percutaneous double balloon valvuloplasty for severe tricuspid stenosis. ahj 1989

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  • 8/10/2019 Percutaneous Double Balloon Valvuloplasty for Severe Tricuspid Stenosis. AHJ 1989

    1/3

    Volume 118

    Number 2

    Brief Communications

    4 17

    Fig. 3.

    VVI bradycardia support systemworking after automatic defibrillator.

    Top

    Device sensing.

    Mid-

    dle,

    Intracavitary electrogram.

    Bottom,

    Surface electrocardiogram.

    D,

    Defibrillation.

    pacemaker unction wasnormal, and no ventricular tachy-

    cardia was observed on Holter monitoring.

    A multiprogrammable telemetric implantable automatic

    defibrillator with a multiprogrammable VVI bradycardia

    support system Telectronics Guardian, model4201,Telec-

    tronics, Inc., Englewood, Colo.) was implanted and the

    original VVI pacemakerwas explanted. The defibrillation

    threshold was 18 oules. The device wasprogrammedwith

    an initial energy of 28 joules (650 V, 8 msec) (safety

    margin = 10 joules). The tachycardia detection interval

    was 320 msec (Fig. 2) and after defibrillation, the VVI

    bradycardia support systemcould be observed Fig. 3). One

    month after implant, ventricular fibrillation was nduced

    and automatic defibrillation wassuccessful. he patient is

    doing well 4 months after implant. No dischargehasbeen

    registeredand the bradycardia support of the defibrillator

    is working as a VVI pacemaker.

    This is the first report of an automatic implantable de-

    fibrillator in Chagasdisease.Becausebifascicular or total

    atrioventricular block are present in almost 50 of cha-

    gasic patients who present with sustained ventricular

    arrhythmias, automatic defibrillators with pacemaker

    function will probably be very useful in selectedcasesof

    chronic Chagasicmyocarditis.

    REFERENCES

    1. Mirowski M. The automatic mplantable ardioverter-de-

    fibrillator. An overview.J Am Co11 ardiol

    985;6:461-6.

    2. PrataA. Natural historyof chagasicardiomyopathy.n: Pan

    AmericanHealthOrganization, d.American rypanossomia-

    sis esearch.Washington,C: Pan American Health Organi-

    zation 1975:191.

    3. Lopes ER Chapadeiro E. Morte subita em ara endemica de

    doenca e Chagas. ev Sot BrasMed Trop 1983;16:79-83.

    4. Mendoza , Camardo , Moleiro F, Castellanos, MedinaV,

    Gomez J Acquatella H Casal H Tortoledo F Puigbo J. Sus-

    tained ventricular tachycardia in chronic chagas ic myocardi-

    tis: electrophysiologic and pharmacologic characteristics. Am

    J Cardiol 1986;57:423-7.

    Percutaneous double balloon valvuloplasty

    for severe tricuspid stenosis

    Irvin F. Goldenberg, MD, Wes Pedersen,MD,

    Jeanne Olson, RDMS, JamesD. Madison, MD,

    Michael R. Mooney, MD, and Fredarick L. Gobel, MD.

    Minneapolis, Minn.

    Balloon valvuloplasty hasemergedasan alternative treat-

    ment to surgicalvalvotomy for somepatients with congen-

    ital or acquired pulmonic, aortic, or mitral stenosis.1-4x-

    perience with balloon valvuloplasty for the treatment of

    tricuspid stenosis,however, hasbeen imited.5 We present

    a caseof tricuspid balloon valvuloplasty in a patient with

    rheumatic mitral, aortic, and tricuspid stenosis.

    A 40-year-old womanpresentedwith complaints of pro-

    gressively increasing fatigue and minimal dyspnea on

    exertion. Clinical examination revealed jugular venous

    distention and a diastolic rumble that increased with

    inspiration, consistent with tricuspid stenosis.On exami-

    nation this patient alsohad a diastolic rumble and opening

    snap at the left sternal border, consistent with mitral

    stenosis;a blowing diastolic murmur, consistent with aor-

    tic insufficiency; and a grade II/VI systolic ejection mur-

    mur. A chest roentgenogram evealed cardiomegaly, but

    there were no signsof pulmonary congestion.An electro-

    cardiogram revealed atria1 fibrillation with a frontal plane

    QRS axis of +30 degrees.Doppler echocardiography ex-

    amination revealed severe tricuspid valve stenosis,mild

    mitral valve stenosis,and minimal aortic valve stenosis.

