fascial space infections
TRANSCRIPT
FASCIAL SPACE INFECTIONS
Dr. Smijal GMMDS 2016
CONTENTS
• Introduction• Definition and classification-Fascial space infections• Various fascial space infections in head and neck • Conclusion• References
INTRODUCTION• Oral cavity consists of more than 500 bacterial taxa, several fungal
species, few protozoal genera and many viruses as normal residents• Occurrence of infectious disease is determined by the interaction of
host, the organism and the environment• In healthy state there is a balance among these factors and when the
balance is lost disease occurs
• An oral infection can originate in the dental pulp and extend through the root canals of the tooth into the periapical tissues or
• It may originate in the superficial periodontal tissues and subsequently disperse through the bone.
• Later it may perforate the outer cortical bone and spread in various tissue spaces or discharge onto a free mucous membrane
• Fascial spaces are fascia lined areas that can be eroded or distended by purulent exudate. These areas are potential spaces that do not exist in healthy people but become filled during infections
FASCIAL SPACESDefinition –
The fascial spaces in head and neck are the potential spaces between the various layers of fascia normally filled with loose connective tissue and bounded by anatomical barriers, usually of bone, muscle or fascial layers.
( Moore-1975)
Shapiro defined fascial spaces as potential spaces between layer of fascia. Its filled with loose connective tissues and various anatomical structures
Compartments Neurovascular structuresClefts Loose areolar connective tissue
• Routes by which an infection can spread are• Lymphatic system• Blood stream• Through tissues
• Local changes:• Pain• Swelling• Restriction of movement• Surface erythema• Pus formation
• Systemic changes• Toxic appearance• Fever • Lymphadenopathy • Malaise • Increased white blood cell count
• Most odontogenic infections arise as a sequel of pulp necrosis caused by caries, trauma, periodontitis, etc.
• They range from periapical abscesses to superficial and deep infections in neck.
• Some resolve with little consequence and some lead to severe infections of head and neck region.
NATURAL HISTORY OF PROGRESSION OF ODONTOGENIC INFECTION• Once bacteria has reached periapical tissue an active lesion is
established• Infection will spread equally in all directions, but preferentially along
the lines of least resistance • Spread through cancellous bone until it encounters a cortical plate• If thin cortical plate, infection erodes through bone and enters soft
tissue• It appears in predictable locations
Location of infection from a specific tooth is determined by• The thickness of bone overlying the apex of the tooth• The relationship of the site of perforation of bone to muscle
attachments of the maxilla and mandible
1vestibular abscess2buccal space3 palatal abscess4sublingual space5submandibular space6maxillary sinus
Cervical Fascia described under :- a) Superficial fascia b) Deep fascia
SUPERFICIAL FASCIA: • Layer of dense CT that courses deep to the SC tissue
throughout the body• SC space is defined as tissues lying superficial to superficial
fascia• Also known as tela subcutanea or hypodermisDEEP FASCIA:• Formed by dense, organized connective tissue• “Invests” deep structures such as muscles• Creates compartments that contain / direct spread of infection• Limits outward expansion of muscles so that veins are
compressed moving blood toward heart (musculovenous pump)
• Superficial fascia• Deep cervical fascia
• Anterior layer/ Superficial/ Investing layer
• Investing fascia• Parotideomasseteric• Temporal
• Middle layer• Sternohyoid-omohyoid
division• Sternothyroid-
thyrohyoiddivision• Visceral division
• Buccopharyngeal • Petracheal • Retropharyngeal
• Posterior layer• Alar division• Prevertebral division
• Abscesses located either superficial to or within the tissue space immediately deep to the superficial cervical fascia are treated by simple incision and drainage
• Superiorly attaches to:• Superior nuchal line of occipital bone(a)• Spinous processes of cervical vertebrae and nuchal
