pes planus by dr. mohammad azhar ud din darokhan
DESCRIPTION
ppt on pes planusTRANSCRIPT
Moderator: Prof. Sudesh Sharma
Presented by: Dr. Mohammad Azhar
2nd year post graduate
Deptt of Orthopaedics
G.M.C Jammu
This truism from a British Medical Journal editorial sums up the problem of ‘normally abnormal’ feet
Also known as FLAT FOOT.
Medial border of the foot is abnormally in contact with the floor during weight bearing
Low or absent medial longitudinal arch
When associated with deformities of the hind, mid and fore foot – pes plano valgus
ANKLE – plantarflexion and dorsiflexion
SUBTALAR (TALOCALCANEAL) – inversion and eversion
MIDTARSAL – adduction and abduction, flexion and extension, supination and pronation
Forefoot - abduction and supination (relative to hind foot)
Talar head - displaced medially, anteriorly and downwards
Calcaneum everts, dorsiflexes - hindfoot is in valgus
Navicular- subluxates dorso-laterally, uncovering the talar head
The medial column of the foot appears to be longer than the lateral column
Navicular, cuneiform, cuboid become wedge-shaped, with apex directed dorso-laterally
Plantar, spring and deltoid ligaments are stretched
Anterior, posterior tibial tendons and plantar muscles are stretched whereas the achilles tendon and peronei become adaptively shortened
Calluses develop over the medial bony prominences
Standing anterior-posterior (AP) (dorsoplantar), lateral, and oblique views Harris-Beath views if a tarsal coalition is suspected
There are basically 3 components that are involved in producing the alignment abnormalities of symptomatic adult flatfoot:
1. Collapse of the longitudinal arch
2. Hindfoot valgus3. Forefoot abduction
Meary’s angle - between long axis of talus and long axis of first metatarsal on a standing lateral X ray
long axis of the talus should nearly bisect the navicular and first metatarsal shaft
0 degrees – normal
0 – 15 degrees – mild
15 – 40 degrees – moderate
> 40 degrees – severe
The location of the sag, talo-navicular, naviculo-cuneiform or both can also be determined
Two lines are drawn, one connecting the edges of the articular surface of the talus, and one connecting the edges of the articular surface of the navicular. The angle formed by these two lines is the talonavicular coverage angle
Normally it is less than 7 degrees
This is an indication of forefoot abduction
Calcaneal pitch – A line is drawn from the plantar-
most surface of the calcaneus to the inferior border of the distal articular surface. The angle made between this line and the transverse plane is the calcaneal pitch
Normal 17-32 degrees, in flat foot is decreased May be 0 or negative in case of tightened TA
Normal talar-1st metatarsal angle on AP view. A line drawn trough the mid-axis of the talus passes through the base of the first metatarsal and is angled laterally in relation to the long axis of the shaft of the metatarsal.
Abnormal talar-1st metatarsal angle, angled medial to the first metatarsal.
it indicates pes planus.
The normal range is 25-45 degrees
This is the angle formed by the intersection of a line bisecting the head and neck of the talus and a line running parallel with the lateral surface of the calcaneus. The range of normal for adults is 15 - 30°
Abnormally increased AP talocalcaneal angle, more than 30 degrees indicating hindfoot valgus in pes planus.
is an architectural term designating the union of two curve lines. A normal midtarsal joint should create a smooth cyma between the talonavicular joint and calcaneocuboid joint on both the AP and lateral views
If the cyma line is broken it suggests “shortening” of the calcaneus relative to the talus
This is often just a radiographic shortening possibly due to rotation of the talus on calcaneus (typically seen in a patient with adult flatfoot including loss of the medial arch)
Exact incidence not known???
