Download - Unit 5 f11
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Unit 4 Problems of Cardiac Output and Tissue Perfusion
Lemone and Burke Ch 30-32
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ECG brief intro
P wave QRS T U http://www.youtube.com/watch?
v=rguztY8aqpk&feature=related
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Heart Sounds http://www.youtub
e.com/watch?v=ax9B6g6gEOc
http://www.youtube.com/watch?v=Ge12P7u0aQo&feature=related
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What is Cardiac Output? CO = HR x SV
CO=cardiac output HR= heart rate SV= stroke volume
Factors that affect SV: HR Preload Afterload Contractility
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Assessing CV status
Other than physical assessment History Family History Genetic Risk Personal History Diet History Socioeconomic Status
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Risk Factors Modifiable
HTN Diabetes Hyperlipidemia Cigarette smoking Obesity Physical inactivity Diet
Nonmodifiable Age Gender Genetic Factors
The text also discusses Metabolic Syndrome and Risk factors unique to women on page 964
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CV Assessment
Focused physical assessment General appearance Integumentary system
Color Temperature
Extremeties Blood pressure Edema
Venous flow and arterial pulses
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CV Diagnostic exams
Lab tests: CBC Serum electrolytes BNP
Mark cardiac damage Troponin CK-MB Myoglobin
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Cardiac lab tests
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Diagnostic exams
Chest x-ray Angiography Cardiac Catheterization ECG Nursing interventions???
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Diversity concerns CV client
Clients often fear diseases r/t cardiovascular system
Require good education, opportunity for client and family to voice concerns/fears
Support groups Cardiac rehab referral
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Pathophysiology of Common Cardiac Disorders
Heart Failure Infective Endocarditis Myocarditis Pericarditis
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Risk Factors and Preventive Measures for Cardiac Disorders
Heart Failure Risk factors
Coronary artery disease Cardiomyopathies Hypertension Congenital and valvular heart disease
prevention Education regarding coronary artery
disease and diabetes
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Pathophysiology of Common Cardiac Disorders
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Pathophysiology of Common Cardiac Disorders
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Right vs Left heart failure
Right Peripheral edema Weight gain anorexia
Left SOB Fatigue Crackles on
auscultation of breath sounds
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Heart Failure
Right vs Left Systolic vs
Diastolic High and low
output
Chronic vs Acute
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Risk Factors and Preventive Measures for Cardiac Disorders
Heart Failure Risk factors
Coronary artery disease Cardiomyopathies Hypertension Congenital and valvular heart disease
prevention Education regarding coronary artery
disease and diabetes
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Pulmonary edema
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Classic symptoms of Pulmonary Edema Rapid onset• Extreme shortness of breath or difficulty breathing• A feeling of suffocating or drowning• Wheezing or gasping for breath• Anxiety, restlessness or a sense of apprehension• A cough that produces frothy sputum that may be tinged
with blood• Excessive sweating• Pale skin• Chest pain, if pulmonary edema is caused by heart
disease• A rapid, irregular heartbeat (palpitations)
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Slow onset symptoms of CHF
• Increased shortness of breath when physically active.
• Difficulty breathing with exertion, often when lying flat as opposed to sitting up.
• Awakening at night with a breathless feeling that may be relieved by sitting up.
• Rapid weight gain when pulmonary edema develops as a result of congestive heart failure.
• Loss of appetite.• Fatigue
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Clinical manifestations of Inflammatory Heart Disease
Types of inflammatory diseases: Myocarditis Infective endocarditis Pericarditis Rheumatic Carditis
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Risk Factors and Preventive Measures for Cardiac Disorders
Myocarditis Risk factors are any thing that alters
immune response Advanced age Malnutrition Alcohol use Immunosuppression Exposure to radiation Stress
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Anatomy, Physiology, and Functions of the Heart
The Pericardium Double-layered fibroserous membrane
surrounding the heart Anchors the heart to surrounding structures
Space between layers is filled with pericardial fluid Lubricates heart muscle Helps to cushion the heart
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Anatomy, Physiology, and Functions of the Heart
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Infective endocarditis
An infection of the endocardium Common in clients who abuse drugs,
had valve replacements, systemic infections or structural cardiac defects
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Risk Factors and Preventive Measures for Cardiac Disorders
Infective Endocarditis Risk factors
Congenital deformities Tissue damage due to ischemic disease Valve prosthesis Intravenous drug use Invasive catheters Dental procedures or poor dental health Recent heart surgery
Prevention Education is key Prophylactic antibiotics
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Infective endocarditis Most common complication is heart failure 50% have embolic complications due to
vegetation Common to have clients with petechia and
splinter hemorrhages Diagnosed with positive blood culture or
echocardiogram Treat with antibiotics Often need antibiotics before dental
procedures
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Petechiae and splinter hemorrhage
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Pericarditis vs endocarditis
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Pericarditis Often follows a respiratory infection Often presents with pain in supine position
releived by sitting or leaning forward May hear friction rub with stethoscope Treated with NSAIDS relieved within 48 hrs.
depends on cause for further treatment Short term course of illness (2-6 weeks) for
acute Chronic may require surgery
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Pericardiocentesis
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Risk Factors and Preventive Measures for Cardiac Disorders
Rheumatic Fever and Rheumatic Heart Disease Risk factors
Crowded living conditions Malnutrition Immunodeficiency Poor access to health care Genetic factor may be present
Prevention Prompt identification, treatment Importance of finishing medications
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Ultimately when the heart fails, the patient will have shock
Chapter 11 Lemone and Burke
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Cellular Homeostasis and Basic Hemodynamics
Homeostatic regulation maintained primarily by cardiovascular system
Four physiologic components Sufficient cardiac output Uncompromised vascular system Sufficient blood volume and blood
pressure Tissues that are able to extract and use
oxygen
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Types of Shock Hypovolemic Shock
Affects all body systems Most common type of shock
Cardiogenic Shock Loss of pumping action of the heart
Obstructive Shock Impaired diastolic filling (pericardial tamponade,
pneumothorax)
Distributive Shock Also known as vasogenic shock
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Shock
Hypovolemic Too little circulating blood causes
decrease in MAP thus not meeting the body’s total need for oxygen
Internal hemorrhage GI bleed
External hemorrhage trauma
Dehydration
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Shock
Cardiogenic Heart muscle is unhealthy or pumping is
impaired Causes a decrease CO, afterload and
reduces MAP
This is seen with an MI
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Shock Obstructive Affects the heart muscles ability to
pump effectively The heart itself is normal however
manifestations outside the heart affect filling or contraction Cardiac tamponade Tension pneumothorax Pulmonary embolism
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Shock
Distributive Loss of sympathetic tone Vasodilation
Leaky capillaries Spinal cord injury Sepsis Anaphylaxis
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Interventions for Clients Shock
Medications Inotropic: increases cardiac contractility Vasopressors: used to treat neurogenic,
septic, or anaphylactic shock Opioids: used to treat pain Immunizations: tetanus prophylaxis
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Shock
Look at the patient Compensated vs uncompensated
Blood pressure Urine output HR RR
Mental status
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Emergency
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Cardiac Emergency
Crash cart BLS Systematic Approach Code Team
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Rapid Response Team
ABC Act Before Code
Early intervention Early recognintion = increased survival
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What if it happens to me?
Don’t panic Activate the code Blue
Call for help if no button exists
BLS (you know this) Get in there!
Record your findings
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Review your ABG’s
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Questions??
Mid Term Exam Next Week.Cumulative to include everything to now. You will have the whole class time to take the exam.