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Brought to you by Similiacare.comBrought to you by Similiacare.com
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Day IIIWithDr.K.N.Ashok Kumar &

Dr.S.G.BIJU

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RECAPRECAP

You can add art to theScience by

Miasmatic correctionof

Pathological

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Investigationsshall be

interpreted interms ofMiasms

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StudyingDisease in

terms of

Miasm is

easy

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UrineUrine

Write down yourinvestigation

schemas in Urine

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Vanillyl Mandilic Acid inurine

Metabolite of

catecholamines

Pheochromocytoma &Neuroblastoma

Miasm

i iY Di i

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Your DiagnosisYour Diagnosis

ASA +Ve

HBe Ag + ve

ASO Titer 800

Anti Ds DNA + ve

Increased PSA

Leukocytosis (50,000)&

Increased K Level(10)Morethan 5

IHBR Dilated

ST Elevation & TWave Inversion

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StoolStool

Write down yourinvestigation

schemas in Stool

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Exercise - IV

Y Di iY Di i

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Your DiagnosisYour Diagnosis

Giardia

Taenia (Tape Worm)

Enterobis (Pin

Worms)

AscarisOccult blood

Cyst of Trichuria(whip)

Ova, Cyst ofAncylostoma

EH Cyst

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SyphilisPsoraTUB

Schistoso

miasis

(Urinarytract

Large

intestine

Liver

Spine

Taenia

Solium(Pork tapeWorm)

TaeniaSaginata

(Beef tapeWorm)

Hydatid

(Dog tape

Round Worm

(Ascaris

Lumbricoids)

Hook Worm

(Ancylostoma

Duodenal)Whip Worm

(Trichuria)

Pin worm(enterobiusVermicularis)

Filariasis

(Tissue

TREMATODSCESTODESNEMATODS
http://similiacare.com/http://similiacare.com/http://similiacare.com/

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Learn Pathology

in

Terms of Miasm

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Metabolic

Disorders

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ExerciseExercise

Write Down The

MetabolicDisorders with its

Miasmatic

Interpretation

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GLYCOGEN

PURINE

LIPID &

AMINO ACIDS

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Miasmatic InterpretationMiasmatic Interpretation

LIPID Syc Syphilitic

GLYCOGEN - Psoric SycoticSyphil

PURINE Tub Sycotic psoricSyph

AMINO ACIDS Tub Syphil

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DM

(Psoric Syco Syphil)

Bronzed DM(Hemochromatosis)

(Sycosis Syphilis)

Wilsons Disease

(Sycosis Syphilis)

Porphyrias

(TUB)

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HyperlipoprotenemiaHyperlipoprotenemia

The liver synthesizestriglycerades from

surplus carbohydrateobtained from diet. Thetriglycerides are

esterified and releasedin to circulation as Very

Low Density Lipoprotein

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TGL SPLIT OFF AND ENTERS

THE ADIPOCYTES.

VLDL remnant which contain mainlycholesterol esters gets physically

transformed in to Low Density

Lipoprotein (LDL)

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Role of HDL is to

transport cholesteroland remove lipid from

arterial walls. It reducesuptake of LDL by thecells.Increase level of

LDL or VLDL withdecreased level of HDL

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Lipoprotein (a) [LP (a)]Lipoprotein (a) [LP (a)]

LP (a) Lipoprotein A ispredominantly a geneticLipoprotein which will beconstant after puberty. It

composed of 27%protein 65% lipid and

8% carbohydrate.

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LP (a) Lipoprotein (a)LP (a) Lipoprotein (a)

A genetically determinedfraction with less

environmental influence.

High level of TGL and LDL increase the

risk.

LP (a) is highly Thrombogenic,Atherogenic and Antifibrinolytic

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LP (a) is advisable in allperson with Family

History of prematureatherosclerosis.

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Indication of LP (a)Indication of LP (a)

Hyperlipoprotene

mia even withlow fat diet

(Ginko Biloba)

Syco Psoric

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Syco Psoric

Syphilitic nature of Lipoprotein (a)due to

Fibrin Binding property andthus formation of clots (Short

term).

