alcohol and aggression: a meta-analysis on the moderating effects of inhibitory cues ... ·...

Psychological Bulletin1996. Vol. 120. No. 1.60-82

Copyright 19% by the American Psychological Association, Inc.0033-2909/96/S3.00

Alcohol and Aggression: A Meta-Analysis on the Moderating Effects ofInhibitory Cues, Triggering Events, and Self-Focused Attention

Tiffany A. Ito, Norman Miller, and Vicki E. PollockUniversity of Southern California

The authors conducted a meta-analysis of 49 studies to investigate 2 explanations of how alcohol

increases aggression by decreasing sensitivity to cues that inhibit it. Both the level of anxiety and

inhibition conflict moderated the difference between the aggressive behavior of sober and intoxicated

participants, but neither level adequately accounted for variation in effect sizes. Additional analyses

of 3 social psychological moderating variables—provocation, frustration, and self-focused atten-

tion—showed that the aggressiveness of intoxicated participants relative to sober ones increased as a

function of frustration but decreased as a function of provocation and self-focused attention. The

authors also examined the moderating effects of dose.

Statistics on the co-occurrence of alcohol and violent crimessuggest a troubling link between alcohol and aggression. Perpe-trators of violent crimes are more likely intoxicated during thecommission of their crime than are perpetrators of nonviolentcrimes (Murdoch, Pihl, & Ross, 1990). Such findings havefueled a large body of research addressing the relationship be-tween intoxication and aggression. Both qualitative (e.g., Gus-tafson, 1993; Moss & Tartar, 1993;Pihl, 1983; Pihl, Peterson, &Lau, 1993; Taylor, 1983; Taylor & Chermack, 1993; Taylor &Leonard, 1983) and quantitative (Bushman & Cooper, 1990;Steele & Southwick, 1985) reviews have quite clearly shownthat intoxication can increase aggression. A theoretical under-standing of this relationship, however, remains unresolved.

In this article, we use meta-analytic procedures to explore therole of situational factors in moderating the relation betweenalcohol and aggression. Theories emphasizing the way in whichattention is allocated to various situational cues are importantin this context (e.g., Taylor & Chermack, 1993; Taylor & Leo-nard, 1983; Zeichner & Pihl, 1979). Two specific variants are a

Tiffany A. Ito and Norman Miller, Department of Psychology, Uni-

versity of Southern California; Vicki E. Pollock, Departments of Psy-

chiatry and Psychology, University of Southern California.

This work was supported by an American Psychological AssociationMinority Fellowship, National Science Foundation Grant SBR-

9319752, National Institute for Alcohol and Alcoholism Grants RO1-

AA08031 and KO2-AAO146. and Alcohol Beverage Foundation Grant

ABMRF.

We thank Shelley Duval and Virginia Duval for theoretical discus-

sions on self-focused attention; John Musciente and Darren Urada for

assistance in locating the studies, computing effect sizes, and serving as

judges; and Brad J. Bushman, Kate B. Carey, Michael J. Cody, MichaelE. Dawson, Mitchell Earleywine, Blair T. Johnson, Sheila T. Murphy,

and Stephen J. Read for helpful comments on drafts. We presented por-

tions of this article at the 1994 annual meeting of the Research Society

on Alcoholism, Maui, Hawaii.Correspondence concerning this article should be addressed to

Tiffany A. Ito, who is now at the Department of Psychology, Ohio State

University, 1885 Neil Avenue, Columbus, Ohio 43210-1222. Electronicmail may be sent via Internet to [email protected].

model based on alcohol's anxioloytic effect and Steele andSouthwick's (1985) inhibition conflict model. Other situation-ally determined variables that may vary within the context of anaggression-eliciting situation are provocation, frustration, andself-focused attention. For decades, researchers have examinedthe relation between alcohol and aggression using paradigmsthat contain either a provoking or frustrating event. Althoughthe roles of provocation and frustration have figured promi-nently in experimental research on aggression per se, interest-ingly, there has been little explicit theoretical concern abouttheir effects on aggression by intoxicated individuals. Similarly,focus of attention historically has been an important variable inthe social psychological literature on aggression, but very littleattention has been paid to understanding how it may affect theaggressive behavior of intoxicated participants. Consequently,in this meta-analysis, we also assess the moderating role of prov-ocation, frustration, and self-focused attention in alcohol-in-duced aggression.

Why Does Alcohol Sometimes Increase Aggression?

The literature on the effects of alcohol on aggression is ex-tremely large, and an examination of all the facets of it is beyondthe scope of a single article. For instance, behavioral changesfollowing alcohol consumption may be directly attributable toits pharmacological effects, but at this time there is no clearunderstanding of the specific mechanism that causes alcohol toincrease aggression. Among the effects of acute and chronic al-cohol administration are altered permeability of cell mem-branes, as well as disruptions of voltage-gated ion channels(Hunt, 1985) and secondary messengers (Hoffman & Tabakoff,1990). Whereas some research has emphasized the roles of tet-rahydroisoquinolines (e.g., Myers, Melchior, & Swartzwelder,1980) and endorphins (e.g., Gianoulakis & Gupta, 1986) as bi-ological mediators of alcohol effects, other research has focusedon alcohol-induced changes in the neurotransmitters dopamineand serotonin as mechanisms that underlie behavioral rein-forcement resulting from alcohol intake (Koob & Bloom,1988). Despite this extensive animal and human research, how-

60

ALCOHOL AND AGGRESSION 61

ever, there is no known direct causal link between specific bio-

logical and behavioral changes induced by alcohol.

In human behavioral research, a large body of survey data

indicates that social drinkers expect alcohol to increase aggres-

sion (Lindman & Lang, 1994; Rohsenow, 1983; Roizen, 1983;

Southwick, Steele, Marlatt, & Lindell, 1981). It is, therefore,

likely that people possess the expectation not only that alcohol

will facilitate aggression but also that such antinormative be-

havior will be met with greater tolerance if it is displayed by an

intoxicated rather than a sober person (Critchlow, 1986; Lang

& Sibrel, 1989; Sobell & Sobell, 1973; but see Gustafson,

1991b, for an alternative outcome). It has, therefore, been sug-

gested that the increased aggression in intoxicated participants

reflects, at least to some degree, their attempt to fulfill this ex-

pectation or role-play the part of the "aggressive drunk"

(Brown, Goldman, Inn, & Anderson, 1980; Lang, 1983; Mac-

Andrew & Edgerton, 1969; Marlatt & Rohsenow, 1980). More-

over, the appropriateness of this social role may be especially

salient to participants in the experiments reviewed in this meta-

analysis because their procedures are likely to prime the alco-

hol-aggression expectation through the display of aggression-

related cues (e.g., shock apparatus and competitive games). Fi-

nally, the very fact that a scientist studies both alcohol and ag-

gression may reinforce these expectancies in participants. Thus,

under an expectancy-driven conceptualization of alcohol-facil-

itated aggression, it is assumed that people possess the expecta-

tion that alcohol can increase aggression and that such beliefs

facilitate aggression following alcohol consumption.

Despite the expectation people possess for alcohol to increase

aggression, substantial unexplained variability characterizes

the relation between alcohol and aggression. For example, al-

though survey data show that people expect alcohol to increase

aggression, this same data show that aggression is not always

expected to follow intoxication. When respondents were asked

to indicate whether alcohol always, usually, or sometimes makes

them aggressive, only 1% reported that they always expected

aggression (Roizen, 1983). In addition, a meta-analytic assess-

ment of the independent contributions of pharmacological and

expectancy effects on intoxicated aggression revealed signifi-

cant heterogeneity among both sets of effect sizes (Hull & Bond,

1986). These findings suggest that situational factors play an

important role in determining whether and to what degree ag-

gression increases following alcohol consumption. Conse-

quently, we discuss five situational factors that we examined in

our meta-analysis. For this purpose, we group them under the

next two main headings.

Cognitive Theories of Intoxicated Aggression

Alcohol has been observed to cause many forms of cognitive

impairments. Intoxication appears to narrow attention

(Huntley, 1973; Moskowitz & DePry, 1968), interfere with

memory processes (Birnbaum, Johnson, Hartley, & Taylor,

1980; Birnbaum & Parker, 1977; Craik, 1977; Jones & Jones,

1977; Rosen & Lee, 1976), and diminish the ability to engage

in abstract thinking (Tartar, Jones, Simpson, & Vega, 1971).

Applying this array of consequences to the explanation of ag-

gressive behavior, several researchers have proposed what we re-

fer to as cognitive theories of alcohol-induced aggression. These

cognitive theories have also been referred to as attentional

hypotheses (Gustafson, 1993) and cognitive theoretical per-

spectives (Taylor & Chermack, 1993). For example, Pernanen

(1976) argued that intoxication decreases the number of cues

to which a person can attend. An intoxicated person may not

correctly perceive the reasons for other people's behavior, mak-

ing the actions of others appear more arbitrary and provocative

than they would to a sober perceiver. As a consequence, Perna-

nen believed intoxicated individuals are more likely to respond

with aggression. Taylor and colleagues (Taylor & Chermack,

1993; Taylor & Leonard, 1983) argued that alcohol-induced

impairments render intoxicated individuals able to attend to

only the most salient and dominant cues, which are typically

those that instigate an aggressive response. According to this

view, the propensity for aggression is increased when alcohol is

consumed because inhibitory cues are less salient to intoxicated

persons. In addition, Zeichner and Pihl (1979) showed that

when an experimental procedure directly manipulated the cor-

relation between the amount of aversive stimulation partici-

pants received with the amount of aggression they had dis-

played, sober participants decreased their aggressive behavior

relative to a condition in which aggression and aversive stimula-

tion were experimentally controlled to be uncorrelated. By con-

trast, intoxicated participants appeared unaffected by the con-

tingency between own aggression and amount of aversive stim-

ulation received, displaying equally high levels of aggression in

both conditions. This led Zeichner and Pihl to suggest that al-

cohol increases aggression by affecting the ability to correctly

perceive the negative consequences of one's aggression.

The assumption that alcohol impairs a person's ability to per-

ceive and respond to aggression inhibiting cues but has less

effect on one's ability to perceive and respond to instigating

stimuli is common to these cognitive theories. However, alcohol

does not uniformly increase aggression. All people do not be-

come aggressive every time they have a drink. Thus, a crucial

question that these theories leave unanswered is when intoxica-

tion increases aggression.

Anxiolysis: Disinhibilion Theories Revisited

Arousal of anxiety is an inherent feature of situations in

which aggression is elicited; therefore, to understand how and

when alcohol increases aggression, it is necessary to understand

the role of anxiety. Specifically, anxiety can be conceptualized

as a warning signal indicating the potential for aversive conse-

quences (Spielberger, 1972). Aggression-eliciting situations are

typically fraught with such potential. For example, social disap-

proval may ensue if others perceive aggression as unreasonable.

In addition, an aggressive action may elicit fear of retaliation.

Functionally, the experience of anxiety serves to suppress be-

haviors that are associated with its arousal, and this could ac-

count for why aggression is not overtly displayed every time it is

instigated.

There has been debate as to whether alcohol reliably de-

creases anxiety-stress. Sayette's (1993) appraisal-disruption

model proposes that the temporal ordering between alcohol

consumption and exposure to anxiety-eliciting cues determines

whether intoxication is anxiolytic. Specifically, Sayette argued

that alcohol produces anxiolysis by disrupting appraisal of the

62 ITO, MILLER, AND POLLOCK

situation as anxiolytic. Therefore, anxiolysis most likely occurswhen alcohol consumption precedes exposure to the anxiety-eliciting cues. For studies in this dataset, the source of anxiety-provoking cues is the experimental task itself (e.g., cues indicat-ing social disapproval of aggression or possible retaliation), towhich participants are not exposed until after they have con-sumed alcohol. Studies in this review, therefore, meet the con-dition set by Sayette for anxiolysis with respect to the temporalordering of alcohol consumption and exposure to anxiety-provoking cues, suggesting that intoxicated participants inthese studies should experience relatively less anxiety than soberones. In summary, we believe that intoxication dampens thearousal of anxiety in the studies in the present dataset and thatthis, in turn, weakens the suppression of aggression (Gray,1987;Pihletal., 1993; Taylor &Chermack, 1993; Washburne,1956). It is, therefore, predicted that alcohol may increase ag-gression in situations in which anxiety would normally bothbe aroused and act to inhibit it. We refer to this model as theanxiolysis-disinhibition model.

The anxiolysis-disinhibition model leads to the hypothesisthat the greatest difference in aggression between sober and in-toxicated individuals occurs when anxiogenic cues are greatest.If anxiogenic cues are weak, then little suppression of aggressionwould be expected in either sober or intoxicated persons. How-ever, as the intensity of anxiogenic cues increases, sober personsshould be more likely to experience anxiety than those who areintoxicated and, hence, be more inclined to suppress aggres-sion. By contrast, intoxicated persons would be expected to be-have in a fashion relatively free from the effects of such suppres-sion. Therefore, a crucial test of this model is whether thedifference between the aggressive behavior of intoxicated andsober persons increases as a function of the degree to which sit-uational factors induce anxiety.

Inhibition Conflict

Alternatively, Steele and colleagues (Steele & Josephs, 1990;Steele & Southwick, 1985) have suggested inhibition conflictas the mechanism by which alcohol increases the likelihood ofaggressive behavior. Inhibition conflict refers to a type of re-sponse conflict that occurs when a behavior is instigated by oneset of strong cues and, simultaneously, is inhibited by anotherset of strong cues. The two primary tenets of the theory arethat (a) alcohol impairs cognitive processing and, in particular,narrows the range of cues to which a person can attend; and (b)as a consequence of this narrowed attentional range, an intoxi-cated person who is confronted with a situation that elicits high-inhibition conflict is able to process and respond only to themost salient behavioral cues. Consider, for example, an individ-ual who experiences a strong provocation to assault another per-son at work. If sober, this person should be able to process notonly the instigating cue of provocation but also other cues thatsignal the normative constraints that make physical aggressioninappropriate in the workplace. By contrast, an intoxicated per-son whose attentional focus has been narrowed by alcohol maylack the cognitive resources to attend adequately to both sets ofcues. Steele and colleagues argued that because instigating cuesare usually more salient and immediate, whereas inhibitingcues often require the retrieval of personal standards of behav-

ior and cognitively demanding estimations of future conse-quences, the intoxicated person typically will be influencedmore by the instigating cue. Thus, an intoxicated person is morelikely to aggress than a sober one.

The experience of inhibition conflict is one manifestation ofwhat Steele has referred to as alcohol myopia—the general nar-rowing of attention following alcohol consumption (Steele &Josephs, 1990). Alcohol myopia may be responsible for a widerange of behaviors, such as drunken self-inflation and decreasesin psychological stress when intoxicated. We focus only on themore specific concept of inhibition conflict because it is thoughtto moderate alcohol's aggression facilitating effects (Steele &Josephs, 1990).

More important, Steele and colleagues (Steele & Josephs,1990; Steele & Southwick, 1985) argued that only an interac-tion between alcohol's pharmacological effects and features ofthe situation that affect inhibition conflict alter the balance ofcues bearing on a response. Hence, not all situations necessarilyincrease aggression among intoxicated individuals. If a situa-tion is low in inhibition conflict—because the instigating cuesare weak, the inhibiting cues are weak, or both sets of cues areweak—then intoxicated and sober persons process cues sim-ilarly and, therefore, behave similarly. According to Steele andJosephs, in a situation low in inhibition conflict, intoxication"would only block inhibiting cues that are already weak orweaken inhibiting cues against a response tendency that wasweak to begin with" (p. 923). That is, the combination of high-inhibition conflict and intoxication is expected to increase ag-gression (p. 925).

