an introduction to the 12 lead ecg
DESCRIPTION
An Introduction to the 12 lead ECG. By the end of this lecture, you will be able to: Understand the 12 lead ECG in relation to the coronary circulation and myocardium Perform an ECG recording Identify the ECG changes that occur in the presence of an acute coronary syndrome. - PowerPoint PPT PresentationTRANSCRIPT
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An Introduction to the12 lead ECG
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12 Lead ECG Interpretation
By the end of this lecture, you will be able to:
• Understand the 12 lead ECG in relation to the coronary circulation and myocardium
• Perform an ECG recording
• Identify the ECG changes that occur in the presence of an acute coronary syndrome.
• Begin to recognise and diagnose an acute MI.
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What is a 12 lead ECG?
• Records the electrical activity of the heart (depolarisation and repolarisation of the myocardium)
• Views the surfaces of the left ventricle from 12 different angles
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Why do a 12 lead ECG?
• Monitor patients heart rate and rhythm
• Evaluate the effects of disease or injury on heart function
• Detect presence of ischaemia / damage
• Evaluate response to medications, e.g anti dysrhythmics
• Obtain baseline recordings before during and after surgical procedures
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Recording an ECG1. Explain procedure to patient,
obtain consent and check for allergies
2. Check cables are connected
3. Ensure surface is clean and dry
4. Ensure electrodes are in good contact with skin
5. Enter patient data
6. Wait until the tracing is free from artifact
7. Request that patient lies still.
8. Push button to start tracing
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Procedure (cont.)
Before disconecting the leads ensure the recording is -
Free from artifact
Paper speed is 25mm/sec
Normal standardisation of 1mv, 10mm
Lead placement is correctECG is labelled correctly
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Anatomy and Physiology Review
• A good basic knowledge of the heart and cardiac function is essential in order to understand the 12 lead ECG
• Anatomical position of the heart
• Coronary Artery Circulation
• Conduction System
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Anatomical Position of the Heart
• Lies in the mediastinum behind the sternum
• between the lungs, just above the diaphragm
• the apex (tip of the left ventricle) lies at the fifth intercostal space, mid-clavicular line
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Coronary Artery Circulation
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Coronary Artery Circulation
Right Coronary Artery• right atrium• right ventricle• inferior wall of left
ventricle• posterior wall of left
ventricle• 1/3 interventricular
septum
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Coronary Artery Circulation Left Main Stem Artery divides in two:Left Anterior Descending
Artery• antero-lateral surface of
left ventricle• 2/3 interventricular
septum
Circumflex Artery• left atrium• lateral surface of left
ventricle
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Coronary Artery Circulation
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The standard 12 Lead ECG6 Limb Leads 6 Chest Leads (Precordial leads)
avR, avL, avF, I, II, III V1, V2, V3, V4, V5 and V6
Rhythm Strip
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Limb leads Chest Leads
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Limb Leads3 Unipolar leads
• avR - right arm (+)
• avL - left arm (+)
• avF - left foot (+)
• note that right foot is a ground lead
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Limb Leads3 Bipolar Leads
form (Einthovens Triangle)
Lead I - measures electrical potential
between right arm (-) and left arm (+)
Lead II - measures electrical potential
between right arm (-) and left leg (+)
Lead III - measures electrical potential
between left arm (-) and left leg (+)
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Chest Leads6 Unipolar leads
Also known as precordial leads
V1, V2, V3, V4, V5 and V6 - all positive
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Chest Leads
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Think of the positive electrode as an ‘eye’…
the position of the positive electrode on the body determines
the area of the heart ‘seen’ by that lead.