    Doppler examination also showedmoderate tricuspid in-

    From the Minneapolis Heart Institute.

    Reprint requests: Irvin F. Goldenberg MD Minneapolis Heart Institute

    920 E. 28th St. Suite 160 Minneapolis MN 55407.

  • 8/10/2019 Percutaneous Double Balloon Valvuloplasty for Severe Tricuspid Stenosis. AHJ 1989

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    4 1a Brief Communications

    I

    PRE TRICUSPID VALVlJLOPL 4STY

    I

    August 1989

    American Heart Journal

    RV

    Tricuspid Valve Gradient

    2

    = I

    Tricuspid Valve Area 1.64 cm2

    c< rdiac Output

    I

    1

    3.05 L/min

    Fig. 1.

    Simultaneous right atria1 (RA) and right ventricular (RV) pressure recordings before (top panel)

    and after (bottom panel) tricuspid valve balloon valvu loplasty. Following balloon valvu loplasty there is a

    marked reduction in the transtricuspid valve gradient.

    suffic iency and minimal mitral and aortic insuff iciency.

    Cardiac catheterization confirmed these findings. Because

    her prominent lesion was tricuspid stenosis and her pre-

    dominant sympt om was fatigue, we elected to perform

    percutaneous balloon valvuloplasty of the tricuspid valve.

    After informed, written consent was obtained, the pa-

    tient was premeditated with 10 mg of diazepam. Aortic

    pressure was monitored by placing a 5F catheter in the de-

    scending aorta from the lef t femoral artery. Using the right

    and lef t femoral veins, a 7F Swan-Ganz catheter (Baxter

    Healthcare Corp., Edwards Division, Santa Ana, Calif. )

    was placed in the right atrium and right ventricle . Trans-

    tricuspid mean and end-diastolic pressure gradients, car-

    diac output, and tricuspid valve area (Gorlin formula) were

    calculated before and after tricuspid valvu loplasty. A right

    ventriculogram was performed with a 7F Berman catheter

    (Arrow, Reading, Pa.) before and after balloon valvulo-

    plasty. After obtaining initial baseline hemodynamic mea-

    surements, a 14F introducer was placed into both femoral

    veins. Using a 7F Berman balloon wedge catheter, an

    0.038-inch Teflon-coated exchange guide wire (250 cm

    long) was advanced into the main pulmonary artery f rom

    each femoral vein. A Mansfield 20 mm

    x

    3 cm balloon

    catheter (Mansfield Scientific Inc. , Mansfield, Mass.) was

    threaded over each guide wire and the balloons were posi-

    tioned across the tricuspid valve. The first three inflations

    were performed with only one balloon across the valv e. The

    next eight inflations had both balloons across the valve.

    The balloons were inflated to a maximum of 4 atm. The

    mean transvalvu lar gradient decreased from 7 to 2 mm Hg

    and the transtricuspid end-diastolic gradient decreased

    from 5 to 0 mm Hg (Fig. 1). Tricuspid valv e area increased

    from 0.82 to 1.64 cm2. Tricuspid insuffic iency that was

    moderate initially did not change. The patients functional

    status improved immediately following valvuloplasty, from

    New York Heart Association class III to New York Heart

    Association class II. The patient was discharged 48 hours

    after admission. During a 3-month follow-up period, the

    patients clinical improvement persisted.

    Prior studies have shown that percutaneous balloon val-

  • 8/10/2019 Percutaneous Double Balloon Valvuloplasty for Severe Tricuspid Stenosis. AHJ 1989

    3/3

    Volume 118

    Number 2

    Brief Communications

    4 19

    vuloplasty offers a promising alternative to surgical val-

    votomy in somepatients with pulmonic, aortic, and mitral

    stenosis.le4 his casedemonstrates hat tricuspid stenosis

    can also espond o this treatment modality. The improve-

    ment in tricuspid valve area and symptoms n our patient

    are similar to those reported after tricuspid valve

    commissurotomy.6n our case,however, improvement

    was

    obtained without the morbidity and cost of surgery. Al-

    though we have demonstrated he feasibility of percutane-

    ous balloon valvuloplasty for the treatment of severe

    tricuspid stenosis,additional investigation will be needed

    to assesshe long-term efficacy of this new therapeutic

    modality.