Steele and Southwick (1985) meta-analytically assessed thepredictive utility of the inhibition conflict model. They con-firmed that situations likely to elicit high, as compared withlow, levels of inhibition conflict were associated with a largerdifference in the degree to which the extremity of the behaviorof intoxicated participants exceeded that of controls. BecauseSteele and Southwick were interested in the effect of alcohol onsocial behaviors in general, the researchers of the 34 studies intheir dataset investigated a wide range of behaviors (e.g., gam-bling and eating) not solely aggression. In the present analysis,we specifically assessed the effects of inhibition conflict on ag-gression. Whereas our sample of studies was culled from 49 re-ports that assessed aggressive behaviors under intoxication,fewer than half of the 34 studies reviewed by Steele and South-wick were concerned with aggression.

Both the anxiolysis-disinhibition and inhibition conflictmodels assume that responsiveness to inhibiting cues is affectedby intoxication, but the explanations they offer for this differsomewhat. The anxiolysis-disinhibition model is concernedwith the specific inhibiting cue of anxiety, whereas the inhibi-tion conflict model does not specify any one source of inhibitingcues. Moreover, the inhibition conflict model focuses on the rel-ative strength between inhibiting and instigating cues. There-fore, each model may provide specific insight into how aggres-sion is increased when alcohol is consumed.

Social Psychological Moderators of Aggressive Behavior

We next address whether other social psychological vari-ables—namely, provocation, frustration, and self-focused at-


tention—which have been previously shown to moderate ag-

gressive behavior in sober individuals, similarly affect intoxi-

cated participants.

Provocation

A provoking stimulus can be very effective in increasing ag-

gression for several reasons. By thwarting or angering a person,

it may directly elicit negative affect and angry or emotional ag-

gression (Averill, 1982; Berkowitz, 1989; Feshbach, 1964). Al-

ternatively, or in parallel with this emotional responding, cogni-

tive factors such as the belief in "an eye for an eye" may lead

people to respond in a tit-for-tat fashion to provocation

(Axelrod, 1984;Gouldner, 1960). In fact, actually experiencing

physical discomfort as a result of an attack may not even be

required. Merely knowing that someone intended to attack can

be sufficient to induce aggression (Greenwell & Dengerink,

1973).

Another way in which provocation can increase aggression is

by serving as a suitable external justification for a behavior that

normally is considered inappropriate. Concern about violating

such normative proscriptions may be associated with anxiety,

the awareness of which serves to inhibit aggressive behavior.

Consequently, people usually behave in a nonaggressive man-

ner, unless a suitable external justification for aggression can be

found. Thus, attack or provocation by another person can serve

as a triggering event that provides justification for aggressive re-

taliation. Because the norm of reciprocity sanctions aggression

in retaliation to another's attack, provocation frees the individ-

ual to aggress. Provocation can, therefore, serve the dual roles

of instigation and excuse for aggression.

The effect of provocation on aggression has long been inves-

tigated in sober participants; although it also frequently appears

as a central feature of experimental paradigms in research on

aggression and alcohol (e.g., Kelly, Cherek, Steinberg, & Rob-

inson, 1988; Richardson, 1981; Shuntich & Taylor, 1972), rel-

atively little attention has been paid to its theoretical impor-

tance in this latter literature. Pernanen (1976) and Gustafson

(1993) have both emphasized that level of provocation should

be considered in interpreting findings in the alcohol and aggres-

sion literature. In fact, in his analysis of the role of provocation,

Gustafson suggested that alcohol facilitates aggression only

when provocation is present and that, in the absence of provo-

cation, sober and intoxicated persons will behave similarly. Ac-

cording to Gustafson, then, absence of provoking stimuli may

serve as a boundary condition on alcohol's aggression facilitat-

ing effects.

Inherent in Gustafson's (1993) view on the importance of

provocation in alcohol-related aggression is the assumption that

intoxicated individuals inhibit their aggression in the absence

of provocation in the same manner as sober participants. Spe-

cifically, it is assumed that intoxicated individuals will aggress

only if a triggering provocation is experienced. Gustafson based

this conclusion on a somewhat informal review of results and,

unfortunately, provides no theoretical explanation for his con-

clusion. Moreover, this prediction is not consistent with evi-

dence showing that alcohol increases aggression among intoxi-

cated participants, even in low-provocation conditions (e.g.,

Gustafson, 1985a, 1986a; Taylor & Gammon, 1976; Taylor &

Sears, 1988).

Extant research, therefore, supports a prediction quite

different from that of Gustafson (1993). Moreover, theoretical

analyses suggest instead that the difference between the aggres-

sive behavior of intoxicated and sober persons should be great-

est when provocations are absent. Under conditions of low prov-

ocation, sober participants are likely to observe normative con-

straints and behave relatively nonaggressively. By contrast,

intoxicated participants seem less responsive to these normative

constraints (perhaps because violating these norms is not asso-

ciated with anxiety) and instead tend to display more aggression

than sober participants, even in circumstances under which

controls do not experience provocation.

A similar theoretical argument was used by Bettencourt and

Miller (1996) to account for the decrease in gender differences

in aggression under provocation. Their meta-analysis shows

that men are consistently more aggressive than woman when

provocation is absent, but this gender difference diminishes

when provocation is introduced. At only moderate levels of

provocation, the strong gender difference found in its absence is

no longer seen. Although social roles dictate that aggression by

women is more unacceptable than that by men, provocation

apparently frees women from this social role proscription by

providing an external justification, thereby enabling them to

display levels of aggression more comparable with that of men.

Consequently, we hypothesize that the aggressive behavior of

intoxicated individuals will exceed that of sober ones under con-

ditions of low provocation. When provoked, sober participants

will behave more aggressively than when unprovoked. Intoxi-

cated participants may also respond to provocation with in-

creased aggression; but, because they are already inclined to

behave in a relatively aggressive manner, the additional instiga-

tion and justification provided by provocation should not in-

crease their aggression as much as it does for sober participants

(i.e., a type of ceiling effect). Thus, the difference between the

aggressive behavior of intoxicated and sober individuals seems

likely to diminish as provocation increases.

Frustration

Just as provoking events are a frequent feature of paradigms

investigating the effects of intoxication on aggression, these par-

adigms also frequently include frustrating events. The role of

frustration in aggression also has a long history in the social

psychological literature, stimulated by the publication of the

frustration-aggression hypothesis (Dollard, Doob, Miller,

Mowrer, & Sears, 1939). Although much of this research has

been dedicated to refuting the assertion that aggression is an

invariant consequence of frustration, this body of evidence,

nevertheless, persuasively indicates that frustration can insti-

gate aggression (for a review, see Berkowitz, 1989). Frustration,

defined as blocking an ongoing goal-directed behavior, may op-

erate in a manner similar to provocation and serve both as an

instigator and an external justification for violating normative

constraints against aggression. Consequently, we predict thatfrustration will be related to alcohol-potentiated aggression in

the same manner as provocation. Specifically, we expect that the

aggressive behavior of intoxicated participants will exceed that

of sober ones when frustrations are minimal, but as frustration

increases and the aggressive behavior of sober participants in-

64 ITO. MILLER, AND POLLOCK

creases correspondingly, intoxicated and sober participants will

behave more similarly.

In his statement on the role of provocation in alcohol-facili-

tated aggression, Gustafson (1993) did not distinguish between

provocation and frustration. Rather, he considered frustration

to be a form of provocation (p. 23). Even though we predict

that provocation and frustration will have similar directions of

influence on the difference in aggression between sober and in-

toxicated participants, we nevertheless believe that it is theoret-

ically important to distinguish between them and to assess their

influences separately. Hence, we constrain the use of the term

provocation to a negative affect that arises as a direct result of

being attacked, or perceiving attack, by another person. In keep-

ing with most prior discussion, we use the term frustration to

refer to the blocking of ongoing goal-directed behaviors.1 In the

research on aggression, frustration is often situationally or task

induced (Carlson & Miller, 1988).

Self-Focused A Mention

Whatever their differences, the anxiolysis-disinhibition and

inhibition conflict models both predict that intoxication typi-

cally is associated with decreased responsiveness to inhibiting

cues and subsequent increases in aggression. Inducing self-

focused attention, however, may increase attention to inhibiting

cues. Self-focus refers to a state in which a self-regulatory pro-

cess is initiated, personal standards of appropriate behavior be-

come salient, and attempts are made to comply with these stan-

dards (Carver, 1979; Carver & Scheier, 1981, 1990; Duval &

Wicklund, 1972). When salient, these standards inhibit impul-

sive, self-indulgent behavior. Self-focused attention in sober par-

ticipants is associated with increased prosocial behavior

(Duval, Duval, & Neely, 1979; Gibbons & Wicklund, 1982; M.

Rogers, Miller, Mayer, & Duval, 1982; also shown meta-analyt-

ically by Carlson, Charlin, & Miller, 1988; and Carlson & Miller,

1987) and less antisocial behavior such as aggression (Carver,

1975; Scheier, Fenigstein, & Buss, 1974) and cheating (Diener

& Wallbom, 1976). Similarly, deindividuation, which is char-

acterized by a loss of individual identity and a subsequent lack

of concern with personal standards of behavior (Diener, 1980),

has been associated with increased aggression (Lightdale &

Prentice, 1994; Prentice-Dunn & Rogers, 1980; R. W. Rogers &

Prentice-Dunn, 1981).

It is unclear, however, whether the self-regulatory processes

associated with self-focused attention operate in the same man-

ner in intoxicated as in sober individuals. In particular, Hull

(1981; Hull, Levenson, Young, & Sher, 1983; see also Wash-

burne, 1956) argued that alcohol interferes with the likelihood

of focusing attention on self by inhibiting the encoding of self-

relevant information. In support of this view, when asked to

make a speech about what they like and dislike about their bod-

ies, intoxicated participants used fewer self-relevant pronouns

than those sober (Hull etal., 1983). Hull argued that a desire to

decrease self-focused attention, and thereby avoid both assess-

ments of how well one is meeting behavioral standards and the

ensuing guilt associated with failure to meet them, may moti-

vate people to drink (Hull & Young, 1983a, 1983b; Hull, Young,

& Jouriles, 1986). Heatherton and Baumeister (1991 ) advo-

cated a similar position.

Despite these findings, Hull and Reilly (1983) suggested that

self-focusing manipulations can counteract the intoxication-

induced tendency to avoid the focus on self. This position is

consistent with data obtained by Bailey, Leonard, Cranston,

and Taylor (1983), who increased self-focus in half of their par-

ticipants by placing a mirror and video camera in the experi-

mental room (common manipulations used to induce self-

focus ), telling them their session would be videotaped and mak-

ing a point of calling them by their names. Self-focused partici-

pants, both in the placebo (nonalcoholic beverage) and alcohol

conditions, chose to administer a lower intensity of shocks to

their competitive opponent than non-self-focused participants.

The effects of self-focusing manipulations were also investi-

gated by Ross and Pihl (1988), who assessed the performance

of intoxicated and sober participants on a complex reaction

time task in which behavioral regulation presumably would

lead participants to strive for better performance. Both sober

and intoxicated participants who performed the task in front of

mirrors and a video camera responded faster and made fewer

errors than those who did not receive the self-focusing manipu-

lation. Self-focus also appeared to interact with alcohol in-

gestion such that performance was most improved for intoxi-

cated self-focused participants.

Ross and Pihl (1988) speculated that their self-focused intox-

icated participants, who knew they had received alcohol, were

attempting to compensate for its deleterious effects. Self-focus

presumably made salient to them the behavioral standard of not

appearing drunk and impaired and, thereby, motivated them to

exhibit superior performance. If this explanation is correct,

then it suggests that the intoxicated participants overcompen-

sated for the effects of intoxication in that their absolute perfor-

mance levels exceeded that of sober participants. An alternative

interpretation is that the narrowed attentional focus accompa-

nying intoxication served to intensify their self-focused state by

decreasing sensitivity to other stimuli that had the potential to

distract attention from self. Sober participants may be relatively

less affected by self-focusing cues because their wider range of

attentional focus allows them to attend to other stimuli that or-

dinarily compete with self for attention.2

These prior outcomes suggest alternative hypotheses about

the relationship of self-focus and aggressive behavior after alco-

hol consumption. According to Hull (1981), intoxicated par-

ticipants are less likely to be affected by situational factors that

ordinarily encourage self-focused attention in sober partici-

pants because alcohol interferes with the encoding of self-rele-

1 We note that the dimension of human agency may also be an im-portant factor in aggression-inducing events. Meta-analytic evidence

suggests that interpersonal sources of instigating events induced twicethe magnitude of aggression (relative to control conditions) as that pro-duced by nonpersonal sources, even when intensity of the negative eventwas controlled (Carlson & Miller, 1998). We note also that the defini-

tion of aggression-eliciting events excludes a class of negative events,namely, nonhuman provocations that do not necessarily interfere witha goal-directed activity (e.g., extremes in temperature). Although thiscategory logically exists, and is represented in the larger literature onaggression, it was not present among the studies in this dataset. We,therefore, do not consider them in our analysis.

2 We thank Blair Johnson for suggesting this line of thought.


vant information. This view leads to the prediction that when

compared with the aggressive behavior of sober participants,

intoxicated ones will be relatively unaffected by level of self-

focus. The difference between the aggressive behavior of intoxi-

cated and sober participants should, therefore, increase as situ-

ational factors encouraging self-focused attention increase. This

may be especially likely when subtle self-focusing cues are pres-

ent. Alternatively, the data collected by Bailey et al. (1983) and

Ross and Pihl (1988) suggest that self-focused attention de-

creases aggression in intoxicated participants as well as sober

ones. Moreover, in Ross and Pihl, the performance of intoxi-

cated participants was more strongly affected by self-focusing

manipulations than that of sober participants, suggesting that

although aggressive behavior may be less likely when attention

is focused on self, it may be even more unlikely among intoxi-

cated self-focused persons.

Method

Literature Searches

We used three methods to locate studies for the meta-analysis. First,Psyclnfo (1967-1994), Medline (1966-1994), and Current Contents

(1989-1994) databases were searched through May 1994 using the key-words alcohol, intoxication, and ethanol, cross-referenced with aggres-

sion, antisocial behavior, anger, and attack. Second, we reviewed the ref-erence lists of three previous meta-analyses on the effects of alcohol on

human behavior(Bushman&Cooper, 1990; Hull & Bond, 1986;Steele& Southwick, 1985). Third, we examined relevant journals (e.g., Quar-

terly Journal of Studies on Alcohol) published in June, July, and August1994.

Criteria for Inclusion

We included studies in the meta-analysis if they fulfilled four criteria.First, studies had to be published in a journal. Second, they had to in-clude at least one measure of aggression. Third, data necessary for com-

puting an effect size had to be included in the published report. Finally,studies had to compare at least one experimental condition in whichalcohol was consumed with a condition in which it was not. This com-parison condition could be a placebo condition (participants expect toconsume alcohol but actually receive a nonalcoholic beverage), a con-

trol condition (participants both expect and receive a nonalcoholicbeverage), or a condition in which no drink was consumed. Although itcould be informative to analyze effect sizes separately as a function of

the type of control group used, two considerations argue against doingso. First, such an analysis would have low power because few studiescompared an alcohol condition with one in which no drink was con-

sumed. Second, we found type of control group was confounded withother potentially important variables such as dose of alcohol. For thesereasons, we collapsed across type of control group (cf. Bushman & Coo-per, 1990; and Steele & Southwick, 1985), which is conservative be-

cause variation among control groups decreases the likelihood of detect-ing significant effects, relative to analyses in which control groups arehomogeneous.