ligament (b)• Mastoid processes of temporal bones ©• Zygomatic arches (d)• Inferior border of mandible (e)• Hyoid bone (f)
• Inferiorly attaches to:• Manubrium (g)• Clavicles (h)• Acromion (i)
• Just above sternum this layer splits around the anterior and posterior surfaces of manubrium forming the Suprasternal space
• Completely surround the neck• Forms floor of submandibular space• Envelopes
• Sternocleidomastoid• Trapezius• Submandibular• Parotid
ANTERIOR LAYER
• Middle layer divided into three divisions. The first two are sternohyoid-omohyoid and the sternothyroid-thyrohyoid divisions
• Surround the corresponding strap muscles of the neck between the hyoid bone and the clavicle
• Primary surgical significance of these layers is that they must be divided in the mid line in a surgical approach to the trachea or thyroid gland
• Not directly involved in head and neck infections because they do not lie on the major routes that an orofacial infection may follow to the mediastinum or chest wall
MIDDLE LAYER
Visceral layer of deep cervical fascia
• Lies deep to the infrahyoid muscles, following their origin behind the sternum, and splits to enclose the thyroid, trachea, pharynx, and oesophagus
• Attached superiorly to cricoid cartilage (e), thyroid cartilage(d), and hyoid bone(f)
• Attached posteriorly to prevertebral fascia• Is referred to pretracheal anteriorly (a) and
retrovisceral (c) posteriorly
POSTERIOR LAYER
Vertebral layer of Deep cervical fascia
• Forms a tubular sheath for the vertebral column and the muscles associated with it extending from the base of the skull to T3 vertebrae
• Begins from cervical spinous processes (a) and the ligamentum nuchae(b)
• Covers the floor of the posterior triangle of the neck
• Attaches to the transverse processes (c) and divides into two layers/ laminae as it passes behind the esophagus
Alar fascia• Anterior subdivision of prevertebral fascia that
bridges between the transverse processes (a)• It blends with the (retro) Visceral fascia( posterior
fascia of the esophagus) at the level of T2 vertebral body. This seals inferiorly the (retro)pharyngeal space
• It runs from base of skull to the superior mediastinum
• Infections of the retropharyngeal space may rupture the alar fascia, thus entering the danger space, which is continuous with the posterior mediastinunm
Carotid sheath• Formed by all three layers of deep
fascia• Contains carotid artery, internal
jugular vein and the vagus nerve• Lincon’s Highway:- A viscerovascular
space coined by mosher- is the carotid sheath from the jugular foramen & carotid canal at the base of the skull to the middle mediastinum
Danger space• Immediately posterior to the retropharyngeal space and immediately
anterior to the prevertebral space• Spread within the danger space tends to occur rapidly because of the
loose areolar tissue that occupies this region. This spread can lead to mediastinitis, empyema, and sepsis
CLASSIFICATION OF FASCIAL SPACES
• Hollinshead’s classification(1958)Infrahyoid spaces -1.Visceral compartment
A) Pretracheal / previsceralB) Retrovisceral
2. Visceral space3. Other space
I. Cavity within carotid sheathII. Space between 2 layers of prevertebral fascia
SPACES OF HEAD AND NECKGrodinsky and Holyoke in 1938 described
Space 1: superficial to the platysma as well as between the latter and the deep fascia.
Space 2: lies in the anterior triangle between the superficial layer of deep fascia and the deep surface of the sternothyroid sheath, thus including the sternohyoid-omohyoid fascia with its contents and the sternothyroidthyrohyoid muscles with the anterior portion of their sheaths.
We have designated as space 2A the corresponding space in the posterior triangle between the superficial layer of the deep fascia and the posterior belly of the omohyoid muscle with its sheath (
Space 3: between the visceral fascia and the sternothyroid-thyrohyoid layer anteriorly, the carotid sheath laterally, and the alar fascia posteriorly.
Space 3a: Lincoln’s highway
• Space 4: also known as danger space, lies between alar & prevertebral fascia.
• Space 4a: posterior triangle of neck
• Space 5: prevertebral space.