One of the most common orthopedic deformities
Affects 15 - 20% of adults, mostly asymptomatic
Of this 2/3rd have flexible flatfoot , 1/4th have a contracted tendo-achilles associated with a
flexible flatfoot and the remainder have rigid flatfoot the most common
cause being tarsal coalition
The medial longitudinal arch normally develops during the first decade of life
Therefore flatfeet are usual in infants, common in children and rare in adults
Flatfoot in an infant is actually a ‘fat foot’ as the excessive amount of fat obscures the arches
An arched foot is a distinctive feature of man
A) Two longitudinal arches
Medial longitudinal arch
Lateral longitudinal arch
B) Transverse arch
Supports body weight in upright posture
Acts as a lever to propel the body forwards in walking, running and jumping
Acts as a shock absorber
Concavity of the arches protects the soft tissues of the sole against pressure
Ends : Anterior : 1-3 MT heads Posterior : Medial tubercle of calcaneum
Summit: Superior articular surface of body of talus
Pillars : Anterior: Talus, navicular, 3 cuneiforms, 1-3 MTPosterior: Medial half of calcaneum
Shape of bones: wedge shaped with apex pointing downwards. The talus acts as a key-stone
Intersegmental ties: ligaments and muscles
Spring ligament
Dorsal ligaments - interosseus talocalcaneal ligament
Tendinous extensions of tibialis posterior
Tie beams or bow strings : connect two ends of an arch
Medial part of plantar aponeurosis
Medial part of the FDB
Abductor hallucis, FHL, FHB
Medial part of FDL.
FACTORS RESPONSIBLE FOR MAINTENANCE OF MEDIAL ARCH (as compared to stone bridge)
FLEXIBLE
Depending on mobility of tarsal joints
RIGID
PHYSIOLOGIC – due to ligamentous laxity in 1st decade
HYPERMOBILE FLATFOOT – excessive ligamentous laxity – familial, Down’s, Marfan’s, Ehlers-Danlos, Osteogenesis Imperfecta
BONY ABNORMALITIES – hypoplasia of sustentaculum tali, hypoplastic calcaneum
OCCUPATIONAL OBESITY
MOTOR WEAKNESS – PTTD, accessory navicular, muscular dystrophy, peripheral nerve lesions, cerebral palsy, spinal cord conditions like polio, myelodysplasia, Werdnig – Hoffman disease, spina- bifida
SECONDARY TO ANATOMIC DEFECTS ELSEWHERE :
Ext. rotation of the limb
Genu valgum
Equinus deformity of the ankle (tight tendo- achilles)
Varus deformity of the foot
Congenital
Tarsal coalition
Congenital vertical talus
Acquired
Inflammatory arthrosis, Traumatic arthrosis
Charcot foot
Residua of clubfoot
Contractures of peronei or TA - Rheumatoid arthritis, Gout, Degenerative arthritis, Infection, Acute sprain, Osteochondral fracture, Foot tumors especially osteoid osteoma
Hereditary condition
Marked ligamentous laxity
Deformity disappears when feet are freed of weight bearing
Weight bearing axis - shifted medial to normal position
Prolonged weight bearing in the everted foot - Heel cord
contractures ( flexible flatfoot associated with tight heel cord)
No broad consensus
Unstable architecture of tarsal bones
Congenitally short tendo achilles
Weakened muscle power
Ligamentous laxity
Age of presentation: adolescence
Usually bilateral and asymptomatic
Family history of flatfeet and joint hyper mobility
Pain, discomfort, burning sensations and fatigue on activity and prolonged standing, cramping at night
Felt around the navicular, talocalcaneal joint, below the medial malleolus or at the ant. or post. extremities of the plantar ligaments
Flatfoot only on weight bearing
Deformity correctable on tip toe standing
Jack’s (great toe extension) test - the arch can be restored by simply dorsiflexing the great toe – suggests that sag is at the naviculocuneiform level
Examine the tendo-achilles for tightness (TA contracture tends makeS flexible flatfoot symptomatic)
Short tendo-achilles: limited dorsiflexion(not able to walk on heels)
Harris and Beath documented that presence or absence of the longitudinal arch did not corelate with the disability and a flatfoot was compatible with normal function unless associated with a tight tendo-achilles
Examine ROM of ankle,subtalar, midtarsal joints
Examine the gait
Generalized ligamentous laxity
Hypermobility of the subtalar and mid-tarsal joints: the forefoot can be bent outwards and upwards to an unusual degree
Spine, hips and knees should be examined
General examination for neuromuscular abnormalities
Don’t forget to examine the shoes
shoes show excessive wear along the medial border
Pedobarography A record of pressures can be obtained by making the patient
to stand and walk on a force plate. Mainly used to compare pre and post operative function
Footprints made with the aid of an ink pad show the difference between normal sole contact andflat-footed contact. (a) Normal footprint, showing the main contact areas across the anterior metatarsal arch, thelateral border of the foot and the heel, with a ‘hollow’ corresponding to the medial arch.
