opener a “smart drug”. figure 17.1 one gene, one enzyme (part 1)
Post on 19-Dec-2015
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Opener A “Smart Drug”
Figure 17.1 One Gene, One Enzyme (Part 1)
Figure 17.1 One Gene, One Enzyme (Part 2)
Figure 17.2 Hemoglobin Polymorphism
Figure 17.3 Genetic Diseases of Membrane Proteins (Part 1)
Figure 17.3 Genetic Diseases of Membrane Proteins (Part 2)
Figure 17.3 Genetic Diseases of Membrane Proteins (Part 3)
Figure 17.3 Genetic Diseases of Membrane Proteins (Part 4)
Figure 17.4 Prion Proteins
Figure 17.5 A Fragile-X Chromosome at Metaphase
Figure 17.6 Strategies for Isolating Human Genes
Figure 17.7 RFLP Mapping (Part 1)
Figure 17.7 RFLP Mapping (Part 2)
Figure 17.8 5-Methylcytosine in DNA Is a “Hot Spot” for Mutagenesis (Part 1)
Figure 17.8 5-Methylcytosine in DNA Is a “Hot Spot” for Mutagenesis (Part 2)
Figure 17.9 The CGG Repeat in the Fragile-X Gene Expands with Each Generation
Figure 17.10 Genetic Screening of Newborns for Phenylketonuria (Part 1)
Figure 17.10 Genetic Screening of Newborns for Phenylketonuria (Part 2)
Figure 17.11 DNA Testing by Allele-Specific Cleavage
Figure 17.12 DNA Testing by Allele-Specific Oligonucleotide Hybridization (Part 1)
Figure 17.12 DNA Testing by Allele-Specific Oligonucleotide Hybridization (Part 2)
Figure 17.13 A Cancer Cell with Its Normal Neighbors
Figure 17.14 Dividing Cells Are Especially Susceptible to Genetic Damage
Figure 17.15 Oncogene Products Stimulate Cell Division
Figure 17.16 The “Two-Hit” Hypothesis for Cancer
Figure 17.17 Tumor Suppressor Gene Products Inhibit Cell Division and Cancer
Figure 17.18 Multiple Mutations Transform a Normal Colon Epithelial Cell into a Cancer Cell (1)
Figure 17.18 Multiple Mutations Transform a Normal Colon Epithelial Cell into a Cancer Cell (2)
Figure 17.18 Multiple Mutations Transform a Normal Colon Epithelial Cell into a Cancer Cell (3)
Figure 17.19 Strategies for Killing Cancer Cells
Figure 17.20 Gene Therapy: The Ex Vivo Approach (Part 1)
Figure 17.20 Gene Therapy: The Ex Vivo Approach (Part 2)
Figure 17.21 Two Approaches to Sequencing DNA
Figure 17.22 The Human Genome
Figure 17.23 Is This the Future of Medicine?
Figure 17.24 Proteomics (Part 1)
Figure 17.24 Proteomics (Part 2)
Table 17.1 Human Cancers Known To Be Caused by Viruses