blok 4.2 2015 - kegawatdaruratan ipd - endokrin metabolik
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Blok 4.2 2015 - Kegawatdaruratan IPD - Endokrin MetabolikTRANSCRIPT
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Dr. Eva Decroli SpPD , KEMD FINASIMSUB BAGIAN ENDOKRIN METABOLIK
BAGIAN ILMU PENYAKIT DALAMRSUP M.DJAMIL
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PENDAHULUAN
TUJUAN
AREA :1. KOMA HIPOGLIKEMIA2. KOMA HIPERGLIKEMIA3. KRISIS TIROID
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ETIOLOGI GAMBARAN KLINIS TATA LAKSANA MONITORING
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KOMA KETOASIDOSIS KOMA HIPERGLIKEMIA NON KETOTIK
ETIOLOGI : GAMBARAN KLINIK TATA LAKSANA MONITORING
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Ketosis
Hiperglikemia Asidosis
DKA
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FAKTOR PENCETUS GAMBARAN KLINIS TATA LAKSANA
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SSPRingan: agitasi (10)Sedang: delirium, letargi, psikosis (20)Berat : koma, kejang (30)
Suhu37,2-37,7 C (5)37,8-38,3 C (10)38,4-38,8 C (15)38,9-39,4 C (20)39,5-39,9 C (25) ≥ 40 C (30)
GITSedang: diare, mual/muntah, nyeri perut (10)Berat : ikterus (20)
Kardiovaskular• Takikardi: 90-109 (5)
110-119 (10) 120-129 (15) 130-139 (20) ≥ 140 (25)CHF: edema tungkai (5) ronkhi bendungan (10) edema paru (15)AF (10)
Pencetus (10)Indeks Burch-Wartofsky
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Penatalaksanaan:1. PTU → blok sintesis hormon baru2. Lugol → blok pelepasan hormon baru3. Kortikosteroid → blok konversi T4 perifer4. Propranolol → blok konversi T4 menjadi T35. Antipiretik6. Atasi faktor pencetus
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