damage to “where” pathway€¦ · • acalculia • language • agraphia • apraxia....
TRANSCRIPT
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Damage to “where” pathway
Abnormal motion processing & Visuspatial neglect
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Akinetopsia
• Clinical features• Can’t see moving objects
(as if under strobe lights); can see still objects– People appear suddenly
• Neuropathology– BL lesion to area MT (V5;
T-O-P junction)– UL lesions cause subtle
defects
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Akinetopsia
• Clinical features– Can’t see moving objects (as
if under strobe lights); can see still objects
– People appear suddenly
• Neuropathology– BL lesion to area MT (V5;
T-O-P junction)– UL lesions cause subtle
defects
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Topographagnosia
– Inability to navigate routes using familiar landmarks -deficit in familiar scene perception
– right ventral temporo-occipital lesions like Right lingual gyrus
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Spatial relationshipsdistorted
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Parietal lobe
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Parietal lobe
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Neglect syndrome
(right parietal association cortex)
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Left parietal
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Parietal lobe
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• Acalculia• Language• Agraphia• Apraxia
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Bilateral Parietal Damage (Balint's Syndrome)
• Impaired control over the focus of visual attention due to inattentional amnesia
• Complex defects in perception of visual object structure, motion and depth.
• Neglect (hemifield)
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Bilateral Parietal Damage (Balint's Syndrome)
• SIMULTANAGNOSIA : Inability to interpret the totality of a picture scene (can identify individual portions of the whole picture)
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Bilateral Parietal Damage (Balint's Syndrome)
• Simultanagnosia: Inability to interpret the totality of a picture scene (can identify individual portions of the whole picture)
• Optic ataxia: Defects of visually guided hand movement
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