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9/7/14
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Evaluation of a child with a limp / Slipped
Capital Femoral Epiphysis (SCFE)
Lee S. Segal, MD
Chief, Division of Pediatric Orthopedics American Family Children’s Hospital
Department of Orthopedics and Rehabilitation, University of Wisconsin School of Medicine and
Public Health Madison, WI
Evaluation of a child with a limp / Slipped Capital Femoral Epiphysis (SCFE)
I have no financial relationships related
to disclose
Introduction
• common presentation to pediatric orthopaedic office
• limp can be caused by pain, weakness, or mechanical factors
• requires thorough history and physical examination
Evaluation of the limping child
narrow differential dx based upon
1. Gait 2. Location 3. Age
– Toddler (<4y) – Child (4-‐10y) – Adolescent (>10y)
4. Pain
Normal gait Abnormal gait
• Trendelenburg • equinus (toe-‐walking)
• circumduction/ vaulting
• steppage
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Non-‐antalgic gait
• Equinus: CP, congenitally tight heel cord, LLD, clubfoot
• Trendelenberg: LCP, DDH, SCFE, Muscular dystrophy
• Circumduction/Vaulting: CP, LLD, any stiffness of ankle/knee
• Steppage: CMT, CP, Spina Bifida
History
• Is limp associated with or without pain? • PQRST-‐ characterize pain
– Provokes, Palliates – Quality – Radiate, Referred – Severity – Timing
• PMH • Family history
Physical examination
• gait • table-‐top exam (supine, prone)
• Take age of child into consideration with exam
Imaging • radiographs
(comparison, oblique)
• bone scan • ultrasound • CT • MRI
• CBC with Diff • ESR / CRP • Lyme titer • rheum (ANA, RF) • joint aspiration (cell count, etc)
Lab tests Critical Questions Flynn J, Widmann RF: The Limping Child: Evaluation and Diagnosis. JAAOS 2001
• Is the limp due to pain? • Did the pain occur suddenly or gradually,
or has it always been there? • Is the child systemically ill? • Does the child exhibit a specific gait pattern? • Can the pain be localized?
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Toddler (<4y) • Toddler’s fracture • DDH • osteomyelitis, septic
arthritis • JA, Lyme • tumor (benign/
malignant) • Non-‐accidental trauma
DDH: 3+8 month old ♀
In toddler, not uncommon to present with a LLD and limp
Risk Factors • 4 “F’s”: Breech, +FH, 1st born,
female (6:1) • Uterine compression syndrome • Associated structural
abnormalities • Ethnic groups • * majority of infants with DDH
don’t have a risk factor • * safe swaddling
Treatment depends on age of presentation
0-‐6 months • Pavlik harness 6-‐18 months • Hip arthrogram, +/-‐
adductor tenotomy, closed/open reduction and hip spica cast for 12-‐18 weeks
> 3 years late discovery DDH
open reduction with femoral shortening + pelvic osteotomy
Child (4-‐10y) • Pediatric bone and
joint infection / synovitis
• Legg-‐Calve-‐Perthes • fracture • OCD • Foreign body • Tumor • Lyme disease
• Transient synovitis • Septic arthritis • Osteomyelitis
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Transient Synovitis • Hip joint most often affected • Mild pain , fever, stiffness, lab abnormalities
(T<101, ESR< 50, WBC< 15,000) • Tolerate movement of hip if done cautiously • U/S shows minimal fluid in joint • TX-‐ Rest and NSAIDS • Improvement is usually within a few days to 2 wks • Wax and wane, recurrence
Septic arthritis hip joint commonly involved
§ Child appears toxic, irritable § Severe pain with joint ROM § U/S demonstrate joint effusion § Neonate often has multiple
joint involvement, may not mount inflammatory response (fever, ESR, WBC)
§ Don’t want to miss septic hip, severe sequelae
§ Confirm with aspiration
normal
fluid
Transient synovitis vs septic arthritis of hip
Prediction of septic arthritis is 93% with 3 or more of the following (Kocher criteria)
• Fever • NWB
• ESR >40 • WBC >12,000
* Does not replace the clinical exam
Septic arthritis of the hip
• Infant with septic arthritis of the left hip
• Hip held rigidly in the classic position of flexion, abduction, and external rotation
• position that maximizes capsular volume
• Infant relatively comfortable as long as the hip remains immobile in this position
Septic Arthritis
• Diagnosis is made via hip joint aspiration (US or in OR)
• Treatment is drainage and irrigation, followed by antibiotics
• Early treatment is important for a good result
• Identify the cause, determine which antibiotic, duration, route
Legg–Calve-‐Perthes Disease
• Most common in children 4 to 8 yrs old
• Males > females affected Insidious limp is most common presentation
• Limited hip abduction and internal rotation on exam
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LCP -‐ etiology Unknown – source of frustration,
2 prevailing theories
(1) disruption of blood supply * disturbance clotting mechanism * hyperviscosity Factor Leiden V, Protein S and C deficiency
(2) underlying systemic disorder primary disorder of epiphyseal cartilage? delayed skeletal maturation, shorter stature, ADHD
Perthes Disease temporal classification -‐ xray
Initial Fragmentation Reossification Healed 9 Mo 32 Mo 10 yr
classification
Extent of involvement
• Catterall • Lateral Pillar
prognosis
• Age at presentation (<6 years) • Extent of involvement • Perfusion MRI • Duration of disease process • Clinical ROM
Treatment principles goal – maintain sphericity femoral head