2. Clots due to lipoprotein

properties (long Term).

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LP(a) is ten

times moreaccurate andspecific for

prediction of

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Allied complaints withAllied complaints with

increased LP (a)increased LP (a)

Hypertension

DM

Kidney DiseaseCollagen Diseases

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P iPrimary

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PrimaryPrimary

Group I- TC (300 400) + TGL WNL +

Increased LDL (High Saturated fat diet +

Smoking < condition)

IHD in 50% cases after the age of 50.

Xanthomas (Tuberosum Bony prominence,Tendinosum tendo achillles) and Xanthelisma.

Group II Normal TC + IncreasedTriglycerides (Common association are Obesity,

DM, and Gout) Tendency to MI. Cancer Miasm

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SecondarySecondary

Increased LDLDM, (Syphilitic)

Hypothyroidism,(Sycoric)Nephrotic Syndrome, (Sycotic)

Biliary Obstruction, (Sycotic)

pancreatitis (Psoric)

Drug induced (Corticosteroids)

Sycotic

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Iron

Metabolism

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(Bronzed Diabetes)Hemochromatosis

Metabolic Disorderassociated with marked

increase in iron store inthe body.

Hepatocytes and kupffer cells

show stainable iron. Cirrhosis

Pigmentation + DM gives thePigmentation + DM gives the

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Pigmentation + DM gives thePigmentation + DM gives the

name Bronzed DMname Bronzed DM

Lethargy

Loss of libido

impotence

pigmentations

hepatomegaly

diabetes.

Testicular atrophy

Complication. HCC.Death with in 5 years

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Investigate if there

is

Hepatic Cirrhosis + DM+ Pigmentation

(+ Cardiac

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Serum Iron (Above 175)

S.Ferritin (Above 1000)

TIBC (Total Iron BindingCapacity)

= Serum Iron + Serum

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UIBC

(Unsaturated Iron-Binding Capacity)

= Subtracting Serum Iron from TIBC.

TIBC elevated when Total Body IronStores are low.

Sycotic Phase of the disease

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Copper

Metabolism

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Wilsons DiseaseWilsons Disease

Inborn error of metabolismof Copper.

Copper absorbed from

intestine bound withalbumin and then with

ceruloplasmin.

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In Wilsons disease

Absorption of copper isnormal or increased

but because of lack ofceruloplasmin in

plasma S.Copper isloosely bound to

albumin and get

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Copper get deposited inLiver

BrainCornea

Kidney

HeartMuscles

Cirrhosis of liverand destruction of baselganglia and renal tubules developed later.

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Relapsing Jaundice

Prolonged Jaundice

Portal HypertensionChorea (incordination),

Dystonia

hepatic failure.

Dementia

convulsions

Osteomalacia

renal rickents.Kayser Fleischer ring Deposition of copper

in cornea. loss of vision.

Fatality with in 5 14 years.

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InvestigationInvestigation

Liver Biopsy for copper.

S Ceruloplasmin Low, (Normal18-65 Mg/dl),

Total Serum Copper is below80u/Dl. Unbound copper is higher

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PorphyriasPorphyrias

A Metabolic error

involvingenzymes

concerned withtheheme

3 Types3 Types

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3 Types3 Types

Neurological

Cutanious

Cuto-neurological.

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Ulceration and scarring offace

ulceration with mutilation

of ears

mutilation of hands andfingers.

Resembles

AIP (A t I t itt tAIP (A t I t itt t

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AIP (Acute IntermittentAIP (Acute IntermittentPorphyria).Porphyria).

An inherited autosomal disease.Acute abdomen with behavioral

disturbances

depressionsuicidal tendencycoma.Severe Constipation or diarrhea

leads to GI Fluid losses anddehydration..

Portwine discoloration of freshly

passed urine on standing.