• Space 5a: fascia enclosed by prevertebral fascia
BASED ON MODE OF INVOLVEMENT
1. Direct Involvement. (Primary Spaces)• Maxillary Spaces – Canine, buccal infratemporal• Mandibular Spaces – Submental, Submandibular,
Sublingual, Buccal2. Indirect involvement (Secondary Spaces)
• Masseteric• Pterygomandibular• Superficial and deep temporal• Lateral and retro pharyngeal• Prevertebral, parotid, carotid sheath,peritonsillar and danger
spaces.
• Based on clinical significance- (TOPAZIAN)
Face- Buccal, canine, parotid, masticatory.
Suprahyoid- Sublingual, submental, submandibular,
lateral pharyngeal, peritonsillar.
Infrahyoid- Pretracheal.
Spaces of total neck- Retropharyngeal, space of carotid
sheath.
Based on aetiology
General classification• Odontogenic• Traumatic• Implant surgery• Reconstructive surgery• Infection arising from contaminated needle puncture• Others (including from factors such as infected antrum,
salivary gland afflictions)• Secondary to oral malignancies
On the basis of causative organisms• Bacterial infection• Odontogenic• Non odontogenic
• Tonsillar infection• Nasal infection• Furuncle overlying skin
Buccal space
BOUNDARIES:-
• ANTERIORLY-Corner of mouth • POSTERIORLY-Masseter muscle, Pterygomandibular space• SUPERFICIAL- skin and Subcutaneous tissue
• DEEP- Buccinator muscle • SUPERIORILY- Maxilla, Infraorbital space• INFERIORLY- Lower border of mandible.
The buccal space occupies the portion of subcutaneous space between the fascial skin and buccinator muscle.
• CONTENTS OF BUCCAL SPACE:-• Buccal pad of fat• Stensons (Parotid duct)• Anterior and transverse facial artery and vein.
MUSCLE RELATED – Buccinator muscle
Neighboring spaces- Infraorbital, pterygomandibular, infratemporal space
BUCCAL SPACE INFECTION-Cause Infection from maxillary
premolars, molars andmandibular premolars if the roots are localized
above the buccinator muscle insertion (upper jaw) or below insertion (lower jaw)
infection spread into the soft tissues of the cheek → along anatomical planes toward the infratemporal or pterygopalatine fossa (pterygomandibular raphe!)
Relation of root with buccinator muscle
Clinical features:Dome shaped swelling on the anterior aspect of cheek extending anteroposteriorly from corner of mouth to angle of mandible and superoinferiorly from level of zygomatic arch to inferior border of mandible.
TREATMENTIncision & Drainage: Incision-Horizontal incision
through oral mucosa of cheek in the premolar, molar region.
Extra oral – below the lower border of mandible
Canine space / Infraorbital space
Boundaries –Anteriorly –Oribicularis orisPosteriorly- Buccal space Superficially – Quadratus labi superioris Deep- Lavator anguli oris, anterior
surface of maxillaMedially – Levator labi superioris alaque
nasiLaterally – Zygomaticus major, Superiorly – Quadratus labi superioris Inferiorly - caninus muscle
• Contents – Angular artery and vein, Infraorbital nerve
• Neighboring spaces – Buccal space
CANINE SPACE INFECTIONETIOLOGY:
• Maxillary canine, rarely from maxillary first premolar.• Rarely from nasal & upper lip infections.
Clinical Features-Swelling of cheek, lower eyelid & upper lip.Drooping of angle of mouth.Nasolabial fold obliterated.Edema of lower eyelid
TREATMENT:- Incision & Drainage: incision – through mucosa of buccal vestibule: of
lateral incisor & canine.
Drainage – A curved mosquito forceps is inserted superior to caninus muscle, pus is evacuated and drain is secured.
Complication- Cavernous sinus thrombosis
PALATAL ABSCESS
Involvement: palatal roots of posterior teeth, abscesses from palatal pocket - cause infection.
