tests in pediatric neurology(2)

7/23/2019 Tests in Pediatric Neurology(2)

http://slidepdf.com/reader/full/tests-in-pediatric-neurology2 1/23

Direction of Pediatric Neurology of Pediatric Department of Tbilisi

State Medical University

Tests in Pediatric Neurology

//// The cause of seizures in early childhood is:

// Deficiency of Piridoxine

/// Deficiency of Calcium Panthothenate

/// Excessive amount of Riboflavine

/// Deficiency of Folium acid

//// The most frequent reason of eilesy in childhood is:

/// !eurosis

// Residual encehaloathy

/// "ller#y/// Poor feedin#

//// The tri##erin# factors of eilesy in early childhood is follo$in#% excet:

/// &nfection

/// Trauma

/// Radiation

// Poor nursin#

//// The simle absence is defined as:

// 'rief loosin# of consciousness

/// "brut droin#

/// Tonic(clonic seizures $ithout losin# of consciousness

/// &nvoluntary urination and defecation

)//// *est syndrome usually occurs in:

/// !eonate eriod

// + to , year of a#e

/// -chool eriod/// Pre(school eriod

.//// The transient Todds aresis is associated $ith:

/// 0eneralized eilesy

/// -imle absence

/// Comlex absence

// Partial eilesy

1/// 'y means of electroencehalo#rahy $e can not:

/// Establish the dia#nosis of eilesy

/// Define the localization of eiletic foci

,



/// "ssess the efficiency of theraeutic mana#ement

// Define the etiolo#y

2//// The follo$in# dru#s are recommended for the treatment of absences:

// -uccinimides and 3alroic acid

/// 4idantoins

/// 'arbiturates

/// Carbamazeine

5//// 6ennox(0astaut -yndrome is defined as:

// Frequent% sometimes serial olymorhic seizures $ith mental retardation7

/// 8nly febrile seizures

/// 8nly artial seizures/// Disease $ith beni#n course

//// The follo$in# dru#s are recommended for the treatment of *est syndrome:

// 4ormones and 3alroic acid or 3i#abatrine

/// Phenobarbital

/// Dihenine

/// Diazeame

//// 0eneralized eilesies are the follo$in#% excet:

/// "bsences

/// 9yoclonic seizures

/// Tonic(clonic seizures

// ac;sons motor seizures

//// Partial seizures are the follo$in#% excet:

/// &dioathic < a#e(related=

/// -ymtomatic

/// Cryto#enic// *est syndrome

//// EE0 attern of tyical absences is:

/// Temoral shar $aves

// > 4z7 -i;e($ave comlexes

/// 4i#h amlitude #eneralized activity

/// )(. 4z7 -i;e($ave comlexes

//// The tyical EE0 attern of *est syndrome is:

// 4ysarrhythmia

?



/// Focal slo$ $ave activity

/// > 4z7 -i;e($ave comlexes

/// 8nly shar $aves

//// The tyical EE0 attern of uvenile absence eilesy is:

/// Diffuse slo$ $ave activity

// 0eneralized bilateral synchronous >%)(@ 4z7 -i;e($ave comlexes7

/// 6o$ amlitude activity

/// 4ysarrhythmia

//// The tyical EE0 attern of atyical absence is the follo$in#:

/// Diffuse slo$ $ave activity// A%)(?%) 4z7 -i;e($ave comlexes

/// 4ysarrhythmia

/// Focal slo$ $ave activity

//// The simle artial seizures are the follo$in#% excet:

/// 9otor

/// Clonic

// "bsences

/// 3e#etative

//// The reserved consciousness is the si#n of:

/// 6ennox(0astaut syndrome

/// *est syndrome

/// "bsences

// -imle artial seizures

//// The follo$in# are not related to #eneralized seizures:

/// "brut onset/// Pro#ressive seizures

// Preserved consciousness

/// "mnesia of seizure eisode

//// The follo$in# are related to the tyical absences:

