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Acknowledgement
We humbly recognize the limitedness of our young minds thus, we had to consult
different people concerning this case presentation, people who are more knowledgeable than us.
Now we see this case study printed, we would like to express our utmost gratitude to our
significant others whom have inspired and helped us dedicate ourselves in the success of our
case study.
First, to the Almighty God, for giving and showering us blessings, further, strengthening
our spirit to face the inherent demands of the task assigned, thereby, molding us to become
useful citizens of this land.
To the family of our patient, for their willingness to be the subject for our case study, for
being responsive and open to inquiries during the interview process and for being cooperative in
everything that we did.
To our wonderful Clinical Instructors, Mrs. Mary Hazel Facundo, RN MN, Ms. Grace
Guitguiten, RN, MN, Mrs. Dinna Rose Bayog, RN MN, and Mrs. Yvonne Kuan, RN, MN, who
imparted their best knowledge for us to be able to work our best as student nurses, may they all
find it in their hearts to keep going and keep teaching the eager young minds of tomorrow.
To the residents-on-duty, staff, and nurses-on-duty at Southern Philippines Medical
Center Pediatrics Ward, for accommodating us and for being understanding and patient with us
during the whole time of our duty.
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Lastly, to our parents and family who never left us and remained with us during our hard
times, may they remain in our hearts all through the years as we finish and reach our goals.
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INTRODUCTION
We, the group 3 of Section 2A of the Ateneo de Davao University, conducted an
interview and physical assessment last February 9 and 10, 2012 at the Pediatrics Ward of the
Southern Philippines Medical Center as part of our case study in relation to Pediatrics
Abnormalities concept. Our case is about Errshen, who was admitted to the Pediatrics-Sick
Neonate ward, with a medical diagnosis of neonatal sepsis and suspected patent ductus
arteriosus.
The circulatory system is a vital system in the human body. Without it, there will be no
transporter of nutrients, water, and oxygen to our billions of body cells and carrier away of
wastes such as carbon dioxide that body cells produce.It is an amazing highway that travels
through your entire body connecting all sour body cells. (hes.ucfsd.org). But what if the system
itself has defect, will we still survive?
The first condition of our patient is septicemia or more commonly known as sepsis.
Septicemia is a serious systemic illness caused by bacteria and bacterial toxins circulating in the
bloodstream. During the past 30 years, it has become an increasingly common condition among
hospitalized patients. Of newborns with early-onset infection, 85% present within 24 hours, 5%
present at 24-48 hours, and a smaller percentage present within 48-72 hours
(www.emedicine.medscape.com). According to the World Health Organization, neonatal sepsis
accounts for 33% of over 40,000 newborns death in the Philippines each year.
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Onset is most rapid in premature neonates. Early-onset sepsis syndrome is associated
with acquisition of microorganisms from the mother. Transplacental infection or an ascending
infection from the cervix may be caused by organisms that colonize in the mother's genitourinary
tract, with acquisition of the microbe by passage through a colonized birth canal at delivery.
The second condition of our patient is Patent ductus arteriosus (PDA) which is one of the
most common congenital heart defects. The ductus arteriosus is a remnant of the distal sixth
aortic arch and connects the pulmonary artery at the junction of the main pulmonary artery and
the origin of the left pulmonary artery to the proximal descending aorta just after the origin of the
left subclavian artery. Most typically, it is a left aortic remnant. In PDA, abnormal blood flow
occurs between two of the major arteries connected to the heart. These arteries are the aorta and
the pulmonary artery. A right-sided patent ductus arteriosus can occur, or the ductus arteriosus
can be present on both the right and the left. Although a left ductus arteriosus is a normal
structure during normal fetal development, the presence of a right ductus arteriosus is usually
associated with other congenital abnormalities of the cardiovascular system, most typically
involving the aortic arch or conotruncal development. The presentation widely varies. Depending
on the size of the patent ductus arteriosus, the gestational age of the neonate, and the pulmonary
vascular resistance, a premature neonate may develop life-threatening pulmonary overcirculation
in the first few days of life. Conversely, an adult with asmall patent ductus arteriosus may
present with a newly discovered murmur well after adolescence.
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The incidence of this anomaly is twice in females than males. After Ventricular septal
defect the Patent Ductus Arteriosus is the commonest congenital heart defect in children. Around
the world, it accounts for approximately 10% in newborns (www.clevelandclinic.org). In the
Philippines, the case rises at an unacceptable increasing level. The patent ductus arteriosus
incidence best estimates for the Philippines suggest that approximately 0.25% children are
affected by this. Those cases that were left untreated have led to death by 78% (The Mindanao
Daily Mirror, 2009).
The group chose the case of Errshen which are neonatal sepsis and patent ductus
arteriosus primarily because we already have the prior knowledge on thess type of pediatric
illnesses, thus requiring us to apply this knowledge in the actual setting. This would serve as a
good avenue for us to develop our skills in relation to the facts and information that we have
already learned in the university.
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OBJECTIVES
General:
Within our 2 days of duty at the Pediatrics Ward of the Southern Philippines Medical
Center, the goal of the group is to be able to provide holistic nursing care to our client and to be
able to apply the knowledge we have gained in the university to the actual clinical situation.
Specific:
Cognitive
Formulate objectives to be followed on the course of making this case study;
Gather pertinent data of the past and present health history of the patient through
interview and assessment;
Collect necessary information about our clients personal data regarding patients profile,
family background, social status and nutritional status;
Draw the family genogram of the patient to trace disease inheritance;
Ascertain the patients developmental status using the theories of Robert Havighurst, Erik
Erikson and Jean Piaget;
Provide a precise cephalocaudal assessment obtained from the client;
Define the complete diagnosis of the patient by directly citing it from three different
sources;
Discuss the anatomy and physiology of the affected body systems related to the case of
our patient;
Identify and rationalize the signs and symptoms associated with the disease
Trace and explain the pathophysiology of the disease
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Present the doctors orders and make rationales for each order;
Obtain, analyze and interpret laboratory and diagnostic procedures done on the patient
and include the normal and abnormal values and findings for comparison, and the
specific nursing responsibilities associated with each diagnostic procedure;
Make a drug study on the medications given to our patient that includes the generic and
brand names, classification, indication, contraindication, action, adverse effects, side
effects, and nursing responsibilities.
Relate the clients condition to three nursing theories
Correlate the different nursing theories with the nursing care plans that are presented in
this case study;
Make a specific, measurable, attainable, realistic, and time-bounded nursing care plans
for the patient;
Validate patients prognosis according to onset of illness, duration of illness, precipitating
factors, willingness to take medications and treatment, age, environmental factors and
family support;
Make a discharge plan for the patient with the use of M.E.T.H.O.D.S.;
Evaluate the enhancement of the clients condition from the interventions rendered.
Psychomotor
Find a patient who will be the subject of our case presentation;
Perform a thorough physical assessment which will serve as our baseline data;
Identify clients health problems and provide care based on the various nursing care plans
formulated by the researchers;
Give the family health teachings to provide wellness for the betterment of the patient;
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Share information about Patent Ductus Arteriosus and the related factors that may be of
concern to the patient.
Affective
Establish a trusting relationship with the patients family to gain necessary information
regarding the patients status.
Approach the patient and the significant others in a non-judgmental demeanor.
Respect the patients familys right for confidentiality and their religious, cultural, and
personal beliefs.
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HEALTH ASSESSMENT
PATIENTS DATA
BIOGRAPHICAL:
Patients Name: Errshen
Address: Purok 5, Buda, Marilog District, Davao City
Date of birth: January 15, 2012
Parents Religion: Christianity (Roman Catholic)
Parents Occupation:
Mother: Housewife
Father: Security Guard
Occupation: None
Health Insurance: PhilHealth (under fathers name)
Source and Reliability of Information: Mother
Profile:
Age: 14 days old
Gender: Female
Parents Race/Ethnic Background: Bagobo
Chief Complaint: Referral from a local hospital (German Doctors Hospital);
episode of respiratory arrest, cyanosis
Date of Admission
January 29, 2012
Healthcare institution
Southern Philippines Medical Center
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Admitting Diagnosis: Neonatal Sepsis R/O CHD probably PDA
Admitting Physician: Dr. Mardigrace B. Puracan
Admitting Clerk: Daisy Mae Maniquit
Vital Signs upon admission:
Pulse Rate: 133 bpm
Respiratory Rate: 54 cpm
Temperature: 36.7C
B.PRESENT HEALTH HISTORY
Last January 20, 2012, according to the mother, the patient experienced convulsions
around 10:30 in the evening and was rushed to German Doctors Hospital. The patient was
admitted at that hospital for continuous care. By January 29, 2012 at around 9:00 AM, the
mother noticed that her child was pale and had difficulty in breathing. Later on, the child had
cyanosis and they immediately rushed to the doctor. The patient was diagnosed to have
congenital heart defect, which is probably patent ductus arteriosus. Since the institution cannot
provide the childs needs for care, the patient was referred to SPMC for continuous recovery. At
around 1:00 PM, they have arrived at the institution.
