locomotion ppt
TRANSCRIPT
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Care of Clients With Problems inLocomotion
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Bone
Provides a frameworkto support the body.
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Bones
Mechanical Function
Support of body tissue
Protection of body organs
Movement, affected by contraction of musclepulling on bones that provide leverage formotion.
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Bones
Other Functions:
Storage of calcium
Morrow produces RBC
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Bones
Composed of Living Intracellular Material
Osteoblast (bone forming cells)
Osteoclast (absorbs and remove osseous
tissue) Osteocytes (mature osteoblast embedded in
bone matrix)
Non-living Intracellular Materials
Mucopolysaccharides
Collagen (insoluble protein that combine toform white glistening inelastic fibers of
tendons, ligaments and fascia
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Bones
4 types of bones according to shape
Long
Short
Flat
irregular
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Haversian Units
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Bones
Long Bone is composed of:
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Bones
Periosteumwhitefibrous layer thatcovers the bone,
except on its articularspaces.
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Bones
Endosteumlines themarrow-filledmedullary cavity and
the Haersian canals
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Callus formation
Callusnew growth of bone.
5 stages of callus formation
1. Hematoma formation
2.Fibrin meshwork formation (semisolid character ofblood clots)
3. Invasion by osteoblast
4. Callus formation5. Remodelling
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The Muscle
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Muscle
1. Skeletal (Striated,Voluntary)
- Produces movement,
maintains posture,stabilises joints andgenerates heat
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Muscle
2. Visceral ( smooth,involuntary)
- found in the walls of
hollow organs likeyour intestines andstomach. They workautomatically without
you being aware ofthem.
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Muscle
3. Cardiac Muscle
- Cardiac musclecontracts to squeeze
blood out of yourheart, and relaxes tofill your heart withblood.
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Different types of MuscleContraction
1. Tonic
- a continual partial contraction that is vital inmaintenance of posture.
2. Isotoniccontraction in which tension andlength of the muscle change.
3. IsometricTension within the muscle
increases but the muscle doenot shorten.4. Twitcha jerky reaction to a single stimulus.
5. Tetanica more sustained contraction of
individual fibers.
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Different types of MuscleContraction
6. Fibrillationasynchronous contraction ofindividual fibers.
7. Convulsiveabnormal coordinated tetanic
contraction occuring in varying groups ofmuscles.
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Other Structure ofMusculoskeletal System
1. Cartilagefibersembedded in a firmgel. These reducefriction in the joint.
2. Ligamentstheseare bands of densefibrous tissue that areflexible and tough.
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Other Structure ofMusculoskeletal System
3. Tendonsbonds offibrous tissue that areattached to the bone.
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Other Structure ofMusculoskeletal System
4. Fascia this is asheet of looseconnective tissue thatmaybe found directlyunder skin or sheet ofdense, fibrousconnective tissue
making up sheet ofmuscles, nerve andblood vessel.
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Other Structure ofMusculoskeletal System
5. Bursaesmall sacsof connective tissuelocated wheneverpressure is exertedover moving parts.
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Joints
- provides flexibility within skeletal framework.
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Classifications of Joints
1. Synarthroses (fibrousjoints)allow nomovement.
2. Amphiarthroses(cartilaginous joints)allow little movement.
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Classifications of Joints
3. Diarthroses ( synovialjoints)allow freemovement.
A t f Cli t ith
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Assessment of Clients withMusculoskeletal Disorders
History
Biographic data: age, sex
Chief complaints:
Pain Joint Stiffness
Swelling
Deformity and Immobility
Sensory changes
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Assessment of Clients withMusculoskeletal Disorders
Medical History
Childhood and Infectious Diseases
Major illnesses, Hospitalizations
Medications
Family History
Psychosocial History and Lifestyle
Daily activities: Occupation; Habits
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Assessment of Clients withMusculoskeletal Disorders
Review of systems
Muscle pain, spasm, tenderness
Joint pains, swelling, redness
Weakness, limited movement, clumsiness
Crepitus, backache, changes in joints andbones.