// "brut onset

/// The #radually onset

/// A%)(?%) 4z7 -i;e($ave comlexes

/// Duration of seizures >A seconds and more

>



//// The follo$in# is related to atyical absences:

/// "brut onset

// The #radually onset

/// >(>%) 4z7 -i;e($ave comlexes

/// Duration of seizures less than ,)seconds//// "ura can be:

/// 3isual

/// "uditory

/// 0ustatory

// "ll of them

//// Febrile seizure can occur:

// 'efore . years of a#e

/// .(,? years of a#e/// Puberty eriod

/// + to ?A years of a#e

//// -imle febrile seizure is not characterized by:

/// Duration B ,) minutes

// Reetition rate ?@ hours

/// Rare eisodes

/// "bsence of neurolo#ical symtoms after seizure

//// -imle febrile seizure is characterized by:

/// Duration ,) minutes

/// Focal aroxysm

/// Reetition rate ?@ hours

// Rare eisodes

//// Comlex febrile seizure is characterized by:

/// Duration B ,) minutes// Duration ,) minutes

/// Rare eisodes

/// Preserved consciousness in ictal eriod

//// Comlex febrile seizure is not characterized by:

// Rare eisodes

/// Duration ,) minutes

/// Reetition rate ?@ hours

/// Focal aroxysms

@



//// "bsence seizure is:

/// "dversive seizure

/// Tonic seizure

/// Tonic(clonic seizure

// *est syndrome

//// "bsence seizure is not:

/// *est syndrome

// "dversive seizure

/// 6ennox(0astaut syndrome

/// -alaami seizures

//// Partial seizures can not be:

/// "dversive/// Tonic

/// Clonic

// *est syndrome

//// &nfantile sasms can occur:

/// > years of a#e

// >(1 months of a#e

/// &n neonatal eriod

//// &n uberty eriod

//// &nfantile sasms first $as described by:

/// 6ennox

/// 0astaut

// *est

/// ac;son

//// &nfantile sasm is defined as:

// Flexor(extensor sasms% mental retardation% hysarrhythmia on EE0

/// !ormal sychomotor develoment

/// -imle artial seizures

/// > 4z7 -i;e($ave activity on EE07

//// &nfantile sasms occur durin# the follo$in# conditions:

// -evere dama#e of C!-

/// 4yerthermia

/// &ntoxication

/// &nfection

)



//// The recommended daily dosa#e of "CT4 for infantile sasm is:

/// ?A(>A unit

// 2A(,AA unit

/// 6ess than ,A unit

/// ) unit

//// The initial dosa#e of Prednisolone used for the treatment of infantile sasms is:

// ? m#/;#/d

/// A%) m#/;#/d

/// @ m#/;#/d

/// , m#/;#/d

//// The reduction of dosa#e of hormones in infantile sasms can be started:

/// *hen seizures become rare

// "fter cessation of seizures and hysarrhythmia on EE0

/// "fter imrovin# the sychomotor functionin#

/// "fter imrovin# the EE0 atterns

//// The dru# used in infantile sasms is:

// 4i#h doses of Piridoxine

/// 3itamin C

/// 3itamin D

/// 3itamin E

7//// The dru# not recommended for the treatment of infantile sasms is:

/// "CT4

/// &mmuno#lobulin

/// Piridoxine

// !ivalin

//// The dru# not recommended for the treatment of infantile sasms is:

/// !ivalin

// "CT4

/// Phenobarbital

///Piracetam

@, The comlex absence is not characterized by:

// "brut onset

/// Tonic comonent

/// Clonic comonent/// 0radual onset

.