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Surfaces of the Left Ventricle
• Inferior - underneath
• Anterior - front
• Lateral - left side
• Posterior - back
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Inferior Surface• Leads II, III and avF look UP from below to the inferior
surface of the left ventricle
• Mostly perfused by the Right Coronary Artery
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Inferior Leads
–II
–III
–aVF
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Anterior Surface• The front of the heart viewing the left ventricle and the
septum
• Leads V2, V3 and V4 look towards this surface
• Mostly fed by the Left Anterior Descending branch of the Left artery
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Anterior Leads
– V2
– V3
– V4
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Lateral Surface• The left sided wall of the left ventricle
• Leads V5 and V6, I and avL look at this surface
• Mostly fed by the Circumflex branch of the left artery
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Lateral Leads
V5, V6, I, aVL
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Posterior Surface• Posterior wall infarcts are rare
• Posterior diagnoses can be made by looking at the anterior leads as a mirror image. Normally there are inferior ischaemic changes
• Blood supply predominantly from the Right Coronary Artery
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Inferior II, III, AVF
Antero-SeptalV1,V2, V3,V4
Lateral I, AVL, V5, V6
Posterior V1, V2, V3
RIGHT LEFT
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ECG Waveforms• Normal cardiac axis is downward and to the
left• ie the wave of depolarisation travels from the
right atria towards the left ventricle• when an electrical impulse travels towards a
positive electrode, there will be a positive deflection on the ECG
• if the impulse travels away from the positive electrode, a negative deflection will be seen
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ECG Waveforms
• Look at your 12 lead ECG’s
• What do you notice about lead avR?
• How does this compare with lead V6?
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An Introduction to the 12 lead ECG
Part II
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Heart beat originates in the SA node
Impulse spreads to all parts of the atria via internodal pathways
ATRIAL contraction occurs Impulse reaches the AV node
where it is delayed by 0.1second
Impulse is conducted rapidly down the Bundle of His and Purkinje Fibres
VENTRICULAR contraction occurs
Basic electrocardiography
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•The P wave represents atrial depolarisation
•the PR interval is the time from onset of atrial activation to onset of ventricular activation
•The QRS complex represents ventricular depolarisation
•The S-T segment should be iso-electric, representing the ventricles before repolarisation
•The T-wave represents ventricular repolarisation
•The QT interval is the duration of ventricular activation and recovery.
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ECG Abnormalities
Associated with ischaemia
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Ischaemic Changes
• S-T segment elevation
• S-T segment depression
• Hyper-acute T-waves
• T-wave inversion
• Pathological Q-waves
• Left bundle branch block
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ST Segment• The ST segment represents period between ventricular
depolarisation and repolarisation.
• The ventricles are unable to receive any further stimulation
• The ST segment normally lies on the isoelectric line.
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ST Segment Elevation
The ST segment lies above the isoelectric line:
• Represents myocardial injury• It is the hallmark of Myocardial Infarction• The injured myocardium is slow to repolarise and
remains more positively charged than the surrounding areas
• Other causes to be ruled out include pericarditis and ventricular aneurysm
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ST-Segment Elevation
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Myocardial Infarction
• A medical emergency!!!
• ST segment curves upwards in the leads looking at the threatened myocardium.
• Presents within a few hours of the infarct.
• Reciprocal ST depression may be present
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ST Segment DepressionCan be characterised as:-
• Downsloping
• Upsloping
• Horizontal
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Horizontal ST Segment Depression
Myocardial Ischaemia:• Stable angina - occurs on exertion, resolves with
rest and/or GTN• Unstable angina - can develop during rest. • Non ST elevation MI - usually quite deep, can be
associated with deep T wave inversion.• Reciprocal horizontal depression can occur during
AMI.
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Horizontal ST depression
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ST Segment Depression
Downsloping ST segment depression:-
• Can be caused by digoxin.
Upward sloping ST segment depression:-
• Normal during exercise.
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T waves
• The T wave represents ventricular repolarisation
• Should be in the same direction as and smaller than the QRS complex
• Hyperacute T waves occur with S-T segment elevation in acute MI
• T wave inversion occurs during ischaemia and shortly after an MI
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T waves
Other causes of T wave inversion include:• Normal in some leads• Cardiomyopathy• Pericarditis• Bundle Branch Block (BBB)• Sub-arachnoid haemorrhage
• Peaked T waves indicate hyperkalaemia
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Hyperacute T waves
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Inferior T-wave inversion
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T wave inversion in an evolving MI
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QRS Complex
May be too broad ( more than 0.12 seconds)
• A delay in the depolarisation of the ventricles because the conduction pathway is abnormal
• A Left Bundle Branch Block can result from MI and may be a sign of an acute MI.