A total of 49 studies satisfied the inclusion criteria (and are indicatedin the reference section by an asterisk). Dates of publication rangedfrom April 1972 to August 1994. One article (Rohsenow & Bachorow-ski, 1984) contained three independent experiments, each satisfied theinclusion criteria. Of the 47 articles from which the 49 studies wereextracted, 22 were included in Bushman and Cooper's (1990) meta-analysis, 13 were included in Steele and Southwick's (1985) meta-anal-ysis, and 3 were included in Hull and Bond's (1986) meta-analysis (see

Appendix A for the list). These figures represent an overlap of 8 studiesbetween Bushman and Cooper and Steele and Southwick and 1 study

between Steele and Southwick and Hull and Bond. Eighteen studies(38%) are uniquely represented in our meta-analysis.

Rating of Potential Moderator Variables

We used judges' ratings, based on the information contained inblinded copies of each study's method section and made without knowl-edge of the study's results or corresponding effect sizes, to assess thecontextual level of each moderator variable within each study. We maderatings of the five theoretical variables—anxiety, inhibition conflict,provocation, frustration, and self-focused attention—for all studies by

two judges (Tiffany Ito and a graduate student in psychology) on 9-point scales possessing no descriptive adjectives other than that a valueof 9 indicated a high level of the variable in question. We present the

operational definitions used by the judges in Appendix B. In addition,Appendix B includes examples (not given to the judges before the

ratings) of a study that received the most extreme rating for each end-point of the rated variables. Judges first independently rated a subset ofeight studies and discussed discrepancies with the other authors of thisarticle. To avoid systematic bias, we made ratings for each of the five

variables across studies before proceeding to the next variable. We ran-domly determined the order in which each judge made his or her ratingsfor the five variables, and the order was different for each judge. Addi-

tionally, the studies were in a different random order for each judge.These procedures correspond to those discussed and recommended inMiller and Carlson (1990) and Miller, Lee, and Carlson (1991).

The judges also rated a sixth variable, task complexity—defined asthe degree to which response measures and features of the experimentaltask require habitual, routine, low level, familiar, easy mental activityas opposed to effortful, complex, novel, mentally difficult activity. Anal-

yses revealed no significant effects of degree of task complexity on effectsize. We did not, therefore, give it further consideration.

We instructed the judges to take within- and between-subject experi-

mental manipulations into account when making their ratings. There-fore, it was possible for a single study to contribute more than one ratingper variable if the judges determined that the level of that variable was

manipulated within the study. For example, the judges made 50 ratingsof self-focused attention. For 48 of the studies, the judges agreed thatthe level of self-focus did not vary across conditions within the same

study; therefore, they assigned a single rating of self-focused attentionto each study. However, self-focused attention was manipulated in one

study, so they assigned two ratings on the self-focus variable. They used

similar procedures to rate within-study differences for the other theo-retical variables. In total, the judges made 77 ratings of anxiety, 54 rat-

ings of inhibition conflict, 91 ratings of provocation, and 74 ratings offrustration.

Descriptive statistics and reliabilities of the judges' ratings are shownin Table 1. Although the validity of these ratings could not be assessed

(e.g., by correlating judges' ratings with manipulation check effectsizes) because the data necessary for such an assessment were not in-cluded in the primary sources, these rating procedures have been shownto have convergent and construct validity in other research (e.g., Carl-son et al., 1988; Carlson & Miller, 1987; Eagly & Steffen, 1986; Miller

&Carlson, 1990; Miller et al., 1991). Moreover, the reliabilities of thejudges' ratings, as defined by the Pearson product-moment correlation

coefficient, were generally high in magnitude, and all exceeded .75 (seeTable I ) . In view of this, we performed moderator analyses on the aver-age of the ratings made by the two judges.

Coding of Study Characteristics

We coded information on 10 characteristics of each study. We codedthree of these originally as continuous variables but later made them


Table 1

Descriptive Statistics and Reliabilities of Judges'Ratings for Moderator Variables

Excluding the two mostAll studies

Variable

AnxietyInhibition conflictProvocationFrustrationSelf-focused attention

M

6.435.713.953.883.41

SD

2.383.162.732.671.81

Reliability

.88***

.94"*

.90***

.78***

.81***

k

7754917450

M

6.395.713.933.943.42

extreme outliers

SD

2.393.192.732.681.85

Reliability

.88***

.93***

.90***

.79***

.78***

Nole. Ratings were made of 49 separate studies. Multiple ratings per study were possible, with the totalnumber shown as k. Reliability is shown as the Pearson product-moment correlation coefficient of the twojudges' ratings. All variables were rated on 9-point scales, with a value of 9 indicating the highest levels ofanxiety, inhibition conflict, provocation, frustration, and self-focused attention.***p<.001.

categorical because of the characteristics of their respective distribu-

tions. These variables are (a) dose of alcohol (two categories), (b) timeallowed for ingestion (three categories), and (c) time allowed for ab-

sorption (four categories). We coded the remaining seven study charac-teristics into either dichotomous or trichotomous categories. The di-chotomous variables are (a) presence of nonaggressive response alterna-tives, (b) possibility of retaliation, and (c) gender. The trichotomous

variables are (a) experimenter knowledge of drink content, (b) distrac-tion of participants during beverage consumption, (c) participants'

drinking history, and (d) type of response measure. Two coders inde-pendently extracted the information from a subset of 26 studies.Agreement between them was 97%, by assessing the number of

agreements divided by the total number of characteristics extracted.Disagreement between the coders typically occurred over whether a cer-tain piece of information could be reliably determined. For example,many studies did not clearly state how long the alcohol absorption pe-

riod was, but it might have been possible to estimate this value. Dis-agreement occurred when one coder estimated the value but the other

coded the study as missing this information. Ultimately, we adopted theconservative strategy of coding a study as missing the information, un-less the relevant information was clearly and unambiguously stated inthe method section. Given the high agreement between coders and our

decision to adopt a conservative strategy in cases of ambiguous infor-mation, only one coder extracted study characteristics from the remain-ing studies.

Computation of Effect Sizes

Following Hedges {1981) and Hedges and Olkin (1985), we used d asthe effect size index, representing the difference between the experimen-tal and control group mean and divided by the pooled standard devia-tion (SD). We corrected these d values for bias resulting from small

sample size (Hedges & Olkin, 1985). In this meta-analysis, a positiveeffect size indicates that more aggressive behavior was displayed by par-ticipants who were administered alcohol relative to those in the controlcondition. The pooled SD was based on individual cell SDs whenever

possible, but when not reported, we obtained estimates of pooled SDsfrom analysis of variance results. We performed computations with themeta-analytic program DST4r(Johnson, 1993).

Frequently, studies reported enough data to compute multiple effect

sizes. There were three different sources of multiple effect sizes: (a) com-parison of more than one dose of alcohol with a control group (e.g., alow-dose and a high-dose group), (b) data on more than one dependentmeasure (e.g., no. of shocks administered and intensity of shocks), and(c) manipulation of other independent variables in addition to the alco-

hol-no alcohol variable. Indiscriminantly including multiple within-

study effect sizes in the meta-analysis would violate assumptions abouttheir independence and could, thereby, bias outcomes. We adopted thefollowing strategies, therefore, for all datasets used in the analyses re-

ported herein. If a study's researchers administered more than one al-cohol dose (as was the case for seven studies), we randomly selected

one of the doses and included in the analysis only those effect sizes thatreflected the comparison of the selected dose with the control condition.

If a study reported data on multiple dependent measures, we calculatedeffect sizes for all possible measures, then averaged them together into a

single index.For the last source of multiple effect sizes, manipulation of other in-

dependent variables besides dose of alcohol, we considered the randomselection and averaging strategies inadequate because the different con-ditions within a study often had bearing on the theoretical questionsaddressed in this analysis. For example, a study might cross a manipu-

lation of alcohol dose with a manipulation of provocation. Because wehypothesize that provocation will moderate effect size, it would be in-

appropriate to average effect sizes that represent different levels of prov-ocation. Similarly, important information on the effects of different lev-

els of provocation would be lost if we only randomly selected one effectsize from such studies.

As a result, we used a shifting unit of analysis approach (Bushman &Cooper, 1990; Cooper, 1989), which represents a compromise betweenthe independence assumption and the desire to retain as much informa-tion as possible. More specifically, use of the shifting unit of analysis

means that, for each of the five variables of theoretical interest, we in-cluded all of the effect sizes relevant to that analysis. As previously indi-cated, judges were instructed to take within-study conditions into ac-

count when making their ratings, resulting in 77, 54, 91, 74, and 50separate ratings of anxiety, inhibition conflict, provocation, frustration,

and self-focused attention, respectively. We created five separate data-sets, one for each variable with effect sizes that corresponded to theconditions rated for that particular variable. For example, in the self-focused attention dataset, where 48 of the 49 studies were judged to lackwithin-study variation in level of self-focused attention, the 48 studiescontributed one effect size each. For the remaining study, which con-tained within-study variation in self-focused attention, we entered twoeffect sizes into the dataset. One represented the difference between so-ber and intoxicated participants in a condition low in self-focus, and theother represented the difference between participants sober and intoxi-cated in a condition high in self-focus. We used similar procedures forthe other four datasets.

We created a sixth dataset in which each study was allowed to con-tribute only one effect size by averaging together all effect sizes from the


same study. We used this dataset to obtain the mean effect size across all

studies, irrespective of potential variation in any of the five variables of

theoretical interest. We also used this dataset for the categorical analyses

of study characteristics (e.g., whether participants were distracted dur-

ing the beverage consumption period) because these characteristics dis-

played no within-study variation. The only exception was the analysis

on participant gender. It was possible in three studies to compute sepa-

rate effect sizes for male and female participants, and a fourth study

included only female participants. The analysis on gender was, there-

fore, based on 50 effect sizes.

Distributions of Variables

The distribution of ratings of anxiety was negatively skewed and that

of self-focused attention was positively skewed. We used logarithmic

transformations to normalize these variables. Provocation, frustration,

and inhibition conflict were each bimodally distributed, and conse-

quently were dichotomized. Low values ranged from 1 to 4, 1.5 to 4.5,

and I to 4; high values ranged from 5 to 9,6 to 9, and 6 to 9 for provo-

cation, frustration, and inhibition conflict, respectively. The dichotomi-

zation of inhibition conflict is consistent with results from Steele and

Southwick (198 5), who reported that a 2-point scale for inhibition con-

flict yielded the most reliable judgments. Also, as suggested earlier, the

distribution of alcohol dose was bimodal. Dose was, therefore, dichoto-

mized, with low doses ranging from 0.23 to 1.00 ml of alcohol/kg of

body weight and high doses ranging from 1.20 to 1.30 ml/kg. We used

these transformed variables in all subsequent analyses.

Data Analysis Strategy

We used regression analyses to assess the effect of the continuouslydistributed moderator variables (anxiety and self-focused attention) on

effect size. The specific regression analyses used were designed for meta-

analytic data (Hedges & Olkin, 1985) and provide a test of model spec-

ification, Q,, in which a significant value indicates that the model is

not sufficient to account for heterogeneity among the effect sizes. For

inhibition conflict, provocation, and frustration, we performed categor-

ical analyses. We first categorized effect sizes as a function of the mod-

erator variable, then we assessed the degree of within- and between-

category homogeneity. A well-specified model in which variability in

the effect sizes is explained by the category distinctions would result in

homogeneity within each category but heterogeneity between the cate-

gories. Between-category homogeneity is assessed by Q,, which has an

approximate x2 distribution with degrees of freedom (df) equal to p -

1, where p is the number of categories in the model. When there are only

two categories in the model, Q, is analogous to a pairwise comparison.

Within-category homogeneity is assessed by gw, which has an approxi-mate x2 distribution with m - 1 df, where m is the number of effect

sizes in each category (Hedges & Olkin, 1985). Both statistics test the

assumption of homogeneity; significant values are indicative of hetero-

geneity in the effect sizes.

Regardless of whether we used regression or categorical models, we

performed all analyses on effect sizes that were weighted by the inverse

of their variance (Hedges & Olkin, 1985). In addition, we included dose

of alcohol administered to participants in the experimental condition

as a factor in each analysis. All significance tests are two-tailed, unless

stated otherwise.

Results

Overall Effect of Alcohol on Aggressive Behavior

The mean effect size of 0.54 across the 49 studies had a 95%confidence interval (CI) that excluded 0, CI = 0.45-0.63, indi-

cating that intoxication increased aggression. By Cohen's(1988) standards, this is considered a medium effect size.(Effect sizes of d = 0.20, 0.50, and 0.80 are considered small,medium, and large in magnitude, respectively.) In addition,there was significant heterogeneity among these effect sizes, Q(48 ) = 285.19, p < .0001. Most effect sizes ranged from -1.0 to1.0, but two substantially exceeded this range, d = 7.81 and5.99, respectively. When we removed these two extreme values,the difference in aggressive behavior between intoxicated andsober participants remained significant: The mean effect sizewas 0.47, and the 95% CI again excluded 0, CI = 0.38-0.56.Removing these extreme values did not result in homogeneityamong effect sizes, Q (46) = 78.92, p < .005. Although the gen-eral pattern of results did not change when we removed the twomost extreme outliers, the possibility that their large magnitudewas due to unique features of the original experiments led us toexclude them from subsequent analyses. Despite the substantialindependence of our dataset from that of Bushman and Cooper(1990), our mean effect size is similar to the mean of 0.43 ob-tained in their analysis.

As indicated in the criteria for study inclusion, we extractedall effect sizes in this analysis from published reports. Rosenthal(1979) and Wachter (1988) have argued that because studiesobtaining null results are less likely to be published, analyzingdata from only published reports biases results in favor of ob-taining a mean effect size that differs significantly from 0. Thishas been labeled the file drawer problem. A fail-safe n, whichrepresents the number of studies obtaining null results thatwould be needed to render the obtained mean effect size non-significant, was calculated to assess threat of the file drawerproblem in this analysis. Rosenthal (1991) suggested that a con-servative tolerance level for the fail-safe n is 5A + 10, where k isthe number of effect sizes. Thus, for 49 effect sizes, it shouldexceed 255. The obtained fail-safe n of 2,826 based on the meaneffect size of the 49 studies (or 1,863 when the two most extremeeffect sizes are removed) suggests that a file drawer problem isunlikely. In other words, there would need to be 2,826 unpub-lished studies with null results to render the overall mean effectsize obtained in this meta-analysis equal to 0.

Given the significant mean effect size indicating that alcoholincreases aggression, it is reasonable to expect that dose of alco-hol consumed is related to effect size, with larger effect sizesassociated with higher doses. To test this, we compared the effectsizes of studies that administered low as opposed to high doses.We omitted from this analysis the two most extreme outliersand one additional study (Boyatzis, 1974) that did not specifydose. As expected, higher alcohol doses were associated withlarger effect sizes, M = 0.53, CI = 0.41 -0.65, than lower doses,M = 0.35, CI = 0.21-0.50, Q. = 3.43, p < .03, one-tailed.3 As

3 We performed comparable analyses on the five other datasets as

well. With the exception of anxiety, all analyses were consistent with

the conclusion that alcohol-induced aggression increases when larger

alcohol doses are consumed. That is, in the inhibition conflict, provo-

cation, frustration, and self-focused attention datasets, higher doses

were associated with significantly larger mean effect sizes than lower

alcohol doses. The only exception was the anxiety dataset, in which we

entered anxiety and dose into a regression equation. Dose was not a

significant predictor of effect size in this analysis, B - -0.11, p < .20. It


evidenced by the exclusion of 0 from both CIs, even low doses

of alcohol (0.23 to 1.00 ml/kg) increased aggression.