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C. PAST HEALTH HISTORY
According to the mother, the child didnt have any communicable disease or any illness except
for the childs convulsions and heart defect. Her first hospitalization was at German Doctors
Hospital due to convulsions. The child has not encountered any injuries or accidents. For the
childs immunizations, only Hepatitis B vaccine was given to her when s he was delivered. She
didnt undergo any surgery or blood transfusions yet.
Medication Reconciliation
Medication Indication as known
by the parents
Dosage Prescribing Health
Care Provider
Cefotaxime
Amikacin
Ceftriaxone
Both the parents
doesnt know about
the medications are
for.
300g IVTT
45g IVTT OD
15g IVTTq 12
Dr. Mardigrace B.
Puracan
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D. Family Health History
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E. Social History
The mother verbalized that upon their stay inside the hospital, mother-child bonding was
implemented. The mother usually breastfeeds the child, changes the diapers and clothes, and
provides comfort to the patient. The mother verbalized ginapunasan nako ni siya adlaw-adlaw
gamit ang labakara. The mother usually watches over the patient. She never left the hospital
since their child was admitted. The father works as a security guard in a hardware located at
Uyanguren and earns 4,500 pesos in a month. He works daily except for Sundays from 8 in the
morning to 5 in the afternoon. He visits their child every after work. He provides the needs of
the mother like clothes and food. To support the childs needs aside from using his own money,
he asked for a cash advance in order to pay for the hospital expenses and the childs medications,
except for the childs lumbar puncture since they cant afford it. The mother verbalized
ginasuportahan man namo sya sa mga gastuhunon pareho anang magpalaboratory ug kanang
makaya lang namo bayaran. They both ensure the wellbeing of their child. Inside the ward, the
patient shares a bed with another patient. The room was crowded with things which make it look
untidy. Regarding the childs condition, the mother verbalized ang nahibal-an lang namo kay
naa siyay buslot sa dughan, wala gyud mi kabalo kung unsa to siya.
F. Health Maintenance Activities
According to the mother, her child wakes up to breastfeed. The doctor ordered
breastfeeding with strict aspiration precaution and is advised to have nothing per orem if
respiratory rate is greater than 60. After breastfeeding, the child will immediately go to sleep.
The mother tries to let the child burp, but she couldnt do it. The child is also lethargic;
sometimes the child suddenly goes to sleep while shes breastfeeding. Whenever the child is
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experiencing difficulty or fast breathing, the mother would administer oxygen to the child via
face mask, 4 liters per minute as ordered.
G. Nutritional Assessment
Height: 54 cm
Weight: 3.5 Kg
BMI: 12.00
Head circumference: 35 cm
Chest circumference: 36 cm
Abdominal Circumference: 37 cm
Diet History
The mother verbalized that the baby was purely breastfed; she complies with the doctors
order that she should limit the childs intake both orally and intravenously due to the possible
cardiac overload that may happen.
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DEVELOPMENTAL DATA
Developmental task theory of Robert Havighurst
Is a task which arises at or about a certain period in the life of an individual , successful
achievement of which leads to his happiness and to success with later tasks, while failure leads to
unhappiness in the individual, disapproval by society, and difficulty with later tasks
(Havighurst, 1972, p.2).
Havighurst also identified Six Major Stages in human life covering birth to old age which are the
following:
Infancy & early childhood (Birth till 6 years old)
Middle childhood (6-12 years old)
Adolescence (13-18 years old)
Early Adulthood (19-30 years old)
Middle Adulthood (30-60years old)
Later Maturity (60 years old and over)
Our patient is in Infancy and early childhood because at this age he learns to walk, crawl, eat
solid food, talk, eliminate body waste, and differentiate sexuality. Forming concepts and
learning language to describe social and physical reality.
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DEVELOPMENTAL
TASK
ACHIEVED OR NOT
ACHIEVED
JUSTIFICATION
Learning to Walk Not achieved The patient is only 14 days old and hasnt
learned to walk. The child is only being
carried.
Learning to take solid
food
Not achieved The patient is purely breastfed by the
mother. Also, she is not yet on the weaning
phase (not less than 4 months, not more
than 6 months.)
Learning to Talk Not achieved The child cannot talk. Because the infants
brain is not yet at the age of learning or
expressing. The child only cries whenever
she needs something.
Learning to control the
elimination of bodywastes
Not achieved The child was unable to defecate and
urinate. She depends on diaper usage for herto eliminate body wastes.
Achieving psychological
stability
Not achieved The child is only 14 days old. At that age,
infants still depend on crying to express of
what they feel and need.
Forming simple concepts
of social and physical
reality
Not achieved The child at the age of 14 days old does not
have the awareness of the environment
around her; may it be her family or the
physical environment around her.
Learning to relate
emotionally to parents,
siblings, and other people
Not achieved At her age of 14 days old, ways of
expression is dependent on crying.
Learning to distinguish
right from wrong and
developing conscience
Not achieved 14 day old infants cannot distinguish
between right and wrong because at that
age, infants do what they want and they
something were to hinder, they would
express by crying.
Learning Sex differences
and sexual modesty
Not achieved The child is only 14 days old. Awareness of
sex and sexual differences is beyond her
understanding.
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Psychosocial theory of Erik Erickson
Eriksons theory proposes that life is a sequence of developmental stages or levels of
achievement. Each stage signals a task that must be accomplished. The resolution of the task can
be complete, partial, or unsuccessful. Erikson believed that the more success an individual has at
each developmental stage, the healthier the personality of the individual.
Stages of Eriksons Psychosocial Theory are as follows:
Infancy Birth-18 months Trust vs. mistrust
Early childhood 1-3 years Autonomy vs. Shame
Late childhood 3-6 years Initiative vs. Guilt
School age 6-12 years Industry vs. Inferiority
Adolescence 12-18 years Identity vs. Role Confusion
Young adulthood 18-30 years Intimacy vs. Isolation
Adulthood 30-65 years Generativity vs. Stagnation
Maturity 65 years to death Integrity vs. Despair
Being an infant at the age of 3 days old, he is considered as a part of the Trust versus
mistrust stage of Erikson.
Stage Description Result Justification
Infancy
(Birth-18
months)
TRUST
VERSUS
MISTRUS
T
Erikson also referred to infancy as
the Oral Sensory Stage (as anyone
might who watches a baby put
everything in her mouth) where the
major emphasis is on the mother's
positive and loving care for the
child, with a big emphasis on visual
contact and touch. If we pass
successfully through this period of
life, we will learn to trust that life is
basically okay and have basicconfidence in the future. If we fail to
experience trust and are constantly
frustrated because our needs are not
met, we may end up with a deep-
seated feeling of worthlessness and
a mistrust of the world in general.
Achieved The infant is only 14 days old
and is fully dependent to her
parents. Whenever the child is
hungry, her mother would
breastfeed her. Her mother also
does the changing of her diapers
from time to time whenever it is
wet and full. The mother also
provides comfort to her child
whenever she is crying and
whenever she goes to sleep. Themother always gives what her
child needs from her.
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Cognitive Theory of Piaget
Cognitive Theory refers to the manner in which people learn to think, reason, and use
language. It involves a persons intelligence, perceptual ability, and ability to process
information. Cognitive development represents a progression of mental abilities form illogical to
logical thinking, from simple to complex problem solving, and from understanding concrete
ideas to understanding abstract concepts
Piagets Phases of Cognitive Development
Phases and Stages Age Significant Behavior
Sensorimotor Phase Birth to 2 years The first is centered on the
infant trying to make sense of
the world. During the
sensorimotor stage, an infants
knowledge of the world is
limited to their sensory
perceptions and motor
activities. Behaviors are
limited to simple motor
responses caused by sensory
stimuli. Children utilize skills
and abilities they were bornwith, such as looking, sucking,
grasping, and listening, to
learn more about the
environment.
Stage 1 Use of reflexes Birth to 1 month Most action is reflexive
Stage 2 Primary Circular
reaction
1 to 4 months Perception of events is
centered on the body
Stage 3 Secondary Circular
reaction
4 to 8 months Acknowledges the external
environment
Stage 4 Coordination ofsecondary schemata
8 to 12 months Can distinguish a goal frommeans of attaining it
Stage 5 Tertiary Circular
reaction
12 to 18 months Tries to discover new goals
and ways to attain goals.