(b) Flat-footed contact, across the sole to the medial side of the foot
Physiological flexible flatfoot with full ROM is asymptomatic
It does not cause pain or disability Xrays are not indicated and
treatment is not required Child should be left alone If symptomatic always look for
associated causes most commonly tight heel cord
Mainstay of treatment as This is what is required in majority
Condition is essentially benign
Only symptomatic treatment possible
No change in ultimate shape of the foot – it is the parents and grand parents who need
treatment and not the child
Conservative treatment should always be tried first
Arch supports, rubber inserts, Plastizote
Whitman valgus brace
UCBL (University of California Biomechanics Laboratory) heel inserts
Shoe modifications –Thomas heel or a 1\4 inch wedge on the inner border
Custom molded orthotics
Medial heel wedge
Do not alter underlying structural fault
Do not encourage redevelopment of the arch
Running sports shoes have been found to be as effective as traditional orthoses and are more socially acceptable
They reduce shoe wear and are said to be more effective in treating shoes rather than feet
Excercises are designed to improve the strength of invertors and the plantar flexors
Toe-walking and multiple toe-ups If tendo-achilles is contracted, stretching it actively and
passively is an important form of management Grasping marbles with toes Heel to toe walking Playing in sand Ballet dancing Walking on a supination board
There is no scientific study evaluating the effectiveness (or lack of it) of these exercises
Reserved for patients with intractable symptoms unresponsive to shoe or orthotic modifications and who are unable to modify pain producing activity
Limitation of daily activities is an indication for surgery
Surgery for flexible flatfoot should not be performed for cosmetic reasons
Arthrodesing procedures should be delayed until 10 and preferably 15 years
Before 10 years arthrodesis is difficult because of excessive cartilaginous component of tarsal bones
Subsequent bony growth is retarded
Patient must be prepared to accept permanent loss of inversion-eversion motion
Achilles tendon lengthening is included if the ankle lacks at least 10 degrees of dorsiflexion with the knee extended
If patient has severe enough symptoms to warrant surgery, then heel cord lengthening should be part of a comprehensive procedure to reconstruct the arch
TECHNIQUE 3 small insicions( 2 medial, 1 lateral) along the length of the
tendon Tendon is cut from midline outwards Tendon sheath is repaired to prevent scarring Closure is done with knee extended and ankle dorsiflexed Long leg cast with ankle in neutral is given for 6 weeks
TECHNIQUE Elevation of tibialis posterior tendon Elevation of osteoperiosteal flap from proximal
to distal Naviculocuneiform arthrodesis Advancement of osteoperiosteal flap Advancement of tibialis posterior
Displacement of the posterior half of the calcaneus medially
Reestablishes the weight bearing line
Indicated in cases with excessive heel valgus
Koutsogiannis calcaneal osteotomy
Osteotomy is fashioned in a coronal plane 1.5 cm
posterior to the calcaneocuboid joint between the anterior and middle facets
This is not a simple opening wedge osteotomy, but rather a lengthening distraction wedge osteotomy, and it requires a trapezoid graft
Tricortical iliac crest graft is inserted between the anterior and middle facets of the calcaneus
Additional internal fixation is required
Nonunion of calcaneal graft
Displacement of the graft requiring revision
Diplacement of the calcaneocuboid joint
Recurrence of deformity or pain
Indicated for correction of residual deformities in flat foot
Forefoot supination is corrected by a plantar medial closing wedge osteotomy of first cuneiform
The goal is to plantar flex the first ray down to the level of the fifth metatarsal to restore Cotton's normal “tripod” configuration.
Tarsal coalition (peroneal spastic flatfoot, congenital rigid flatfoot) [most common cause]
Heel cord tightening
Accessory navicular
Vertical talus
Cannot be passively manipulated without causing pain
Feet are flat - regardless of weight bearing / position
Pain is usually a prominent symptom.