A i A id M t b liAmino Acid Metabolism

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Amino Acid MetabolismAmino Acid Metabolism

Mental

Retardation andshortened life

span.

H Ph l Al i i

Hyper Phenyl Alaninemia

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Hyper Phenyl AlaninemiaHyper Phenyl Alaninemia(Phenyl Ketonure _PKU)(Phenyl Ketonure _PKU)

An infantile Metabolicerror.

Hypopigmentation ofSkin and hairs.

Severe MentalRetardation,

Microcephaly Milk t (B f h l l i

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Milk < ts (Because of phenyl alaninein milk)

Phenyle alanine hydrylase is absentwhich is necessary for conversion ofPhenyle alnine to tyrosin.

So Phenyle alnine will be already inexcess.

Investigations : S.Phenylalanine(Above 20Mg/Dl)

MIASM ?

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Tubercular BecauseTubercular Because

Milk

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or More of the followingor More of the following

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or More of the followingor More of the following

criteria for more thancriteria for more than 6 weeks6 weeks

is Diagnosticis Diagnostic Morning Stiffness extendingover 1 hour

Arthritis of 3 or more Joints

Arthritis of Hand Joints

Symmetrical Arthritis

Rheumatoid Nodules Positive Rhumatoid factor

Radiological abnormalities.

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What is Your InvestigationWhat is Your Investigation

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What is Your InvestigationWhat is Your Investigation

Profile inProfile in

Undeveloped breast.Immaturity of

external genitalia.Poor deposition of fat

in buttocks, thighs &decreasedendometrial

thickenin

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E2 EstradiolE2 Estradiol

When You suspect

Retardedgrowth ofUterus,

Fallo ian Tube

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Normal rangeFollicular Phase 60 160 Pg/Ml

Mid Cycle 30 - 150 Pg/MlLuteal phase 60 200 Pg/Ml

TUBERCULINUM CALC PHOS

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> (More than)

Normal levelIndicates

Overian tumor

SYCOTIC LYC < Normal Level also indicates< Normal Level also indicates

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Normal Level also indicatesTurners Syndrome (AutosomalTurners Syndrome (Autosomal

Recessive- DNA) ResistantRecessive- DNA) Resistant

Ovary Syndrome (Sycotic Ovary Syndrome (Sycotic Thuja)Thuja)

Primary Hypofunction of Overy Primary Hypofunction of Overy Premature Menopause due toPremature Menopause due toAutoimmune EndocrinopathiesAutoimmune Endocrinopathies

((Syphilitic SYPHILSyphilitic SYPHIL))

Toxicity after radiation orToxicity after radiation orChemotherapyChemotherapy

-Cancer Miasm - Kali Phos Se .

E2 E t di l

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E2 Estradiol

is essential in patients

ART(Assisted Reproductive

Technology)


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Indication in Male Gynecomastia(Chloramphenicolum)

Increased PrecociousPuberty

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Unconjugated Estriol E 3Unconjugated Estriol E 3

It has a potential ProtectiveIt has a potential ProtectiveProperty againstProperty against

production of Cancer cells.production of Cancer cells.An investigation in case ofAn investigation in case of

habitualhabitual Abortion andAbortion and

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UnconjugatedUnconjugated

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UnconjugatedUnconjugated

Estriol E 3Estriol E 3

Persistently low orrapidly fallingE3 indicates

fetal distress andSEPIA!

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Low E3 indicatesfetal anencephaly,

placentalinsufficiency andDown SyndromeAnd Increased during normal

pregnancy No Need of Medicine.

ASABASAB

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BTB (Blood Testis Barrier)is a natural protectivemechanism that protects

the sperms from immunesystem. Tight connectionsbetween the cells liningmale reproductive tractkeep immune cells from

gaining entry to sperm Causes for development ofCauses for development of

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ppASABASAB

Vericocele (Sycotic) - LycTorsion of testis (syphilitic) Meny, Sil

Congenital Absence of Vasdifference (Syphilitic)

Testicular Biopsy (mechanical-

Psoric?) Arnica?Cryptorchism (Syphilitic) - Aur

Ca Testis (Cancer) - Con

Orchitis (Psoric) - S on TheThe Syco-

Syco-Syphlitic

Syphlitic ASAB!ASAB!

