beat the heart blocks - cherylherrmann.com · av nodal blocks first degree av block second degree...
TRANSCRIPT
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Beat the Heart Blocks
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Beat the Blocks
• Complete RBBB
• Complete LBBB
• Bifascicular,
Trifascicular Blocks
• 1st degree Heart Block
• 2nd degree Heart Block
• 3rd degree Heart Block
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The Electrical Conduction System
SA node 60-100 BPM
AV node 40-60 BPM
Purkinje cells 20-40 BPM
Creates an electrical impulse and transmits it in an
organized manner to the rest of the myocardium
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Basic Components of the Complex
Deflections & Segments
• P Wave– Rounded, < 2-3 mm, in hypertrophy
• QRS Segment– < .12 sec. & > 5 mm, transition occurs V3 or V4
– presence of Q normal in children/elderly
– Q wave sig. > 0.5 mm
• T Wave – < 5-10 mm, peaked in K+
• U Wave– Follows T wave, present in K+
• ST Segment– Isoelectric, sig. If > +1.0 above or below
baseline
– Depression = ischemia
– Elevation = injury• Bundle Branch Blocks
are identified by duration
of QRS complex
• Normal QRS duration
60 – 100 msec
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Right Bundle Branch Block
RBBB
Source: Garcia 12 Lead EKG 13:2 & 13:3
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Right Bundle Branch Block
RBBB
Causes
• Chronically increased right ventricular pressure, as in
cor pulmonale
• Right ventricular hypertrophy
• A sudden increase in right ventricular pressure with stretch, as in pulmonary embolism.
• Congenital heart disease (atrial septal defect)
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RBBB Criteria
• QRS > 0.12 sec or 120msec
• Slurred S wave leads I & V6
• RSR’ pattern V1
Easy way:
• V1 = Positive, QRS > 0.12
sec
• Rabbit Ears
Source: Garcia. 12 Lead ECG
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RBBB
• Half a rabbit ear
• QRS mostly postive
Source: Garcia. 12 Lead ECG
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Slurred S wave Lead I & V6Positive V1
QRS = 136 ms
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QRS = 134 ms
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Left Bundle Branch Block
LBBB
Source: Garcia 12 Lead EKG 13:19
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Left Bundle Branch Block
LBBB
Causes
• Dilated cardiomyopathy
• CAD
• Hypertension
• Infiltrative diseases of
the heart
• Benign or idiopathic
causes
• Higher mortality
than RBBB
• Most often seen in
large Anterior MIs
• Lower EFs
• Often seen in later
stages of Heart
Failure
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LBBB Criteria
• QRS > 0.12 sec or 120msec
• Broad, monomorphic R
waves in I & V6, with no Q
waves
• Broad, monomorphic S
waves in V1; may have a
small r wave
Easy way
• QRS > 0.12 sec
• Negative V1 = Carrot
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QRS = 140 ms
Negative V1Broad, monomorphic R waves in I & V6,
with no Q waves
Broad, monomorphic S waves in V1
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QRS = 144 ms
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LVH, LBBB, LAD
QRS = 134 ms
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BBB = QRS > 0.12sec or 120msec
• LBBB = QRS > 0.12
sec, Negative QRS
in V1 (carrot)
• RBBB = QRS >
0.12sec; Positive
QRS in V1 (rabbit
ears)
LBBB
RBBB
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Incomplete Bundle Branch Block
QRS in no man’s land
Incomplete LBBB
• QRS 100 – 110 msec
• LBBB pattern
Incomplete RBBB
• QRS 100 – 110 msec
• RBBB pattern
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QRS = 108ms
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QRS = 110 ms
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QRS = 108 ms
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QRS = 106 ms
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AV Nodal Blocks
First Degree AV Block
Second Degree AV Block Type I
Second Degree AV Block Type II
Third Degree AV Block
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The Electrical Conduction System
SA node 60-100 BPM
AV node 40-60 BPM
Purkinje cells 20-40 BPM
Creates an electrical impulse and transmits it in an
organized manner to the rest of the myocardium
Heart Blocks occur due
to AV node disease
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Basic Components of the Complex
Deflections & Segments
• P Wave– Rounded, < 2-3 mm, in hypertrophy
• QRS Segment– < .12 sec. & > 5 mm, transition occurs V3 or V4
– presence of Q normal in children/elderly
– Q wave sig. > 0.5 mm
• T Wave – < 5-10 mm, peaked in K+
• U Wave– Follows T wave, present in K+
• ST Segment– Isoelectric, sig. If > +1.0 above or below
baseline
– Depression = ischemia
– Elevation = injury• AV node correlates to
the PR Interval
• Normal PR interval
120 – 200 msec
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First-Degree AV Block
• Atrioventricular (AV) Block occurs when the AV Node fails to properly conduct the impulses from the atria to the ventricles
• Conduction to the ventricles will occur every time……it….will……… just….be………….. delayed.