Thin or thick bar composed of bone (synostosis), cartilage (synchondrosis) or fibrous tissue (syndesmosis) connects tarsal bones
Failure of embryonic segmentation
Calcaneum is held in eversion
An irritative focus is produced which causes painful spasm of the peronei
Impossible for the patient to walk on the lateral border of the foot due to limited inversion
Mechanics of the tarsus is impaired and abnormal stresses result casing sec. degenerative arthritis
Symptoms : do not develop until ossification of the fibrous syndesmosis or the cartilagious synchondrosis
Syndesmosis and synchondrosis are usually more troublesome than synostosis
Symptoms – vague active adolescents with dorsolateral foot pain around the sinus tarsi, difficulty in walking on uneven surfaces, foot fatigue, painful limp
Tenderness is present along the bar
The condition is known to run in families
Auto. dominant inheritance with variable penetrance
50% bilateral
Incidence - 0.4-6%
Symptomatic at 8 – 12 yrs
Varying loss of subtalar motion
Best seen on a 45 degree lat oblique projection
Beaking of dorsal articular margin of talus is uncommon
CT is usually not required
Middle facet talocalcaneal coalition is most common
Symptomatic at 12 – 16 yrs of age
Marked reduction or absence of subtalar motion (cardinal sign)
Best seen on a Harris Beath axial calcaneal view – posterosuperior oblique projection
Talar beaking is commonly seen – traction spur and not a sign of degenerative arthritis
CT is usually needed for diagnosis(in coronal plane at 3mm increments)
Harris axial calcaneal view for middle facet coalition
Most patients respond to conservative treatment – Rest
Shoe inserts (arch supports)
Orthotics (AFO, Plastizote, UCBL insert)
Shoe modifications (high top shoes, Thomas heel, Whitman plate) 4-6 weeks of immobilization in a short leg walking cast with the foot
plantigrade may provide lasting relief of symptoms
Splintage with an outside iron and inside T-strap
Resection of the bar and interposition of muscle, fat or gelfoam – should be performed before secondary degenerative changes have set in
Calcaneal osteotomy can be combined to to correct hind foot valgus
Subtalar arthrodesis
Triple arthrodesis
Extensive talocalcaneal coalition
Multiple coalition
Development of sec. degenerative arthritis
Ball and socket ankle joint
When the coalition involves more than 50% articular surface of talocalcaneal joint or more than 50% of the posterior facet
The concept
“limiting the ability of the calcaneus to externally rotate and the talus to internally rotate”
maintenance of correction of the arch was possible.
inserted in a screw fashionbetween the lateral process of the talus and the anteriorprocess of the calcaneus and prevents external rotation of the calcaneus on the talus
intraarticular device that is inserted under the lateral processof the talus in the lateral most portion of the subtalar joint andelevates the lateral aspect of the talus
is inserted in bone in the floor of the sinus tarsi and acts in a similar fashion to the selflockingwedge by preventing external rotation of the calcaneusunder the talus
In summary, after review of the literature, several findings seem to be consistent:
1. Insertion of a sinus tarsi blocking implant, whichever design is used, seems consistently to reduce the pes planus deformity and, at least in short-term follow-up studies
2. A significant incidence of sinus tarsi pain requiring implant removal has been noted, and this pain does not always resolve with removal of the implant.
3. Follow-up can be characterized as midterm at best with no truly long-term studies available at this point.
4. Further studies are needed before these devices can be recommended for general use.
5. The literature appears to indicate that the best use of these implants is in children with symptomatic pes planus who have combined neuromuscular disorders
First described by Bauhin in 1605
Also called prehallux, accessory scaphoid, os tibiale externum, os naviculare secondarium and navicular secundum
Separate ossification center for the tuberosity of the navicular
Prevalance 5-10%
Cause and effect relationship with flatfoot has not been shown
3 types
Round sesamoid bone within TP tendon - rarely symptomatic
8-12 mm ossicle connected to the navicular by a synchondrosis. This is the type that is usually symptomatic as the synchondrosis is at risk of disruption from traction injury / shear forces
Navicular beak / Cornuate navicular -fusion of acc. navicular with the primary navicular.