Boundaries: inferiorly:- bounded by cortical plates of hard palatesuperiorly: overlying periosteium. laterally: alveolar process of maxilla & teeth
Clinical features: i) well defined circumscribed fluctuant swelling, confined to one side of palateii) offending tooth is tender on percussion.
Incision & Drainage. incision- through the mucosa down to the bone, care should be taken as to avoid cutting of greater palatine nerves & blood vessels.
The submucosal portion of the hard palate contains neurovascular bundle, minor salivary glands a lymfoid tissue
the rich innervation of thePeriosteumpainful
the course of the palatineArterybleeding
Infratemporal SpaceBoundaries-• Superiorly: infratemporal surface of
greater wing of sphenoid.• Inferiorly: lateral pterygoid muscle.• Laterally: temporalis tendon ,ramus of
mandible• Medially: lateral pterygoid plate,muscle
& medial pterygoid plate• Posteriorly: parotid gland• Anteriorly: infratemporal surface of
maxilla & posterior surface of zygomatic bone.
Contents-• Pterygoid plexus of veins.• Internal maxillary artery.• Mandibular nerve & its branches.Etiology-• Infected buccal roots of maxillary 3rd molars and 2nd molars• Infected needles or contaminated LA solution in the area of
tuberosity• Spread from other space infections• infection may ascend into the cavernous sinus (through
venous plexus in the ovale and spinosum foramen), orbita, temporal fossa, pterygopalatine fossa
Infratemporal space
Clinical features:
Extraoral: trismus, bulging of temporalis muscle, swelling of the face on affected side in front of the ear, eye is often closed
Intraoral: swelling in the tuberosity area, elevated temp: upto 104 F
TREATMENTIncision & Drainage
• Intraoral approach: no trismus opposite to 2nd & 3rd molar
• Extraoral approach: in severe casesincision made at upper & posterior edge of temporalis muscle. A sinus forceps is directed upwards and medially
• If failure in mouth openingtemporalis myotomy / excision of coronoid process.
Submandibular SpaceBoundaries-• Superiorly: mylohyoid muscle, inferior border of mandible.• Inferior: anterior & posterior belly of digastric.• Laterally: deep cervical fascia, platysma, superficial fascia & skin.• Medially: hyoglossus,styloglossus,mylohyoid muscle.• Posteriorly: to hyoid bone.• Anteriorly: submental space.Contents-• Submandibular salivary gland.• Proximal portion of Wharton’s duct.• Lingual & hypoglossal nerves.• Branches of facial artery- palatine,tonsillar,glandular,submental.
Submandibular Space Infection
Etiology-• Infected mandibular 2nd & 3rd molars.• From submental,sublingual spaces.• infecting mandibular lymphnodes & salivary glands• Involvement from middle 3rd of tongue, posterior part of
sublingual space, submental space, maxillary teeth, cheek, maxillary sinus and palate.
Clinical features
Extraoral: firm swelling in submandibular region some degree of tenderness redness of overlying skin Intraoral: teeth sensitive on percussion mobility of teeth dysphagia moderate trismus
TREATMENTIncision & drainage
Incision made of 1.5 to 2 cm below border of mandiblesinus forceps inserted superior & posteriorly on lingual side of mandible below mylohyoid – pus- drained & sutured
Submental SpaceBoundaries-Roof: mylohyoid muscle.Inferior: deep cervical fascia, platysma, superficial fascia & skin.Laterally: anterior belly of digastric.Posteriorly: submandibular space.
• MUSCLE RELATED – mentalis muscle
• CONTENTS – submental lymph nodes and anterior jugular vein.
SUBMENTAL SPACE INFECTION
ETIOLOGY:-• From lower anteriors.• Secondarily due to infection from submental lymph
nodes which drain lower lip, skin overlying chin, anterior part of floor of the mouth, tip of the tongue & sublingual tissues.
• Symphysis fracture.
Clinical features:
Extra oral: distinct , firm swelling in midline beneath chin, skin is taunt.
