mvp handouts
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7/23/2019 MVP Handouts
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Mitral valve prolapse
Commonest MV lesion.
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Billowing of 1 or 2 leaflets into LA beyond AV junction in systole.
•
With more severe prolapse MR occurs.
•
Most cases are mild, asymptomatic, benign.
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Children with MVP rarely have MR or needsurgery.
MVP
• MVP may be congenital but is often
diagnosed in the teens. May be familial.
• May be caused by problems affecting part
of leaflet,1 or 2 leaflets, chordae tendinae, papillary muscle or valve ring.
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MVP
associated conditions
• Connective tissue disorders e.g. Marfan’s
• Inflammation – RF, IE.
• Myocardial ischaemia/infarct – affect
papillary muscle.
• Rupture of chordae in IE.
• IDIOPATHIC
• Associated with other CHD in children
MVP
• Prevalence 1-2% in children, 5-15% in
adolescents and young adults.
• In some studies F:M = 2:1.
• Associated with neuro endocrine imbalance
adrenaline/noradrenaline increased.
MVP
symptoms
• Most asymptomatic.
• Chest pain, palpitations, dyspnoea and
fatigue on exertion, anxiety attacks,
syncope.
• Associated skeletal abnormality – asthenic
build, increased arm span, arachnodactyly,
scoliosis, pectus excavatum.
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MVP
signs
• Mild cases of mitral valve prolapsesyndrome have only mid-systolic click.
• When MR occurs there is a late systolicmurmur.
• Murmur occurs earlier in systole on sittingup.
• If MR severe - signs of LA and LVenlargement +/- CCF.
MVP with MR
MVP
Investigations
• 2Decho parasternal long axis view shows prolapse of valve leaflets which may bethickened/redundant. Valve ring may be
enlarged.• MR may be demonstrated.
• ECG may show arrhythmias or non-specificST and T wave changes or chamberhypertrophy.
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MVP
management
• For asymptomatic or with only anxiety –
reassure, follow clinically and repeat 2DE
every 3 years. The prognosis is good.
• Patients with MR should be reviewed at
least annually
• For severe MR – surgery, usually repair.
MVP
management
• Beta blockers for palpitations, syncope,
chest pains.
• Appropriate CCF treatment including
ACEI.