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Wide QRS (LBBB)
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QRS Complex
• May be too tall.• This is caused by an increase in muscle mass in
either ventricle. (Hypertrophy)
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Q Waves
Non Pathological Q waves
Q waves of less than 2mm are normal
Pathological Q waves
Q waves of more than 2mm
indicate full thickness myocardial
damage from an infarct
Late sign of MI (evolved)
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Pathological Q waves
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Any Questions?
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ECG Interpretation in
Acute Coronary Syndromes
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The ECG in ST Elevation MI
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The Hyper-acute Phase
Less than 12 hours
• “ST segment elevation is the hallmark ECG abnormality of acute myocardial infarction” (Quinn, 1996)
• The ECG changes are evidence that the ischaemic myocardium cannot completely depolarize or repolarize as normal
• Usually occurs within a few hours of infarction
• May vary in severity from 1mm to ‘tombstone’ elevation
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The Fully Evolved Phase
24 - 48 hours from the onset of a myocardial infarction
• ST segment elevation is less (coming back to baseline).
• T waves are inverting.
• Pathological Q waves are developing (>2mm)
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The Chronic Stabilised Phase
• Isoelectric ST segments
• T waves upright.
• Pathological Q waves.
• May take months or weeks.
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Reciprocal Changes
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Reciprocal Changes
• Changes occurring on the opposite side of the myocardium that is infarcting
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Reciprocal Changes
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The ECG in Non ST Elevation MI
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Non ST Elevation MI
• Commonly ST depression and deep T wave inversion
• History of chest pain typical of MI
• Other autonomic nervous symptoms present
• Biochemistry results required to diagnose MI
• Q-waves may or may not form on the ECG
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Changes in NSTEMI
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The ECG in Unstable Angina
• Ischaemic changes will be detected on the ECG during pain which can OCCUR AT REST
• ST depression and/or T wave inversion
• Patients should be managed on a coronary care unit
• May go on to develop ST elevation
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Unstable AnginaECG during pain
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Any Questions?
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Quick Quiz
How well have you listened?
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Quick Quiz
Mr Jones is diagnosed as having had an anterior MI. On which leads would you expect to see the main changes?
(a) II, III and avL.
(b) I and avL.
(c) V2 - V4.
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Quick Quiz
The Right Coronary Artery mainly supplies:
(a) The inferior surface of the heart?
(b) The anterior surface of the left ventricle?
(c) The lateral surface of the heart?
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Quick Quiz
Mr Jackson has ECG changes suggestive of an MI on leads II, III and avF. Which surface of his heart is affected?
(a) The anterior surface.
(b) The lateral surface.
(c) The inferior surface.
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Quick Quiz
The Circumflex artery mainly supplies:
(a) The right ventricle?
(b) The lateral surface of the heart?
(c) The ventricular septum?
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Quick Quiz
The Left Anterior Descending Artery mainly
supplies:
(a) The right ventricle?
(b) The anterior and septal surfaces of the left ventricle?
(c) The right atrium?
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Quick Quiz
Mrs Brown requires PTCA to her Circumflex artery after complaining of unstable angina symptoms. Her 12 lead ECG shows ST depression and T wave inversion in what leads?
(a) I, avL, V5 and V6
(b) II, III and avL
(c) V3 and V4
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A 55 year old man with 4 hours of “crushing” chest pain.
Acute inferior myocardial infarction (with reciprocal changes)
ST elevation in the inferior leads II, III and aVF
reciprocal ST depression in the anterior leads
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A 63 Year Old woman with 10 hours of chest pain and sweatingCan you guess her diagnosis?
Acute anterior-lateral myocardial infarction
ST elevation in the anterior leads V1 - 6, I and aVL
reciprocal ST depression in the inferior leads
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Which one is more tachycardic during this exercise test?
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Any Questions?
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Thanks for paying attention.
I hope you have found this session useful.