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yy ypyp ASAB immobilizes the spermASAB immobilizes the sperm

and thus prevent migrationand thus prevent migrationto Fallopian Tube. (Sycotic)to Fallopian Tube. (Sycotic)

ASAB attract destructiveASAB attract destructivephagocytes that attack andphagocytes that attack anddestroy the sperm.destroy the sperm.(Syphilitic)(Syphilitic)

ASABASAB binding to head ofbinding to head of

spermsperm and preventand preventpenetration of sperm in topenetration of sperm in toegg. (Syco-syphilitic)egg. (Syco-syphilitic)

ASAB interfere with theASAB interfere with the

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All About

All Diseases

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424 Diseases !50 Doctors

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Welcome Dr.SarathWelcome Dr.Sarath

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Welcome Dr.SarathWelcome Dr.SarathChandranChandran

How

homoeopathicMedicines are

Working in

pathological UTILIZATION STRATEGIESUTILIZATION STRATEGIES

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UTILIZATION STRATEGIESUTILIZATION STRATEGIESOF REPERTORIES.OF REPERTORIES.

W lW l

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WelcomeWelcome

T f i tiT f i ti

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Types of prescriptionTypes of prescription

1.Aeitiologicalprescription

a. Exciting cause

b.Maintaing causec.Fundamental

E iti CE iti C

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Exciting CauseExciting Cause

Aphorism 5 Useful to the physician in assisting him to cure are the

particulars of the most probable exciting cause of theacute disease, as also the most significant points in thewhole history of the chronic disease, to enable him todiscover its fundamental cause, which is generally dueto a chronic miasm.

In these investigations, the ascertainable physicalconstitution of the patient (and intellectual character, his

occupation, mode of living and habits, his social anddomestic relations, his age, sexual function, ..etc., are tobe taken into consideration.

E iti CE citing Ca se

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Exciting CauseExciting Cause

Physical cause

Nervous causeMechanical cause

Physical causePhysical cause

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Physical causey

APPU 12M/c

GENERALS -WEAKNESS - sea-

bath, after(1) Mag.m

N CNer o s Ca se

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Nervous CauseNervous Cause

Lekshmi 4F/c

FEVER - FRIGHT; after) chen-a

(chenopodium anthelminticum)

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maintaining causemaintaining cause

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maintaining causemaintaining cause

Manoj34M, Painter

ABDOMEN - PAIN -

cramping, griping -

lead poisoning;from

2.Patholagical prescription2.Patholagical prescription

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g p pg p p

THUMP Index MethodTHUMP Index Method

BEENA GEORGE 30F,Staff Nurse

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Gadha 4 Gadha 4

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Gadha 4 Gadha 4

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A Case of Hiccough

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A Case of Hiccough

STOMACH - HICCOUGH continued

(1) vert.v A case of Haemorrhoids

ABDOMEN - PAIN - hemorrhoidal flow;

suppressed (1) NUX.V

A case of Eczema

MIND - ADMIRATION, excessive (1) cic (cicuta virosa )

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Please observePlease observe

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Please observePlease observe

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Case of CroupCase of Croup

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Case of CroupCase of Croup

TEETH - GRINDING - fear; from -children; in

(1) kali-br

A Case of HeadacheA Case of Headache

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A Case of HeadacheA Case of Headache

HEAD - PAIN - menses suppressed

(5)PULS ,Acon,alum,sep,vert.v

ShajiShaji

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ShajiShaji

SHAJI 39MSHAJI 39M

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SHAJI 39MSHAJI 39M

SHAJI 39MSHAJI 39M

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SHAJI 39MSHAJI 39M

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Observe From The

Society

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School HeadacheSchool Headache