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AV node’s function is to slow down the conduction from the SA node to allow the
ventricles to fill up with blood.
In 1st degree block, the AV node is slowing down the conduction a bit too much.
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First-Degree AV Block
PR interval > 200 msec
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First-Degree AV Block
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Characteristics: 1st Degree
PR 320 msec
✓ P waves present with same morphology✓ QRS are normal✓ P:QRS has a 1:1 ratio (no missing QRS)✓ Atrial Rate is regular✓ Ventricular Rate is regular✓ PR > 200 msec and remain constant✓ Looks like a normal sinus rhythm
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✓ P waves present with same morphology✓ QRS are normal✓ P:QRS has a 1:1 ratio (no missing QRS)✓ Atrial Rate is regular✓ Ventricular Rate is regular✓ PR > 200 msec and remain constant✓ Looks like a normal sinus rhythm
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Possible Causes
• Acute myocarditis
• Acute MI
• Cardiomyopathy
• Chronic Aortic Regurgitation
• Acute conduction system disease
• Hypothyroidism
• Hyperkalemia
• Mild digitalis toxicity
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Treatment
If pulse is normal, the patient will usually be asymptomatic so no treatment is needed.
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Marriage
Relationship
and the Blocks
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Second-Degree AV Block
Two main types• Mobitz I or Wenckebach
• Mobitz II
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The Electrical Conduction System
SA node 60-100 BPM
AV node 40-60 BPM
Purkinje cells 20-40 BPM
Creates an electrical impulse and transmits it in an
organized manner to the rest of the myocardium
Heart Blocks occur due
to AV node disease
![Page 40: Beat the Heart Blocks - cherylherrmann.com · AV Nodal Blocks First Degree AV Block Second Degree AV Block Type I Second Degree AV Block Type II Third Degree AV Block. The Electrical](https://reader033.vdocuments.net/reader033/viewer/2022050413/5f8a1591ee3887455b4b09d1/html5/thumbnails/40.jpg)
Second Degree Type I
This block is also a delay in the AV node/AV junction
but instead of having the same delay, it gradually gets longer then drops a
longer…..longer………drop!it’s a Wenckebach!
Mobitz I AV Block
Wenckebach
Second Degree AV Block
Type I
QRS
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Mobitz I: 2nd Degree AV Block or Wenckebach
• PR interval progressively lengthens until a P wave is eventually blocked.
• PR interval after dropped QRS is the shortest• Irregular Regular --- pattern
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Examples of Mobitz I Second-Degree AV Block
Wenckebach
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longer longer
drop!
Characteristics Wenckebach
✓ P waves present with same morphology
✓ QRS are normal and narrow
✓ Not a 1:1 ratio (missing QRS complexes)
✓ Atrial Rate is regular
✓ Ventricular Rate is irregular
✓ PR not constant (gets progressively longer)
Look forthe
pattern
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Possible Causes
• Primary conduction system disease
• Ischemic heart disease
• Inferior wall MI
• Cardiomyopathy
• Rheumatic Fever
• Intense vagal stimulation
• Electrolyte imbalance
• Beta or Calcium channel blockers
• ***Digitalis toxicity
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TreatmentUsually benign – no treatment needed
Treat Only if patient is symptomatic
SOB, hypotension, weakness, dizziness
AtropineFirst dose: 0.5mg bolus
Repeat Q3-5 minMax 3mg
Transcutaneous PacingAtropine
•Blocks vagal effects on the SA & AV nodes•Enhances conduction through AV node
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Marriage
Relationship
and the Blocks
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Indicates a problem below AV node/junctional area
Bundle of His or Bundle Branches
Mobitz II AV Block
Second Degree Type II
Second Degree AV Block Type II Classical
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There’s no warning!