@? The simle absence is characterized by:

/// Fixation of #aze

// 3e#etative manifestation

/// "brut be#innin# and finishin#

/// d7 "bsolutely imaired consciousness

@> The simle absence is characterized by:

/// 0radual be#innin# and finishin# of aroxysms

/// Duration of seizure more t)han >A seconds

/// 9ioclonic comonent

// "brut cessation of motor activity

@@ The comlex artial seizure is characterized by:

/// Preserved consciousness

// Comlete or incomlete loss of consciousness/// 8nly motor comonent

/// "bsence of amnesia

@) 0eneralized tonic(clonic seizure is not characterized by:

/// 6oss of consciousness

/// "brut droin#

// Preserved consciousness

/// Tonic(clonic sta#e

@. !eonatal seizure occurred:

/// "fter , month of a#e

// ,(@ $ee;s of a#e

/// ?(@ months of a#e

/// "fter , years of a#e

@1 The cause of neonatal seizures are the follo$in#% excet:

/// 4yoxic(ischemic dama#e of C!-

// 8enheims myotonia/// &ntracranial hemmora#e

/// 9icroelements imbalance

@2 The cause of neonatal seizures are the follo$in#% excet:

/// &ntrauterinal infections

/// Piridoxine deficiency

// 3erdin#(4offmans sinal amyotrohy

/// &nherited metabolic disorders

1



@5 !eonatal seizures can not be:

/// !ot manifested

/// Tonic

/// 9ioclonic

// "tonic

)A The reason of seizure syndrome caused by calcium deficiency are the follo$in#% excet:

/// The lo$ level of arathyroid hormone

// 4i#h level of bilirubin in blood

/// Disorder of vitamin D metabolism

/// 4yoma#nemia

), Eiletic status is defined as:

/// !ormal consciousness bet$een aroxysms

// Permanent aroxysms $ith duration of >A minutes or more/// "mnesia of seizure eisode

/// "rterial hyertonia

)? Eiletic status is not defined as:

/// Permanent aroxysms $ith duration of >A minutes or more

/// "mnesia of seizure eisode

// !ormal consciousness bet$een aroxysms

/// Eiletic atterns on EE0

//// The mana#ement of eiletic status does not imly:

/// 9onitorin# of vital functions

/// +r#ent anticonvulsive theray

/// Exress dia#nosis

// Per os administration of anticonvulsants

//// The follo$in# dru#s are recommended for the mana#ement of eiletic status:

// Diazeam or 6orazeam

/// -uccinimedes/// 6amotri#ine

/// 3i#abatrine

//// The dru# not recommended for the mana#ement of eiletic status is:

/// Phenobarbitale ,Am#/;#

/// Pentobarbitale .(2 m#/;#

/// 6idocaine ,(? m#/;#

// 6amotri#ine ?%) m#

2



//// The dru# not recommended for the mana#ement of eiletic status is:

/// Diazeame or 6orazeam

/// Phenitoine

// 3i#abatrine

/// Phenobarbitale

//// The dru# administration in eilesy lasts for:

/// ,(> months

/// >(. months

// >() years

/// , years

)2//// The anticonvulsive theray is conducted:

/// "s a short eriod course for ,(? years

// Permanently% $ith administration of dru# for several years/// 'y usin# of broad sectrum anticonvulsant for . months

/// 'y administration of dru# for , year

)5//// The anticonvulsive theray is recommended to start by:

// -in#le anticonvulsant

/// T$o or three anticonvulsants

/// 3itamins

/// "ntibiotics

.A //// The dru#s of first choice in febrile seizure are the follo$in#:

/// Phenobarbitale

// 3alroic acid

/// 6amotri#ine

/// Toiramate

.,//// The dru# recommended to use for the treatment of artial seizures is:

// Carbamazeine

/// -uccinimide/// ' #rou vitamins

/// 4ormone

.?//// The dru# not recommended to use for the treatment of artial seizures is:

/// Carbamazeine

/// 3alroic acid

// -uccinimnides

/// Phenobarbitale

5



.> The dru# recommended to use for the treatment of absence seizures is:

/// Carbamazeine

/// Phenobarbitale

// 3alroic acid

/// Phenitoine

.@ The dru# not recommended to use for the treatment of is:

/// -uccinimides

/// Clonazeame

/// 3alroic acid

// Carbamazeine

.) The follo$in# dru#s are used for the treatment of 6ennox(0astou syndrome:

// 6amotri#ine

/// Phenobarbitale/// Piridoxyne

/// "ntibiotics

.. The dru#s not used for the treatment of 6ennox(0astaut syndrome is:

/// 9onotheray $ith 6amictal

// Phenobarbitale

/// 3alroic acid

/// Clonazeame

.1 The anticonvulsants of ne$ #eneration is:

/// Phelbatole

/// 8xcarbamazeine

/// "cetazolamide

// Phenobarbitale

//// The basic anticonvulsant is not:

// 0abaentine

/// Phenobarbitale/// Carbamazeine

/// 3alroic acid

.5 The basic anticonvulsant is :

/// Toiramate

/// 3a#abatrine

// Carbamazeine

/// 9idazolam

,A



1A 'esides the dru#s the aroach also used for the treatment of intractable eilesy is:

/// Physiotheray

// eto#enic diet

/// 8ccuational theray

/// Psychotheray

//// For the treatment of intractable eilesy is used:

/// Electrohoresis

/// 8ccuational theray

// -timulation of 3a#us

/// Psychotheray

1? For the treatment of intractable eilesy is used:

// !eurosur#ical oerations on brain

/// 8ccuational theray/// Psychotheray

/// Electrohoresis

1> Porencehaly often causes the follo$in# form of cerebral alsy:

/// -astic dile#ia

/// -astic tetraaresis

// 4emiaretic form

/// 4yer;inetic form

1@ "lasia of basal #an#lia and nuclear aundice cause the follo$in# form of cerebral alsy:

// 4yer;inetic form

/// 4emiaretic form


/// -astic dile#ia

1) 6ittly disease is resented as the follo$in# form of cerebral alsy:


// -astic dile#ia/// 4yer;inetic form

/// "tonic(astatic form

1. The disorders of extremities in sastic dile#ia are exressed:

/// 8n one side

// Disorder is symmetric

/// +er extremities are affected more

/// +er and lo$er extremities are affected on one side

//// &n cerebral alsy the symmetric reflex from head to trun; and extremities is:

,,



/// &ncreased

// &s increased in neonate eriod and inhibited in re(school eriod

/// &s absent

/// &s al$ays chan#ed

12 &n cerebral alsy of aralitic form the muscle atrohy is exressed by the follo$in# $ays:

/// &s absent

/// 8ccurred only in later residual eriod

/// 8ccurred only durin# muscle hyotonia

// &s mildly exressed and has diffuse manifestation

15 The revalence of cerebral alsy er ,AAA is:

/// A%) and less

// ,%) and ?

/// ) and more/// ,A and more

2A &n cerebral alsy of sastic tetrale#ia form is exressed:

// Tetraaresis more affected the uer extremities

/// 4emile#ia on one side and hemiaresis on other side

/// Tetraaresis occurred only in severe cases

/// Disorders only in lo$er extremities

2, &n cerebral alsy of tetrale#ia form the muscle tone is:

/// "l$ays decreased

/// -li#htly increased

// &s increased si#nificantly

/// &s increased only in later residual eriod

?//// &n cerebral alsy of sastic dile#ia form are affected:

/// 8nly internal casule on one side

/// Cerebral eduncles

// Cerebral hemisheres% esecially cortex/// 'rainstem

2> &n cerebral alsy of sastic tetrale#ic form atient start $al;in#:

// &n very rare cases

/// 8n the third year of life

/// "fter )(. year of a#e

/// 8nly in the case of intact mental field

@//// &n cerebral alsy of hyer;inetic form is affected the follo$in# structures:

,?



