eps-os
DESCRIPTION
epsTRANSCRIPT
![Page 1: EPS-OS](https://reader033.vdocuments.net/reader033/viewer/2022051621/55cf9309550346f57b9b2fe7/html5/thumbnails/1.jpg)
ERYTHRO PAPULOSQUAMOUS ERUPTION
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Erythro-Papulo-Squamous (EPS)
CHARACTERIZED BY ERYTHEMA,
PAPULES OR PLAQUES AND SCALLING
TRUE EPS & EPS-LIKE
![Page 3: EPS-OS](https://reader033.vdocuments.net/reader033/viewer/2022051621/55cf9309550346f57b9b2fe7/html5/thumbnails/3.jpg)
A. TRUE EPS1. PSORIASIS
2. P. ROSEA
3. SEBORRHEIC DERMATITIS
4. ERYTHRODERMA
5. PARAPSORIASIS
6. PITYRIASIS RUBRA PILARIS
7. LICHEN PLANUS
8. LICHEN STRIATUS
9. LICHEN NITIDUS
![Page 4: EPS-OS](https://reader033.vdocuments.net/reader033/viewer/2022051621/55cf9309550346f57b9b2fe7/html5/thumbnails/4.jpg)
B. EPS - LIKE
1. DERMATOFITOSIS
2. T. VERSIKOLOR
3. DRUG ERUPTION
4. SYPHILIS II
5. LUPUS ERYTHEMATOSUS
6. MORBUS HANSEN
7. MYCOSIS FUNGOIDES
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PSORIASISPSORIASIS
* IS A COMMON PAPULO SQUAMOUS DISEASE
* E/ ?
* SHOWING WIDE VARIATION IN SEVERITY & IN
DISTRIBUTION
* CHRONIC
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EPIDEMIOLOGY :
- PSORIASIS IS FOUND ALL OVER THE WORLD
- MALES FEMALES
- THE ONSET OF THE DISEASE IS LESS COMMON
IN THE VERY YOUNG & THE ELDERY
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ETIOLOGY & PATHOGENESIS
AT THE CELLULAR LEVEL IT IS ACCEPTED THAT
PSORIATIC KERATINOCYTE DIFFERS FROM THE
NORMAL KERATINOCYTE GENETICALLY IN ITS
RESPONSE TO VARIOUS STIMULI
ENDOGENOUS & EXTERNAL STIMULI
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CLINICAL MANIFESTATIONS
- A SHARPLY DEFINED BORDER, A BRIGHT RED
COLOR & A SILVERY - WHITE SCALE DELINEATE
THE LESION OF PSORIASIS
- SITES OF PREDILECTION: THE ELBOWS &KNEES,
THE SCALP & LUMBO SACRAL SKIN
- SUBTLE DISTORTIONS OF NAILS, MUCOSAL
CHANGES, ISOMORPHIC PHENOMENON
![Page 9: EPS-OS](https://reader033.vdocuments.net/reader033/viewer/2022051621/55cf9309550346f57b9b2fe7/html5/thumbnails/9.jpg)
CLINICAL FORMS OF PSORIASIS
- COMMON PLAQUE OR NUMULAR PSORIASIS
- INVERSE OR FLEXURAL PSORIASIS
- GUTTATE PSORIASIS
- FOLLICULAR PSORIASIS
- PALMAR PSORIASIS
- PUSTULAR PSORIASIS
- EXFOLIATIVE PSORIASIS
- PSORIATIC ARTHRITIS
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HISTOPATHOLOGY
- ACANTHOSIS WITH ELONGATION OF THE RETE RIDGES
- ELONGATION OF THE DERMAL PAPILLAE
- PARAKERATOSIS
- MUNRO’S MICROABSCESSES
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TREATMENT
- TOPICAL : * SALICYLIC ACID
* TARS : LCD
* CORTICOSTEROIDS
* SUN - UV LIGHT THERAPHY
ANTHRALIN GOECKERMAN TECHNIQUE
& THE INGRAM TECHNIQUE
* PUVA/ NARROW BAND UVB
* CALCIPOTRIOL
* TACROLIMUS
* PIMECROLIMUS
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- SYSTEMIC : * ANTIMITOTIC AGENTS : METHOTREXATE
* ETRETINATE
* CYCLOSPHORIN
- DIALYSIS
PROGNOSIS QUO AD VITAM TYPE OF PSORIASIS
QUO AD FUNCTIONAM AD BONAM
QUO AD SANATIONAM DUBIA AD MALAM
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![Page 14: EPS-OS](https://reader033.vdocuments.net/reader033/viewer/2022051621/55cf9309550346f57b9b2fe7/html5/thumbnails/14.jpg)
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SEBORRHEIC DERMATITISSEBORRHEIC DERMATITIS
CHRONIC DERMATOSIS CHARACTERIZED BY
REDNESS & SCALING
ITS OCCURS IN THE AREAS OF THE SKIN IN WHICH
THE SEBACEOUS GLANDS ARE MOST ACTIVE:
FACE, SCALP, IN THE BODY FOLDS, PRESTERNAL REGION
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ETIOLOGY ?