Intra oral: non-vital/ fractured teeth discomfort on swallowing mobility & tenderness on percussion in offending tooth
Incision & drainage:
Transverse incision symphysis of mandible kelly’s forceps/sinus forcepsupward & backward directiondrained & sutured
Sublingual SpaceBoundaries-• Superiorly: mucosa of floor of mouth.• Inferior: mylohyoid muscle. • Posteriorly: body of hyoid bone.• Anteriorly & laterally: inner aspect of mandibular body.• Medially: geniohyoid,styloglossus,genioglossus muscle.Contents-• Deep part of Submandibular gland.• Wharton’s duct.• Sublingual gland.• Lingual & hypoglossal nerves.• Terminal branches of lingual artery.
SUBLINGUAL SPACE INFECTION
Involvement:Infection spreads from mandibular incisors, canine,
premolars sometimes first molar, perforates lingual cortical plate & passes into sublingual space
Clinical features
Extraoral: little or no swelling enlarged & tenderness of lymphnodes discomfort & pain on deglutition speech affectedIntraoral: pain on affected side of floor of mouth
floor of mouth raised tongue pushed superiorly airway obstructed
TREATMENTIncision & drainage
Intraorally: incision made close to lingual cortical plate, sinus forceps inserted, pus drained & sutured
Extraoral: incision done in case were both submental & sublingual spaces are affected , incision made at submental region
Ludwig´s angina = the right and left submandibular, sublingual and submental spaces are infected
A fulminant infection can spread rapidly to pharyngeal and retropharyngeal space
Submandibular space
Submental space
Sublingual space
• The original description of the disease was given by Wilhelm Friedrich von Ludwig.
1. Rapidly spreading gangrenous cellulitis.
2. Originates in the region of submandibular gland but never involves one single space and
3. Arises from extension by continuity and not by lymphatics
4. Produces gangrene with serosanguinous, putrid infiltration but very little or no frank pus.
Ludwig’s Angina
Etiology
Odontogenic- acute dentoalveolar abscess, periodontal, pericoronal- mandibular 2nd & 3rd teeth
Iatrogenic- contaminated needleTrauma- mandibular fractures, punctured woundsOsteomyelitisSubmandibular & sublingual sialadenitisSecondary infections of oral malignancies
CLINICAL FEATURES
Patient looks toxic, difficulty in swallowing, impaired speech, marked pyrexia
Extraoral- severe tenderness, firm brawny swelling in submandibular & submental region, muscle spasm- trismus, airway obstruction , dilatation of alae nasi, cyanosis, death
Intraorally- raises the floor of mouth, tongue maybe raised, difficulty in swallowing,drooling of saliva
TREATMENT• Establishment & maintenance of adequate airway• Extraction of affected teeth, prolonged I.V antibiotics,
hydration, incision & drainage• Antibiotic regime-penicillin, metronidazole or clindamycin.• Tracheostomy
Incision for surgical drainage of Ludwig’s Angina
Classic method – Not used nowadaysBilateral through and though drainage of spaces
COMPLICATIONS • Death due to airway compromise• septicemia• mediastinitis• carotid blow out
Masticatory spaceThere are 5 masticatory spaces .1. Superficial temporal space2. Infratemporal space3. Deep temporal space 4. Submassetric space5. Pterygomandibular space
SubMasseteric SpaceBoundaries-Superiorly: zygomatic arch.Inferiorly: inferior border of
mandible.Laterally: medial surface of
masseter muscle.Medially: lateral surface of
ramus of mandible.Posteriorly: parotid gland & its
fascia.Anteriorly: buccal space &
buccopharyngeal fascia.
CONTENTS -• Masseteric artery and vein
Neighboring spaces-• Buccal, pterygomandibular,
superficial temporal, parotid space
SUBMASSETERIC SPACE INFECTION
• ETIOLOGY:-• Pericoronitis related to vertical or distoangular
impaction of third molars- Fracture of angle of mandible
Clinical features
External facial swelling is moderate
Tenderness over angle of mandible
Limited mouth opening
Pyrexia & malaise.