Mechanisms of Increased Aggression in Intoxicated

Individuals

Role of anxiety. We have suggested that the increased ag-

gression of intoxicated participants might be due to their de-

creased sensitivity to anxiety-provoking situational cues, which

in a sober participant would serve to inhibit aggression. Spe-

cifically, the aggressive behavior of intoxicated participants is

expected to most greatly exceed that of sober ones as the inten-

sity of anxiety-provoking cues increases. We tested this with a

regression analysis. To account for the effects of alcohol dose,

we entered dose into the analysis with the anxiety ratings. The

regression coefficients and test of model specification are shown

Table 2. As expected, level of anxiety was a significant predictor

of effect size, such that the difference between intoxicated and

sober participants increased as level of anxiety increased, B =

0.58, p < .001. Nonetheless, the test of specification for this

model was significant, indicating that heterogeneity among the

effect sizes was present, Q, = 141.07, p< .001.

To explicitly assess whether the influence of anxiety-provok-

ing cues differs as a function of dose, we regressed anxiety onto

effect size separately for studies in the high- and low-dose sub-

sets. Regression coefficients and tests of model specification for

these analyses are also shown in Table 2. In both subsets, the

regression coefficient for anxiety was positive. In the high-dose

subset, the coefficient was significant, B = 0.70, p< .001, but

the coefficient in the low-dose subset reached only marginal sig-

nificance, B = 0.43, p < .08, two-tailed. Despite the absolute

difference between these coefficients, they did not differ signifi-

cantly from each other, t = 0.49. Also, in the high-dose subset,

no significant heterogeneity was present in the model, Qe =

23.17, but heterogeneity remained in the low-dose subset, Q, =

117.22, .p<.001.

Inhibition conflict. Table 3 shows the mean effect size and

CIs as a function of both level of inhibition conflict and alcohol

dose. The difference in mean effect size between studies with

high and low levels of inhibition conflict was significant, Q, =

17.46, p< .001, such that larger effect sizes were associated with

higher levels of inhibition conflict. It should be noted, however,

that the CIs for both means excluded 0. Thus, even when inhi-

bition conflict was low, intoxicated participants exhibited more

aggression than sober ones. Significant heterogeneity was pres-

ent among only the low-inhibition conflict effect sizes, (3» =

52.98, p<.001.

To assess potential moderating effects of dose, we separated

the dataset into low- and high-dose subsets and examined the

means as a function of inhibition conflict (see Table 3, lower

panel). Dose did not moderate the tendency for effect size to

increase with higher levels of inhibition conflict. High-inhibi-

tion conflict was associated with larger effect sizes than low-

should also be noted that the effect of dose in the self-focus datasetreached only marginal significance with a two-tailed test, p < . 10. (Theregression coefficients for dose when entered into the anxiety and self-focus regression equations are shown in Tables 2 and 6, respectively.)

Table 2

Regression Analyses of Anxiety and Alcohol Dose

Dataset and predictor

All studiesAnxietyDose

Low-dose subsetAnxiety

High-dose subsetAnxiety

B

0.58"*-0.11

0.43

0.70"'

P

0.32-0.11

0.16

0.56

QE

141.07***

117.22***

23.17

k

74

39

35

Note. Dose was dichotomized, with low values ranging from 0.23 to1.00 ml/kg and high values ranging from 1.20 to 1.30 ml/kg. B denotesunstandardized regression coefficient. 8 denotes standardized regres-sion coefficient. GE denotes test of model specification. Significant QE

values indicate that the model is inadequate to account for heterogene-ity among effect sizes. In addition, the two studies contributing the mostextreme outliers were excluded. In the low-dose subset, the regressioncoefficient for anxiety was marginally significant, p < .08, two-tailed.***p<.001.

inhibition conflict within both the high-dose, Q, = 7.52, p <

.006, and low-dose subsets, Q, = 5.67, p < .02.

Other Social Psychological Moderators

The analyses of anxiety and inhibition conflict suggest that

both factors may have predictive utility in explaining how alco-

hol increases aggression. However, neither accounted for all of

the variation in effect sizes. We obtained significant heterogene-

ity in the overall regression analysis of anxiety. According to

Steele and Josephs (1990), intoxication should increase aggres-

sion only when higher doses of alcohol have been administered

and when inhibition conflict is high. In our analyses, however,

intoxication increased aggression even at low doses and low lev-

els of inhibition conflict. Moreover, as mentioned, dose did not

moderate the effect of inhibition conflict, and there was signifi-

cant heterogeneity among the low-inhibition conflict studies.

These outcomes suggest that the relationship between alcohol

and aggression may depend on other factors. We examine next

the role of three such factors: provocation, frustration, and self-

focused attention.

Provocation. We predicted that the difference in aggression

between sober and intoxicated participants would decrease as

intensity of provocation increased. As shown by the means and

CIs in Table 4, this was the pattern we obtained. Lower levels of

provocation were associated with larger effect sizes than higher

levels of provocation, Q, = 4.10, p < .05. Note, however, that

both CIs exclude 0. Hence, under low as well as high provoca-

tion, intoxicated participants behaved more aggressively than

did those who were sober.

To assess whether the effect of provocation was moderated by

alcohol dose, we divided the dataset as a function of dose, and

analyses on provocation were repeated. The lower panel of Ta-

ble 4 shows that, in the low-dose subset, lower levels of provoca-

tion were associated with larger effect sizes, Q, = 6.23, p < .01.

For the high-dose subset, the means were in the same direction,

but the difference was not significant, Q, = 0.63. Even though

the aggressive behavior of intoxicated and sober participants be-


Table 3

Categorical Analysis of Inhibition Conflict and Alcohol Dose

95% CI for d,

Dataset and variable class k M d+ Lower Upper Qw QB

All studiesLow-inhibition conflictHigh-inhibition conflict

Low-dose studiesLow-inhibition conflictHigh-inhibition conflict

High-dose studiesLow-inhibition conflictHigh-inhibition conflict

2229

168

521

0.270.65

0.240.64

0.220.65

0.140.53

0.080.35

-0.050.52

0.400.78

0.390.93

0.500.79

52.98***17.61

46.71***6.34

2.8411.26

17.46***

5.67*

7.52**

Note. d+ denotes effect sizes weighted by the reciprocal of their variances. CI denotes confidence interval.Qv tests within-category homogeneity; QB tests between-category homogeneity. Significant values indicaterejection of the hypothesis of homogeneity. One study (Boyatzis, 1974) lacked dose information, so it wasexcluded from all analyses involving dose. In addition, the two studies contributing the most extreme outli-ers were excluded. Significance tests are two-tailed.*p<.05. **p<.0\. ***/>< .001.

came more similar as the intensity of provocation increased Self-focused attention. The results of the regression analysis

(viz., smaller mean effect sizes), intoxicated participants con- on self-focused attention are shown in Table 6. Following the

sistently remained more aggressive than sober ones. This latter same strategy as with the analysis of anxiety, we included dose

effect is revealed by the exclusion of 0 from all CIs, even those as a predictor in the equation with self-focused attention. The

in which intensity of provocation was high. results indicate that self-focused attention was a significant pre-

Frustration. Table 5 shows that both low and high levels of dictor of effect size, B = —0.42, p < .05. The negative sign of

frustration were associated with mean effect sizes that differed the regression coefficient indicates that as self-focused attention

from 0 (i.e., the 95% CIs excluded 0). However, contrary to increased, sober and intoxicated participants behaved more

our expectation that sober and intoxicated participants would similarly.

behave more similarly when frustration was high, the results To assess whether the effect of self-focused attention was

show that higher levels of frustration were associated with larger, moderated by dose, we conducted separate regression analyses

not smaller, mean effect sizes, Q, = 6.69, p < .01. We observed on the low- and high-dose subsets of the studies. The results

this same pattern in the low-dose subset, Q, = 16.48, p < .001. from the low-dose subset replicate those in the overall sample

Such a comparison could not be made within the high-dose sub- (see Table 6); higher levels of self-focused attention were asso-

set, however, because there was only one effect size in the high- ciated with smaller effect sizes, B = -0.77, p < .001. By con-

frustration-high-dose cell. trast, we observed the opposite relationship within the high-

Table 4

Categorical Analysis of Provocation and Alcohol Dose

95% CI for rf+

Dataset and variable class k M d+ Lower Upper gw QB

All studiesLow provocationHigh provocation

Low-dose studiesLow provocationHigh provocation

High-dose studiesLow provocationHigh provocation

5533

2412

3021

0.650.50

0.540.23

0.700.63

0.560.40

0.390.04

0.590.50

0.740.61

0.700.42

0.820.76

77.75*59.44**

52.32***33.28***

22.6414.32

4.10*

6.23**

0.63

Note. d+ denotes effect sizes weighted by the reciprocal of their variances. CI denotes confidence interval.2w tests within-category homogeneity; QB tests between-category homogeneity. Significant values indicaterejection of the hypothesis of homogeneity. One study (Boyatzis, 1974) lacked dose information, so it wasexcluded from all analyses involving dose. In addition, the two studies contributing the most extreme outli-ers were excluded.*/><.05. **/x.01. ***/p<.001.


Table 5

Categorical Analysis of Frustration and Alcohol Dose

95%CIford+

Dataset and variable class Lower Upper <2w

All studiesLow frustrationHigh frustration

Low-dose studiesLow frustrationHigh frustration

High-dose studiesLow frustrationHigh frustration

5220

2819

231

0.420.66

0.230.67

0.580.54

0.330.50

0.110.50

0.45-0.35

0.510.82

0.360.83

0.701.43

86.38**54.17***

55.33**54.10***

15.38NA

6.69**

16.48"*

NA

Note. d+ denotes effect sizes weighted by the reciprocal of their variances. CI denotes confidence interval.Qw tests within-category homogeneity; Qz tests between-category homogeneity. Significant values indicaterejection of the hypothesis of homogeneity. One study (Boyatzis, 1974) lacked dose information, so it wasexcluded from all analyses involving dose. The two studies contributing the most extreme outliers wereexcluded.**p<.Q[. ***p<.001.

dose subset of the studies. In this latter set, higher levels of self-

focused attention were associated with larger effect sizes, B =

0.95, p < .05. That is, when higher alcohol doses were adminis-

tered, greater levels of self-focus were associated with larger

differences in the aggressiveness of intoxicated and sober partic-

ipants. The coefficients for the low- and high-dose subsets

differed reliably, f(43) = 4.lO,p < .01.

Study Characteristics

Categorical moderator analyses of study characteristics are

shown in Table 7. Although ancillary to the major theoretical

questions, we summarize these results for descriptive purposes.

Presence of nonaggressive-response alternatives. Many

studies used paradigms that forced participants to engage in

some type of aggression. For example, participants could

Table 6

Regression Analyses on Self-Focused Attention

and Alcohol Dose

Dataset and predictor

All studiesSelf-focused attentionDose

Low-dose subsetSelf-focused attention

High-dose subsetSelf-focused attention

B

-0.42*0.16

-0.77***

0.95*

0

-0.220.19

-0.44

0.44

CE

70.77**

43.52***

16.71

k

47

20

27

Note. Dose was dichotomized, with low values ranging from 0.23 to1.00 ml/kg and high values ranging from 1.20 to 1.30 ml/kg. B denotesunstandardized regression coefficients, g denotes standardized regres-sion coefficient. gE denotes test of model specification. Significant val-ues indicate that the model is inadequate to account for heterogeneityamong effect sizes. The two studies contributing the most extreme out-liers were excluded. The regression coefficient for dose was marginallysignificant, p < .10, two-tailed."f < .05. **p < .01. *"p < .001.

choose how intense a shock to deliver but lacked the option of

not delivering a shock. Gustafson (1993) suggested that in-

creased aggression following intoxication may be restricted to

situations in which aggression is the only response alternative

available. As shown in Table 7, studies lacking a nonaggressive-

response alternative yielded a significantly larger mean effect

size than those in which a nonaggressive-response alternative

was available, Q, = 10.70, p < .001. However, intoxicated par-

ticipants behaved more aggressively than sober ones, even when

a nonaggressive-response alternative was available (i.e., the CI

excluded 0). Caution should be used when interpreting these

results because subsequent follow-up analyses revealed that

studies in which aggression was the only response alternative

also tended to administer higher alcohol doses than studies in

which nonaggressive alternatives were available, ((31)= 5.65, p

<.OOOI.

Possibility of retaliation. Studies in which it was possible

for the target of the participant's aggression to retaliate were

associated with a larger mean effect size than those in which

retaliation was not possible. This finding is somewhat difficult

to interpret, however, because possibility of retaliation tended

to covary with other theoretically important variables. For ex-

ample, a concern about retaliation was the source of inhibiting

cues in many studies that received high ratings of inhibition

conflict. Similarly, fear of retaliation often contributed to rat-

ings of high anxiety.

Experimenter knowledge of drink content. To examine the

consequence of the experimenter's knowledge of drink content,

we classified studies into one of three categories: experimenter

blind to drink content, experimenter not blind, and not enough

information reported to determine the experimenter's knowl-

edge. Although all three categories were associated with positive

mean effect sizes and CIs that excluded 0, the mean effect size

was smaller in the experimenter blind category, compared with

categories in which the experimenter was not blind or not

enough information was provided, x 2 (2) = 10.41, p < .005,

and % 2 (2 ) = 16.88, p < .0001, respectively. This outcome, also


Table 7

Tests of Categorical Models of Study Characteristics

95%CIford+

Variable and class

Presence of nonaggressive responsealternatives

AvailableNot available

Possibility of retaliationPossibleNot possible

Experimenter knowledge of drink contentBlindNot blindNot clearly stated

Distracted during consumption of alcoholDistractedNot distractedNot clearly stated

Time allowed for ingestion (in min)5-17

20-30Not clearly stated

Time allowed for absorption (in min)10-1520-2530-45Not clearly stated

Drinking historyLight-moderate socialHeavy socialMissing

Type of response measureDirect behaviorVerbal-questionnaireBoth

Gender"MenMen and women

k

1730

2917

138

26

156

26

21179

1413164

214

22

4241

464

A/rf+

0.27,

0.58b

0.64,0.23b

0.21,

0.65b

0.63b

0.24, c0.56c;d0.63M

0.37«0.47c,d0.74W

0.47C

0.31, ,0.63b.c0.69Cib

0.62.-0.05b

0.62.

0.56,0.02h

0.51

0.49,0.27,

Lower

0.120.47

0.520.09

0.060.430.49

0.100.290.51

0.22

0.320.53

0.280.160.47

0.33

0.48-0.26

0.47

0.45-0.21

0.10

0.39-0.06

Upper

0.420.70

0.760.37

0.360.870.76

0.390.840.76

0.49

0.610.94

0.66

0.450.791.04

0.750.170.77

0.660.250.92

0.580.60

(2w

41.28"*26.94

17.0242.17'**

30.09**2.86

26.19

40.25***6.05

16.42

55.18"12.95

1.70

33.65**18.5415.900.54

26.051.61

11.33

56.645.01

—

76.237.69

QB

10.70***

18.80***

19.78***

16.91"*

9.10"

10.29"

28.39***

17.27*"

1.60

Note. d+ denotes effect sizes weighted by reciprocal of their variances. CI denotes confidence interval.Cw tests within-category homogeneity; QB tests between-category homogeneity. Significant values indicaterejection of the hypothesis of homogeneity. Qx is not reported for those categories consisting of only onecase. The two most extreme outliers were excluded from all analyses. Therefore, k = 47. Within eachcategory dimension, means with different subscripts differ at p < .05.a Separate effect sizes for men and women were calculated from three separate studies. In addition, onestudy included only female participants. As a result, k - 50 for the analysis of gender.V<.05. **p<.01. "*p<.001.

obtained by Bushman and Cooper (1990), is consistent with the

literature on experimenter expectancy effects (e.g., Rosenthal,

1976).