Stage 6 Inventions of new
means
18 to 24 months Interprets the environment by
mental image. Uses make-
believe and pretend to play
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Preconceptual Phase 2 to 4 years Uses an egocentric approach
to accommodate the demands
of an environment
Intuitive Though Phase 4 to 7 years Egocentric thinking
diminishes. Thinks of one idea
at a time. Includes others in
the environment.
Words express thoughts
Concrete Operations Phase 7 to 11 years Solves concrete problems.
Begins to understand
relationships such as size.
Understands right and left.
Cognizant of viewpoints
Formal operations phase 11 to 15 years Uses rational thinking.
Reasoning is deductive andfuturistic
Phase and Stage Description Justification
Stage 1 Use of
Reflexes
Achieved. The child was able to achieve:
rooting reflex with weak response. The child turned
her head toward the side of the face stroked;
sucking reflex because the child was able to suck
her thumb and was able to breastfeed frequently;
palmar grasp reflex with strong, immediate
response; flexion of the hands and feet were
present;
tonic neck reflex with weak response; the childs
head turned to one side and the arm and leg on the
same side extended;
babinski reflex with weak response; the big toe
rose and the remaining toes fanned out upon
stroking the sole of the foot from heel to toe;
moro reflex with quick response, where suddenextension and abduction of extremities were noted.
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ETIOLOGY
As a baby develops in the womb, a vascular connection (ductus arteriosus) between two
major blood vessels leading from the heartthe aorta and pulmonary arteryis a normal and
necessary part of your baby's blood circulation while in the womb. The ductus arteriosus diverts
blood from the lungs of the fetus while they aren't being used. The fetus receives oxygen from
the mother's circulation. But, the ductus arteriosus is supposed to close within two or three days
after birth once the newborn's heart adapts to life outside the womb. In premature infants, the
connection often takes longer to close on its own. If the connection remains open, it's referred to
as a patent ductus arteriosus.
The abnormal opening causes too much blood to circulate to the lungs and heart. If not
treated, the blood pressure in the lungs may increase (pulmonary hypertension) and the heart
may enlarge and weaken.
Congenital heart defects arise from problems early in the heart's development but there's
often no clear cause. Genetics and environmental factors may play a role.
Predisposing
Factors
Present/
AbsentRationale Justification
Being born too soon
(prematurity)
Absent Patent ductus arteriosus
(PDA) occurs more
commonly in babies
who are born too early
than in babies who bornfull term because the
infants heart was not
fully developed before
delivery.
The mothers LMP was on
April 16, 2011 and the baby was
born on Jan 15, 2012. Therefore
the baby was exactly born
9months and 1 day. Proving thatshe is not in a preterm state.
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Having other heart
defects
Absent Babies who have other
heart problems when
they're born (congenital
heart defects) are also
more likely to have a
patent ductus arteriosus.
Because other defects
such
Other than Patent Ductus
Arteriosus, no other heart defect
has been noted/diagnosed.
Family history and
other genetic
conditions.
Present If you have a family
history of heart defects,
it's more likely your
child may have a patent
ductus arteriosus. Other
genetic conditions, suchas heart problems or
chromosomal
abnormalities, also have
been linked to an
increased chance of
having a PDA.
Tracing back to the family
history, the grandmother of the
baby has myocardial infarction
in which may be a factor for her
to have PDA.
Having a female baby Present PDAs are much more
common in girls than in
boys.
The baby was born as a female
as evidenced by the presence of
only female genitalia.
Precipitating
Factors
Present/
AbsentRationale Justification
Use of Teratogens
during pregnancy
Absent Teratogens are any
chemical, substance, or
exposure that could possibly
cause birth defects in a
developing fetus. Exposure
to teratogens seems to result
in malformations especially
when it occurs during the
fourth and tenth week of
pregnancy.
Only FeSO4 was taken by the
mother during her pregnancy
with patient Errshen.
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Smoking Present A shortage of oxygen can
have devastating effects on
your baby's growth and
development. On average,
smoking during pregnancy
doubles the chances that a
baby will be born too early
or weigh less than 5 1/2
pounds at birth. Smoking
also more than doubles the
risk of heart diseases on
infants
The mother told us that she
smoked during the first
trimester of her pregnancy;
however, she stopped smoking
when she knew that she was
pregnant.
Chromosomal
abnormalities
Absent Trisomy 21, the
chromosomal abnormality
associated with Downsyndrome, is associated
with cardiac abnormalities
in 50 percent of babies.
Chromosomal abnormalities
The infant may have PDA, but
no signs of Chromosomal
abnormalities, like simianscrease, lethargic eyes, short
neck, Brushfield spots, and
others was seen.
Maternal
infections (esp.
Rubella/German
measles)
Absent If you have rubella in the
first trimester, you have
about a 25% risk of having
a birth defect, known as
congenital rubella
syndrome. The defects can
include:
heart deformities
hearing loss
mental retardation
eye deformities
others
The mother told us that she had
no diagnosis of infections
before and during her
pregnancy.
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PHYSICAL ASSESSMENT
General Survey
The patient is 14 days old. She looked appropriate to her age. The patient has no
obvious physical deformities; her body is symmetrical and normal for her age. No body
odor or breath odor was noted. Skin integrity is interrupted due to IVTT insertion of
D5IMB 500 cc infusing @ 13cc/hr at the left metacarpal vein.
The patient was awake and was carried by the mother upon assessment. The
patient was in respiratory distress manifested by fast breathing with a respiratory rate of
100 cycles per minute. Oxygen was administered to her via face mask at 4 liters per
minute.
Vital signs
Vital Signs Upon
Admission:
4PM 8PM
Blood Pressure ---- ---- ----
Cardiac Rate 133 bpm 171 bpm 120 bpm
Respiratory Rate 54 cpm 100 cpm 58 cpm
Temperature 36.7C 38.3C 37.2C
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Skin, Hair, Nails Assessment
Skin
The skin is smooth, thin, and pale; no edema and tenderness noted. The patient has a brown
complexion in all areas. No bleeding or lesions was noted. The skin is warm to touch and has
good skin turgor. The child has a birthmark on her left leg at about 5 cm in length and 3 cm in
width. Milia was not present.
Hair
The hair is black in color, thin, and is evenly distributed. Her scalp is light brown, with no signs
of lesions. There is presence of lanugo at the back of the patient.
Nails
The nails were round, hard. Nails were intact and pale in color with a capillary refill of
3-4 seconds. The nails are clean and smooth with clubbing noted. No lesions were observed.
Head, Neck, and Regional Lymphatic Assessment
Head
The head of the patient is normocephalic and symmetrical. The head circumference of the
patient is 35 cm. The mother verbalized pag mag-utong ni siya, mamula jud ni iyang agtang
which was observed upon assessment. Upon palpation, the skull is smooth, soft, and fused except
for the fontanels. Her facial features and expressions are symmetrical and the shape of the head
is round. There are no obvious deformities. She could open and close her mouth without any
discomfort.
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Neck
Her neck is short and warm to touch. Her neck movement is coordinated; the patient can
move her head from side to side without signs of discomfort.
Regional Lymphatics
There were no visible lymph nodes upon inspection. No palpable lymph nodes were
present. There were also no abnormalities in her thyroid gland both anteriorly and posteriorly.
Eye Assessment
Upon assessment, eyebrows are thin, smooth, and symmetrically aligned. The eyelashes
are evenly distributed and curled slightly outward. No noted discharges were present in the
lacrimal apparatus. The color of her iris is dark brown. The iris and pupils were round and equal
on both sides. Pupillary reflex is not yet developed. The sclera is clear. Whenever the child cries,
tears are not present since the childs tearing ability is not yet fully developed.
Ears, Nose, Mouth, and Throat assessment
Ears
Both of her ears are symmetrical in size and are located. There is no presence of foreign
bodies, redness, deformities, or lesions. Inflammation and nodules were not visible. The external
auditory canal curves upward and is short and straight. Minimal amount of cerumen in the ear
canal was observed. The ears were physically symmetrical in size; pinna is in line with the outer
canthus of his eyes. Upon palpation, ears are smooth and no tenderness is noted. No bleeding or
masses were noted. No pain or tenderness was noted.
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Nose
Nose was located symmetrically in the midline of the face with no evidence of swelling,
bleeding, lesions, and masses. Nasal flaring was noted. No tenderness noted upon palpation.
