
Many people who experience the vomiting and diarrhea that develop from these types of infections or irritations think they have "food poisoning," which they may, or call it "stomach flu," although influenza has nothing to do with it.
Travelers to foreign countries may experience "traveler's diarrhea" from contaminated food and unclean water.
The severity of infectious gastroenteritis depends on your immune system’s ability to resist the infection. Electrolytes (these include essential elements of sodium and potassium) may be lost as you vomit and experience diarrhea.
Most people recover easily from a short bout with vomiting and diarrhea by drinking fluids and easing back into a normal diet. But for others, such as babies and the elderly, loss of bodily fluid with gastroenteritis can cause dehydration, which is a life-threatening illness unless the condition is treated and fluids restored.
This Concept Map, created with IHMC CmapTools, has information related to: Acute
Gastroenteritis (lower GI bleed), Decreased coronary blood flow: angina . Myocardial infarction: pulmonary edema, Accumulation of blood in GI tract . Digestion of blood proteins, Compensatory constriction of peripheral arteries . Compensatory failure, Compensatory constriction of peripheral arteries . Metabolic acidosis, Mesenteric insufficiency: abdominal pain . Liver necrosis, Lactic acidosis . Anoxia, Mesenteric insufficiency: abdominal pain . Bowel infarction, Nubain . Mesenteric insufficiency: abdominal pain, Metabolic acidosis . Decreased blood flow to GI structures, Blood volume depletion . Decreased systolic blood pressure, Anoxia . Decreased blood flow to brain anxiety, confusion, stupor, coma, Decreased systolic blood pressure , Compensatory constriction of peripheral arteries, D5 1/2 NS . Blood volume depletion, Risk for constipation/diarrhea r/t inadequate diet/fluid intake . Increased peristalsis and diarrhea, Metabolic acidosis . Lactic acidosis, Acute Gastroenteritis Lower GI Bleed . Blood volume depletion, Decreased blood flow to skin: pallor . Decreased blood flow to kidneys: decreased urine output, Increased: MCV, MCH, and Lymphocytes . Blood volume depletion, Acute Gastroenteritis Lower GI Bleed . Accumulation of blood in GI tract, Risk for decreased cardiac output r/t decreased blood flow to heart muscle . Decreased cardiac output
Gastroenteritis (also known as gastric flu or stomach flu, although unrelated to influenza) is inflammation of the gastrointestinal tract, involving both the stomach and the small intestine and resulting in acute diarrhea. It can be transferred by contact with contaminated food and water. The inflammation is caused most often[citation needed] by an infection from certain viruses or less
often[citation needed] by bacteria, their toxins, parasites, or an adverse reaction to something in the diet or medication. Current death rates have come down significantly to approximately 1.5 million deaths annually in the year 2000, largely due to the global introduction of oral rehydration therapy [1] and is a leading cause of death among infants and children under 5.[2]
At least 50% of cases of gastroenteritis due to foodborne illness are caused by norovirus.[3] Another 20% of cases, and the majority of severe cases in children, are due to rotavirus. Other significant viral agents include adenovirus [4] and astrovirus.
Different species of bacteria can cause gastroenteritis, including Salmonella, Shigella, Staphylococcus, Campylobacter jejuni, Clostridium, Escherichia coli, Yersinia, Vibrio cholerae, and others. Some sources of the infection are improperly prepared food, reheated meat dishes, seafood, dairy, and bakery products. Each organism causes slightly different symptoms but all result in diarrhea. Colitis, inflammation of the large intestine, may also be present.
Risk factors include consumption of improperly prepared foods or contaminated water and travel or residence in areas of poor sanitation. It is also common for river swimmers to become infected during times of rain as a result of contaminated runoff water.[5]
Gastroenteritis Overview
Gastroenteritis is a condition that causes irritation and inflammation of the stomach and intestines (the gastrointestinal tract). An infection may be caused by bacteria or parasites in spoiled food or unclean water. Some foods may irritate your stomach and cause gastroenteritis. Lactose intolerance to dairy products is one example.