Distraction during consumption of alcohol. There were

three categories of studies in this analysis: participants defi-

nitely distracted, participants definitely not distracted, and not

enough information provided for an accurate classification. Al-

though all yielded CIs that excluded 0, studies in which partici-

pants were distracted during the alcohol consumption period

had a significantly smaller mean effect size than those studies

for which no clear information was provided, x2(2) = 15.71, p

< .0005.

Time allowed for ingestion and for absorption. Q. values for

both analyses of time allowed for ingestion and for absorption

were significant, indicating that these variables moderate the

degree to which alcohol augments aggression. In the case of in-

gestion time, this effect seems to be primarily due to the differ-

ence between studies allowing 5 to 17 min for ingestion and

those in which no accurate information on time allowed for in-

gestion could be obtained, x2(2) = 9.08, p < .01. Larger effect

sizes were associated with studies that did not clearly report the

time for ingestion. The effect of time allowed for absorption

seems most attributable to the difference between studies allow-

ing 20 to 25 min for absorption and those allowing 30 to 45 min,

X 2 (2) = 9.08, p< .01, with larger effect sizes in the latter group.

However, it should be noted that time for absorption tended to

be confounded with dose, such that studies administering

higher doses tended to allow more time for absorption.


Drinking history, type of response measure, and gender. Al-

though the significant Q, values for drinking history and type

of response measure suggest that these variables also moderate

effect size, we refrain from conclusions concerning them be-

cause of their substantial covariation, as well as the small num-

ber of studies within some of the variable classes. In addition,

both variables tended to covary with participant gender, such

that a history of heavy social drinking among the participants

and use of verbal measures were associated with the inclusion

of female participants. Thus, we were unable to make an assess-

ment of the individual moderating effects of each variable.

Discussion

Anxiety and Inhibition Conflict

Many formulations take the view that increased aggression

following alcohol consumption is a consequence of decreased

sensitivity to cues that inhibit aggression (e.g., Taylor & Cher-

mack, 1993; Taylor & Leonard, 1983; Zeichner& Pihl, 1979).

One model focuses on the role of anxiety in inhibiting aggres-

sion. It argues that to the extent that intoxication is associated

with anxiolysis, intoxication blocks responsivity to this behav-

ioral inhibition system. Similarly, the inhibition conflict model

proposed by Steele and colleagues (Steele & Josephs, 1990;

Steele & South wick, 1985) describes how decreased sensitivity

to inhibiting cues, in conjunction with intoxication, facilitates

aggression.

Our results provide some support for both models. Consider-

ing the anxioloysis-disinhibition model first, this model argues

that when anxiety arousing cues are very low in intensity, they

have little inhibiting effect on either sober or intoxicated partic-

ipants, thus yielding small effect sizes. When instead such cues

are stronger in intensity, their effects in sober participants is to

strongly inhibit their aggression. Therefore, if intoxicated par-

ticipants ignore these latter cues, there is a large difference in

the aggressiveness of the two groups, reflected by large effect

sizes. As predicted, effect size (reflecting the greater aggression

of intoxicated participants relative to that of sober controls) in-

creased as intensity of anxiety-provoking cues increased. There

was also a tendency for the effect of anxiety to be strongest in

those studies administering relatively high-alcohol doses. This

is consistent with the interpretation that at lower alcohol doses.

and presumably lower levels of alcohol-related cognitive im-

pairments, both intoxicated and sober participants are rela-

tively responsive to anxiety-provoking cues that serve to inhibit

aggression. As alcohol dose increases and various aspects of cog-

nitive functioning are correspondingly impaired, intoxicated

participants may experience anxiolysis. Their behavior would,

therefore, be relatively free from anxiety-related inhibition,

leading to increased aggression in intoxicated relative to sober

persons.The analysis of inhibition conflict revealed that increased lev-

els of inhibition conflict were associated with larger effect sizes.

However, our results do not completely support the inhibition

conflict model because Steele (Steele & Josephs, 1990; Steele &

Southwick, 1985) argued that the aggressive behavior of intox-

icated persons should exceed that of sober ones only when both

inhibition conflict and alcohol dose are high. Our results show

that the combination of high-inhibition conflict and high dose

is associated with increased aggression. In conflict to other pre-

dictions of this model, however, intoxicated participants were

also significantly more aggressive than sober ones when inhibi-

tion conflict was high but alcohol dose was low and when both

inhibition conflict and dose were low. Indeed, as seen by inspec-

tion of Table 3 and contrary to the inhibition conflict model,

there is no hint that dose moderates the effect of inhibition

conflict.

The conceptual independence of these two models also war-

rants consideration. Some of the study characteristics judged to

be anxioloytic were also judged as contributing to high levels of

inhibition conflict. One such example, as mentioned earlier, is

the degree to which the experimental paradigm suggests the

possibility of retaliation. It could be argued that the specifica-

tion of anxiolysis is simply one way in which the potency of

aggression inhibiting cues is decreased in situations of high-

inhibition conflict. However, the anxiolysis-disinhibition model

is distinct from, and perhaps preferable to, the inhibition con-

flict model because it allows for the possibility that inhibiting

cues are attended to as fully as instigating cues but that aggres-

sion nevertheless increases in intoxicated people. In other

words, in contrast to inhibition conflict, the anxiolysis-disinhi-

bition model is more parsimonious because it only requires the

assumption that the psychological effect of the inhibiting cues

differs depending on whether one is sober or intoxicated.

Zeichner, Pihl, Niaura, and Zacchia (1982) illustrated this

difference between the anxiolysis-disinhibition and inhibition

conflict models. In their study, intoxicated and sober partici-

pants were given the opportunity to shock a "bogus" partner,

and the partner was said to have the ability to deliver aversive

stimulation to the participants in the form of irritating noise.

No partner in fact existed. One third of both groups of partici-

pants were given attentional instructions that required them to

write down after each trial the level of shock they selected for

their partner and the level of noise they received back from their

partner. Because the level of noise that participants received was

programmed to correlate perfectly with the intensity of shock

they chose to deliver to their partners, the attentional instruc-

tions should have forced these participants to attend to the con-

sequences of their aggressive behavior. This, in turn, should

have inhibited aggression. Instead, intoxicated participants in

this condition were more aggressive than their sober counter-

parts. As Pihl et al. (1993) summarized,

the acutely intoxicated participants were not more aggressive be-cause they were unaware, verbally or cognitively, of what they weredoing and of the consequences of their behavior but because thatknowledge no longer served an inhibitory function, perhaps be-cause it no longer produced fear. (p. 132)

The inhibition conflict model seemingly predicts the opposite

pattern of results. Forcing participants to attend to the negative

consequences of their aggressive behavior should make the in-

hibiting cues relatively more salient than instigating cues. Con-

sequently, with a narrowed range of attentional focus, intoxi-

cated participants would be expected to attend more exclusively

to these salient inhibiting cues (Steele & Josephs, 1990, Foot-

note 1). The inhibition conflict model would, therefore, predict

that intoxicated participants in the forced attention condition


would behave less aggressively than those in the other two con-ditions, which clearly was not the case. Alternatively, even ifforcing them to attend to the negative consequences of their ag-gressive behavior only made the salience of the inhibiting cuesmore similar to that of the provoking cues, these participantsshould have behaved less aggressively or should not havediffered from sober controls, leaving the outcome still contraryto the inhibition conflict model.

Although intensity of anxiety-provoking cues and level of in-hibition conflict did moderate effect size, neither completely ac-counted for the relation between aggression and intoxication.Significant heterogeneity among effect sizes remained in theanalyses of anxiety and inhibition conflict. Consequently, weconclude that although knowledge of the intensity of anxiety-provoking cues and level of inhibition conflict provides somepredictive utility the effect of alcohol on aggression is a complexprocess with multiple determinants. A full understanding of therelation between alcohol and aggression, therefore, requires at-tention to other moderating variables.

Provocation and Frustration

The presence and intensity of provocation is one additionalfactor that can moderate the tendency for intoxicated people tobehave more aggressively than sober ones. Gustafson (1993)has argued that provocation moderates the effect of alcohol onaggression, such that alcohol increases aggression only in situa-tions in which provocation is present. Two aspects of our analy-sis challenge that conclusion. First, the obtained direction ofeffect is opposite to that predicted by Gustafson and is in accordwith our theoretically derived prediction. That is, the differencein aggression between intoxicated and sober participants actu-ally decreased rather than increased as intensity of provocationincreased.

Second, it appears that Gustafson's (1993) conclusion wasbased on the assumption that provocation affects the differencein behavior between intoxicated and sober participants throughits effects on the behavior of those intoxicated. By contrast, wesuspect that provocations have their strongest effect on soberpeople because intoxicated people display relatively greater lev-els of aggression, even in the absence of provocation. As evi-dence of this, we calculated the mean effect size of those studiesthat received a provocation rating of 1 (the lowest possiblerating). The mean of these 26 effect sizes was 0.56, CI = 0.43-0.69, indicating that even among studies with the lowest level ofprovocation, the aggressive behavior of intoxicated participantsreliably exceeded that of sober ones. Hence, it is likely that thesmaller effect size associated with higher levels of provocation islargely the result of an increased display of aggression by soberparticipants in response to provocation.

When we examined this difference in effect size as a functionof provocation separately within the low- and high-dose studies,the same direction of effect was obtained in both subsets. Inthe low-dose studies, the greater aggressiveness of intoxicatedparticipants relative to that of sober ones was reliably reducedunder higher levels of provocation. In the high-dose studies,however, although provocation produced a similar direction ofeffect, it did not evidence reliability. In summary then, provo-cation reduces the difference in aggression between intoxicated

and sober participants, although the source of these effects re-mains unspecified. They may be due to the provocation's insti-gating or angering effects or because it provides a suitable ratio-nale for aggression, which might otherwise be considered an in-appropriate behavior.

Originally, we expected provocation and frustration to havesim ilar directions of influence on effect size, but this was not thecase. Whereas higher levels of provocation were associated withsmaller effect sizes, higher levels of frustration were associatedwith larger effect sizes. In retrospect, we believe that these op-posing effects become theoretically understandable when theyare linked to paradigm characteristics associated with the high-frustration as compared with high-provocation studies in thisdataset. Aggressive behaviors can be categorized into two types:(a) instrumental and affective or (b) hostile (e.g., Baron, 1977;Feshbach, 1964; Geen, 1990). The main goal of affective ag-gression is to inflict harm, whereas instrumental aggression isenacted in the service of attaining some other goal, such asmoney or status. Of course, any aggressive act may contain ele-ments of both intent to harm and instrumentality. Nevertheless,one motive can dominate.

In this dataset, many studies in which high levels of provoca-tion were induced used the reaction time-competition para-digm. The male participant in this paradigm believed that hewas competing with another participant who was said to be lo-cated in a separate room (e.g., Jeavons & Taylor, 1985). Theloser of each competitive trial received a shock, the intensity ofwhich was determined by his opponent. For trials on which theparticipant won, he received no shock but was given feedbackabout the intensity he would have received had he lost. No op-ponent in fact existed, and the shock intensity feedback was pre-programmed by the experimenter. This feedback was manipu-lated to indicate that the opponent sometimes selected relativelyhigh-intensity shocks for the participant. We believe that expe-riencing such provocative behavior from a stranger with whomno prior interaction has occurred, and in particular no priornegative interaction has occurred, would serve to anger and ir-ritate the participant. Aggression displayed by the participantagainst the opponent is, therefore, likely motivated by a desireto harm the opponent and retaliate against him for the pain hehas actually or intended to inflict.

By contrast, the studies judged as high in frustration tendedto be teacher-learner studies, or modifications of the paradigm,which we believe are more likely to elicit instrumental aggres-sion. The participant, in the role of teacher (or supervisor, inthe modified paradigm), was thought to experience frustrationto the extent that a partner performed poorly and jeopardizedthe participant's likelihood of obtaining a monetary incentive.4

4 The only difference between a teacher-learner study and what we

refer to as the "modified version" is that in the latter, participants are

explicitly told that the task performed by their bogus partner requires

concentration but no learning (e.g., Gustafson, 1986a). We retain the

term teacher-learner to refer to both versions of this paradigm, regard-less of whether learning is involved, for two reasons: (a) to link the mod-

ified paradigm with the larger body of work that is descended from

Buss's (1961) original development of the paradigm and (b) the two

versions of the paradigm do not differ in the feature that is of theoretical

interest, namely, the extent to which a participant's valued goal isblocked.


In many cases, this monetary incentive was quite sizable (e.g.,

approximately $80 in Gustafson, 1986a), so the motivation toobtain it should be substantial. In addition, participants weresometimes explicitly informed that their aggression could be in-strumental in obtaining their monetary reward (e.g., Gustaf-son, 1985b).

The increased aggression among intoxicated participants un-der conditions of high frustration may, therefore, be attributableto differences in perceived instrumental value between soberand intoxicated participants. After all, there is the potential thateven low-intensity shocks could be effective in improving thelearner's performance. Sober and less impaired participantsmay be more likely to consider this. Moreover, sober partici-pants who do use higher intensity shocks may be more likelythan intoxicated ones to notice that their relatively aggressivebehavior is not having the intended effect (i.e., is not improvingthe learner's performance). As a consequence, their aggressivebehavior may become less responsive to increases in frustration(i.e., continued poor performance by the learner). By contrast,intoxicated participants may be less likely to realize that theircontinued and escalating use of aggression is relatively in-effective. If so, the difference in aggression between intoxicatedand sober participants should increase as a function of frustra-tion. The likelihood that aggression would begin to lose its in-strumental value for sober participants is particularly high inthis dataset, given the fact that participants' goal-directed be-havior was always completely thwarted. That is, no matter howmuch aggression participants displayed, they were never able toreach their desired goal.

Additionally, in a teacher-learner study, the participant ischarged with the task of teaching or supervising another person.To some extent then, poor performance by the partner reflectspoorly on the participants's ability as a teacher-supervisor(Rule & Percival, 1971). Sober participants, as relatively lessimpaired, are likely to be more sensitive to this contingencythan their intoxicated counterparts. They may appear to be lessresponsive to increases in frustration because they do not wantto be seen as severely punishing another person for somethingfor which they themselves are partly responsible.

Finally, the different effects we obtained for frustration andprovocation may be attributable to differences in the perceivedappropriateness of aggression in response to these two events.As we have already argued, the belief in an eye-for-an-eye maymake aggression in retaliation to a provocation acceptable.However, there may be greater ambiguity as to whether it is ap-propriate to take one's frustrations out on another person. Frus-trated individuals may, therefore, be more likely to wrestle withtheir conscience than those provoked. The greater aggressiondisplayed by intoxicated, frustrated participants may reflecttheir decreased sensitivity to these concerns.5

Paradigm differences between high-frustration and high-provocation studies may, therefore, explain the different effectsobtained as a function of provocation and frustration. In par-ticular, paradigm characteristics that elicit angry as comparedwith instrumental aggression appear important. It should benoted that, whereas high frustration appeared to be associatedwith instrumental aggression in the current dataset, this covari-ation does not necessarily appear in the larger literature on ag-gression. Inductions of task frustration may typically lack the

interpersonal aspects that elicit instrumental aggression and in-stead evoke affective aggression. For example, frustration elic-ited by one's inability to complete an ostensibly easy task in-creases aggression relative to individuals who experience tasksuccess (Geen, 1968). This aggression could in no way facilitatecompletion of the task, rendering an instrumental motivationfor it unlikely. Thus, it may be that had the research paradigmsof the studies in our meta-analysis invoked noninterpersonal,task frustration, our parallel predictions for frustration andprovocation would have been upheld.