A t f Cli t ith
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Assessment of Clients withMusculoskeletal Disorders
Physical Examination
Inspection and Palpation of
Muscle masses (symmetry, involuntarymovements, tenderness, tone, strength and size)
Weakness (polyneuropathy, electrolyteimbalance)
Joints for symmetry, crepitus, tenderness or pain,ROM
Bone deformity
Assessment of Clients with
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Assessment of Clients withMusculoskeletal Disorders
Note for:
Assessment of Clients with
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Assessment of Clients withMusculoskeletal Disorders
Posture: normalcurvature of the spineis convex through thethoracic and concavethrough the cervicaland lumbar portion.
Assessment of Clients with
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Assessment of Clients withMusculoskeletal Disorders
Observe the gait for smoothness and rhythm
Unsteadiness or irregular movement, Limpingmotion
Spastic hemiparesis gait Steppage gait
Shaffling gait
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Laboratory and Diagnostic Test
1. Blood Test
a. Erythrocyte Sedimentation Rate (ESR)
- useful in detecting inflammation in the body that may
be caused by infection, some cancers, and certainautoimmune diseases such as rheumatoid arthritis,lupus, and Kawasaki disease.
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Laboratory and Diagnostic Test
b. Rheumatoid factor
- a blood test that measures the amount of the RFantibody in the blood.
- diagnosed rheumatoid arthritis
c. Antinuclear Antibodies (ANA)
- is ordered to help screen for autoimmune disordersand is most often used as one of the tests to
diagnose systemic lupus erythematosus (SLE).
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Laboratory and Diagnostic Test
d. C-reactive Protein
- is produced by the liver. The level of CRP rises whenthere is inflammation throughout the body.
e. Uric Acid
- Uric acid is a chemical created when the bodybreaks down substances called purines.
f. Minerals
Calcium Alkaline hosphatase
Phosphoros
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Laboratory and Diagnostic Test
Muscle enzymes
Aldolase - a protein (called an enzyme) that helpsbreak down certain sugars into energy. It is found inhigh amounts in muscle tissue.
AST - is an enzyme found in high amounts in heartmuscle and liver and muscle cells. It is also found inlesser amounts in other tissues.
CK-MM (creatine Phosphokinase)elevated intraumatic injuries.
LDH
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Laboratory and Diagnostic Test
2. X-raydetect bone fractures.
3. Bone Scan
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Laboratory and Diagnostic Test
3. Arthroscopy
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Laboratory and Diagnostic Test
4. Arthrocentesis
- removal of synovialfluid, blood or pus
from a joint.
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Laboratory and Diagnostic Test
5. Myelography
- lumbar puncture isdone to withdraw a
small amount of CSF,which is replaced witha radiopaque dye,used to detect tumors
or herniatedintravertebral disc.
L b t d Di ti T t
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Laboratory and Diagnostic Test
6. Electromyography(EMG)
- measures and records
activity of muscles inresponse to electricalstimulations.
N i di
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Nursing diagnoses
- Pain
- Altered peripheral tissue perfusion
- Impaired physical mobility
- Risk for infection
- Risk for injury
- Self-care deficit
- Knowledge deficit
- anxiety
I l t ti
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Implementation
a. Perform a neurovascular assessment (6 Ps)
1. Assess pain (signals the beginning of muscle
ischemia)
2. Assess pulses (disruption of arterial blood flow)
3. Assess for pallor (disruption of blood flow)
4. Assess for paresthesia (nerve function may bedisruptive by nerve compression)
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5. Assess for paralysis (inceasing edema causesnerve compression)
6. Assess for polar (coldness)disrupted arterial
blood flow
Infectious/ Inflammatory and
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ect ous/ a ato y a dDegenarative Disorders
1. Osteomyelitis
- acute or chronicinfection of the bone
and surrounding softtissues.
Cause?
