bojana gardijan 4th year march 16, 2010 mentor: a. Žmegač horvat

Bojana Gardijan4th year

March 16, 2010Mentor: A. Žmegač Horvat

destruction of heart tissue resulting from obstruction of the blood supply to the heart muscle

part of acute coronary syndrome, ACS non- STEMI

non ST-elevation MI STEMI

ST-elevation MI

diabetes hyperlipoproteinemia,

especially high LDL and low HDL

high blood pressure family history of

ischemic heart disease

obesity BMI>30 kg/m² age M>45, F>55 stress alcohol

a manifestation of coronary artery disease, also called ischemic heart disease

most common triggering event: disruption of an atherosclerotic plaque in an coronary artery clotting cascade sometimes results in total occlusion of the artery

chest pain (typically radiating to the left arm or left side of the neck)

shortness of breath (dyspnea) nausea, vomiting palpitations sweating anxiety

history and physical examination EKG CBC cardiac markers, preferably troponin T

(and complete biochemistry) differential diagnosis includes pulmonary

embolism, aortic dissection, pericardial effusion causing cardiac tamponade, tension pneumothorax, pancreatitis and esophageal rupture

MONA as soon as possiblemorphine, oxygen, nitrates, aspirin

clopidogrel, heparin, eptifibatide procedure of choice : PCI (percutaneous

coronary intervention) if unavailable: fibrinolysis

postinfarctial therapy: SAABstatins, ACEI, aspirin, beta blockers

varies greatly, depending on a person’s health, the extent of the heart damage and the treatment given

time to reperfusion is of great importance

a quick reaction saves lives

http://en.wikipedia.org/wiki/Myocardial_infarction

http://www.escardio.org