We conducted an internal analysis to test the hypothesis thatprovocation and frustration would have similar directions ofeffect when frustration was likely to elicit affective aggression.We divided studies on the basis of the predominant type of ag-gression likely instigated (affective or instrumental), then com-pared effect sizes as a function of level of provocation and frus-tration. The determining characteristics of aggression type werewhether (a) features of the experimental paradigm providedparticipants with a desirable goal and (b) aggression could beperceived as useful in achieving the goal. We coded the aggres-sion as instrumental in nature if both conditions were met. Oth-erwise, we considered the aggression primarily affective in na-

ture. The mean effect sizes as a function of level of frustrationin Table 8 show that the unexpected reverse outcome obtainedin the main analysis—namely, smaller, rather than larger, effectsizes under low levels of frustration—was substantially attenu-ated when the frustrating event elicited affective aggression.Moreover, the absolute magnitude of the reverse frustrationeffect obtained in the main analysis (as compared with Table 5)is even larger among the subset of studies limited to include onlythose in which frustration elicited instrumental aggression.

To provide a conceptual comparison, we conducted this sameanalysis as a function of level of provocation (see Table 8).When we only considered those studies that elicited affectiveaggression, as expected, the effect of provocation replicates thatobtained in the main analysis: Low provocation was associatedwith larger effect sizes than high provocation. By contrast, whenwe examined the subset of studies that elicited instrumental ag-gression, the absolute magnitude of the difference between lowand high provocation was smaller and nonsignificant. Althoughthese outcomes do not completely confirm our hypothesesabout the role of type of aggression, they are consistent with it.We are cautious in our interpretation, however, due to the factthat there were only two effect sizes in the high-provocation,instrumental aggression cell (Lang, Goeckner, Adesso, & Mar-latt, 1975; Zeichner, Allen, Giancola, & Lating, 1994)andonlythree in the high-frustration, affective aggression cell (Cherek,Steinberg, & Manno, 1985; Kelly, Cherek, Steinberg, & Robin-son, 1988; Taylor, Schmutte,& Leonard, 1977).

These opposing directions of effect for provocation and frus-tration were interesting although unexpected. In the larger ex-perimental literature on aggression per se (wherein only the be-havior of sober participants is considered), the magnitude ofthe effects may differ, but provocation and frustration typicallyoperate in a similar direction (meta-analysis; Carlson & Miller,1988; Geen, 1990). Their opposite effects on the difference be-

3 We thank a reviewer for this suggestion.


Table 8

Categorical Analysis of Frustration and Provocation as a Function of Type of Aggression

Instrumental aggression

95% CI for <t

Variable and class

FrustrationLowHigh

ProvocationLowHigh

k

2017

152

Md+

0.230.66

0.370.26

Lower

0.080.50

0.19-0.18

Upper

0.380.83

0.540.70

6w

35.75*54.04***

35.12***2.18

a, k14.67***

313

0.183931

Md+

0.510.57

0.750.52

Affective aggression

95% CI font

Lower

0.40-0.10

0.640.41

Upper

0.621.23

0.860.63

Gw GB

40.59***40.51

0.048.77*

29.3556.08*

Nole. </+ denotes effect sizes weighted by reciprocal of their variances. CI denotes confidence interval. Qw tests within-category homogeneity;tests between-category homogeneity. Significant values indicate rejection of the hypothesis of homogeneity.*p<.05. *"*p<.001.

tween sober and intoxicated aggression in the current dataset

suggest that greater attention needs to be paid to the unique

aspects of the paradigms in which they are induced. We, there-

fore, echo a concern—first voiced by Pernanen (1976) and later

by Gustafson (1993)—that features of the paradigms that

affect frustration and provocation need to be considered when

interpreting results of studies on the alcohol-aggression rela-

tion. We also think that it may be useful to attend to the motives

for aggression that are aroused in different situations (i.e., in-

strumental as compared with affective aggression).

Self-Focused AI tent ion

The negative relationship between effect size and self-focus in

the overall dataset and in the subset of low-dose studies indicates

that the tendency for intoxicated participants to behave more

aggressively than sober ones is reduced when attention is fo-

cused on self. In other words, intoxicated participants behave

more similarly to those sober as self-focus increases. Because

intoxication generally increases aggression, this finding suggests

that self-focus differentially exerts greater inhibitory effects on

intoxicated participants. That is, self-focused participants are

seemingly able to overcome the aggression-inducing conse-

quences of alcohol ingestion. There are at least two possible ex-

planations for such a pattern. First, intoxicated participants

have been shown to overcome alcohol-induced impairments

when motivated to concentrate on their performance. For ex-

ample, intoxicated participants who were told to "try to stay

sober" showed better motor and cognitive performance than

those not given this instruction (Young & Pihl, 1980; see also

Gustafson & K.allmen, 1990a, 1990b, 1990c; Myrsten, Lamble,

Frankenhaeuser, & Lundberg, 1979; Ross & Pihl, 1988; Wil-

liams, Goldman, & Williams, 1981). Ross and Pihl argued that

focusing attention on self can induce such a compensatory pro-

cess. To the extent that intoxicated participants are more influ-

enced by self-focusing cues than sober participants, as our re-

sults indicate, it appears that they actually overcompensate and

regulate their behavior even more stringently than sober partic-

ipants. Alternatively, the narrowed attentional focus accompa-

nying intoxication may intensify a self-focused state by decreas-

ing sensitivity to other, potentially distracting stimuli. Sober

participants may be relatively less affected by self-focusing cues

because their wider range of attentional focus exposes them to

cues that compete with self for attention. These explanations

are not incompatible; both processes may occur.

The fact that effect size and self-focus were positively related

in the high-dose subset of studies may be a function of the rela-

tively weak strength of the self-focusing cues in this dataset. Ex-

plicit manipulations of self-focus were rare in the dataset, and

we made ratings of self-focus on the basis of subtle variations in

situational cues. Not surprisingly, on average, self-focus was low

(viz., below the scale midpoint in 70% of the studies). In this

dataset, it is therefore reasonable to expect that as alcohol dose

and the cognitive impairment associated with it increased, in-

toxicated participants would be less affected by the relatively

weak self-focusing cues. The unimpaired sober participants

may still be influenced by the self-focusing cues, leading them

to behave less aggressively than those intoxicated who had been

given higher doses.

The different effect of self-focus as a function of dose may

also be explained by the finding that intoxication typically de-

creases the likelihood of focusing attention on self (Hull et al.,

1983). The self-focusing cues in this dataset may have been

sufficient to overcome this general tendency in those intoxicated

participants who had consumed lower doses. Because they may

have started out at a lower level of self-focus than sober ones,

the aggressive behavior of intoxicated participants may have

been more greatly affected than that of sober ones. This would

account for the negative relation between effect size and self-

focus when lower doses had been consumed. As dose increased,

the likelihood of focusing on self may correspondingly decrease,

such that stronger self-focusing cues are required if attention is

to be redirected to self. The relatively weak self-focusing cues in

this dataset may, therefore, have had little effect on the intoxi-

cated participants who had received higher doses. These same

cues should still have effected sober ones, resulting in a positive

relation between effect size and self-focus when higher doses are

involved.6 It should be noted that the mean judges' ratings of

self-focus did not differ as a function of dose (p > .05), indicat-

' We thank a reviewer for this suggestion.

76 [TO, MILLER, AND POLLOCK

ing that the different relation observed between self-focus and

effect size as a function of dose is not attributable to differences

in the intensity of self-focusing cues between high- and low-dose

studies.

Study Characteristics

This review has focused on a number of potentially impor-

tant moderators of the relation between alcohol and aggression.

One issue that needs to be stressed is that it is often not possible

in the existing literature to assess separately the effect of other

theoretically relevant factors because the variables of interest

tend to covary within studies. Our analysis has revealed many

instances of this. For example, the display of aggression differs

as a function of gender (Eagly & Steffen, 1986); but, in the al-

cohol and aggression literature, inclusion of female participants

has tended to covary with a history of heavier social drinking

among the participants and use of verbal measures of aggres-

sion—both of which could influence effect size. We, therefore,

found it impossible to assess the independent influences of these

three variables. Because each of these variables is of potential

theoretical and practical importance, systematic investigation

of their independent effects would be welcomed.

Similarly, we were unable to reach a strong conclusion about

certain paradigmatic features, such as the influence of the avail-

ability of nonaggressive-response alternatives. Gustafson (1993)

argued that alcohol increases aggression only in situations in

which participants are forced to make an aggressive response.

We attempted to test this hypothesis by dividing studies as a

function of whether nonaggressive-response alternatives were

present in an experiment. Unfortunately, these two categories

differed significantly in mean dose of alcohol given to the par-

ticipants, thereby precluding assessment of whether observed

differences in effect size were due to dose effects or to the type

of response alternatives.

Implications for Future Research

The analyses on the moderator variables in this meta-analysis

produced a number of interesting results. However, like any

meta-analytic research whose researchers examine the effects of

moderator variables by comparing subsets of studies or ordering

studies in terms of their judged or coded level on the moderator

of interest, the obtained relation between the moderator (e.g.,

anxiety) and effect size (e.g, the difference in aggression be-

tween sober and intoxicated participants) is correlational in na-

ture. Hence, it needs to be followed up by experimental re-

search. In this section, we briefly suggest future experimental

research linked to the specific effects we have obtained.

Anxiety and inhibition conflict. Although not strongly em-

phasized in our report, our judges' ratings of anxiety and inhi-

bition conflict showed strong covariation. Indeed, in an analysis

in which each study was allowed to contribute one rating per

variable, the correlation between ratings of anxiety and inhibi-

tion conflict was so high as to suggest singularity of the two con-

cepts (r = .91, p < .001, when corrected for reliability of the

judgments). Despite this, we have tried to outline differential

predictions made by the anxioloysis-disinhibition and inhibi-

tion conflict models. Before such predictions can be adequately

tested, however, independent manipulations of the two models

are required, assuming that they do indeed possess discrimina-

tive construct validity. Frankly, we are at a loss to generate inde-

pendent manipulations. Operationalizations of anxiety (e.g.,

presence of a high-status observer who has control over the par-

ticipant with respect to an important outcome) that come to

mind would also serve as inhibiting cues that increase inhibi-

tion conflict, and vice versa. The conceptual independence of

anxiety and inhibition conflict and their relation in moderating

alcohol-induced aggression, therefore, requires greater atten-

tion. As previously discussed, the possibility of retaliation often

contributed to ratings of both high-anxiety and high-inhibition

conflict in this dataset. A wider range of Operationalizations of

anxiety and inhibition conflict should be explored to eliminate

the possibility that the obtained effects are unique to threat of

retaliation. In summary, at the very least, a wider variety of ma-

nipulations of anxiety and inhibition conflict and an elaborated

conceptual analysis of their potential discriminative construct

validity is warranted.

Provocation and frustration. In our analysis of provocation

and frustration, it was apparent that the distinction between

these concepts was confounded with other variables. In partic-

ular, our discussion suggests the need to separately manipulate

the type of aggressive responses elicited by provocation and

frustration manipulations. Specifically, it calls for experimental

researchers to examine separately the effects of alcohol for prov-

ocations experimentally designed to elicit instrumental versus

affective aggression and, in parallel, for inductions cXfrustration

designed to elicit instrumental versus affective aggression. Per-

haps also important is the need for experiments whose research-

ers orthogonally examine these effects when the instigatory

source is human versus environmental. With respect to this lat-

ter call, it is worth noting that in our dataset, many of the frus-

tration inductions depended on human agency (a "stupid"

partner in the teacher-learner paradigm), as opposed to envi-

ronmental circumstances (e.g., extreme temperatures or task

failure). Provocation may also be induced either by human

agency or environmental circumstances. It may be important

that the definition of provocation given to our judges con-

strained it to instances of human agency.

We also note that many of the paradigms used to study alco-

hol and aggression are fairly provoking (e.g., a shock machine

is present, a participant is involved in competition). As a result,

studies that received the lowest rating on the provocation di-

mension most likely reflected a condition of low, as opposed to

no, provocation. It would be of interest to compare the aggres-

sion of intoxicated and sober participants under conditions in

which provocation was judged below threshold, so as to deter-

mine whether the relation between provocation and effect size

holds in the absence of provocation. Similarly, some amount of

anxiety and inhibition conflict likely existed even in those stud-

ies that received the lowest ratings on these variables. Hence,

our results do not specifically address the relationship between

alcohol and aggression under situations of no anxiety or no in-

hibition conflict.

Self-focus. The concept of self-focus contains two compo-

nents: One refers to personal standards for ideal behavior,

whereas the other represents impulsive inclination. It would be

useful to develop separate measures of each component, so as


to determine whether they are effected equally as a consequence

of alcohol ingestion and its dose. It is possible that the atten-

tional deficits induced by intoxication primarily interfere with

the encoding of one, the other, or both of these components.

Alternatively, or in addition, intoxication may interfere with the

high order cognitive process of comparing an adequate cogni-

tive representation of each.

Other issues. It would also be useful to determine whether

changes in motivation or ability are responsible for the results

obtained herein. On the one hand, proscriptions against drink-

ing and driving imply that alcohol has inevitable capacity re-

duction consequences. On the other hand, Ross and Pihl (1988)

have concluded that the

pharmacological action of alcohol may be counteracted when sub-

jects are motivated to "act sober," either by experimenters setting

a behavioral standard of sober performance, or by manipulations

aimed at forcing the subjects to take personal responsibility for

their performance, (p. 116)

The influence of self-focused attention at lower doses is consis-

tent with this conclusion. Clearly, however, at high enough

doses, alcohol exerts behavioral effects that cannot be overcome

by increased motivation. The data that we reviewed are not

sufficient to address this issue, but determination of the relative

contributions of changes in ability and motivation could help

clarify the effects of alcohol on behavior in general, and on ag-

gressive behavior in particular.

Conclusion

In closing, we emphasize that although this meta-analysis

confirms the causal role of alcohol in increasing aggression (see

also Bushman & Cooper, 1990; and Steele & Southwick, 1985),

we do not view aggression as an inevitable consequence of in-

toxication. Laboratory studies such as those in this analysis are

specifically designed to facilitate aggression by creating situa-

tions in which participants feel comfortable displaying it should

they desire to do so. By contrast, most real-world settings con-

tain many fewer aggression-instigating cues and many more in-

hibiting ones.

References

References marked with an asterisk indicate studies included in the

meta-analysis.

Averill.J. R. (1982). Anger and aggression: An essay on emotion. New

York: Springer-Verlag.Axelrod, R. (1984). The evolution of cooperation. New \brk: Basic

Books.

'Bailey, D. S., Leonard, K. E., Cranston, J. W., & Taylor, S. P. (1983).Effects of alcohol and self-awareness on human physical aggression.

Personality and Social Psychology Bulletin, 9, 289-295."Bailey, D. S., & Taylor, S. P. (1991). Effects of alcohol and aggressive

disposition on human physical aggression. Journal of Research on

Personality, 25, 334-342.Baron, R. A. (1977). Human aggression. New "York: Plenum Press.

•Bennett, R. M, Buss, A. H., & Carpenter, J. A. (1969). QuarterlyJournal of Studies on Alcohol. 30, 870-877.

Berkowitz, L. (1989). Frustration-aggression hypothesis: Examination

and reformulation. Psychological Bulletin, 106, 59-73.

Bettencourt, B. A., & Miller, N. (1996). Gender differences in aggres-

sion as a function of provocation: A meta-analysis. Psychological Bul-

letin, 179,422-447.

Birnbaum, I. M., Johnson, M. K.., Hartley, J. T, & Taylor, T. H.(1980).

Alcohol and elaborative schemas for sentences. Journal of Experi-

mental Psychology: Human Leaning and Memory, 6, 293-300.