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Cause: staphylococcus aureus
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Cause: staphylococcus aureus
- affects person with low resistance or withdecreased blood flow to the trauma site
- Blood borne (hematogenic) osteomyelitis ismore common in children after throat infection
- Osteomyelitis resulting from trauma ororthopedic procedures is more common inadults
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Osteomyelitis
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Osteomyelitis
Clinical Manifestations:
Malaise, fever
Pain and tenderness of bone, redness and
swelling over bone Difficulty with weight bearing
Drainage from wound site may be present
Osteomyelitis
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Osteomyelitis
Diagnostic Test:
CBC (increased)
ESR (increased)
Osteomyelitis
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Osteomyelitis
CollaborativeManagement:
Collaborative mgnt
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Collaborative mgnt
-analgesics
-antibiotics
-Dressing change in sterile technique
-maintain body alignment and change positionfrequently to prevent deformities
-immobilization of affected part
-psychological support
Osteomyelitis
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Osteomyelitis
Surgeries:
Surgeries
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Surgeries
If needed:
- incision and drainage of bone abscess
- Sequestrectomy- removal of the dead, infected
bone and cartilage- Bone grafting after repeated infections
Nsg mngt
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Nsg mngt
1. Adminster prescribed medication
2. Protect the affected extremity from furtherinjury and pain
3. Promote healing and tissue growth-local treatments (warm saline soaks, wet to
dry dressing)
4. Prepare patient for surgical treatment e.g. bonegrafting, debridement etc
5. Provide health teachings
Septic Arthritis
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Septic Arthritis
A closed -spacedinfection caused byinavasion of thesynovial membrane by
pus-forming bacteriaor other pathogen.
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Septic Arthritis
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Septic Arthritis
Cause: Neisseriagonorrhoeae.
Commonly affected:Knee, hip, shoulder,wrist and ankle.
Septic Arthritis
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Septic Arthritis
Clinical Manifestations: Pain, swelling, warmth and tenderness in a
single joint.
Low grade fever, malaise High grade fever and chills
Septic Arthritis
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Septic Arthritis
Collaborative Management:1. Synovial fluid culture
2. Antibiotic therapy
3. Decompression of the infected joint
4. Synovectomy
Systemic Lupus Erythematosus(SLE)
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(SLE)
- Chronic multisystem,collagen disorder.
- higher incidence infemale (15 to 40 y.o)
Cause: UNKNOWN
Systemic Lupus Erythematosus(SLE)
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(SLE)
Diagnostic Test: CBC
ESR
ANA
Anti- DNA
Systemic Lupus Erythematosus(SLE)
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(SLE)
Clinical Manifestations:
Systemic Lupus Erythematosus(SLE)
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(SLE)
Systemic Lupus Erythematosus(SLE)
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(SLE)
Clinical manifestations
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1. Musculo-skeletala. Arthralgia and arthritis (synovitis)
b. Joint edema and tenderness
c. Pain on movement and morning sickness
2. Integrumentary
a. Butterfly rush across the bridge of the nose andcheeks
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b. Oral ulcers of the mucosa and hard palate andmay be accompanied by skin lesions
3. Cardiovasculara. Pericarditis (up to 30% of patients)
b. Papular, erythematous and purpuric lesions on
fingers, elbows, toes, fore-arms and hands
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4. Respiratory systema. Pleural effusion
b. Pleuritis
5. Neurologic
a. Subtle changes in personality and cognitive
ability
b. Depression and psychosis often occur
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6. Other systemsa. Glomeruli of the kidneys usually affected
b. lymphadenopathy
Systemic Lupus Erythematosus(SLE)
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(SLE)
Collaborative Management:1. Rest
2. ROM exercises
3. Prevent infection
4. Avoid exposure to sunlight
5. Pharmacotherapy (NSAIDS, Salycilate for joint
pains, oral/topical cortecosteroids for skinrashes)
6. Apply warm packs to relieve joint pains and
stiffness
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7. Provide meticulous mouth care8. Maintain skin integrity by keeping skin clean
and dry, using mild soaps and lotions, andinspect for vascular lesions
Rheumatoid Arthritis
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- Chronic systemicdisease characterizedby inflammatorychanges in joints and
related structures,specially the synovialmembrane.
Rheumatoid Arthritis
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Cause: Unknown May be autoimmune process or genetic
Causative factors include fatigue, cold, emotionalstress, infection
Joint distribution is symmetrical
Women > men
Between 20-55 years old
Complications (infections, osteoporosis, vasculitis,carpal tunnel syndrome)
Rheumatoid Arthritis
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Clinical Manifestations: Fatigue, anorexia, weight loss, slight
temp.elevation
Painful, warm, swollen joints with limited motion,stiffness in the morningand after period ofinactivity.