Necrosis of muscle
TREATMENTIncision & drainageIntraoral approach- incision done at lower part of
ramussinus forcepsdownward &backwarddrain inserted and secured
Extraoral approach- approach-incision is placed behind angle of mandiblehilton’s methodrubber drain inserted & secured
Pterygomandibular SpaceBoundaries-• Superiorly: lower head of lateral pterygoid
muscle.• Laterally: medial surface of ramus.• Medially: medial pterygoid muscle.• Posteriorly: deep part of parotid.• Anteriorly: pterygomandibular raphe.Contents-• Inferior alveolar neurovascular bundle.• Lingual & auriculotemporal nerves.• Mylohyoid nerve & vessels.
Etiology-• Infected mandibular 3rd molars(mesioangular/horizontal)• Pericoronitis.• Infected needles or contaminated LA solution.Clinical Features-• Absence of extra-oral swelling.• Severe trismus.• Difficulty in swallowing.• Anterior bulging of half of soft palate & tonsillar pillars with deviation of
uvula to unaffected side.
Spread of Infection-• Superiorly to infratemporal space.• Medially to lateral pharyngeal space.• To submandibular space.
TREATMENT:
Intraoral- vertical incision(1.5cm) made on anterior & medial aspect oframus of mandiblesinus forceps opened & closed & with drawnPus drained & sutured.
Extraoral- incision made at angle of mandiblesinus forceps Pus drained & sutured
Temporal Spaces• Superficial temporal-Laterally: temporalis fascia.Medially: temporalis muscle.• Deep temporal-Laterally: temporalis
muscle.Medially: temporal bone &
greater wing of sphenoid.
Temporal space infectionClinical featuresPain , trismus, swelling may/ may not
be present
Incision & drainage
Extraoral- incision at temporal region, haemostat inserted above & below temporalis muscle
Lateral Pharyngeal SpaceBoundaries-Shape of an inverted cone or pyramid, the
base is at sphenoid bone and the apex at hyoid bone.
Anteriorly: pterygomandibular raphe. Posteriorly: extends to prevertebral fascia.Laterally: fascia covering medial pterygoid
muscle, parotid & mandible.Medially: buccopharyngeal fascia on lateral
surface of superior constrictor muscle.Styloid process divides the space into
anterior muscular and posterior vascular compartment.
Contents-
Anterior compartment: loose areolar connective tissue, lymph nodes and facial artery
Posterior compartment: carotid sheath(carotid artery,internal jugular vein,vagus nerve), cranial nerves IX through XII.
LATERAL PHARYNGEAL SPACE INFECTION
Clinical features
Extraoral-brawny induration of face
Intraoral- marked trismus, lateral pharyngeal wall maybe swollen, dysphagia
TREATMENTIncision & drainage
Extraoral- incision made at ant: border of sternocleidomastoid Muscle extending to the middle of sub-mandibular glandhemostat inserted into mandible until cavity is reached
Intraoral approach
Incision made at pterygomandibular rapheforceps insertedpus drained
Retropharyngeal SpacePosteromedial to lateral pharyngeal space and anterior to
the prevertebral space .
Boundaries-Anterior: posterior pharyngeal wall. Posterior: prevertebral fascia. Superior: skull base.Inferior: mediastinum.Laterally: lateral pharyngeal space.
.
RETROPHARYNGEAL SPACE INFECTIONInvolvement- lateral pharyngeal space
Clinical features
Infection of throatSnoring, chocking , dyspnoeaSwelling in lower portion of pharynx
TREATMENTIncision & drainage
Suprahyoid portion Infrahyoid portion
spaces is approached through lateral pharyngeal space
Peritonsillar space infection
Clinical evaluation:• Pharyngitis .• Severe sore throat, dysphagia, and
referred otalgia.• The speech is muffled and classically
described as hot potato voice.• Trismus is not present
Treatment • According to recent literature,needle aspiration is done instead
of incision and drainage .• (JOMS,Vol 51,2009
PAROTID SPACE BOUNDARIES:-• superiorly zygomatic arch• Inferiorly lower border of mandible• Anteriorly posterior border of the mandible• Posteriorly Retromandibular region
• Space formed by splitting of the superficial layer surrounding the parotid gland and lies posterior to the masticator space.