/// The uer arts of brainstem

/// The cerebral cortex

/// Cerebellum

// -trioaidal system

//// 4earin# more frequently is imaired durin# the follo$in# forms of cerebral alsy :

/// -astic dile#ia

/// -astic tetrale#ia

/// 4emiaretic form

// 4yer;inetic form occurrin# after nuclear aundice

2.//// The sychiatric disorders in cerebral alsy of atonic(astatic form occur:

/// never occur

// in case of frontal lobes disorders

/// in case of cerebellum disorders/// in case of disorders of cerebellum ath$ays

1 //// The muscle tone in cerebral alsy of atonic(astatic form is chan#ed by the follo$in# $ays:

/// -astic

/// Ri#id

// 4yotonia

/// Distonia

22 The course of cerebral alsy is:

/// Pro#redient

// Re#redient in case of adequate treatment

/// Re#redient only in early eriod of life

/// Pro#redient only in reschool a#e

25 The ro#nosis of cerebral alsy deends on the follo$in# issues:

/// 8nly eriod of dama#e

/// 8nly eriod of startin# of treatment

/// The somatic condition of child// Time and de#ree of dama#e and eriod of startin# of treatment

5A Contractures and deformation in cerebral alsy :

// 9ay increase to#ether $ith a#e

/// &s stable

/// 9ay decrease to#ether $ith a#e

/// 9ay increase only in case of sastic tetrale#ia

5, The eilesy of childhood is often caused by:

/// &nfection and hyotrohy

// 0enetic redisosition and ne#ative exo#enous factors

,>



/// 9inimal cerebral dysfunction

/// Defects of carin#

//// The bloody saliva in eiletic seizure is the result of:

/// !asal bleedin#

/// Pharyn#eal bleedin#/// Disorder of blood a##re#ation system

// Dama#e of ton#ue and chee;s

5> The reventive vaccination in case of eilesy can be conducted only in the follo$in#

cases:

/// "fter ,(? years from the last seizure

// "fter . months from the last seizure to#ether $ith increasin# the anticonvulsant

theray

/// "fter ? years from the last seizure

/// 8nly $hen the EE0 atterns are normal

5@ The #radual decreasin# of antieiletic treatment is ossible in the follo$in# cases:

/// "fter ,(? years from the last seizure

/// *hen there is imrovement on EE0

/// 8nly in uberty eriod

// "fter >() years from the last seizure and in case of normal EE0 atterns

5) The etiolo#ical factors of neurosis is:

/// -omatic condition

/// "#e

/// Premorbid bac;#round

// Psychotrauma

5. &n re( and uberty eriod fear is often accomanied by:

/// 4allucinations

/// -eizures

// Exressed ve#etative reactions

/// &nvoluntary urination

//// 6o#oneurosis is often occurred in the follo$in# eriods:

// 'efore ) years of a#e

/// early school eriod

/// 6ate school eriod

/// Puberty eriod

52 The neuro#enic tics often occur in the follo$in# eriods:

/// 'efore > years

,@



/// >() years

// )(,? years

/// "fter ,. years

55 The main aroach in the treatment of neurosis is:

/// Physiotheray/// Dru#s

/// Comlex aroach containin# dru#s and hysiotheray

// Psychotheray

,AA The inherited unconditioned automatism reflexes are:

/// The reflex of suc;in#

/// The reflex of automotive climbin#

/// The reflex of #rasin#

// "ll of them

//// The follo$in# are not the reflexes of oral automatisms:

/// The reflex of suc;in#

/// The reflex of see;in#

/// The reflex of Gelehant trun;H

// The reflex of defense

,A? The follo$in# are not the inherited myelencehalic reflexes of neonatal eriod:

/// The tonic symmetric nec; reflex

/// The tonic asymmetric nec; reflex <9a#nus(leins=

// The reflex of #rasin#

/// The symmetric and asymmetric tonic reflexes

//// The suc;in# reflex doesnt disaear:

/// +ntil ? months

/// +ntil . months

// +ntil ,2 months

/// +ntil ,? months

//// The #rasin# <Robisons= reflex in normal neonates disaears:

/// "t ? months of a#e

/// "t >(@ months of a#e

// "t , year of a#e

/// "fter , year

,A) The diffuse decrease of muscle tone in neonates can occur in the follo$in# cases:

/// Dama#e of cerebellum

/// *erdni#(4offmanns sinal amyotrohy

/// 8enheims myotonia

// Cerebral alsy of hemiaretic form,A. The feature of *erdni#(4offmanns amyotrohy is:

,)



/// Disaearance of ;nee reflex

/// The ro#redient course of disease

/// Diffuse hyotonia

// 4yereflexia

,A1 The inherited unconditioned reflexes of neonates disaears at the follo$in# a#es:

/// 'efore , month

/// 'efore ,(? months

// 'efore >() moths

/// "fter , years

//// 6andau inherited reflex in neonates disaears:

/// "t , month of a#e

/// "t ,(> months of a#e

/// "t . months of a#e// &t never disaears

,A5 The follo$in# are not T8RC4 infection:

// Rubella infection

/// Cytome#alovirus infection

/// 4eres -imlex infection

/// Toxolasmosis

//// &n con#enital rubella infection microcehaly occurs:

/// &n ,AAI of cases

// &n ,A(?AI of cases

/// &n )AI and more

/// &t never occurs

,,, 4ydrocehaly% enohthalmy% sastic aresis% eiletic seizures% chorioretinitis associated

$ith fetal heatitis in neonates are the features of:

/// Rubella infection

// Toxolasmosis

/// Cytome#alovirus infection/// 6isteriosis

//// The most frequent form of develomental defect of brain is:

/// 9yelocele

/// 9acrocehaly

// 9icrocehaly

/// Dysostosis of s;ull and face

,,> 9icrocehaly is divided by the follo$in# $ays:

// Primary and secondary

/// Con#enital and acquired

,.



/// Postinfectious and osttraumatic

/// Diffuse and focal

,,@ &n microcehaly occur the follo$in# rocesses:

/// The brain is si#nificantly small in size comared to s;ull

// The small size s;ull is correlated to reduced size brain/// 8nly brain art of s;ull is reduced comared to face art

/// The face and brain art of s;ull are roortional

,,) 9orholo#ical determinant of microcehaly is:

/// The roortionally reduction of the $hole brain

// The severe defects of develoment

/// 8nly macro( and micro#yria

/// 8nly disorder of s;ull

//// The secondary microcehaly occurs in:

/// Prenatal eriod

/// 8nly ostnatal eriod

// Perinatal eriod and in the first months of life

/// "ny a#e

,,1 The sizes of s;ull in microcehaly at the moment of birth is:

/// "l$ays reduced by >(@ cm

/// Reduced by ) cm and more

/// 9ay be increased

// 9ay be normal

//// The motor develoment in microcehaly is:

/// "l$ays normal

// &s al$ays delayed

/// &s delayed in case of combination $ith cerebral alsy

/// &s delayed only in rimary microcehaly

5 The ro#nosis of microcehaly can be redicted by:

/// The rate of increasin# of s;ull sizes

/// -;ull sizes

// The level of mental retardation

/// The time of adequate treatment

The increasin# of intracranial ressure is the si#n of:

// Craniostenosis

/// Primary microcehaly

/// -econdary microcehaly/// 9icrocehaly associated $ith eilesy

,1



,?, The sontaneous comensation of con#enital hydrocehaly may occur:

/// 'y imrovin# the cerebrosinal fluid absortion

// Durin# the atrohy of choroidal lexuses and by reducin# the cerebrosinal fluid

roduction

/// 'y movin# a$ay of s;ull sutures

/// Durin# the liquorrhea

,?? 4ydrocehaly:

/// &s al$ays secondary and is the result of brain atrohy

/// &s al$ays rimary

// 9ay be rimary or secondary

/// "l$ays occurs durin# orencehaly

//// &n con#enital hydrocehaly the follo$in# chan#es occur on the fundus of eye:

/// Con#estion/// "n#ioathy

// The decoloration of otic dis;

/// The neuritis of otic nerve

,?@ The non(comensatin# hydrocehaly occurs:

/// 8nly durin# the viral infection

/// 8nly durin# the enterovirus infection

/// 8nly durin# the neuroinfection

// Durin# the C!- trauma or infection

//// The sur#ical treatment of hydorcehaly is recommended in order to:

/// &mrove venous circulation

/// !ormalize the cerebral hemodinamics

// 9a;e the collateral circulation of cerebrosinal fluid

/// Reduce the secretion of cerebrosinal fluid

//// The erinatal eriod is defined as the eriod:

/// From the first movement of fetus to moment of birth// From ?2 $ee;s of #estation to first 1 days of life

/// From ?2 $ee;s of #estation to moment of birth

/// The first 1 days of life

,?1 The traumatic inury of sinal cord in neonates often occurs in the follo$in# se#ments:

/// The uer cervical

// &ntumenscentia cervicalis

/// Thoracal

/// &ntumenscentia lumbaris

//// The obstetrical aralysis of hand of total tye occurs durin# the dama#e of the follo$in#

se#ments:

,2



/// C)(C.

/// C1(C2

/// D,(D?

// C)(D,

//// The obstetrical aralysis of hand of roximal tye occurs durin# the dama#e of the follo$in#

se#ments:

// C)(C.

/// C1(C2

/// D,(D?

/// C)(D,

,>A The obstetrical aralysis of hand of distal tye < Deerin(lum;e= occurs durin# the

dama#e of the follo$in# se#ments:

/// C)(C.

/// C1(C2// D,(D?

/// C)(D,

131 The structural defect of the brain in cerebral alsy affects the follo$in# functions:

/// 8nly motor develoment

/// 8nly seech develoment

// The develoment of the $hole brain

/// &t doesnJt affect on brain develoment

//// The cerebral alsy in relation to hyoxic(ischemic encehaloathy is:

/// &ndeendent disease

/// 8rdinary outcome

/// The have no relation

// 8ne ossible variant of outcome

,>> &n cerebral alsy of sastic dile#ia form occurs:

/// +er sastic araaresis

/// -astic hemiaresis

// 6o$er araaresis/// Tetraaresis

//// The sastic tetraaresis form of cerebral alsy is defined as:

/// Tetraaresis $ith mild mental retardation

// Tetraaresis $ith severe mental retardation

/// The eriheral aresis in hands and sastic aresis in le#s

/// The sastic aresis in hands and eriheral aresis in le#s

,>) &n cerebral alsy of atonic(astatic form are imaired the follo$in# structures:

/// 'oth internal casules

/// 'asal #an#lias

,5



// Cerebellum

/// The brain hemisheres

//// The ossible reason of traumatic inury of facial nerve in neonates is not:

/// Face resentation

/// Difficult labor /// The fractures of temoral bone

// -ubarachnoidal hemmora#e

//// The reason of sinal cord trauma can be:

// Pelvic resentation

/// Cross birth

/// !ormal cehalic resentation

/// 'ro$ resentation

//// The sinal inury in neonates occurs in the follo$in# cases:

/// &ntrauterine hyoxia

/// !atal eriod ashyxia

/// !atal eriod infection

// !atal eriod trauma

//// The severe dama#e of C!- can be defined by "#arJs scale by the follo$in# scores:

/// A(,

// ,(@

/// )(.

/// .(1

,@A The moderate dama#e of C!- can be defined by "#arJs scale by the follo$in# scores:

/// A(,

/// ,(@

// )(.

/// .(1

//// The mild dama#e of C!- can be defined by "#arJs scale by the follo$in# scores:

/// A(,

/// ,(@

/// )(.