Usually asymptomatic, noticed incidentally
Presentation - adolescence
Pain over an enlarged area at the medial aspect of the navicular just at the insertion of the tibialis posterior tendon
Pain aggravated by wearing tight-fitting shoes
Accessory navicular is best seen on the external oblique view
Accessory navicular ossifies even later than a normal navicular which is the last tarsal bone to ossify
CT can identify an accessory navicular
Bone scan can identify a hot accessory navicular
Soft pads, avoid wearing tight fitting shoes
Special shoes, valgus correcting shoe inserts( UCBL devise)
Steroid and analgesic injections
Strenghening of tibialis tendon and treatment of tendonitis
Immobilization in a short leg cast
Simple excision of the accessory navicular shelling it out of the post. tibial tendon
Navicular is resected until it is slightly depressed relative to the talus and cuneiform
Bone wax is applied to the to prevent regrowth
Good or excellent result in 93% cases
Involves excision of the accessory navicular with re-routing of the central slip of the tibialis posterior laterally onto the plantar surface of the navicular, where it is sutured under tension to the surrounding ligaments
Gives no added advantage in short term and long term follow up and therefore the simpler procedure is preferred
Congenital rigid flat foot, rocker bottom foot, convex pes valgus or teratologic dorsolateral dislocation of
the talo-naviculo-cuneiform joint
First description by Henken in 1914
Characteristic features described by Lamy and Weissman
X-ray shows the vertical talus pointing downwards towards the sole and the other tarsal bones rotated around the head of the talus
after bilateral operative correction at age 14 months in which transverse circumferential approach was used.
Congenital dislocation of talonavicular joint such that the talus is disposed vertically with its head forming the most prominent part of the sole
The navicular is displaced dorsolaterally firmly lodged on to the neck of the talus, preventing reduction. The navicular abuts the ant. surface of the tibia
The calcaneum is displaced posterolaterally in relation to the talus, is rigidly locked into equinus and in contact with the distal fibula
The angle between the long axis of the talus and calcaneum is markedly increased
The forefoot is deviated outwards and dorsally and hence the sole has a convex contour
Dorsolateral dislocation or extreme subluxation of calcaneocuboid joint might occur
Abnormal relationship of tarsal bones remain constant whether the foot is plantar flexed or dorsiflexed, this is in contrast to congenital flexible flatfoot
Achilles tendon is contracted, ant. tibial and peroneal tendons are taught
The subtalar joint is abnormal with the anterior facet absent and the middle facet hypoplastic
Adaptive changes occur in the tarsal bones with weight bearing
The talus becomes shaped like an hour glass, with its longitudinal axis almost same as the tibia
Only the posterior 1/3rd of the superior articulating surface of the tibia articulates with the tibia
Anterior part of the plantar surface of the calcaneus becomes rounded
Callosities develop beneath the anterior end of the calcaneus and along the medial border of the foot superficial to the head of the talus
Muscle imbalance
Intra-uterine compression
Arthrogryposis
Autosomal dominant transmission
Arrest of fetal development of the foot between 7th and 12th weeks of gestation
Usually bilateral Sole is characteristically convex at birth, so that it
resembles the bottom of a rocking chair and hence the name
Dorsolateral fold is deep and situated at the mid-tarsal area Talar head is prominent over the medial and plantar
aspects Deformity from the outset is rigid Deformity may be so severe that heel might not touch the
ground at all Gait is awkward and resembles a waddle Shoes are rapidly worn out over the inner sides Pain - at adolescence or soon thereafter
Idiopathic flatfoot
Paralytic flatfoot
Spurious correction of clubfoot
Talipes calcaneovalgus (benign condition easily amenable to correction)
Tarsal coalition
Calcaneus is held in eversion by contracted interosseous ligament, bifurcated ligament and calcaneofibular ligament
Calcaneus is fixed in equinus by contracted posterior capsule and achilles tendon
Dorsal capsules of talonavicular, calcaneocuboid joints and tibio-navicular portion of the deltoid ligament are markedly contracted and prevent reduction
Tibialis anterior, long toe extensors, peroneus brevis and triceps surae are contracted