Both left and right nares were patent, with no discharges; air could freely move in and out when
the patient breathes. The nasal septum was at the midline without perforation, lesions, or
bleeding.
Mouth
Her lips are pale and smooth. Upon inspection of the mouth, oral mucosa was pinkish and
moist. The tongue is located at the midline without lesions present. The gums were pinkish and
bleeding is absent. Her tonsils were free from inflammation. The patients teeth are not yet
developed; teeth were absent upon inspection.
Breast and Regional Nodes
The patients breasts were flat and symmetrical. Areolar areas and nipples were pale in
color. No thickening or edema was found. No fixed, firm, immobile, irregular lymph nodes were
present.
Thorax and lungs
Her respiration is irregular. Rapid, shallow breathing was noted. The patients respiratory
rate is 100 breaths in one full minute. Upon auscultation, breath sounds were low in pitch with
snoring quality, indicating rhonchi. No tenderness and masses noted upon palpation. No sputum
was present.
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Heart and Peripheral Vasculature
The patients cardiac rate was 171 beats per minute. Presence of abnormal heart sounds
was noted. Upon auscultation, machine-like murmurs were heard, which indicates heart disease.
The apical pulse is easily palpable. Her skin was warm upon palpation and capillary refill time is
3-4 seconds.
Upper Extremities
The patients upper extremities are symmetrical. Body fats are evenly distributed. Upper
extremities were pale and warm to touch. 5 fingers were present on both hands. The palm is
lighter in color than the hands.
Abdomen
Upon inspection, the abdomen is cylindrical, round, and soft. The patients
abdomen has same color with his ches. The umbilicus is medially located and shows no signs of
inflammation or abnormal discharges. Bowel sounds were heard upon auscultation. The
abdomen rises with inspirations and falls with expiration.
Genitalia
The genitals are warm, dry, smooth, and soft, with good skin turgor. Labia majora covers
labia minora. There were no discharges present.
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Lower Extremities
The patients lower extremities are symmetrical. No lesions were noted. Legs have equal
length. 5 toes were present on both legs. Cyanosis was noted; lower extremity is cold to touch
and was pale. The soles are lighter in color than the legs.
Anus
Anal patency is present; the child was able to defecate once during the shift.
Musculoskeletal System
The extremities resist when extended and return to flexed state when released. Sutures of
the head are palpable. The neck moves freely and holds the head in midline position. Clavicles
are symmetrical and intact. The spine is flexible and rounded. Palm was able to stay in both
prone and supine in a good manner without difficulty. She was able to exhibit strong hand grip
on both arms. No tenderness was visible upon moving the patient. Reflex on the upper extremity
was good. No hand tremors noted.
Neurological System
Rooting Reflex: Achieved; weak response. The child turns head toward side of face stroked.
Sucking Reflex: Achieved; the child was able to suck her thumb and was able to breastfeed
frequently.
Palmar Grasp Reflex: Achieved; flexion of hands and feet were present; the grasp was strong;
the child was able to grasp immediately.
Tonic Neck Reflex: Achieved; weak response; the childs head turned to one sid e, the arm and
leg on the same side extended.
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Stepping Reflex: Not achieved; the child wasnt able to execute this reflex since the child was
asleep and carried by the mother.
Babinski Reflex: Achieved; with weak response; upon stroking the outer sole of foot from heel
to toe, the big toe rise and the remaining toes fan out.
Moro Reflex: Achieved with quick response; sudden extension and abduction of extremities
noted.
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PATIENTS DIAGNOSIS
Definition of the complete diagnosis
Neonatal Sepsis:
A life threatening illness defined as a suspected or known plus the systemic inflammatory
response syndrome
F. J. Domino; The 5-minute Clinical Consult 2011
Neonatal sepsis is defined as a clinical syndrome of bacteremia with systemic signs and
symptoms of infection and a positive culture from central body fluid.
D.K. Guha; Guhas NEONATOLOGY Principles and Practice
Sepsis means putrefaction, i.e., the decomposition of organic matter (by bacteria or fungi)
resulting from an interaction between germs and host
J.L. Vincent; Intensive Care Medicine
Patent Ductus Arteriosus:
Patent Ductus Arteriosus (PDA) is the persistence in postural life of the normal fetal vascular
conduit between the central pulmonary and systemic arterial systems.
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M. William Schwart; Five-minute pediatric consult
Patent Ductus Arteriosus (PDA) is the failure of the ductus arteriosus to close after birth.
F. J. Domino; The 5-minute Clinical Consult 2011
A patent ductus arteriosus is a permanent defect in the muscle wall of the duct and is unlikely to
close spontaneously
A.A. Fanaroff, T. Lissauer; Neonatology at a Glance
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ANATOMY AND PHYSIOLOGY
Patent Ductus Arteriosus is a heart problem that occurs soon after birth in some babies.
In PDA, abnormal blood flow occurs between two of the major arteries connected to the heart.
Superior vena cava
Brings de-oxygenated blood from the head, neck, arm and chest regions of the body to the right
atrium.
Inferior vena cava
Brings de-oxygenated blood from the lower body regions (legs, back, abdomen and pelvis) to
the right atrium.
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Right Atrium: The right upper chamber of the heart. The right atrium receives
deoxygenated blood from the body through the vena cava and pumps it into the
right ventricle which then sends it to the lungs to be oxygenated.
Tricuspid valve:
Valves are flap-like structures that allow blood to flow in one direction. The tricuspid
valve is located between the right atrium and the right ventricle. Prevents the back flow
of blood as it is pumped from the right atrium to the right ventricle
Right ventricle
the relatively thin-walled chamber of the heart that pumps blood received from the right atrium
into the pulmonary arteries to the lungs for oxygenation. The right ventricle is shorter and
rounder than the long conical left ventricle. The chordae tendineae of the tricuspid valve of the
right ventricle are finer than the coarse strands of the chordae tendineae of the left ventricle.
Pulmonary semilunar valve:
a semilunar valve between the right ventricle and the pulmonary artery; prevents blood from
flowing from the artery back into the heart
Pulmonary artery
The pulmonary artery is the vessel transporting de-oxygenated blood from the right ventricle to
the lungs. A common misconception is that all arteries carry oxygen-rich blood. It is more
appropriate to classify arteries as vessels carrying blood away from the heart.
http://www.medterms.com/script/main/art.asp?articlekey=5974http://www.medterms.com/script/main/art.asp?articlekey=5984http://www.medterms.com/script/main/art.asp?articlekey=5984http://www.medterms.com/script/main/art.asp?articlekey=5974 -
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Pulmonary vein
The pulmonary vein is the vessel transporting oxygen-rich blood from the lungs to the left
atrium. A common misconception is that all veins carry de-oxygenated blood. It is more
appropriate to classify veins as vessels carrying blood to the heart.
Left Atrium
The left atrium receives oxygenated blood from the lungs through the pulmonary vein. As the
contraction triggered by the sinoatrial node progresses through the atria, the blood passes through
the mitral valve into the left ventricle.
Mitral Valve
The mitral valve separates the left atrium from the left ventricle. It opens to allow the oxygenated
blood collected in the left atrium to flow into the left ventricle. It closes as the left ventricle
contracts, preventing blood from returning to the left atrium; thereby, forcing it to exit through
the aortic valve into the aorta.
Left Ventricle
The left ventricle receives oxygenated blood as the left atrium contracts. The blood passes
through the mitral valve into the left ventricle. The aortic valve leading into the aorta is closed,
allowing the ventricle to fill with blood. Once the ventricles are full, they contract. As the left
ventricle contracts, the mitral valve closes and the aortic valve opens. The closure of the mitral
valve prevents blood from backing into the left atrium and the opening of the aortic valve allows
the blood to flow into the aorta and flow throughout the body.
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Aortic Valve:
The aortic valve separates the left ventricle from the aorta. As the ventricles contract, it opens to
allow the oxygenated blood collected in the left ventricle to flow throughout the body. It closes
as the ventricles relax, preventing blood from returning to the heart.
Aorta
The aorta is the largest single blood vessel in the body. It is approximately the diameter of your
thumb. This vessel carries oxygen-rich blood from the left ventricle to the various parts of the
body.
Chondrae Tendinae
The chordae tendineae are tendons linking the papillary muscles to the tricuspid valve in the
right ventricle and the mitral valve in the left ventricle. As the papillary muscles contract and
relax, the chordae tendineae transmit the resulting increase and decrease in tension to the
respective valves, causing them to open and close. The chordae tendineae are string-like in
appearance and are sometimes referred to as "heart strings."