Many people who experience the vomiting and diarrhea that develop from these types of infections or irritations think they have "food poisoning," which they may, or call it "stomach flu," although influenza has nothing to do with it.
Travelers to foreign countries may experience "traveler's diarrhea" from contaminated food and unclean water.
The severity of infectious gastroenteritis depends on your immune system’s ability to resist the infection. Electrolytes (these include essential elements of sodium and potassium) may be lost as you vomit and experience diarrhea.
Most people recover easily from a short bout with vomiting and diarrhea by drinking fluids and easing back into a normal diet. But for others, such as babies and the elderly, loss of bodily fluid with gastroenteritis can cause dehydration, which is a life-threatening illness unless the condition is treated and fluids restored.
V. ANATOMY AND PHYSIOLOGY
Anatomy of the Digestive System
If a human adult’s digestive tract were stretched out, it would be 6 to 9 m (20 to 30 ft) long.
In humans, digestion begins in the mouth, where both mechanical and chemical digestion occur. The
mouth quickly converts food into a soft, moist
mass. The muscular tongue pushes the food against the teeth, which cut, chop, and grind the food.
Glands in the cheek linings secrete mucus, which lubricates the food, making it easier to chew and
swallow. Three pairs of glands empty saliva into the mouth through ducts to moisten the food. Saliva
contains the enzyme ptyalin, which begins to hydrolyze (break down) starch—a carbohydrate
manufactured by green plants.Once food has been reduced to a soft mass, it is ready to be swallowed.
The
tongue pushes this mass—called a bolus—to the back of the mouth and into the pharynx. This cavity
between the mouth and windpipe serves as a passageway both for food on its way down the
alimentary canal and for air passing into the windpipe. The epiglottis, a flap of cartilage, covers the
trachea (windpipe) when a person swallows. This action of the epiglottis prevents choking by
directing food from the windpipe and toward the stomach.
MouthThe mouth plays a role in digestion, speech, and breathing. Digestion begins
when food enters the mouth. Teeth break down food and the muscular tongue pushes food back
toward the pharynx, or throat. Three salivary glands—the sublingual gland, the submandibular gland,
and the parotid gland—secrete enzymes that partially digest food into a soft, moist, round lump.
Muscles in the pharynx swallow the food, pushing it into the esophagus, a muscular tube that passes
food into the stomach. The epiglottis prevents food from entering the trachea, or windpipe, during
swallowing.
Esophagus
The presence of food in the pharynx stimulates swallowing, which squeezes the food into the
esophagus. The esophagus, a muscular tube about 25 cm (10 in) long, passes behind the trachea and
heart and penetrates the diaphragm (muscular
wall between the chest and abdomen) before reaching the stomach. Food advances through the
alimentary canal by means of rhythmic muscle contractions (tightenings) known as peristalsis. The
process begins when circular muscles in the esophagus wall contract and relax (widen) one after the
other, squeezing food downward toward the stomach. Food travels the length of the esophagus in two
to three seconds.
A circular muscle called the esophageal sphincter separates the esophagus and the stomach.
As food is swallowed, this muscle relaxes, forming an opening through which the food can pass into
the stomach. Then the muscle contracts, closing the opening to prevent food from moving back into
the esophagus. The esophageal sphincter is the first of several such muscles along the alimentary
canal. These muscles act as valves to regulate the passage of food and keep it from moving
backward.
Stomach
The stomach, located in the upper abdomen just below the diaphragm, is a saclike structure
with strong, muscular walls. The stomach can expand significantly to store all the food from a meal
for both mechanical and chemical processing. The stomach contracts about three times per minute,
churning the food and mixing it with gastric juice. This fluid, secreted by thousands of gastric glands
in the lining of the stomach, consists of water, hydrochloric acid, an enzyme called pepsin, and
mucin (the main component of mucus). Hydrochloric acid creates the acidic
environment that pepsin needs to begin breaking down proteins. It also kills microorganisms that
may have been ingested in the food. Mucin coats the stomach, protecting it from the effects of the
acid and pepsin. About four hours or less after a meal, food processed by the stomach, called chyme,
begins passing a little at a time through the pyloric sphincter into the duodenum, the first portion of
the small intestine.