// .(1

,@? The etiolo#ical factor of hyoxic(ischemic encehaloathy is not:

/// 4yoxia

/// Trauma

/// Toxic rocess// Con#enital factor

?A



//// The #ood ro#nosis has the follo$in# form of hydrocehaly:

/// 8cclusive

// 8en% $ith slo$ rate of ro#ression

/// 8ccurrin# durin# intrauterine infections

/// 8ccurrin# after delivery trauma

,@@ The mental retardation in severe hydrocehaly is resented by the follo$in# $ays:

/// -evere retardation

// Ran#in# from mild form to idiotia

/// 8ften in normal ran#e

/// -li#htly exressed retardation

//// The reason of 0reheJs symtom in hydrocehaly is the follo$in#:

/// The irritation of cilliosinal symathic nucleus

/// Defects of vision/// &ncreasin# of intraorbital ressure

// Deformation of orbitas and exohthalmus

,@. The head circumference starts to increase in the follo$in# eriods:

// The first month of life

/// >() months of life

/// "fter . months of life

/// "t , year of life

//// The follo$in# conditions occur in severe hydrocehaly:

/// 8tical nerve neuritis

// 4ydroanencehaly

/// Pyramidal deficiency

/// 6esion of cerebellum and its connections

//// The most frequent reason of con#enital hydrocehaly is:

/// Porencehaly

/// "#enesia of corus callosum

/// "lasia of cerebellum

// 8cclusion of cerebral aquaductus

,@5 The child $ith con#enital hydrocehaly may has:

// !ormal or sli#htly increased head circumference

/// &ncreased head circumference by @() cm

/// Reduced circumference of head

/// &ncreased head circumference by )(2 cm

//// The most frequent form of hydrocehaly is:

?,



/// Traumatic

/// Toxic

/// 4yoxic

// Con#enital

,), Dilatation of subarachnoid saces occurs in the follo$in# forms of hydrocehaly:

/// &nternal

// External

/// 'asal

/// Convexital

//// Dilatation of brain ventricles and subarachnoid saces occurs in the follo$in# forms of

hydrocehaly:

/// &nternal

/// External

// 8en

/// Convexital

,)> Dilatation of only ventricles occurs in the follo$in# forms of hydrocehaly:

// &nternal

/// External

/// 'asal

/// Convexital

//// 'y the eriod of develoment hydrocehaly can be divided in the follo$in# forms:

/// Primary and secondary

// Con#enital and acquired

/// Pre( and ostnatal

/// Early < before . month= and late < after . month=

//// Passive hydrocehaly occurs in the follo$in# cases:

// 'rain atrohy or ro#ressive sclerosis of brain

/// Disorder of cerebrosinal fluid absortion

/// 3enous stasis

/// Excessive roduction of cerebrosinal fluid

,). The basic si#n of hydrocehaly is:

/// &ncreasin# of intracranial ressure

// &mbalance bet$een the roduction and absortion of cerebrosinal fluid

/// Dilatation of ventrical system and cerebrosinal circulation $ays

/// &ncreased head circumference

//// The clinical feature of hyertensive(occlusive crisis imlies:

??



// Common cerebral hyertensive si#ns and focal neurolo#ical symtoms

/// 9enin#eal si#ns

///0eneralized seizures

/// 8nly focal si#ns

//// The reason of hyertensive(occlusive crisis is:

/// Excessive roduction of cerebrosinal fluid

// 8cclusion of cerebrosinal fluid circulation $ays

/// -ubarachnoid hemmorra#e

/// Difficulty of cerebrosinal fluid absortion

//// The K(ray si#ns of hydrocehaly are not:

/// "trohy of s;ull bones

/// Flattenin# of s;ull base bones

/// Dilatation of sella turcica// Existence of intracranial calcification

4ead of !eurolo#ical Direction

of Pediatric Deartment of

Tbilisi -tate 9edical +niversity%

Professor !ana 0eladze

tests in pediatric neurology(2)

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