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Ductus arteriosus:
The ductus arteriosus is part of the normal fetal circulatory system. This vessel connects the aorta
and the pulmonary artery. Prior to birth the ductus arteriosus allows for antegrade flow from the
right ventricle to the aorta. Following birth the ductus arteriosus normally closes
Ductus venosus:
in the fetus, the ductus venosus shunts approximately half of the blood flow of the umbilical
vein directly to the inferior vena cava. Thus, it allows oxygenated blood from the placenta to
bypass the liver. In conjunction with the other fetal shunts, the foramen ovale and ductus
arteriosus, it plays a critical role in preferentially shunting oxygenated blood to the fetal brain. It
is a part of fetal circulation.
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Foramen ovale:
Blood from the mother enters the placenta and comes in close proximity to the fetal blood that
has returned from the fetus to the placenta through the umbilical arteries . once the two
circulations are in close proximity in the placenta , the oxygen and nutrients, like sugar , protein
and fat molecules can move from maternal to fetal blood, carbon dioxide and waste products can
move from fetal to maternal blood. The maternal blood returns from the placenta to the mothers
veins for her systems to take care of the waste. The new nourished fetal blood returns to the baby
through the umbilical vein.
Fetal circulation:
- Oxygenated blood from the placenta enters the fetus through the umbilical vein
- Most of the newly oxygenated blood bypasses the liver via the DUCTUS VENOSUS and
combines with deoxygenated blood in the inferior vena cava
- Blood then joins deoxygenated blood from the superior vena cava and empties into the
right atrium
- Since pressure in the right atrium is larger than pressure left atrium, most blood will be
shunted through the foramen ovale
- Some blood does travel from the right atrium to the right ventricle through the pulmonary
trunk but most blood bypasses the pulmonary arteries and moves directly to the aorta via
the ductus arteriosus
-Deoxygenated blood returns to the placenta via the umbilical arteries originating from the
internal iliacs near the bladder
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Oxygenated blood from the placenta is transported through the umbilical vein into the
body of the fetus. Blood bypasses the liver by traveling through the ductus venosus. The ductus
venosus provides a direct communication between the umbilical vein and inferior vena cava.
Oxygenated blood from the ductus venosus combines with deoxygenated blood in the inferior
vena cava and continues to the heart.
Blood travels to the heart through the inferior vena cava and mixes with deoxygenated
blood returning from the superior vena cava. Blood enters the right atrium of the heart. Because
the fetal lungs are not functional, most blood will bypass the right ventricle and be shunted to the
left atrium via the foramen ovale. Blood will then travel into the left ventricle and be distributed
throughout the body via the aorta. Some blood will enter the right ventricle from the right atrium
and proceed into the pulmonary trunk. However, most of this blood will be shunted away from
the pulmonary arteries and into the aorta via the ductus arteriosus.
Blood then circulates through the body and returns to the placenta via the umbilical
arteries. These arteries are carrying deoxygenated blood back to the placenta .
The placenta reoxygenates blood returning from the umbilical arteries and repeats the
fetal cardiovascular cycle by recycling newly oxygenated blood to the fetus through the
umbilical vein.
Postnatal Circulatory Changes:
- With the first breath, increased alveolar oxygen pressure causes vasodilation in the
pulmonary vessels
- Obstetrical clamping induces spontaneous constriction and changes of the umbilical vein
to ligamentum teres and umbilical arteries to medial umbilical ligaments
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- Within 10 -15 hours after birth, the ductus arteriosus constricts and will become the
ligamentum arteriosum
- Increased left atrial pressure and decreased right atrial pressure causes the foramen
ovale to close and become fossa ovalis.
- The ductus venosus also constricts and will become the ligamentum venosum.
Obstetrical clamping leads to spontaneous constriction of the umbilical vessels and
eventually leads to the conversion of the ductus venosus to the ligamentum venosum. Changes
due to increased alveolar pressure in the lungs lead to anatomical and physiological alterations
in the circulatory system.
Usually within 10 -15 hours after birth, the ductus arteriosus will constrict and change to
the ligamentum arteriosum.
The umbilical vein and umbilical arteries become the ligamentum teres and medial
umbilical ligaments, respectively.
Normal Heart and Heart With Patent Ductus Arteriosus
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ETIOLOGY:
As a baby develops in the womb, a vascular connection (ductus arteriosus) between two
major blood vessels leading from the heartthe aorta and pulmonary arteryis a normal and
necessary part of your baby's blood circulation while in the womb. The ductus arteriosus diverts
blood from the lungs of the fetus while they aren't being used. The fetus receives oxygen from
the mother's circulation. But, the ductus arteriosus is supposed to close within two or three days
after birth once the newborn's heart adapts to life outside the womb. In premature infants, the
connection often takes longer to close on its own. If the connection remains open, it's referred to
as a patent ductus arteriosus.
The abnormal opening causes too much blood to circulate to the lungs and heart. If not
treated, the blood pressure in the lungs may increase (pulmonary hypertension) and the heart
may enlarge and weaken.
Congenital heart defects arise from problems early in the heart's development but there's
often no clear cause. Genetics and environmental factors may play a role.
Etiology for PDA:
Predisposing
FactorsPresent/ Absent Rationale Justification
Being born too soon
(prematurity)
Absent Patent ductus arteriosus
(PDA) occurs more
commonly in babies who
are born too early than in
babies who born full term
because the infants heart
was not fully developed
before delivery.
The mothers LMP
was on April 16,
2011 and the baby
was born on Jan 15,
2012. Therefore the
baby was exactly
born 9months and 1
day. Proving that she
is not in a preterm
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state.
Having other heart
defects
Absent Babies who have other
heart problems when
they're born (congenital
heart defects) are also more
likely to have a patent
ductus arteriosus. Because
other defects such
Other than Patent
Ductus Arteriosus, no
other heart defect has
been
noted/diagnosed.
Family history and
other genetic
conditions.
Present If you have a family
history of heart defects, it's
more likely your child may
have a patent ductusarteriosus. Other genetic
conditions, such as heart
problems or chromosomal
abnormalities, also have
been linked to an increased
chance of having a PDA.
Tracing back to the
family history, the
grandmother of the
baby has myocardialinfarction in which
may be a factor for
her to have PDA.
Having a female baby Present PDAs are much more
common in girls than in
boys.
The baby was born as
a female as
evidenced by the
presence of onlyfemale genitalia.
Precipitating FactorsPresent/
AbsentRationale Justification
Use of Teratogens
during pregnancy
Absent Teratogens are any
chemical, substance, or
exposure that could
possibly cause birth defects
in a developing fetus.
Exposure to teratogens
seems to result in
malformations especially
Only FeSO4 was
taken by the mother
during her pregnancy
with patient Errshen.
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when it occurs during the
fourth and tenth week of
pregnancy.
Smoking Present A shortage of oxygen can
have devastating effects on
your baby's growth and
development. On average,
smoking during pregnancy
doubles the chances that a
baby will be born too early
or weigh less than 5 1/2
pounds at birth. Smoking
also more than doubles the
risk of heart diseases on
infants
The mother told us
that she smoked
during the first
trimester of her
pregnancy; however,
she stopped smoking
when she knew that
she was pregnant.
Chromosomal
abnormalities
Absent Trisomy 21, the
chromosomal abnormality
associated with Down
syndrome, is associated
with cardiac abnormalities
in 50 percent of babies.
Chromosomal
abnormalities
The infant may have
PDA, but no signs of
Chromosomal
abnormalities, like
simians crease,
lethargic eyes, short
neck, Brushfield
spots, and others was
seen.
Maternal infections
(esp. Rubella/German
measles)
Absent If you have rubella in the
first trimester, you have
about a 25% risk of having
a birth defect, known as
congenital rubella
syndrome. The defects can
include:
heart deformities
hearing loss
mental retardation
eye deformities
others
The mother told us
that she had no
diagnosis of
infections before and
during her
pregnancy.
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Etiology for Neonatal Sepsis:
Etiology Sepsis
Predisposing
Factors
Present or absent Rationale Justification
Preterm baby Absent Experimental as
well as
epidemiological
studies have shown
that low birthweight
is also a
predisposing factor
for abnormalities
such as
atherosclerosis,
cardiovascular
diseases, sepsis,
renal disease, non-
insulin diabetes,
hypertension,
obesity or the
metabolic
syndrome. Indeed,
the human fetus
The mothers LMP
was on April 16,
2011 and the baby
was delivered on
Jan. 17, 2012.
Hence the baby was
born 9 months and
a day. Proving that
she was not a
preterm baby.