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School HeadacheSchool Headache

MIND - AILMENTS FROM indignation(25) STAPH ,PULS

MIND - AILMENTS FROM mortification

(64) COLOC ,STAPH IGN,NAT-M

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SHAJI . DyspSHAJI . Dysp

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S J yspJ y p

MIND ADULTEROUS MIND - AMBITION -increased fame; for

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MATHEW M JAMES 18MMATHEW M JAMES 18M

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JJ

MOUTH - SPEECH - stammering MIND - EXCITEMENT - stammers when

talking to strangers

MOUTH - SPEECH - stammering - fast;when talking

HEAD - DANDRUFF

GENERALS ALLERGIC constitution

LAC-C

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BINU 28MBINU 28M

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MIND - IMPATIENCE MIND - SQUANDERING - money

MIND - SQUANDERING - boasting, from

KIDNEYS STONES

NUX-V

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ARATHI GOPI 17FARATHI GOPI 17F

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MIND - ABRUPT, rough MIND - AVERSION - persons - certain, to

MIND - HURRY, haste

MIND TALKING-loud indisposed to talk

HEAD LARGE SIZE

FEMALE GENITALIA/SEX - MENSES - irregular

GENERALS - FOOD and DRINKS - sweets -

desire CALC

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MIND EFFEMINATE (5) PULS,Plat MIND - MANNISH - girls; mannish looking

(7) Nat.m

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MIND - TOUCHING everything; impelledto - children, in (2) Cina, carc

MIND - TOUCHING everything; impelled

to (10) Merc, Thuj MIND - TOUCHED - aversion to be -children; in (5) ant.c,ant.t,cina,cham,cupr

MIND - LOOKED AT; to be - cannot bearto be looked at

children;in(4)ant.c,ant.t,cham,cina

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MIND - AFFECTATION MIND - BOASTER, braggart

MIND - CURSING

MIND - EXAGGERATING

MIND - EXAGGERATING - symptoms; her

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ObservationObservation

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MIND - FROWN, disposed to (14) NUX-V,CHAM

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MIND - FEIGNING - sick; to be (16)Puls,Taret

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MIND - AILMENTS FROM - domination -children; in - parental control; long history

of excessive (3)Aur-m-n(aurum

muriaticum natronatum)carc,vanad

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Observe the

Society

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HEAD - PAIN -

jaundice, with(1) sep

At time (Not Place!) of StoolAt time (Not Place!) of Stool

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RECTUM - URGING- smoking, while

(2) calad,thuj

Occupation - teacherOccupation - teacher

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NOSE -

SNEEZING -

chalk, from (1)Nat.p

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Simple look of

Stool

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STOOL - MOSS;

like (1) asc-t.(asclepias

tuberosa)

STOOL GREEN

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COUGH - SUGAR -agg. (1) zinc

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GENERALS - COLD

- heat and cold(63)

FL.AC,LYC,NAT.M

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Irvin

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MIND AUDACITYMIND AUDACITY

(21)(21)Arn,Puls,TubArn,Puls,Tub,IGN,IGN

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Dont Close your

EyesIt is your duty to

observe

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Children RubricChildren Rubric

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MIND - JEALOUSY - children - newborngets all the attention; when the - Hys,

ign

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MIND - PRECOCITYof children

LACH,MED,VERAT

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FACE - EXPRESSION - old looking -children; in sars,sil

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Case taking is theultimatum of

Individualization. Butcertain Rubrics are theultimatum of

Individualization

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Mind is the

center core ofeach individual

but mind is nota necessary

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FINAL LAPFINAL LAP

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Knowing Disease isthe need of the

Hour

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FINAL LAPFINAL LAP

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Everything You Heard,Observed and Search

for is there inREPERTORY

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Case taking

starts fromObservations

and ends withinterpretations.

We will meet again

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g

for a wonderful dayexclusively for case

taking andRepertorization on

28th

O t b

Till ThenTill Then

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Good

Bye

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sahya part 16

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