    REFERENCES

    1.

    2.

    3.

    4.

    5.

    6.

    Kan JS, White RI, Mitchell SE, Gardner TJ. Percutaneous

    balloon valvu loplasty: a new method fo r treating congenital

    pulmonary valve stenosis. N Engl J Med 1982;307:540-2.

    Waldman JD,

    Schoen

    J, Fitzpatrick SE, Mathewson JW,

    George L, Lambert JJ. Balloon dilatation of stenotic porcine

    pulmonary valves. Clin Res 1987;35:204A.

    Cribier A, Savin T, Berland J, et al. Percutaneous translumi-

    nal balloon valvu loplasty of adult aortic stenosis: report of 92

    cases. J Am Co11 Cardiol 1987;9:381-6.

    Kveselis DA, Rocchini AP, Beekman R, et al. Balloon angio-

    plasty for congenital and rheumatic mitral stenosis. Am J

    Cardiol 1986,57:348-50.

    Ribeiro PA, Zaibag MA, Kasab SA, Idris M, Halim M, Abdul-

    lah M, Shahed M. Percutaneous double balloon valvotomy for

    rheumatic tricuspid stenosios. Am J Cardiol 198&61:660-l.

    Trace HD, Bailey CP, Wendhos MH. Tricuspid valve com-

    missurotomy with a one-year follow up.

    AM HEART J 1954;

    47:613-17.

    Presbyesophagus masquerading as an

    extracardiac mass on echocardiography

    Brian D. Hoit, MD, and Dave Eppert, RDMS.

    Cincinnati, Ohio

    Two-dimensional echocardiography has proven useful for

    the detection of mediastinal masses.Although the esoph-

    aguscourses hrough the posterior mediastinum with an

    intimate spatial relationship to the heart, cardiac compres-

    sion due to esophageal isease s infrequently reported.2p

    In this report, we describe a case n which an asymptom-

    atic, functional disorder of esophagealmotility presented

    as an extracardiac masson two-dimensional echocardio-

    wvb.

    A 75-year-old man was admitted to University of Cin-

    cinnati Hospital with a history of lethargy and fatigue.

    There was no dysphagia or prior symptoms of upper gas-

    trointestinal disease.On physical examination the patient

    From the University of Cincinnati Medical Center Division of Cardiology.

    Reprint requests: Brian D. Hoit MD University of Cincinnati Medical

    Center D ivision of Cardiology 231 Bethesda Ave. M.L. 542 Cincinnati OH

    45267.

    Fig. 1. Left parasternal long-axis

    (upper panel)

    and

    apical four-chamber

    (lower panel)

    two-dimensional

    echocardiogram.A large, echogenicmass M) compresses

    the posterior wall of the left atrium.

    wasconfused.The vital signsand ugular venouspressure

    were normal. The lungswere clear. The heart soundswere

    normal and there was a 2/6 late systolic ejection murmur

    at the lower left sternal border. The abdomen was dis-

    tended, and the prostate wasenlarged.There wasbilateral

    lower extremity edema. An anteroposterior chest radio-

    gram wasnormal. Electrocardiogram revealed evidence of

    an anteroseptal myocardial infarction of undetermined

    age. Abdominal distension was relieved by insertion of a

    Foley catheter, which drained 1800 cc of urine. The

    patients mental status improved, and an echocardiogram

    wasperformed to evaluate the systolic murmur.

    Two-dimensional echocardiography (Fig. 1) revealed a

    large masscompressing he posterior border of the left

    atrium. A color Doppler study revealed a late systolic et of

    mitral regurgitation that wasdeflected towards the lateral

    wall of the left atrium by the mass.A computed tomogra-

    phy (CT) scanof the chest evealeda distended, luid-filled

    esophagusrom the esophagogastricunction to the upper

    esophageal phincter. The remainder of the mediastinum

    was

    normal. An air-fluid level with a very poor stripping