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adapts to
undernutrition by
redistribution and
infection of blood
flow
infants under 3
months, whose
immune systems
haven't developed
enough to fight off
overwhelming
infections
Present Infants below 3
months have
immune systems
that are still in the
process of
maturation. Thus
being prone to
certain blood
infections
At the age of 14
days old, the
infants immune
system is still
maturing thus
making her prone to
certain infections.
Bloodstream
infection of the
mother(bacteremia)
Absent Mothers with blood
stream infection
also causes the
baby to have
infection for they
share the same
blood during fetal
development
There is no
diagnosis of the
mother about blood
stream infection.
Precipitating Present or absent Rationale Justification
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Factors
Infection Absent Newborn baby can
get infected in many
ways. Infection in
mother can be
transmitted to the
baby. After birth the
umbilical cord can
be source of
infection. Infection
of the skin can also
invade and cause
sepsis. Baby can
catch infection from
other people.
Because the
immune system of
the newborn is not
mature any infection
can potentially
cause sepsis.
Mother was not
diagnosed of
infection during
pregnancy or after
delivery.
Alcoholism Present A woman who
drinks alcohol while
Tracing to the
mothers social
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she is pregnant may
harm her
developing baby
(fetus). Alcohol can
pass from the
mothers blood into
the babys blood. It
can damage and
affect the growth of
the babys cells.
history, she
verbalized that she
has taken alcohol
during the first
trimester. After
knowing of being
pregnant, she has
stopped the alcholol
intake.
immunosuppressive
medications taken
by the mother
Absent Taking of
immunosuppressive
drugs weakens the
immune system of
the mother which
makes her prone to
infections. So if the
mother has
infections, the baby
will also share the
same fate.
Mother has not
taken any
medications aside
from FeSO4.
However she
suspects that it was
the reason for her
hematemesis during
her first 7 months of
pregnancy.
Frequent vaginal
checkups during
Absent Frequent vaginal
check-ups can
Due to financial
pressures, the
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pregnancy. cause infection to
the womens genital
area because
clinical instruments
are foreign to the
body
family only goes to
the check-up in
times of severe
symptoms such
vaginal bleeding, or
abdominal pain
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SYMPTOMATOLOGY
Symptoms Rationale Present/Absent Justification
Rapid breathing The normal respiratory rate
for infants is 40-60 breaths
per minute. Lower than
normal indicates
bradypnea; higher than
normal indicates tachypnea.
Present The respiratory rate of
the patient is 100 cycles
per minute.
Poor feeding
habits
Poor feeding occurs when
infants lack interest in
nursing or cannot obtain
the nutrition necessary for
appropriate weight gain and
other development. Infants
lacking appropriate
nutrition due to poor
feeding can suffer
significant and permanent
delays in mental and
physical development.
Absent The patient was able to
breastfeed every four
hours.
Rapid Pulse The normal pulse rate forinfants is 120-160 beats per
minute. Lower than normal
indicates bradycardia;
higher than normal
indicates tachycardia.
Present The cardiac rate of thepatient is 171 breaths per
minute, which indicates
tachycardia.
Sweating while
feeding
baby likely sweats because
he's warm while
breastfeeding. Being skin
to skin with your baby
raises body temperature,which initiates his natural
cooling system.
Present Sweat was visible upon
breastfeeding.
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PATHOPHYSIOLOGY
Superior vena cava and inferior vena cava with unoxygenated blood
Right atrium (deoxygenated blood flow in right heart)
Right ventricle
Pulmonary arteries
Lungs ( oxygenated blood)
Pulmonary veins
Left atrium
Left ventricle
Aortic valve
Aorta (mixing of blood from aorta (oxygenated) and pulmonary artery (deoxygenated blood))
distributes to the circulatory system
Superior vena cava and Inferior vena cava
- Machine like
murmur
- Fast breathing
- Poor feeding
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Pregnancy
Fetal Development( begins at the 10th week of gestation)
1st
trimester:
- Embryo increases in size to about 1 inches. Bones and muscles begin to round
Out contours of body
- Sex organs begins to form
- Beginning of fetal period
2nd
trimester:
- Hands and feet are well formed.
- Skin appears dark red
- Finger closure is possible
- Reflexes become more active
3rd
trimester:
- Generally the fetus is about 15 inches long and weighs about three pounds
- Fat is formed all over the body
- Fetal heart rate becomes quite rapid
Birth
Maternal age
SmokingIncreased parity
congenital
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Failure of ductus arteriosus to close within 10-15 hours
Blood enters systemic circulation
Through ductus bypassing
The pulmonary system
Systemic pressure greater than
Pulmonary pressure
Left to right shunting from aorta
To pulmonary artery
Right ventricular hypertrophy
If treated: if not treated
Machine like
murmur
Fast breathing
Rapid pulse
Poor feeding
Medical management
-fluid restriction-indomethacin
-cardiac catheterization
-surgical repair
Nursing management
-assessment of vital signs such as
tachycardia, tachypnea-avoid cold stress to infant- position semi-fowlers if necessary
-reduce environmental stimuli
-promote uninterrupted sleep
Bad prognosis
Poor growthPoor feeding
Increased heart size
Prolonged capillary refill time
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Doctors OrdersDATE
ORDERED DOCTORS ORDER RATIONALE REMARKS
February 3,2012
Please admit to SN ward
VSq4
BF with SAP, NPO if RR > 60
Rx:
CBC, UA, LP, 2DECHO,
Crea, Na+, k+, MCHC MCV,
Blood GSCS
To monitor closely the neonatefor any eventualities that may
occur.
Vitals signs should be checked
so that we would have abaseline record for comparison.
Also, to be able to check if
there are any abnormalities in
the persons vital statistics.
Breastfeeding is alwaysrecommended for all neonates,
however since the patient hascardiac problems, he may
develop difficulty of breathing
anytime which may lead toaspiration. If the patient has
tachypnea, the patient is
predisposed to aspiration
especially when feeding.Therefore the patient is advised
to have nothing per orem if theRR >60.
CBC is used as a broadscreening test to check for such
disorders as anemia, infection
and many other diseases. It is
actually a panel of tests thatexamines different parts of the
blood and it includes: WBC
count, RBC count,Hemoglobin, Hematocrit.- The hematocrit is used to
screen for anemia, or is
measured on a person todetermine the extent of anemia.
An anemic person has fewer or
smaller than normal red blood
DONE
DONE
DONE
DONE
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cells. A low hematocrit,
combined with other abnormal
blood tests, confirms the
diagnosis.- Lumbar puncture is done to
determine infection in themeninges. To rule out thepresence of meningitis or any
cerebral infection.
- 2DimensionalEchocardiography is used to
examine the heart. It is capable
of displaying a cross-sectional
"slice" of the beating heart,including the chambers, valves
and the major blood vessels that
exit from the left and rightventricle. For us to be able tosee presence of blockage and
any abnormalities in the
chamber.- Urinalysis is a test to check
for the presence of bacteria,
blood and pus which causeurinary tract infection.
- Serum sodium examination is
done to assess the levels of the
sodium in the blood which isespecially critical in Cardiac
Patients.
- Serum potassium examinationis done to assess the
concentration of potassium in
the blood which is critical in the
functioning of muscular organslike the heart.
- mean corpuscular volume, or
"mean cell volume" (MCV), is
a measure of the average redblood cell size that is reported
as part of a standard complete
blood count to detect anemiaand polycythemia.
- The mean corpuscular
hemoglobin concentration, orMCHC, is a measure of the
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IVF D5IMB 500 cc to run
@ 13cc/hr
Meds:
Ceftriaxone
15g IVTTq 12
Amikacin 45g
IVTT OD
Diazepam 0.6g
IVTT PRN foractive seizure
concentration of hemoglobin in
a given volume of packed red
blood cells. It is reported as part
of a standard complete bloodcount to detect if the patient is
anemic or not.- Blood Gram stain, Culture andSensitivity is to detect what
type of bacteria grows in the
infected blood of the patientand for the Doctors to know
what type of antibiotics is
responsive to the bacteria
present in the patients blood.
- Balanced Multiple
Maintenance Solution with 5%Dextrose was given to thepatient as a maintenance fluid
to increase calories, hydration
and electrolytes in the body.This specific infusion rate is
advised depending on the
weight of the patient and thecondition. Since this patient is
a cardiac patient, we cant givetoo much IV fluid because this
might lead to cardiac overload.