MANY HYPOTHESES HAVE BEEN MADEAS TO ITS CAUSE
- SEBORRHEA
- PITYROSPORUM OVALE INFECTION
- INFECTION BY CANDIDA OR STAPHYLOCOCCI
- EMOTIONAL RESPONSES TO STRESS OR FATIQUE
- ABNORMAL DIET
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EPIDEMIOLOGY
- AGE : * INFANCY
* PUBERTY
* > 50 YEARS
- SEX : MALES
- INCIDENCE : VERY COMMON
- PREDISPOSING FACTOR : OFTEN A GENETIC DIATHESIS
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CLINICAL MANIFESTATIONS
INFANCY * CRADLE CAP
* GLABROUS : FLEXURAL, DIAPER AREA & TRUNK
* GENERALIZED : LEINER’S DISEASE
ADULTS
* SCALP: PITYRIASIS SICCA
* FACIAL, FLEXURAL & TRUNCAL
* GENERALIZED: ERYTHRODERMA
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LABORATORY FINDINGS
HISTOPATHOLOGY
DIFFERENTIAL DIAGNOSIS :
- ATOPIC DERMATITIS
- ALLERGIC AND IRRITANT CONTACT DERMATITIS
- PITYRIASIS ROSEA
- DERMATOPHYTE INFECTION
- CANDIDIASIS
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TREATMENT :* CONSERVATIVE
- SHAMPOO
- EMOLLIENTS & CREAMS
* INTENSIVE
- KETOCONAZOLE CREAM
- TOPICAL STEROIDS
- TAR PREPARATIONS
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PROGNOSIS:
QUO AD VITAM: AD BONAM QUO AD FUNCTIONAM: AD BONAM QUO AD SANATIONAM: DUBIA AD BONAM
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![Page 24: EPS-OS](https://reader033.vdocuments.net/reader033/viewer/2022051621/55cf9309550346f57b9b2fe7/html5/thumbnails/24.jpg)
PITYRIASIS ROSEAPITYRIASIS ROSEAPROBABLY CAUSED BY AN INFECTIOUS AGENT
AGE : 10 - 35 YEARS
DURATION OF LESIONS :
- A “ HERALD PATCH “ PRECEDES THE
EXANTHEMATOUS PHASE
- THE EXANTHEMATOUS PHASE DEVELOPS OVER A
PERIOD OF 1 TO 2 WEEKS
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PHYSICAL EXAMINATION :
-SKIN SYMPTOMS : PRURITUS
ABSENT, MILD OR SEVERE
- SKIN LESIONS
* HERALD PATCH 2 - 5 CM, BRIGHT RED, SCALE
* FINE SCALING MACULES AND PAPULES WITH MARGINAL COLLARETTE
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CHARACTERISTIC PATTERN OF THE LESIONS:
THE LONG AXES OF THE LESIONS FOLLOW THE
LINES OF CLEAVAGE
“CHRISTMAS TREE “ DISTRIBUTION
TRUNK & PROXIMAL OF THE ARMS & LEGS
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DIFFERENTIAL DIAGNOSIS
- DRUG ERUPTIONS
- T. CORPORIS
- SECONDARY SYPHILIS
- T. VERSICOLOR
TREATMENT
- TOPICAL : * POWDER
* CREAM ( CORTICO STEROID )
- SYSTEMIK : ANTIHISTAMINES
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PROGNOSIS :
QUO AD VITAM : AD BONAM
QUO AD FUNCTIONAM : AD BONAM
QUO AD SANATIONAM : AD BONAM
SPONTANEOUS REMISSION IN 6 - 12 WEEKS
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HERALD PATCH
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ERYTHRODERMAERYTHRODERMAREACTION PATTERN OF THE SKIN CHARACTERIZED BY
GENERALIZED, CONFLUENT REDNESS, SCALING &
ASSOCIATED WITH SYSTEMIC SYMPTOMS
AGE ~ ETIOLOGY
ETIOLOGY
- EXTENSION OF PREEXISTING DERMATOLOGIC
DISEASE
PSORIASIS, ATOPIC DERMATITIS, SEBORRHEIC
DERMATITIS
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- DRUGS REACTIONS
- SEZARY SYNDROME
- EXTENSION OF SYSTEMIC DISEASE
LUPUS ERYTHEMATOSUS
SKIN LESION UNIVERSALIS
SKIN IS RED, THICKENED & SCALY
LABORATORY & HISTOPATHOLOGY ~ ETIOLOGY
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TREATMENT ~ ETIOLOGY
- THE PATIENT SHOULD BE HOSPITALIZED
- TOPICAL : EMOLLIENTS
- SYSTEMIC : CORTICOSTEROID
PROGNOSIS ~ ETIOLOGY
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![Page 34: EPS-OS](https://reader033.vdocuments.net/reader033/viewer/2022051621/55cf9309550346f57b9b2fe7/html5/thumbnails/34.jpg)
TRUE EPS
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PITYRIASIS RUBRA PILARIS
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PARAPSORIASIS EN PLAQUES
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PARAPSORIASIS
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LICHEN PLANUS
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LICHEN PLANUS
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LICHEN NITIDUS
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LICHEN STRIATUS
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EPS - LIKE
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TINEA CORPORIS
![Page 44: EPS-OS](https://reader033.vdocuments.net/reader033/viewer/2022051621/55cf9309550346f57b9b2fe7/html5/thumbnails/44.jpg)
TINEA CORPORIS
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TINEA VERSICOLOR
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MORBUS HANSEN
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Drug eruption(erythema multiforme)
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Maculopapular drug eruption e.c ampicillin
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THANK YOU