Birnbaum, I. M., & Parker, E. S. (1977). Acute effects of alcohol on

storage and retrieval. In I. M. Birnbaum & E. S. Parker (Eds.), Alco-

hol and human memory(pp. 99-108). Hillsdale, NJ: Erlbaum.

"Bond, A., & Lader, M. (1987). The effects of alcohol on behavioural

aggression and cardiac and electrodermal activity monitored during

an aversive task. Journal ofPsychophysiology, 1, 229-240.

*Boyatzis, R. E. (1974). The effects of alcohol consumption on the ag-

gressive behavior of men. Quarterly Journal of Studies on Alcohol, 35,959-972.

Brown. S. A., Goldman, M. S., Inn, A.. & Anderson, L. R. (1980).

Expectations of reinforcement from alcohol: Their domain and rela-

tion to drinking patterns. Journal of Consulting and Clinical Psychol-ogy, 48, 419-426.

Bushman, B. J., & Cooper, H. M. (1990). Effects of alcohol on human

aggression: An integrative research review. Psychological Bulletin,

707.341-354.

Buss, A. H. (1961). The psychology of aggression. New York: Wiley.

Carlson, M., Charlin, V., & Miller, N. (1988). Positive mood and help-

ing behavior: A test of six hypotheses. Journal of Personality and So-

cial Psychology, 55, 1-17.

Carlson. M., & Miller, N. (1987). Explanation of the relation between

negative mood and helping. Psychological Bulletin, 102, 91-108.

Carlson, M., & Miller, N. (1988). The differential effects of social and

nonsocial negative events on aggressiveness. Sociology and Social Re-

search, 72, 155-158.

Carver, C. S. (1975). Physical aggression as a function of objective self-

awareness and attitudes toward punishment. Journal of Experimen-

tal Social Psychology, II, 510-519.

Carver, C. S. (1979). A cybernetic model of self-attention processes.

Journal of Personality and Social Psychology, 8, 1251-1281.

Carver, C. S., & Scheier, M. F. (1981). Attention and self-regulation:

A control theory approach to human behavior. New \brk: Springer-

Verlag.

Carver, C. S., & Scheier, M. F. (1990). Origins and functions of positive

and negative affect: A control-process view. Psychological Review, 97,19-35.

•Cherek, D. R., Steinberg, J. L., & Manno, B. R. (1985). Effects of

alcohol on human physical aggressive behavior. Journal of Studies on

Alcohol, 46.321-32$.

Cohen, J. (198 8). Statistical power analyses for the behavioral sciences

(2nd ed). Hillsdale, NJ: Erlbaum.

Cooper, H. M. (1989). Integrating research: A guide for literature re-

views. Newbury Park, CA: Sage.

Craik, F. I. M. (1977). Similarities between the effects of aging and

alcoholic intoxication on memory performance, construed within a

"levels of processing" framework. In I. M. Birnbaum & E. S. Parker(Eds.), Alcohol and human memory (pp. 9-21). Hillsdale, NJ:

Erlbaum.

Critchlow, B. (1986). The powers of John Barleycorn. American Psy-chologist, 41, 751-764.

Current Contents [Database]. (1989-1994, July). Philadelphia: Insti-

tute for Scientific Information [Producer]. Available from: Ovid

Technologies.

Diener, E. (1980). Deindividuation: The absence of self-awareness and

self-regulation in group members. In B. P. Paulus (Ed.), The psychol-

ogy of group influence (pp. 209-242). Hillsdale, NJ: Erlbaum.

Diener, E.,& Wallbom, M. (1976). Effects of self-awareness on antinor-

mative behavior. Journal of Research in Personality. 10, 107-111.


Dollard, J., Doob, L., Miller, N., Mowrer, Q, & Sears, R. (1939). frus-

tration and aggression. New Haven, CT: Yale University Press.

Duval, S., Duval, V. H., &Neely, R. (1979). Self-focus, felt responsibil-

ity, and helping behavior. Journal of Personality and Social Psychol-

ogy, 37, 1769-1778.

Duval, S., & Wicklund, R. A. (1972). A theory of objective self-aware-

ness. New York: Academic Press.

Eagly, A. H., & Steffen, V. J. (1986). Gender and aggressive behavior: A

meta-analytic review of the social psychological literature. Psycholog-

ical Bulletin, 100, 309-330.

Feshbach, S. (1964). The function of aggression and the regulation of

aggressive drive. Psychological Review, 71, 257-272.

*Ganter, A. B., & Taylor, S. P. (1992). Human physical aggression as a

function of alcohol and threat of harm. Aggressive Behavior, 18, 29-

36.

Geen, R. G. (1968). Effects of frustration, attack, and prior training in

aggressiveness upon aggressive behavior. Journal of Personality and

Social Psychology, 9, 316-321.

Geen, R. G. (1990). Human aggression. Pacific Grove, CA: Brooks/

Cole.

Gianoulakis, C, & Gupta, A. (1986). Inbred strains of mice with vari-

able sensitivity to ethanol exhibit difference in the content and pro-

cessing of beta-endorphins. Life Sciences, 39, 2315-2325.

Gibbons, F. X., & Wicklund, R. A. (1982). Self-focused attention and

helping behavior. Journal of Personality and Social Psychology, 43,

462-474.

Gouldner, A. W. (1960). The norm of reciprocity. American Sociologi-

cal Review, 25, 161-178.

Gray, J. A. (1987). The psychology of fear and stress. New York: Cam-

bridge University Press.

Greenwell, J., & Dengerink, H. A. (1973). The role of perceived versus

actual attack in human physical aggression. Journal of Personality

and Social Psychology, 26, 66-71.

•Gustafson, R. (1984). Alcohol, frustration, and direct physical aggres-

sion: A methodological point of view. Psychological Reports, 55, 959-

966.

•Gustafson, R. (I985a). Alcohol and aggression: Pharmacological ver-

sus expectancy effects. Psychological Reports, 57, 955-966.

•Gustafson, R. (1985b). Alcohol-related aggression: A further study of

the importance of frustration. Psychological Reports, 57, 683-697.

*Gustafson, R. (1985c). Frustration as an important determinant of

alcohol-related aggression. Psychological Reports, 57, 3-14.

*Gustafson, R. (1986a). Alcohol, frustration, and aggression: An ex-

periment using the balanced placebo design. Psychological Reports,

59,207-218.

•Gustafson, R. (1986b). Threat as a determinant of alcohol-related ag-

gression. Psychological Reports, 58, 287-297.

•Gustafson, R. (1987). Alcohol and human physical aggression: An

experiment using a "backward" balanced placebo design. Journal of

Social Behavior and Personality, 2, 135-144.

•Gustafson, R. (1988). Beer intoxication and physical aggression in

males. Drug and Alcohol Dependence, 21, 237-242.

•Gustafson, R. (1989). Alcohol and the validation of experimental ag-

gression paradigms: The Taylor reaction time procedure. Drug and

Alcohol Dependence, 23, 49-54.

•Gustafson, R. (1990). Wine and male physical aggression. Journal of

Drug Issues, 20, 75-86.

•Gustafson, R. (I991a). Aggressive and nonaggressive behavior as a

function of alcohol intoxication and frustration in women. Alcohol-

ism: Clinical and Experimental Research, 15, 886-892.

Gustafson, R. (1991b). Alcohol and outcome expectancies: Tolerance

for socially nonacceptable behaviors. Psychology Reports. 69. 67-74.

•Gustafson, R. (1991c). Male physical aggression as a function of alco-

hol, frustration, and subjective mood. International Journal of the

Addictions, 26, 255-266.

•Gustafson, R. (199 Id). Male physical aggression as a function of alco-

hol intoxication and frustration: Experimental results and method-

ological considerations. Alcoholism: Clinical and Experimental Re-

search, 15, 158-164.

•Gustafson, R. (1992). Alcohol and aggression: A replication study

controlling for potential confounding variables. Aggressive Behavior,

IK, 21-28.

Gustafson, R. (1993, September). What do experimental paradigms

tell us about alcohol-related aggressive responding. Journal of Studieson Alcohol (Suppl. 11), 20-29.

Gustafson, R., & Kallmen, H. (1990a). Alcohol and the compensation

hypothesis: A test with cognitive and psychomotor tasks. Perceptual

and Motor Skills, 71, 1367-1374.

Gustafson, R., & Kallmen, H. (1990b). Effects of alcohol on cognitive

performance measured with Stroop's Color-Word Test. Perceptual

and Motor Skills, 71, 99-105.

Gustafson, R., & Kallmen, H. (1990c). Effects of alcohol on prolonged

cognitive performance measures with Stroop's Color-Word Test. Psy-

chological Reports, 67, 645-650.

Heatherton, T. F., & Baumeister, R. F. (1991). Binge eating as escape

from self-awareness. Psychological Bulletin, 110, 86-108.

Hedges, L. V. (1981). Distribution theory for Glass's estimator of effect

size and related estimators. Journal of Educational Statistics, 6, 107-

128.

Hedges, L. V., &Olkin, I. (1985). Statistical methods far meta-analysis.

New \brk: Academic Press.

Hoffman, P. L., & Tabakoff, B. (1990). Ethanol and guanine nucleotide

binding proteins: A selective interaction. FASEB Journal, 4, 2612-

2622.

Hull, J. G. (1981). A self-awareness model of the causes and effects of

alcohol consumption. Journal of Abnormal Psychology, 90, 586-600.

Hull, J. G., & Bond, C. F. (1986). Social and behavioral consequences

of alcohol consumption and expectancy: A meta-analysis. Psycholog-ical Bulletin, 99, 347-360.

Hull, J. G., Levenson, R. W., Young, R. D., & Sher, K. J. (1983). Self-

awareness-reducing effects of alcohol consumption. Journal of Per-

sonality and Social Psychology, 44, 461-473.

Hull, J. G., & Reilly, N. P. (1983). Self-awareness, self-regulation, and

alcohol consumption: A reply to Wilson. Journal of Abnormal Psy-

chology. 92, 514-519.

Hull, J. G., & Young, R. D. (1983a). The self-awareness-reducing

effects of alcohol: Evidence and implications. In J. Suls & A. G.

Greenwood (Eds.), Psychological perspectives on the self (Vol. 2, pp.159-190). Hillsdale, NJ: Earlbaum.

Hull, J. G., & Young, R. D. (1983b). Self-consciousness, self-esteem,

and success-failure as determinants of alcohol consumption in male

social drinkers. Journal of Personality and Social Psychology, 44,

1097-1109.

Hull, J. G., Young, R. D., & Jouriles, E. (1986). Applications of the

self-awareness model of alcohol consumption: Predicting patterns of

use and abuse. Journal of Personality and Social Psychology. 51, 790-

796.

Hunt, W. A. {1985). Alcohol and biological membranes. New York:

Guilford Press.

Huntley, M. J. (1973). Effects of alcohol and fixation-task difficulty onchoice reaction time to extrafoveal stimulation. Quarterly Journal of

Studies on Alcohol, 34, 89-103.

•Jeavons, C., & Taylor, S. P. (1985). The control of alcohol-related ag-gression: Redirecting the inebriate's attention to socially appropriate

conduct. Aggressive Behavior, 11, 93-101.

Johnson, B. T. (1993). DSTAT 1.10: Software for the meta-analytic re-view of research literatures [Manual], Hillsdale, NJ: Erlbaum.


Jones, B. M, & Jones, M. K. (1977). Alcohol and memory impairment

in male and female social drinkers. In I. M. Birnbaum & E. S. Parker

(Eds.), Alcohol and human memory (pp. 127-138). Hillsdale, NJ:Erlbaum.

•Kelly, T. H., Cherek, D. R., & Steinberg, J. L. (1989). Concurrent

reinforcement and alcohol: Effects on human aggressive behavior.

Journal of Studies on Alcohol. SO, 399-405.

•Kelly, T. H., Cherek, D. R., Steinberg, J. L., & Robinson, D. (1988).

Effects of provocation and alcohol on human physical aggressive be-

havior. Drug and Alcohol Dependence, 21, 105-121.

Koob, G. E, & Bloom, F. E. (1988, November 4). Cellular and molec-

ular mechanisms of drug dependency. Science, 242, 715-723.

Lang, A. R. (1983). Drinking and disinhibition: Contributions from

psychological research. In R. Room & G. Collins (Eds.), Alcohol and

disinhibition: Nature and meaning of the link (National Institute for

Alcohol and Alcoholism Research Monograph No. 12, pp. 48-87).

Washington DC: U.S. Government Priming Office.

*Lang, A. R., Goeckner, D. T., Adesso, V. J., & Marian, G. R. (1975).

Effects of alcohol on aggression in male social drinkers. Journal of

Abnormal Psychology, 84, 508-518.

Lang, A. R., & Sibrel, P. A. (1989). Psychological perspectives on alco-

hol consumption and interpersonal aggression: The potential role of

individual differences in alcohol-related criminal violence. Criminal

Justice and Behavior, 16. 299-324.

•Laplace, A. C, Chermack, S. T., & Taylor, S. P. (1994). Effects of alco-

hol and drinking experience on human physical aggression. Person-

ality and Social Psychology Bulletin, 20.439-444.

•Leonard, K. E. (1984). Alcohol consumption and escalatory aggres-

sion in intoxicated and sober dyads. Journal of Studies on Alcohol,

45, 75-80.

•Leonard, K. E. (1989). The impact of explicit aggressive and implicit

n on aggressive cues on aggression in intoxicated and sober males. Per-

sonality and Social Psychology Bulletin, 15, 390-400.

Lightdale, J. R., & Prentice, D. A. (1994). Rethinking sex differences

in aggression: Aggressive behavior in the absence of social roles. Per-

sonality and Social Psychology Bulletin. 20, 34-44.

Lindman, R. E., & Lang, A. R. (1994). The alcohol-aggression stereo-

type: A cross-cultural comparison of beliefs. International Journal of

the Addictions, 29, 1-13.

MacAndrew, C., & Edgerton, R. B. (1969). Drunken comportment.

Chicago: Aldine.

Marlatt, G. A., & Rohsenow, D. J. (1980). Cognitive processes in alco-

hol use: Expectancy and the balanced placebo design. In N. Mello

(Ed.), Advances in substance abuse(Vo\ 1, pp. 159-199). Greenwich,

CT: JAI Press.

Medline [Database]. (1966-1994. July). Bethesda, MD: National Li-

brary of Medicine [Producer]. Available from: Ovid Technologies.

Miller, N.. & Carlson, M. (1990). Valid theory-testing meta-analyses

further question the negative state relief model of helping. Psycholog-

ical Bulletin. 707.215-225.

Miller, N., Lee, J.-Y., & Carlson, M. (1991). The validity of inferentialjudgments when used in theory-testing mcta-analysis. Personality

and Social Psychology Bulletin. 17, 335-343.

Moskowitz, H., & DePry, D. (1968). Differential effect of alcohol on

auditory vigilance and divided-attention tasks. Quarterly Journal of

Studies on Alcohol, 29, 54-63.

Moss, H. B., & Tartar, R. E. (1993). Substance abuse, aggression, and

violence. American Journal on Addictions, 2, 149-160.

•Murdoch, D., & Pihl, R. O. (1985). Alcohol and aggression in a groupinteraction. Addictive Behaviors, 10, 97-101.

Murdoch, D., Pihl, R. O., & Ross, D. (1990). Alcohol and crimes of

violence: Present issues. International Journal of the Addictions, 25,1065-1081.

Myers, R. D., Melchior, C., & Swartzwelder, H. S. (1980). Amine-alde-

hyde metabolites and alcoholism: Fact, myth, or uncertainty. Sub-

stance and AlcoholActions /Misuse, I, 223-238.

Myrsten, A.-L., Lamble, R., Frankenhaeuser, M., & Lundberg, A.

(1979). Interaction of alcohol and reward in an achievement situa-

tion. Psychopharmacology, 62, 211 -215.