Liver
The liver is the largest internal organ in the human body, located at the top of the abdomen on
the right side of the body. A dark red organ with a spongy texture, the liver is divided into right and
left lobes by the falciform ligament. The liver performs more than 500 functions, including the
production of a digestive liquid called bile that plays a role in the breakdown of fats in food. Bile
from the liver passes through the hepatic duct into the gallbladder, where it is stored. During
digestion bile passes from the gallbladder through bile ducts to the small intestine, where it breaks
down fatty food so that it can be absorbed into the body. Nutrient- rich blood passes from the small
intestine to the liver, where nutrients are further processed and stored. Deoxygenated blood leaves
the liver via the hepatic vein to return to the heart.
Small Intestine
Most digestion, as well as absorption of digested food, occurs in the small intestine. This
narrow, twisting tube, about 2.5 cm (1 in) in diameter, fills most of the lower abdomen, extending
about 6 m (20 ft) in length. Over a period of three to six hours, peristalsis moves chyme through the
duodenum into the next portion of the small intestine, the jejunum, and finally into the ileum, the last
section of the small intestine. During this time, the liver secretes bile into the small intestine through
the bile duct. Bile breaks large fat globules into small droplets, which enzymes in the small intestine
can act upon. Pancreatic juice, secreted by the pancreas, enters the small intestine through the
pancreatic duct. Pancreatic juice contains enzymes that break down sugars and starches into simple
sugars, fats into fatty acids and glycerol, and proteins into amino acids. Glands in the intestinal walls
secrete additional enzymes that break down starches and complex sugars into nutrients that the
intestine absorbs. Structures called Brunner’s glands secrete mucus to protect the intestinal walls
from the acid effects of digestive juices.
The small intestine’s capacity for absorption is increased by millions of fingerlike
projections called villi, which line the inner walls of the small intestine. Each villus is about 0.5 to
1.5 mm (0.02 to 0.06 in) long and covered with a single layer of cells. Even tinier fingerlike
projections called microvilli cover the cell surfaces. This combination of villi and microvilli
increases the surface area of the small intestine’s lining by about 150 times, multiplying its capacity
for absorption. Beneath the villi’s single layer of cells arecapillaries (tiny vessels) of the bloodstream
and the lymphatic system. These capillaries allow nutrients produced by digestion to travel to the
cells of the body. Simple sugars and amino acids pass through the capillaries to enter the
bloodstream. Fatty acids and glycerol pass through to the lymphatic system.
Large Intestine
A watery residue of indigestible food and digestive juices remains unabsorbed. This residue
leaves the ileum of the small intestine and moves by peristalsis into the large intestine, where it
spends 12 to 24 hours. The large intestine forms an inverted U over the coils of the small intestine. It
starts on the lower right-hand side of the body and ends on the lower left-hand side. The large
intestine is 1.5 to 1.8 m (5 to 6 ft) long and about 6 cm (2.5 in) in diameter.
The large intestine serves several important functions. It absorbs water— about 6 liters (1.6
gallons) daily—as well as dissolved salts from the residue passed on by the small intestine. In
addition, bacteria in the large intestine promote the breakdown of undigested materials and make
several vitamins, notably vitamin K, which the body needs for blood clotting. The large intestine
moves its remaining contents toward the rectum, which makes up the final 15 to 20 cm (6 to 8 in) of
the alimentary canal. The rectum stores the feces—waste material that consists largely of undigested
food, digestive juices, bacteria, and mucus—until elimination. Then, muscle contractions in the walls
of the rectum push the feces toward the anus. When sphincters between the rectum and anus relax,
the feces pass out of the bod