traumatic cardiac arrest alex king and kaushik nilakant
TRANSCRIPT
• Conventional ACLS based resuscitation focussing on “time on chest” and defibrillation will be a waste of time
• Modified resuscitation might save lives
• Use your judgement and tailor to each case
Reversible causes of cardiac arrest
-Hypoxia-Hypovolaemia-Hyper/hypokalaemia-Hypoglycaemia-Hyper/hypothermia
-Tension pneumothorax-Tamponade-Toxins-Thrombosis - pulmonary-Thrombosis - cardiac
Reversible causes of TRAUMA cardiac arrest
-Hypoxia-Hypovolaemia-Hyper/hypokalaemia-Hypoglycaemia-Hyper/hypothermia
-Tension pneumothorax-Tamponade-Toxins-Thrombosis - pulmonary-Thrombosis - cardiac
A = artificial airway I.e. intubation
B = bilateral finger thoracostomy
C = compression of pelvis and reduce long bone fractures
D = deploy IV fluids (or blood)
E = ECHO
F = FAST
A = artificial airway I.e. intubationHYPOXIA
B = bilateral finger thoracostomyTENSION PNEUMOTHORAX
C = compression of pelvis and reduce long bone fracturesHAEMORRHAGE
D = deploy IV fluids (or blood)HAEMORRHAGE
E = ECHOTAMPONADE
F = FAST
This applies to traumatic cardiac arrest (the cardiac arrest follows the trauma)
What if we cannot be sure of which came first?Are there departmental issues we need to address?