Pernanen, K. (1976). Alcohol and crimes of violence. In B. Kissin &

H. Begleiter (Eds.), The biology ofalcoholism: Vol. 4. Social aspect of

alcoholism (pp. 351-441). New York: Plenum Press.

Pihl, R. O. (1983). Alcohol and aggression: A psychological perspective.

In E. Gottheil, K. A. Druley, T. E. Skoloda, & H. M. Waxman (Eds.),

Alcohol, drugabuse, and aggression (pp. 292-313). Springfield, IL:

Charles C Thomas.

Pihl, R. Q, Peterson, J. B., & Lau, M. A. (1993, September). A bioso-

cial model of the alcohol-aggression relationship. Journal of Studies

on Alcohol (Suppl I I ) , 128-139.

•Pihl, R. O., Smith, M., & Farrell, B. (1984). Alcohol and aggression

in men: A comparison of brewed and distilled beverages. Journal of

Studies on Alcohol, 45. 278-282.

•Pihl, R. O., & Zacchia, C. (1986). Alcohol and aggression: A test of

the affect-arousal hypothesis. Aggressive Behavior, 12. 367-375.

•Pihl, R. Q, Zeichner, A., Niaura, R., Nagy, K, & Zacchia, C. (1981).

Attribution and alcohol-mediated aggression. Journal of Abnormal

Psychology, 90. 468-475.

Prentice-Dunn, S., & Rogers, R. W. (1980). Effects of deindividuating

situational cues and aggressive models on subjective deindividuation

and aggression. Journal of Personality and Social Psychology, 39,

104-113.

PsycINFO [Database]. (1967-1994, July). Washington, DC: Ameri-

can Psychological Association [Producer]. Available from: Silver-Platter.

•Richardson, R. (1981). The effect of alcohol on male aggression to-

ward female targets. Motivation and Emotion. 5, 333-344.

Rogers, M., Miller, N., Mayer, F. A., & Duval, S. (1982). Personal re-

sponsibility and salience of the request for help: Determinants of the

relation between negative affect and helping behavior. Journal of Per-

sonality and Social Psychology, 43, 956-970.

Rogers, R. W, & Prentice-Dunn, S. (1981). Deindividuation and anger-

mediated interracial aggression: Unmasking regressive racism.

Journal of Personality and Social Psychology, 41, 63-73.

Rohsenow, D. J. (1983). Drinking habits and expectancies about alco-

hol's effects for self versus other. Journal of Counseling and Clinical

Psychology, 51, 752-756.

•Rohsenow, D. J., & Bachorowski, J. (1984). Effects of alcohol expec-

tancies on verbal aggression in men and women. Journal of Abnormal

Psychology. 93, 418-432.

Roizen, R. (1983). Loosening up: General population views of the

effects of alcohol. In R. Room &G. Collins (Eds.), Alcohol and dis-

inhibition: Nature and meaning of the link (National Institute of Al-

cohol and Alcoholism Research Monograph No. 12, pp. 236-257).

Washington DC: U.S. Government Printing Office.

Rosen, L. J., & Lee, C. L. (1976). Acute and chronic effects of alcohol

use on organizational processes in memory. Journal of Abnormal


Rosenthal R. (1976). Experimenter effects in behavioral research (rev.

ed.). New York: Appleton-Century-Crofts.

Rosenthal R. (1979). The "file drawer problem" and tolerance for null

results. Psychological Bulletin, 86. 638-641.

Rosenthal, R. (1991). Meta-analysis: A review. Fsychonomic Medicine,5^,247-271.

Ross, D. F., & Pihl, R. O. (1988). Alcohol, self-focus, and complex re-

action-time performance. Journal of Studies on Alcohol, 49, 115-125.

Rule, B. G., & Percival, E. (1971). The effects of frustration and attack


on physical aggression. Journal of Experimental Research in Person-

ality. 5, 111-118.

Sayette, M.A.(1993).An appraisal-disruption model of alcohol's effect

on stress responses in social drinkers. Psychological Bulletin, 114,

459-476.

Scheier, M. E, Fenigstcin, A., & Buss, A. H. (1974). Self-awareness and

physical aggression. Journal of Experimental Social Psychology, 10,

264-273.

•Schmutte, G. T, Leonard, K. E., & Taylor, S. P. (1979). Alcohol and

expectations of attack. Psychological Reports, 45, 163-167.

•Schmutte, G. X, & Taylor, S. P. (1980). Physical aggression as a func-

tion of alcohol and pain feedback. Journal of Social Psychology, 110,

235-244.

*Shuntich, R. J., &Taylor, S. P. (1972). The effects of alcohol on human

physical aggression. Journal of Experimental Research in Personal-

ity, 6, 34-38.

Sobell, M. B., & Sobell, L. A. (1973). Individualized behavior therapy

for alcoholics. Behavior Therapy, 4, 49-72.

Southwick, L., Steele, C. M., Marlatt, A., & Lindell, M. (1981). Alco-

hol-related expectancies: Denned by phase of intoxication and drink-

ing experience. Journal of Consulting and Clinical Psychology, 49,

713-721.

Spielberger, C. D. (1972). Anxiety as an emotional state. In C. D.

Spielberger (Ed.), Anxiety: Current trends in theory and research

(Vol. I , pp. 23-49). New York: Academic Press.

Steele, C. M., & Josephs, R. A. (1990). Alcohol myopia: Its prized and

dangerous effects. American Psychologist, 45, 921-933.

Steele, C. M., & Southwick, L. (1985). Alcohol and social behavior I:

The psychology of drunken excess. Journal of Personality and Social

Psychology. 48, 18-34.

Tartar, R. E., Jones, B. M., Simpson, C. D., & Vega, A. (1971). Effects

of task complexity and practice on performance during acute alcohol

intoxication. Perceptual and Motor Skills, 33, 307-318.

Taylor, S. P. (1983). Alcohol and human physical aggression. In E. Gott-

heil, K. A. Druley, T. E. Skoloda, & H. M. Waxman (Eds.), Alcohol,

drug abuse, and aggression (pp. 280-291). Springfield, IL: Charles

C Thomas.

Taylor, S. P., & Chermack, S. T. (1993, September). Alcohol, drugs, and

human physical aggression. Journal of Studies on Alcohol (Suppl.

11), 78-88.

•Taylor, S. P., & Gammon, C. B. (1976). Aggressive behavior of intoxi-

cated subjects: The effect of third party intervention. Journal of Stud-

ies on Alcohol. 37. 917-930.

•Taylor, S. P., Gammon, C. B., & Capasso, D. R. (1976). Aggression as

a function of the interaction of alcohol and threat. Journal of Person-

ality and Social Psychology, 34. 938-941.

Taylor, S. P., & Leonard, K. E. (1983). Alcohol and human physical

aggression. In R. G. Geen & E. I. Donnerstein (Eds.), Aggression:

Theoretical review. Vol. 2: Issues in research (pp. 77-101). New York:

Academic Press.

•Taylor, S. P., Schmutte, G. T, & Leonard, K. T. (1977). Physical ag-

gression as a function of alcohol and frustration. Bulletin ofthePsy-

chonomic Society, 9, 217-218.

•Taylor, S. P., Schmutte, G. T., Leonard, K. E., & Cranston, J. W.

(1979). The effects of alcohol and extreme provocation on the use of

a highly noxious electric shock. Motivation and Emotion, 3, 73-81.

•Taylor, S. P., & Sears, J. D. (1988). The effects of alcohol and persua-

sive social pressure on human physical aggression. Aggressive Behav-

ior, 14, 237-243.

Young, J. A., & Pihl, R. Q (1980). Self-control of the effects of alcohol

intoxication. Journal of Studies on Alcohol, 41. 567-571.

Wachter, K. W. (1988, September 16). Disturbed by meta-analysis? Sci-

ence, 24, 1407-1408.

Washbume, C. (1956). Alcohol, self and the group. Quarterly Journal

of Studies on Alcohol, 17,108-123.

•Weisman, A. M., & Taylor, S. P. (1994). Effect of alcohol and risk

of physical harm on human physical aggression. Journal of General


Williams, R. M., Goldman, M. S., & Williams, D. L. (1981). Expec-

tancy and pharmacological effects of alcohol on human cognitive and

motor performance: The compensation for alcohol effect. Journal of

Abnormal Psychology, 90, 267-270.

•Zeichner, A., Allen, J. D., Giancola, P. R., & Lating, J. M. (1994).

Alcohol and aggression: Effects of personal threat on human physical

aggression and affective arousal. Alcoholism: Clinical and Experi-

mental Research, 18, 657-663.

•Zeichner, A., & Pihl, R. O. (1979). Effects of alcohol and behavior

contingencies on human aggression. Journal of Abnormal Psychol-

ogy. 88. 153-160.

•Zeichner, A, & Pihl, R. O. (1980). Effects of alcohol and instigator

intent on human aggression. Journal of Studies on Alcohol, 41, 265-

276.

•Zeichner, A., Pihl, R. O., Niaura, R., & Zacchia, C. (1982). Atten-

tional processes in alcohol-mediated aggression. Journal of Studies

on Alcohol, 43, 714-724.


Appendix A

Overlap of Studies With Other Meta-Analyses

The following studies from the present dataset were also included inthe following prior meta-analyses on the relation of alcohol and socialbehavior:

Hull & Bond (1986) Meta-Analysis

Langetal.(1975)Pihletal. (1981)

Rohsenow & Bachorowski (1984)

Bushman & Cooper (1990) Meta-Analysis

Bailey etal. (1983)

Gustafson (1985b)Gustafson(1985c)

Gustafson (1986a)Gustafson (1986b)Gustafson (1987)

Gustafson (1988)Gustafson (1990)

Gustafson (1991c)Jeavons & Taylor (1985)

Pihletal. (1984)

Pihl&Zacchia(1986)

Pihletal. (1981)

Schmutte etal. (1979)Schmutte& Taylor (1980)ShuntichA Taylor (1972)

Taylor & Gammon (1976)

Taylor etal. (1976)Taylor el al.( 1977)

Taylor etal. (1979)Taylor & Sears (1988)Zeichneretal. (1982)

Steele & Southwick (1985) Meta-Analysis

Bennett etal. (1969)

Boyatzis(1974)

Lang etal. (197 5)Pihletal. (1981)

Schmutte & Taylor (1980)Shuntich& Taylor (1972)Taylor & Gammon ( 1976)

Taylor etal. (1976)Taylor etal. (1977)

Taylor etal. (1979)

Zeichner &Pihl( 1979)Zeichner &Pihl( 1980)

Zeichner etal. (1982)

Bushman & Cooper's (1990) Descriptive Summary

(but not in their main meta-analysis)Boyatzis(1974)Gustafson (1984)

Lang el al. (1975)Leonard (1984)Rohsenow & Bachorowski ( 1984)

Appendix B

Attention and Cue Salience Variables Extracted From Each Study

These are the definitions judges used to rate the levels of anxiety, in-hibition conflict, provocation, frustration, and self-focused attention of

each study. All variables were rated on 9-point scales; examples are pro-vided of studies which, according to the ratings obtained from the

judges, represented the endpoints of each scale.

Anxiety

This criteria is the extent to which the overall experimental circum-stances effected the participants by increasing their anxiety, makingthem excited, worried, highly alert, or attentive. Such a state is usually

aroused by the perception of threat to self (either physical or

psychological) or worry over potential or actual negative consequencesfor the self. Endpoints are

2 = participating in a reaction time-competition study in whichthe fear of receiving shocks has been removed (e.g., Ganter & Tay-lor, 1992)

9 = participating in a reaction time-competition study (e.g., Tayloret al., 1977) where there is concern about beating an opponent ona reaction time task and receiving shocks for losing.

Inhibition Conflict

This criteria is the degree to which participant's response is simulta-neously pressured by instigating and inhibiting cues (Steele & South-

wick, 1985). High-inhibition conflict is assigned when conflicting pres-

sures are strong and relatively equal. If inhibiting cues involve the pos-sibility for negative consequences, these consequences should bedirectly linked to the participant's behavior. Endpoints are

1 - ability to negatively evaluate another person without fear ofretaliation (e.g., Rohsenow & Bachorowski, 1984)

9 = participating in a reaction time-competition paradigm (e.g.,

Taylor et al., 1977) where there are cues instigating the delivery ofhigh-intensity shocks to an opponent, while fear of receivinghigh-shock levels from that opponent simultaneously inhibitsaggression.

Provocation

This criteria is the extent to which negative affect would arise as adirect result of being attacked or provoked by another person. It de-


pends on the extent to which the participant perceives malicious or ag-gressive intent (i.e., how the participant perceives the situation, irre-spective of the actual intentions of the actor}. Endpoints are

1 = Trial I in a reaction time-competition study before any high-intensity provoking shocks have been received from an opponent(e.g.,Jeavons& Taylor, 1985)9 = learning that an opponent planned to deliver a shock that wastwice as high as one's pain threshold in a reaction time-competi-tion study (e.g., Taylor et al., 1979).

Frustration

This criteria is the extent to which an ongoing, goal-directed behavioris blocked or thwarted. The source of frustration could be another per-son or an event. Endpoints are

1 - participating in a reaction time-pain perception study inwhich the participant's task is to respond as quickly as possible toatone and in so doing, deliver a shock to one's partner(e.g., Pihl etal., 1984)9 = the crucial trials where the teacher-supervisor in a teacher-

learner study sees that he or she will not win an amount ($80)because the learner-partner has made too many mistakes on a sim-ple task (e.g., Gustafson, 1986a).

Self-Focused Attention

This is the degree to which the participant's attention is focused onself because of circumstances in the experimental setting that cause theparticipant to be the object of his or her attention and the degree towhich experimental situation induces self-evaluation for internal con-cerns or standards {but not self-presentational concerns). Endpointsare

1-2 = participating in a teacher-leaner study (e.g., Gustafson,1986a) where attention is directed toward supervising anotherperson9 = performing a task in front of a mirror and video camera (Baileyetal., 1983).

Received December 21, 1994Revision received August 18, 1995

Accepted August 22, 1995

AMERICAN PSYCHOLOGICAL ASSOCIATIONSUBSCRIPTION CLAIMS INFORMATION Today's Date:_

We provide this form to assisl members, institutions, and nonrnember individuals with any subscription problems. With theappropriate information we can begin a resolution. If you use the services of an agent, please do NOT duplicate claims throughthem and directly to us. PLEASE PRINT CLEARLY AND IN INK IF POSSIBLE.

PRINT FULL NAME OR KEY NAME OF INSTITUTION MEMBERORCT1STOMER NUMBER (MAY BEFOUND ON ANYPASTISSUE [ABED

DATE YOUR ORDER WAS MAILED (OR PHONED)

_PREPA1D CHKCK CHARGECHK:K/CARD CLEARED DATE:.

CITY STATE/roltNTRY

YOl!R NAME AND PHONE NUMBER

TITLE

<lf possible, send a copy, front and back, of yoar cancelled check lo help us in our n-scarvhof your claim, t

ISSUES: MISSING DAMAGED

VOLUME OR YEAR NUMBER OR MONTH

Thank you. Once a claim is received and resolved^ delivery of replacement issues routinely takes 4—6 weeks.

————————— (TO BE FILLED OUT BY APA STAFF) '

DATE RECEIVED:ACTION TAKEN: _STAFF NAME:

DATE OF ACTION; _INV. NO. & DATE:LABEL NO. & DATE:

Send this form lo APA Subscription Claims, 750 First Street, NE, Washington, DC 20002-4242

PLEASE DO NOT REMOVE. A PHOTOCOPY MAY BE USED.

alcohol and aggression: a meta-analysis on the moderating effects of inhibitory cues ... ·...

Documents