more serious because…
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• One or more P waves are blocked• PR intervals are constant throughout the strip.• Dropped QRS will march out with the p waves
Mobitz II Second-Degree AV Block
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Mobitz II Second-Degree AV Block
• One or more P waves are blocked• PR intervals are constant throughout the strip.• Dropped QRS will march out with the p waves
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Examples of Mobitz II Second-Degree AV Block
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✓P waves present with same morphology✓QRS are normal and narrow✓Not a 1:1 ratio✓Atrial Rate is regular✓Ventricular rate is irregular (missing QRS complexes)
✓PR is constant and complex is the same
Characteristics Mobitz Type II
nor
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Possible Causes
• Primary conduction system disease
• Ischemic heart disease
• Inferior wall MI
• Cardiomyopathy
• Rheumatic Fever
• Intense vagal stimulation
• Electrolyte imbalance
• Beta or Calcium channel blockers
• ***Digitalis toxicity
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Treatment
If patient is symptomaticSOB, hypotension, weakness, dizziness
AtropineFirst dose: 0.5mg bolus
Repeat Q3-5 minMax 3 mg
Atropine •Blocks vagal effects on the SA & AV nodes•Enhances conduction through AV node
• Transcutaneous Pacing or at bedside• Stop Beta Blockers, Digoxin and Calcium
Channel Blocker• Stop narcotics• Prepare for temporary or permanent pacer
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Marriage
Relationship
and the Blocks
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Third-Degree or Complete AV Block
Two pacemakers by definition:– One supra-ventricular– One ventricular
***No communication between atria and ventricles
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Third Degree AV Block
Complete Heart Block
SA nodeP waves
Atrial Contracting
60 – 100 bpm
AV Node
Blocked
HIS BundleNarrow QRS complex
Rate 40 – 60 bpm
Junctional Rhythm
Purkinje CellsWide QRS complex
Rate < 40
Idioventricular Rhythm
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The most serious block because no impulses are reaching the ventricles
ventriclesSA Node
Atria
AV Node
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Third-Degree or Complete AV Block
• No communication whatsoever between atria and ventricles
• Two pacemakers by definition:– One supraventricular, one ventricular
• Atrial and ventricular rates are typically different.• Key feature is that PR intervals are completely changing
and have no effect on the ventricular rate.
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p p p p p p p p pp
Characteristics Complete Heart Block
✓ P waves present with same morphology
✓ QRS are narrow or wide
✓ Not a 1:1 ratio
✓ Atrial Rate is regular (p waves march out)
✓ Ventricular Rate is regular (QRS march out)
✓ PR is not constant
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Junctional Escape Rhythm Rate > 40
Narrow QRS as the impulse starts In the AV Node
Ventricular Escape Rhythm Rate < 40Wide QRS as the impulse starts In the Purkinje fibers
To compensate HIS bundle or Purkinje Fibers kick in
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Examples Third Degree AV Block
Third Degree AV Block with Junctional (Narrow) Escape Rhythm
Third Degree AV Block with Ventricular (Wide) Escape Rhythm
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Possible Causes
• Anterior or Inferior MI
• Myocardial ischemia
• Cardiomyopathy
• AV nodal damage
• Rheumatic Fever
• Electrolyte imbalance
• Digitalis/beta blocker/calcium channel blocker toxicity
• Mitral or Aortic valve replacement complications
• Cardiac cath/angioplasty
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TreatmentDepends on location of the block
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Treatment
• If Asymptomatic– Observe and treat causes
– External pacer at bedside
• If Symptomatic SOB, Dizziness, Hypotension
– Increase ventricular rate
– External pacing
– Prepare for temporary or permanent pacing
– Dopamine 2-10mcg/kg/min
– Epinephrine 2-10 mcg/min
–
If the block is lower in Purkinje System area (QRS is WIDE)
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Marriage
Relationship
and the Blocks
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ReviewHEART BLOCKS KEY POINTS
1st DEGREE – not actually a block/merely a delay in
conduction. Appears to be a NSR with a PR interval >0.20
2nd DEGREE TYPE I – each beat is progressively delayed
until one is blocked. PR gets longer and longer until a QRS
complex is dropped.
2nd DEGREE TYPE II –the AV node selectively lets some
beats through and blocks others. MORE SERIOUS of the
two.
3rd DEGREE – complete block at the AV node. 2 separate
pacemakers
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✓P waves present with same morphology✓QRS are normal and narrow✓P:QRS has 1:1 ratio✓Both atrial/vent rate is regular/looks like SR with longer PR✓PR > 0.20 and constant
✓P waves present with same morphology✓QRS are normal and narrow✓Not a 1:1 ratio✓Atrial Rate is regular✓Ventricular Rate is irregular✓PR not constant (gets progressively longer)
✓P waves present with same morphology✓QRS are normal and narrow✓Not a 1:1 ratio✓Atrial Rate is regular✓Ventricular rate is irregular✓PR is constant with Dropped QRS
✓P waves present with same morphology✓QRS are narrow or wide✓Not a 1:1 ratio✓Atrial Rate is regular✓Ventricular Rate is regular✓PR is not constant
1st Degree AV block
2ND DegreeType I
2nd DegreeType II
3rd Degree
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Practice
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Practice & Application Time
For each EKG
– Identify any BBB
present
– Identify any
heart blocks
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EKG 1
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EKG 2
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EKG 3
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EKG 4
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Heart Blocks
5
6
7
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EKG 8
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Answers
1. RBBB
2. LBBB
3. RBBB with ST elevation anterior, septal, lateral leads →
cath lab
4. RBBB + Third Degree (Complete) Heart Block with
ventricular escape rhythm → pacemake3rr
5. Wenckebach
6. 2nd Degree Heart Block, Mobitz II
7. 3rd Degree Heart Block with ventricular escape rhythm
8. Sinus Bradycardia with 1st Degree AV Block and LBBB