• Maintain ROM (PT, NSAID, petrie casts) • Containment (bracing vs surgery) • If >6 yo or lateral pillar B/C, hip subluxation, other
treatment may be considered (femoral or pelvic osteotomy)
• Long term prognosis depends on residual femoral head deformity
Adolescent (>10 yrs) CITMAT
• Sports -‐ related stress fractures (overuse, repetitive), OCD
• Transitional: SCFE, tarsal coalition, Osgood-‐Schlatter
• Inflammation: Juvenile arthritis, • Infection: Osteomyelitis, Lyme
disease • Tumor
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The limping child
• Common musculoskeletal presentation
• Determine gait pattern, associated with or without pain, age of child, and localization
• Establish differential DX, choose appropriate studies to confirm diagnosis
• Be familiar with treatment with relation to condition
Slipped Capital Femoral Epiphysis (SCFE) “the unsolved adolescent hip disorder”
• Most common adolescent hip disorder
• Missed or delay in diagnosis
• Asymptomatic, chronic SCFE “tilt deformity”
• Etiology remains unknown
etiology remains unknown
mechanical
genetic
inflammatory trauma
endocrine
Missed or delay in diagnosis
Kocher et al Pediatrics 2004 • Median delay 8 weeks • Significant relationship: delay in dx à
increase slip severity Matava JPO 1999 • Isolated distal thigh or knee pain
common presentation • Leads to higher rates of missed dx,
unnecessary x-‐rays, and more severe slips
Silent slip Silent slip
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Silent slip Silent slip à tilt / pistol grip deformity
Clinical presentation
• May complain of hip, thigh, or knee pain (Hilton’s Law)
• Extremity externally rotated • + roll test • +/-‐ ability to WB • ** Any older child or adolescent
with knee pain has a SCFE until proven otherwise
Treatment goals
• Preventing progression of the slip
• Promote closure of the growth plate
• Minimizing risks of AVN / chondrolysis
• Prophylactic pinning of contralateral hip controversial in NA
Narrow age range at presentation
• Males -‐ 12.7 yrs • Females -‐ 11.6 yrs • Beyond this range (+/-‐ 2 yrs) – consider endocrine or systemic disorder (atypical)
Atypical SCFE
• Below 10th percentile for height for chronologic age
• Bilateral presentation • Juvenile • Consider associated endocrine disorder :
hypothyroidism (esp. if < 10) • pituitary and parathyroid abnormalities • VDRR • Radiation exposure • GH treatment
Renal osteodystrophy
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smooth k-‐wires/semitubular plate (1996)
kidney transplant (2005)
Epidemiology
• Incidence -‐ 2/100,000 • Ethnic variation • Seasonal variation • Geographic variation (40o latitude)
• Bilateral 25% (?)
40o
classification
• Traditional (temporal) – Acute, acute on chronic, chronic
• Physeal stability • Severity of slip
Acute SCFE
• Onset < 3 weeks • Epiphyseal displacement
without remodeling • High risk AVN >15%
Physeal stability
Loder et al JBJS 75A, 1993 • Clinical evaluation • Unstable -‐ “patient presents with
severe hip pain, and is unable to walk with or without crutches, regardless of the duration of symptoms”
• Fluoroscopic evaluation • *prognostic importance
pathophysiology
• Direction of slip • True posterior • Hypertrophic zone of physis
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Torsional basis / SCFE
• Both coronal + sagittal planes of shear loading
• Displacement of CFE resolved by common torsional force
torsional basis of SCFE
radiographs ** must obtain both AP and
frog-‐lateral x-‐rays of the pelvis
• AP radiograph -‐ Difficult to recognize minimal slip
Clues • Klein’s line • Decreased epiphyseal
height • Blanch sign
AC 7-7-97
AC 9-30-1997 Goals of Treatment (older)
• Prevent further slip* • Promote premature physeal closure* • Minimize risks associated with SCFE
Evolving concepts with SCFE
• FAI – femoral acetabular impingement
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treatment • Always consider dx • Don’t send home • Differentiate stable vs.
unstable • Do not allow further
ambulation • Surgical “urgency” • standard of care North
America : in-‐situ single screw fixation
NORTH AMERICA • CANNULATED SCREW
FIXATION/single • B/L INCIDENCE 25% • OBSERVE
CONTRALATERAL HIP
GERMANY • K–WIRE FIXATION • B/L INCIDENCE 75% • STABILIZE
CONTRALATERAL HIP
Entry site different than pinning hip fracture
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risks • AVN • chondrolysis
• Unstable • Slip severity • Iatrogenic • Segmental vs global
pitfalls
a. Growth disturbance / premature closure b. Subtrochanteric fracture c. Strip screws upon attempt to remove
a.
b.
c. Pin in situ for severe SCFE In past used to consider osteotomies to compensate for deformity
osteotomies
AVN risk higher closer to deformity
A C
New concepts
• Contralateral hip – prophylactic stabilization
• Femoral acetabular impingement (FAI)
• Open surgical hip dislocation
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Femoral Acetabular Impingement (FAI)
CAM FAI
Open surgical hip dislocation Preserve blood supply
Surgical hip dislocation protects blood supply to femoral head
summary
• Any adolescent with knee pain is a SCFE until proven otherwise
• Always obtain x-‐ray of pelvis • Always obtain 2 views (AP/Frog) • Surgical “urgency” – don’t send home • Delay in Dx increases risk of slip severity
Thank you