Crippling deformity
Muscle weakness secondary to inactivity
stages
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a. Firstsynovitis (joint inflammation andswelling)
b. Secondpannus (inflammatory granulationtissue)
c. Thirdtough fibrous connective tissue
d. Fourthcalcification of bone
Rheumatoid Arthritis
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Clinical Manifestations: Sjorgren's syndrome
-excessive dryness of the eyes, mouth and
vagina Felty's syndrome
- leukopenia, splenomegaly
Rheumatoid Arthritis
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Collaborative Management:1. Bed rest during acute phase
2. Passive ROM exercises to prevent
contractures.3. Splint painful joints.
4. Heat and cold application.
5. Warm bath in the morning to relieve morningstiffness.
6. Protect the client from infection.
Rheumatoid Arthritis
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Surgery:1. Osteotomysurgical removal of the wedge of
the joint.
2. Synovectomyremoval of the synovia.3. Arthroplastyreplacement of joints with
prostheses.
Rheumatoid Arthritis
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Pharmacotherapy:1. Aspirin
2. NSAIDS
Indocin (Indomethacin) Butazolidin (Phenylbutazone)
Motrin (Ibuprofen)
Nalfon ( Fenoprofen) Naprosyn( Naproxen)
Rheumatoid Arthritis
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Gold compounds (Chrysotherapy) Injectable forms
Oral forms
Corticosteroids (intra-articular injections)
Health therapyParrafin deep
Osteoarthritis
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Osteoarthritis
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Metabolic Response
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- Chronic, nonsystemic disorder of the jointscharacterized by degeneration of the articularcartilage
- Male = female affected, incidence increased
with age. Related to obesity and joint trauma
- Weight bearing jointsspine, knees, hips andends of the fingers are most commonly affected
Assesment
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-pain aggravated by use and relieve by rest-stiffness of the joints
-Herberdens nodes/Bouchard Nodes bony
overgrowth at terminal interphalangeal joints-Decreased ROM
Collaborative Mngt
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Assess joints for pain and ROMRelieve pain and prevent further trauma to joints
-cane or walker as indicated
-posture and body mechanics, avoid weightbearing and continous standing
- physical therapy to maintain joint mobilty andmuscle strength
-promote comfort/pain relief (NSAIDS,analgesics
-Decreased calories
Gout
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-Disorder of purine metabolism-precipitation of urine cyrstals in the joints (tophi)
-causes of inflammation and pain
-occurs in males, familial
Clinical manifestations
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- Joint pains, redness, heat, swelling (great toeand ankle mostly affected)
- Headache, malaise, anorexia
- Tachycardia, fever, tophi in outer ear, handsand feet
- Uric acid elevated in blood/urine
Collaborative Mngt
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- Drug therapya. Acute attackcolchicine (discontinue with
diarrhea or N/V); NSAIDS (indocin, butazolidin)
b. Prevention
-uricosuric agents
a. Probenecid (Benemid), sulfinpyrazoneincrease renal excretion of uric acid
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b. Allupurinol inhibits uric acid formationc. Encourage fluids to 2-3 L a day when givingantigout drugs to prevent stone formation inkidneys
Food
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- Low purine dieta. foods to avoid
-organ meats, shellfish, legumes, sardines,
mushrooms, sweetbreads, beer/wine
Paget's Disease (OsteitisDeformans)
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- Increase rate of bonetissue breakdown(resorption) byosteoclasts.
- Bone is weak andprone to fracture.
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- Etiology is unkown but may be due to virusacquired 20-40 years prior to onset
- May be familial risks, found 30% with familyhistory
- Older adults are susceptible (3M in US)
- Cultural (more in European Descent, not seenmostly in Asia and Scandinavian countries)
Complications
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- Gout- Hyperhtyroidism
- Renal stones
- Heart failure- If untreated, blindness or deafness
Clinical Manifestations:
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- 80% are asymptomatic- Pain in the lower back and extremeties and
nerves
- Posture changes
- Bowing of long bones
- Enlarged skull
- Pathological fractures- Fatigue
- Flushed warm skin
Collaborative Management
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2. Calcitonin/ Etidronate
3. Cytotoxic Antibiotic
Legg-Calve-Parthe's Disease
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- a.k.a Coxa plana or Osteochondritis deformsjuvenilis.
- Characterized by aseptic necrosis of the head ofthe femur.