Posterior tibial and peroneal tendons may be displaced anteriorly so that they act as dorsiflexors rather than plantar flexors
Forefoot dorsiflexors are contracted
Calcaneonavicular ligament is elongated and attenuated
Posterior tibial tendon becomes attenuated as it passes over the displaced head of talus
If deformity persists into late childhood, alterations in the bony shape develop that encourage redisplacement even after surgery
Talus assumes hour-glass constriction, calcaneus becomes curved dorsally at its anterior end becoming beak shaped and navicular becomes wedge shaped
Difficult to treat tends to recur Serial casting to stretch the foot in
plantarflexion and inversion while counterpressure is applied to the medial aspect of the talus
Reverse Ponseti method Complete correction rarely achieved. Open reduction is generally required
1 - 4 yrs : soft tissue release, open reduction and realignment of the talonavicular and subtalar joints (KUMAR, COWELL, RAMSEY)
Children > 3 yrs with severe deformity generally require navicular excision at the time of open reduction
4 - 8 yrs : soft tissue release and open reduction with Grice-Green subtalar extra-articular arthrodesis
>12 yrs failure of above procedure striple arthrodesis
Should be done before 2 yrs Best done as a single stage release at 1 yrSTEPS Dorso-lateral soft tissue release Medial soft tissue release Reduction of talonavicular and calcaneocuboid
jts Posterior soft tissue release Internal fixation
Kodros and Dias reported a single-stage procedure
in which a threaded Kirschner wire is used as a “joystick”
To manipulate the talus into correct position.
The corrected position is held with threaded Kirschner wires across the talonavicular and subtalar joints
Results are satisfactory if surgery is done before 27 months
All feet have some residual midfoot sag and forefoot abduction and some have decreased motion
Commonest reason for surgical failure is inadequate reduction of the navicular
Aseptic necrosis of the navicular
Aseptic necrosis of the talus
These can be averted by limited amount of dissection
Examination of the flatfoot compares the (A) nonweightbearing and (B) weightbearing arch of the foot. As the arch depresses,(C) the forefoot abducts and (D) the lesser toes become visible upon posterior observation of the foot. The relaxed calcaneal stance position is viewed standing behind the patient. A flatfoot deformity will demonstrate heel eversion that is accentuated with apparent bowing of thetendo-Achilles (Helbing sign). The too many toes sign, indicative of excessive forefoot abduction in the flatfoot, may also be noted.
Most common cause of adult onset acquired flat foot
The components of the deformity are 1. hindfoot valgus, 2. midfoot abduction at the midtarsal joint, 3. forefoot pronation, primarily at the
midtarsal joint.
Chronic tenosynovitis (either traumatic, degenerative, or secondary to inflammatory arthritis), loss of continuity of the tendon (either complete or incomplete), and loss of the normal anatomical relationships of the tendon to its insertion or insertions (the accessory navicular or prehallux syndrome)
may render the posterior tibial tendon insufficient to perform its tasks of plantar flexion and inversion
and stabilization of the medial longitudinal arch.
The classification system originally developed by Johnson and Strom in 1989
Conservative management of stage II disease often is successful, and most patients obtain pain relief withapplication of an orthotic device that has a medial post anda double upright AFO with a medial T-strap.
The brace is configured to allow 20 to 30 degrees of plantarflexion and 10 degrees of ankle extension.
arthrodesis is indicated if conservative measures, including a double upright ankle-foot orthosis, have failed.
For rigid deformities, the procedure of choice usually is arthrodesis of the ankle or tibiotalocalcaneal arthrodesis
In a select group of patients with flexible, reducible deformity, less than 10 degrees of tibiotalar tilt, and minimal lateral ankle joint arthrosis
Jeng et al. described a “minimally invasive”allograft technique for deltoid ligament reconstruction for stage IV flatfoot deformity done in conjunction with triple arthrodesis.
Unilateral deformity that develops rapidly
History of trauma
Young patient- tendon transfer using flexor digitorum longus
Elderly- splintage
If this fails and symptoms are marked triple arthrodesis
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