Ceftriaxone is a third-
generation cephalosporinantibiotic a choice drug for
treatment of bacterial
meningitis. In pediatrics, it is
commonly used in febrileinfants between 4 and 8 weeks
of age who are admitted to the
hospital to exclude sepsis and
since the patient is beingconsidered to be septic,
therefore this drug was
prescribed.Amikacin is anaminoglycoside antibiotic used
to treat different types of
bacterial infections. Amikacinis most often used for treating
DONE
DONE
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55
February 4,
2012
I &O q shift
O2 inhalation
@ 4LPM via
face mask PRN
for tachypnea
Refer
unusualities
Rx:
FU CBC,
FU Na+, K+ ,
S/F cranial CT
scan
severe, hospital-acquired
infections with multidrug
resistant Gram negative
bacteria. Amikacin wasprescribed to the patient to
prevent hospital ward infection.Diazepam is an anti-convulsantor anti-seizure drug given to
patient as a stand-by medication
in case the Patient will haveseizure.
- Intake and Output is
monitored because too much
fluid intake for our patient cancause cardiac overload both
orally and intravenously. While
Urine output is also monitoredto make sure the kidneys arefunctioning normally meaning
whatever is being taken inside
the body should be excreted.
- Patients with cardiac problems
may develop dyspnea anytime
thats why oxygen inhalation isconsidered as a standby
amenity for emergency
purposes.
The patient is for close watch
thus referral is necessary if any
abnormalities occur.
The laboratory tests that are still
for follow-up are really
necessary to establish thediagnosis and treatment of the
patient.
This procedure is requested torule out any other congenitalabnormalities present in the
Nervous System of the neonate
because some PDA patients can
DONE
DONE
DONE
DONE
DONE
DONE
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56
S/F 2DECHO
S/F LP
undecided
IVF same @
1L
Shift cefriaxme
to cefortaxime
tt. 300g IVTT
VSq4
I &O q
have concomitant neurological
defects.
This examination is still forfollow-up because it is
necessary to establish thediagnosis and treatment of thepatient.
This examination is still forfollow-up because this
procedure can detect whether
the patient has bacterial
meningitis or sepsis.
To continue to give the
nutritional and electrolyte needsof the patient and for IVmedications as well.
Ceftriaxone was shifted toCefotaxime because this drug
has a broader spectrum activity
against Gram positive andGram negative bacteria
compared to that first drug.
For close monitoring of thepatient and to check if there arechanges in the Vital signs and
sensorium of the patient.
Intake and Output is monitored
because too much fluid intakefor our patient can cause
cardiac overload both orally
and intravenously. While Urine
output is also monitored tomake sure the kidneys are
functioning normally meaning
whatever is being taken insidethe body should be excreted.
DONE
DONE
DONE
DONE
DONE
DONE
DONE
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57
February 5,
2012
Refer any
unusualities
Rx: FU CBC,
Na+, K+
S/F cranial CT
scan
S/F 2DECHO
S/F LP
\
Continue
monitoring of
VS
Continue IVF
at SR
Continue
meds:
CefotaximeD1
Amikacin- D2
The patient is for close watch
thus referral is necessary if any
abnormalities occur.
The laboratory tests that are still
for follow-up are reallynecessary to establish thediagnosis and treatment of the
patient.
This procedure is requested to
rule out any other congenital
abnormalities present in the
Nervous System of the neonatebecause some PDA patients can
have concomitant neurological
defects
This examination is still for
follow-up because it isnecessary to establish the
diagnosis and treatment of the
patient.
This examination is still for
follow-up because this
procedure can detect whetherthe patient has bacterial
meningitis or sepsis.
For close monitoring of the
patient and to check if there are
changes in the Vital signs and
sensorium of the patient.
To continue to give the
nutritional and electrolyte needs
of the patient and for IVmedications as well.
These meds are still to becontinued to complete the
dosage to treat the severity of
the disease.
DONE
DONE
DONE
DONE
DONE
DONE
DONE
DONE
-
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58
February 6,
2012
I &O q shift
Refer any
unusualities
S/F cranial CT
scan
S/F 2DECHO
S/F LP
IVF @ SR
Cont meds:
CefotaximeD1
Amikacin- D2
Intake and Output is monitored
because too much fluid intake
for our patient can cause
cardiac overload both orallyand intravenously. While Urine
output is also monitored tomake sure the kidneys arefunctioning normally meaning
whatever is being taken inside
the body should be excreted.
The patient is for close watch
thus referral is necessary if any
abnormalities occur.
This procedure is requested to
rule out any other congenitalabnormalities present in theNervous System of the neonate
because some PDA patients can
have concomitant neurologicaldefects
This examination is still forfollow-up because it is
necessary to establish the
diagnosis and treatment of the
patient.
This examination is still for
follow-up because thisprocedure can detect whether
the patient has bacterial
meningitis or sepsis.
To continue to give the
nutritional and electrolyte needs
of the patient and for IV
medications as well.
These meds are still to be
continued to complete thedosage to treat the severity of
the disease.
DONE
DONE
DONE
DONE
DONE
DONE
DONE
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59
February 7,
2012
I &O q shift
Refuse LP
Rx
S/F Cranial CT scan
S/F 2DECHO
shift to Heplock
Cont meds:
CefotaximeD1
Amikacin- D2
Intake and Output is monitored
because too much fluid intake
for our patient can cause
cardiac overload both orallyand intravenously. While Urine
output is also monitored tomake sure the kidneys arefunctioning normally meaning
whatever is being taken inside
the body should be excreted
The mother refused to undergo
this procedure because they
dont have the money to pay forthis procedure. Also, the
Mother didnt think this
procedure was necessary to beundergone
This procedure is requested to
rule out any other congenitalabnormalities present in the
Nervous System of the neonate
because some PDA patients canhave concomitant neurological
defects
This examination is still forfollow-up because it is
necessary to establish the
diagnosis and treatment of thepatient.
The condition of the patient has
improved and the IV line wasremoved therefore heplock was
inserted for the medications of
the patient.
These meds are still to be
continued to complete the
dosage to treat the severity ofthe disease.
DONE
DONE
DONE
DONE
DONE
DONE
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60
February 8,2012
VSq4
I &O q shift
Refer any unusualities
Rx:
S/F Cranial CT scan
S/F 2DECHO
Med:
cefotaxime- D2
AmikacinD3
VSq4
For close monitoring of the
patient and to check if there are
changes in the Vital signs and
sensorium of the patient.
Intake and Output is monitoredbecause too much fluid intakefor our patient can cause
cardiac overload both orally
and intravenously. While Urineoutput is also monitored to
make sure the kidneys are
functioning normally meaning
whatever is being taken insidethe body should be excreted.
The patient is for close watchthus referral is necessary if anyabnormalities occur.
This procedure is requested torule out any other congenital
abnormalities present in the
Nervous System of the neonatebecause some PDA patients can
have concomitant neurological
defects
This examination is still for
follow-up because it is
necessary to establish thediagnosis and treatment of the
patient.
These meds are still to becontinued to complete the
dosage to treat the severity of
the disease
For close monitoring of the
patient and to check if there are
changes in the Vital signs andsensorium of the patient.
DONE
DONE
DONE
DONE
DONE
DONE
DONE
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61
February 9,
2012
I &O q shift
Refer unusualities
S/F cranial CT
scan
S/F 2DECHO
Meds:
CefotaximeD3
AmikacinD4
VSq4
I &O q shift
Intake and Output is monitored
because too much fluid intake
for our patient can cause
cardiac overload both orallyand intravenously. While Urine
output is also monitored tomake sure the kidneys arefunctioning normally meaning
whatever is being taken inside
the body should be excreted.
The patient is for close watch
thus referral is necessary if any
abnormalities occur.
This procedure is requested to
rule out any other congenitalabnormalities present in theNervous System of the neonate
because some PDA patients can
have concomitant neurologicaldefects
This examination is still forfollow-up because it is
necessary to establish the
diagnosis and treatment of the
patient.
These meds are still to be
continued to complete thedosage to treat the severity of
the disease
For close monitoring of thepatient and to check if there are
changes in the Vital signs and
sensorium of the patient.
Intake and Output is monitored
because too much fluid intake
for our patient can causecardiac overload both orally
and intravenously. While Urine
output is also monitored tomake sure the kidneys are
DONE
DONE
DONE
DONE
DONE
DONE
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62
February10, 2012
Refer unusualities
Rx:
S/F 2DECHO on
Thursday
Meds:
CefotaximeD6
AmikacinD5
VSq4
I &O q shift
Refer any unusualities
functioning normally meaning
whatever is being taken inside
the body should be excreted.
The patient is for close watch
thus referral is necessary if anyabnormalities occur
This examination is still for
follow-up because it isnecessary to establish the
diagnosis and treatment of the
patient.