• CONTENTS:• Parotid gland• Parotid lymph nodes• Facial nerve.• Retromandibular vein• External carotid artery
PAROTID SPACE INFECTIONClinical featuresSevere painSwelling at masseter musclePus escapes from stenson’s duct
TREATMENTIncision & drainage
Extraoral approach- retromandibular incision done at posterior border of mandible extending to lobule of sinus forceps inserted & various parts of gland accomplished
Rubber drain is inserted & secured with suture
DRAINAGE OF FASCIAL SPACES• Canine, Sublingual and Vestibular abscesses intraorally.
• Masseteric, Pterygomandibular, Buccal and Lateral
Pharyngeal space abscesses combination of intraoral and
extraoral drainage.
• Temporal, Submandibular, Submental, Retropharyngeal and
Parotid space abscesses extraoral incision and drainage.
Differences between cellulitis and abscess
Characteristics Cellulitis Abscess
Duration. Acute phase Chronic phase
Pain Severe and generalised Localised
Size Large. Small
Localization Diffuse borders Well-demarcated
Palpation Doughy / indurated Fluctuant
Presence of pus No Yes
Degree of seriousness Greater Less
Bacteria. Aerobic Anaerobic/mixed
DIRECTION OF SPREAD OF INFECTION• Infection from any tooth will spread along the path of
least resistance. It can perforate either the buccal cortical plate or lingual / palatal cortical plate depending upon which is thinner
LIFE THREATENING COMPLICATIONS OF OROFACIAL INFECTIONS
Complications of space infection
• Osteomyelitis• Mediastenitis• Meningitis • Brain abscess• Cavernous sinus thrombosis• Ludwig’s angina
BRAIN ABSCESSEtiologyArises from bacteremia – odontogenic infectionsProduces- inflammation, edema and septic thrombosisClinical featuresHeadacheHemiplegia PapilloedemaAphasiaConvulsionsAbducens palsy
Treatment
1) IV chloramphenicol2) Surgery to provide drainage
CAVERNOUS SINUS THROMBOPHLEBITIS
• Occur as a result of superior spread of odontogenic infection via a hematogenous route
• Formation of thrombus in cavernous sinus/ communicating branches
• Unusual occurrence, rarely the result of an infected tooth
• Serious, life threatening infection
• 2 routes• External route (Anteriorly)- infection enters- face & lipsuperior
or inferior ophthalmic veinsuperior orbital fissurecavernous sinus
• Internal route (Posteriorly)- pterygoid plexusemissary veincavernous sinus
Microbiology streptococci & staphylococci & gram –ve bacteria
Clinical featuresSwelling of face, eyesPain in the eyeEdema of conjunctivaPulsating exophthalmos
Treatment
Antibiotic therapyNeurosurgical consultation AnticoagulantsSurgical drainage
CONCLUSION• A clinician must have a working knowledge of all
information regarding signs, symptoms & history ,with results from clinical examination & tests to obtain diagnosis. A methodical & disciplined approach, along with a good measure of patience will help establish an accurate diagnosis & resulting in a good treatment
References • Textbook of oral & maxillofacial surgery : Neelima Malik.• Oral & maxillofacial Infections : Topazian• Textbook of oral & maxillofacial surgery : Laskin• Manuel Grodixsky And Edward A. Holyoke:THE FASCIAK AND FASCIAL
SPACES OF THE HEAD, NECK AND ADJACENT REGIONS• Newman, Takei, Klokkevold & Carranza: Carranza’s Clinical
Periodontology: 11th Edition