Legg-Calve-Parthe's Disease
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Cause: Unknown- Disturbance in circulation to the
Femoral capital epiphysis that causes
Ischemic, aseptic necrosis to femoral head
- incidence: dominant in caucasian males (4-8
years old); 1 in 12,000 in children
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- Can be familial, trauma, inflammation, orcoagulation defects
Clinical Manifestations
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Joint dysfunction
Xray reveals changesin bone.
Manifestations
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- Mild pain in the hip and anterior thigh- Limp aggravated by increased activity and
relieved by rest
- Stiffness in the morning or after rest
- Progressive stages may have limited ROM,weakness, muscle wasting, shortening ofaffected limb
Collaborative Management
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1. Pain relief2. Limit activities (Bed rest)
3. Provide mobility equipment
4. Use of brace5. Cast application
6. Surgery
7. Prevent Trauma to the hips( avoid contactsports, high impact running)
Fibromyalgia
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- Chronic syndrome ofmusculoskeletal tissuecharacterized bywidespread pain.
Fibromyalgia
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Excessresponse tocold
Collaborative Management
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Pharmacologic Management
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Osteoporosis
Systemic skeletal
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- Systemic skeletaldisease characterizedby low bone mass,leading to enhanced
bone fragility and aconsent increase infracture risk.
Risk Factors
Age 50 y o
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Age50 y.o
Low body weight(less than 70kg)
Famiy history
Low physical activity Female
Tobacco use
Estrogen deficiency
Increase with age
Clinical Manifestations
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Collaborative Management
Diet supplementation
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Diet supplementation
Exercise
Avoid tobacco and alcohol
Pharmacotherapy Evista
Calcitonin
Forteo
Carpal Tunnel syndrome
- Form of repetitive stress injury
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Form of repetitive stress injury
- Characterized by progressive compression ofthe median nerve caused by narrowing of thecarpal tunnel in the wrist through overuse
- Women 30-60 years old, is a seriousoccupational health problems
- Complications: partial paralysis, sensory
disturbance, atrophy in the muscles of thepalms and three fingers
Assessment
- numbness, paresthesia
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numbness, paresthesia
- Pain in the hand and fingers progressing up thearm in time
- Inability to clench fist
- May be decreased strength and flexibility of thethumb
- Temp in the palm may be cold; palm may be
discolored
Diagnostic tests
- X-ray to rule out fracture
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X ray to rule out fracture
- Electromyography- electrodes are placed atspecific places along the median nerve toassess diminished nerve conduction of the
elec. Current- Phalens wrist-flexion testclient places the
elbow on the table, letting the wrist drop, + testindicates when carpal tunnel syndrome occursin hand/fingers in 60 second or less
- Tinel sign- percuss the median nerve at the
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e s g pe cuss e ed a e e a ewrist, + sign if tingling occurs in the thumb/3fingers
- Compression testblood pressure cuff is
inflated above systolic pressure on the forearmfor 1.5 min, + test when carpal syndrome ispresent after 1.5 min
Nsg Interventions
a. Immobilize the affected area using splint for 1-2
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g pweeks
b. Apply compression of the affected wrist
c. Administer analgesia or muscle relaxant
d. NSAIDs to decrease pain
e. Instruct patient who has surgical repair that noheavy lifting or direct pressure in the palm is
permitted for 2-3 weeks
Surgical Mgnt
- Decompression is achieved by resecting the
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p y gcarpal ligament and may need synovectomy orremoval of synovium
Trauma
1. Straindamage to
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gtendon due to twistingmotion.
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2. Spraindamage to
Trauma
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ligaments due totwisting motion.
3. Subluxationpartiadisarticulation of joint.
Trauma
4. Fractureany
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impairement in thebone integrity.
Collaborative Management
R- est/ immobilized affected joints
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I- ce application (1st72 hours)
C- ompression(elastic bandage)
E- levate affected joint
Types of Fractures:
1. Completeentire
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circumference of thebone is impaired.
2. Incompleteonypartial circumferenceof bone is impaired.
Types of Fracture:
3. Transversethe lineb k i th
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or break is across thebone.
4. Obliqueline ofbreak goes diagonal
along the bone.
5. Spiralline of breakd th b
Types of Fracture:
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goes around the bone.
6. Greenstickone sideof the bone is
impaired, the other isbent.