These meds are still to be
continued to complete the
dosage to treat the severity ofthe disease
For close monitoring of the
patient and to check if there arechanges in the Vital signs and
sensorium of the patient.
Intake and Output is monitored
because too much fluid intake
for our patient can cause
cardiac overload both orallyand intravenously. While Urine
output is also monitored to
make sure the kidneys arefunctioning normally meaning
whatever is being taken inside
the body should be excreted.
The patient is for close watch
thus referral is necessary if any
abnormalities occur
DONE
DONE
DONE
DONE
DONE
DONE
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Date/
Time
Ordered
February
3, 2012
Diagnostic Test and
Normal Range of
Values
Urinalysis
Findings:
Color:
N: pale yellow to deep
amber
Appearance
Result
Straw( Pale
Yellow)
Clear
Clinical
Significance/Rationale
An unusual urine color is
among the most common
signs of a urinary tract
infection.
Cloudy or foamy urine may
occur occasionally due to
mild dehydration, polyuria
or phosphate in urine.
Nursing
Responsibilities
-provide the
patients mother
or significant
other with urine
container with
lead
-instruct the
watcher or
significant other
to collect sample
of urine
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Specific Gravity
N:1.010-1.020
Albumin
Sugar
Pus cells
1.005
(Normal)
Negative
Negative
0-2
Increased in: dehydration,
fever, profuse sweating,
vomiting, diarrhea,
glycosuria, proteinuria
Decreased in:
overhydration,
hypotension, severe renal
damage, diabetes insipidus
Positive in: renal disorders,
associated with
hypertension, diabetes
mellitus
Positive in:
Hyperglycemia, diabetes
mellitus
High level of pus in urine
is an indication of Urinary
tract infection
preferably arising
-Collect specimen
from infants or
young children
into a disposable
apparatus
consisting of a
plastic bag with
an adhesive
backing around
the opening that
can be fastened to
the perineal area
or the penis to
permit voiding
directly to the
bag. Depending
on the hospital
policy, the
collected urine
can be transferred
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Bacteria
IPD Hematology
CBC
WBC count
N: 5-10
Hemoglobin
N: 115-155
Few
8.35 x10/ uL
133.0 g/L
Numerous amount of
bacteria in urine will lead
to Urinary tract infection
Increased in: Infection,
inflammation, hematologic
malignancy, leukemia.
Decreased in: Aplastic
anemia, B12 or folate,
sepsis (decreased survival)
Increased in:
polycythemia, dehydration,
acute thermal injury
Decreased in: hemorrhage,
bleeding, anemia,
hemolytic anemia, fluid
to an appropriate
specimen
container.
-Cover all
specimens tightly,
label properly and
send immediately
to the laboratory
- identify patient
and check for the
requisition form
with the patients
identification
bracelet
-inform the
watcher or
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66
Hematocrit
N: 0.36-0.48
Red Blood Cells
N: 4.20-6.10
Neutrophils
N: 55-75
0.38
3.81x10^6/uL
30%
overload, fluid retention
Low levels of hematocrit is
an indication of anemia,
acute or chronic bleeding
from the digestive tract,
Nutritional deficiencies
such as iron, folate or B12
deficiency
High level of RBC in the
urine Indicates bleeding at
some point in the urinary
tract
Increased neutrophils:
suggests infection, acute
stress, acute and chronic
inflammations
Decreased neutrophils:
suggests aplastic anemia,
drug-induced neutropenia,
significant other
of the patient that
blood needs to be
drawn from the
designated site.
- ask assistance
from the mother
or the significant
other in handling
the baby in order
for him/her to be
secured.
- the patient may
be seated or in
supine position.
The patients arm
Is in extension.
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Lymphocytes
N: 20-35
Monocytes
N: 2- 10
Eosinophils
N: 1-8
54%
14%
2%
folate or B12 deficiency
Increased lymphocytes:
viral infection, adrenal
insufficiency disease,
chronic infection, drug and
allergic reactions,
autoimmune disease.
Decreased lymphocytes:
immune deficiency
syndrome.
Increased monocytes:
inflammation, infection,
malignancy, TB,
myeloproliferative
disorders.
Decreased monocytes:
depleted in overwhelming
bacterial infection.
Increased eosinophils:
allergic states, drug
sensitivity reaction, skin
disorders, tissue invasion
Post test
-instruct the
mother or the
watcher of the
patient to continue
compression of
the puncture site
for 25 mins or
until the bleeding
stops
-assess the
patients arm to
ensure that the
bleeding has
ceased.
-Apply adhesive
bandage if
necessary.
-if hematoma
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PH
N: 150-400
464x10/uL
by parasites,
hypersensitivity response
to malignancy, pulmonary
infiltrative disease,
Decreased eosinophils:
acute and chronic
inflammation, stress
A highly acidic urine pH
occurs in: Acidosis,
Uncontrolled diabetes,
Diarrhea, Starvation and
dehydration, Respiratory
diseases in which carbon
dioxide retention occurs
and acidosis develops
A highly alkaline urine
occurs in: Urinary tract
obstruction, Pyloric
obstruction, Salicylate
intoxication, Renal tubular
acidosis, Chronic renal
failure
occurs or if there
is still bleeding,
ask the watcher or
the mother of the
patient to continue
compression of
puncture site or
elevate the arm
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MCV
N:79.4-94.8
MCH
N: 25.6- 32.2
MCHC
N:32.2-35.5
99.7
34.9 pg
35.0 g/dL
Increased in: Liver
disease, megaloblastic
anemia (folate, B12
deficiencies), high WBC.
Decreased in: Iron
deficiency, thalassemia;
decreased or normal in
anemia of chronic disease.
increased in: Macrocytosis
, Megaloblastosis (Vitamin
B12 or folate deficiency),
reticulocytosis, liver
disease.
Decreased in:
Microcytosis (iron
deficiency, thalassemia).
Increased in:
hyperchromia, hemolysis
(with spuriusly high Hb or
low MCV or RBC),
Decreased in:
http://en.wikipedia.org/wiki/Reticulocytosishttp://en.wikipedia.org/wiki/Reticulocytosis -
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BLOOD CHEM
Glucose RBC
N: 4.10-6.6
Creatinine
N: 53.00-115.00
4.5
15.40 umol/L
Hypochromic anemia (iron
deficiency, thalassemia)
increased in: Diabetes
mellitus, Cushing's
syndrome, chronic
pancreatitis
Decreased in: Pancreatic
islet B cell disease with
increased insulin, diffuse
liver disease, infant of
diabetic mother, enzyme
deficiency diseases.
Increased in: Acute or
chronic renal failure;
urinary tract obstruction
Decreased in: Reduced
muscle mass
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Sodium
N:136.00-155.00
Potassium
N: 3.5- 5.5
137.70
mmol/L
4.63 mmol/L
=989898988
2.54 mmol/L
increased in: Dehydration,
polyuria , inadequate water
intake
Decreased in: Congestive
heart failure, cirrhosis,
vomiting, diarrhea,
excessive sweating, adrenal
insufficiency, nephrotic
syndrome.
Increased in:
Hyperkalemia, acute
tubular necrosis,Cushing
syndrome(rare),Diabetic
acidosisand other forms of
metabolic acidosis, Kidney
Disorders
Decreased in:
Hypokalemia, adrenal
gland insufficiency,
Gastrointestinal disorders
associated with diarrhea Prior to taking the
http://www.nlm.nih.gov/medlineplus/ency/article/000512.htmhttp://www.nlm.nih.gov/medlineplus/ency/article/000512.htmhttp://www.nlm.nih.gov/medlineplus/ency/article/000512.htmhttp://www.nlm.nih.gov/medlineplus/ency/article/000512.htmhttp://www.nlm.nih.gov/medlineplus/ency/article/000410.htmhttp://www.nlm.nih.gov/medlineplus/ency/article/000410.htmhttp://www.nlm.nih.gov/medlineplus/ency/article/000410.htmhttp://www.nlm.nih.gov/medlineplus/ency/article/000410.htmhttp://www.nlm.nih.gov/medlineplus/ency/article/000320.htmhttp://www.nlm.nih.gov/medlineplus/ency/article/000320.htmhttp://www.nlm.nih.gov/medlineplus/ency/article/000320.htmhttp://www.nlm.nih.gov/medlineplus/ency/article/000320.htmhttp://www.nlm.nih.gov/medlineplus/ency/article/000320.htmhttp://www.nlm.nih.gov/medlineplus/ency/article/000320.htmhttp://