fcps part i mcqs

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Lamellar bodies are found in: A- Clara cells B- Type 1 alveolar cells C- Type 2 alveolar cells D- Macrophages Ans C surfacan conained !ihin he lamellar !hich is released via e"ocyosis# Bes mehod o deec malignancy A- Cyology B- $isopahology C- %mmunochemical Ans B $%& diagnosis is hrough A- !esern blo B- 'lisa C- CD coun ()** Ans A &iamin D causes: A- increased renal Ca re-absorpion B- increased serum phosphae levels Ans A Anihrombin A- $as a srucure similar o hrombin B- is a valine proease inhibior C- +hen binds o proease acion is increased by heparin

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Page 1: Fcps Part i Mcqs

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Lamellar bodies are found in:A- Clara cellsB- Type 1 alveolar cellsC- Type 2 alveolar cells

D- Macrophages

Ans Csurfac an con ained !i hin he lamellar !hich is released viae"ocy osis#

Bes me hod o de ec malignancy

A- Cy ologyB- $is opa hologyC- %mmunochemical

Ans B

$%& diagnosis is hroughA- !es ern bloB- 'lisaC- CD coun ()**

Ans A&i amin D causes:A- increased renal Ca re-absorp ionB- increased serum phospha e levels

Ans AAn i hrombinA- $as a s ruc ure similar o hrombinB- is a valine pro ease inhibi orC- +hen binds o pro ease ac ion is increased by heparin

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Ans Ci is a serine pro ease inhibi or# %n an i hrombin %%% deficiencyheparin,s an icoagulan effec is decreased#

$ead in ury causes paraplegia !i h no sensory loss !hich areis involved.A- Cerebral cor e"B- /pinal cord a T1* levelC- 0onsD- Medulla

Ans ATric y % guess !ha hey are referring o is he involvemenof mo or cor e"AD$A- $as double si3e compared o o"y ocinB- 4eleased on decreased osmolari yC- %s ranspor ed do!n a ached o neurohypophysin

Ans C

$%& infec ion in female !ill causeA- vulvar cancerB- endome rial cancerC- cervical CAD- leu emia

Ans C

5ollo!ing is no a sign of irreversible cell in ury:A- 6arryorhe"isB- 6aryolysisC- Mi ochondrial shrin ageD- myelin figures

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Ans Ds ill reversible a his s age7 bu if he condi ions don,improve irreversible in ury !ill develop

Type of necrosis in omen um:A- li8uefac iveB- coagula iveC- gangernousD- fa necrosis

Ans D

A idney ransplan immedia ely urns blue and is re ec ed!i hin minu es:A- ype 9 sensi ivi yB- Ab# media edC- Cy o o"ic re ec ionD- &$D

Ans Ba 8ues ion of hyper-acu e re ec ion#

+hich of he follo!ing causes viral media ed hos D;Aransfer:

A- Transduc ionB- Transforma ionC- Con uga ion

Ans A

+hich of he follo!ing mos probably has an animalreservoir.A- influen3aB- measlesC- smallpo"

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Ans A&i amin 6 deficiency follo!ing is unusual:A- coumarin herapyB- Bile duc obs ruc ion

C- red mea inges ionD- in ne!born due o lac of ranspor hrough placen a

Ans C

Li8uid dysphagia is due o:A- hypoglossal nerve palsy

B- plummer-vinson syndromeC- pala al palsyD- neuromuscular incoordian ion

Ans D$ein3 bodies are found inA- au oimmune hemoly ic anemiaB- -<-0-D deficiencyC- heredi ary spherocy osisD- pos splenec omy

Ans B

$emoly ic anemia is a fea ure of A- acu e myeloid anemiaB- cirrhosis

C- mycoplasmaD- /L'

Ans AAll he o hers#;ecrosis in omen um isA- fa necrosis

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B- vomi ingC- sei3ures

Ans B

A scenario of child having serum choles erol of <**mg>dl7 hisfa her7 mo her and elder bro her are all having his highlevel of choles erol in heir blood#+ha gene mu a ion isresponsible for his condi ion.A- apo'B- $DL recep ors

C- LDL recep orsD- basal chylomicron ranspor pro eins'- &LDL recep or

Ans CA scenario of child having $b <gm>dl7 +BC is ?2** andpla ele s 1?****>mm)# his elder bro her also has a his ory ofrepe i ive blood ransfusions# o! you come o a diagnosis for

his child.A- CBCB- %mmunocy ochemis ryC- 'lec rophoresisD- /ic ling es'- 5ragile es

Ans C

Diagnosis of a umor is made byA- cy ological e"amina ionB- his opa hological e"amina ionC- immunochemical assayD- en3yme assay'- umor mar er

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Ans B@* of o"ygen is e" rac ed by !hich organ.A- hear

B- s ele al muscleC- lungsD- brain

Ans A

A scenario of pa ien having 5ac or &%%% deficiency# The genes

responsible for his disease are a chromosome.A- 21B- 19C- 11D- '-

Ans DA surgical pa ien is ransfused )-? uni s of blood before andaf er surgery# $e develops abnormal 'C changes noconsis en !i h ischemia# +ha is he cause of he 'Cchanges.A- me abolic al alosisB- hypercalcemiaC- hyper alemia

Ans C

lucagon increases glycogenolysis in liver !hile ACT$ doesno due o.A- ACT$ increase plasma glucoseB- glucagon has differen guanyl cyclase han ACT$C- liver has differen recep ors for ACT$

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D- ACT$ is readily degraded by he liver en3ymes

Ans AACT$ via cor isol causes gluconeogenesis#

%f celiac run is bloc ed !hich of he follo!ing !ill nosuffer from ischemic in ury.A- liverB- pancreasC- spleenD- gall bladder

Ans B$ead of pancreas is in fron of of aor aA- inferior vena cavaB- celiac runC- aor aD- renal ar eries'- superior mesen eric ar ery

Ans A

Carcinoma breas me as asi3es oA- lungB- boneC- brainD- idney

'- liverAns B

Lung con ains me as a ic umor7 his opa hology reveals clearcells# Mos commonly his umor has me as asi3ed fromA- idney

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B- liverC- boneD- adrenal'- breas

Ans A

4egarding saliva ionA- caused by saliva ory nucleus of medulla>ponsB- increases during sleepC- inhibi ed by pilocarpine

D- increases in response o Be a-adrenergic s imula ionAns D

Be a adrenergic s imula ion causesA- mydriasisB- increased blood flo! o s ele al musclesC- increase peris alsis

Ans B

+hich organism is found in urine sampleA- paragonimus !es ermaniB- !uchereria bancrof iC- schis osoma hema obium

Ans C

+hich pus forming organism in lung also causes meningi is.A- s rep ococciB- s aphylococci

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C- bac eriodesD- pneumococci'- psuedomonas

Ans D

Common premalignan condi ion of mou h in adul sA- lichen planusB- hyper era osis

Ans A

Mos le hal premalignan condi ion isA- lichen planusB- leu opla iaC- ery hropla iaD- solar era osis

Ans C