Types of Fracture:
7. Comminutedboned li t d
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ends are splinteredinto two more smallpieces.
8. Impactedone boneenters theintramedullary spaceof another bone end.
Types of Fracture:
9. Closed or simpleN b k i ki
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No break in skinintegrity.
10. Open or compoundwith break in skin.
Clinical Manifestations:
Pain aggravated by motion
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Loss of motion
Edema
Crepitus Shortening of the limbs due to spasm
Obvious deformity
Collaborative Management
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Fracture complications
a. Direct
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1. Alteration in union, delayed union, andmalunion and etiologies include:
1.1 inadequate immobilization
1.2 inadequate reduction
1.3 excess movement
1.4 infection
1.5 poor nutrition
1.6 agehealing time for fractures increases withage.
2. Avascular necrosisdeath of cells from no
blood supply
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blood supply
3. Osteomyelitisbone infection
b. Indirect
1. Associated with blood vessel and nervedamage: etiologies include:
1.1 compartment syndromedistal humerus andproximal tibia are mostly affected affected by thissyndrome
1.2 venous thrombosis
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1.3 fat embolism
1.4 hypovolemic shock
Assessment
a. Immediate localized pain
b D d f ti
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b. Decreased function
c. Inability to use affected part
d. Visible deformitye. Ecchymosis
f. Limited sensation in distal to the site
g. Crepitus or clicking sounds on movement
Diagnostic tests
a. X-ray to confirm fracture
b Bl d t di ifi t th f t
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b. Blood studies specific to the fracture
c. Client-specific history
Nsg Mgnt
a. Maintain anatomic realignment of the bone,
known as reduction
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known as reduction
b. Maintain immobilization to maintainrealignment
c. Restore fxn on the injured part
d. Adminster muscle relaxants or analgesia forpain and muscle spasm
e. High protein, high calorie, high fiber
f. Encourage vit B, C and D
I fl id i t k 2 3 L t t b l
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g. Increase fluid intake 2-3 L to promote bowelmovement and prevent constipation or renalcalculi
h. Apply direct pressure if active bleeding
Surgical Mgnt
a. Immediate re-alignment of bones if possible
b ORIF of fracture if no other realignment method
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b. ORIF of fracture if no other realignment methodcan be completed
Traction
- Application of a pulling force to a part of the body
by pulleys and weights to correct deformities
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by pulleys and weights to correct deformities,improve joint contractures, treat dislocation,immobilization, treat disease processes
Types and Uses
a. Skeletalforce applied directly to the bone by
inserting pins wires or tongs into the bone
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inserting pins, wires or tongs into the bone
b. May have 40 lbs of traction up to 8 weeks at atime
c. Used to reduce fractures
b. Manualcompleted by application of the handsof a health care worker; temporary form of
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of a health care worker; temporary form oftraction until application of a cast to reduce afracture
c. Skinusually 5 lb of traction is applied directly
to the skin; may be used continuously or
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to the skin; may be used continuously orintermittently
Traction type (upper extremeties)
a. Dunlops (sidearm)
1 Used to immobilize the arm to correct
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1. Used to immobilize the arm to correctcontractures or fractures
2. Pull is upward
3. Elbow flexion usually 90 degrees
4. May be skin or skeletal traction
2. Olecranon pin (overhead arm)
1 Used to immobilize arm to prevent contractures
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1. Used to immobilize arm to prevent contracturesor fractures
2. Pull is upward
3. Elbow flexion usually 90 degrees
4. May be skin or skeletal traction
Traction type: lower extremity
a. Bucks (extension)
1 Used to immobilize the leg to correct
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1. Used to immobilize the leg to correctcontractures of the knee and joint or fracture ofthe hip or femoral shaft
2. A form of skin traction
3. Pull on the lower extremity with knee and hipfully extended
b. Russell
1 Used to immobilize the leg generally for a
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1. Used to immobilize the leg generally for afracture of the hip of femur
2. A form of skin traction
3. Exerts pull on the lower extremity
c. Balanced traction with thomas ring splint and
Pearson attachment
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1. Used to immobilize the leg to correct fracturesgenerally of the femoral shaft of the femur, hip
or tibia2. Entire leg is supported with a ring placed at the
groin of the affected leg to support thigh off theleg
3. Skeletal type of traction
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