unit 13 weight control
TRANSCRIPT
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ENERGY BALANCE AND
WEIGHT CONTROL
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ENERGY BALANCE State in which energy intake, in the form
of food and /or alcohol, matches the energy
expended, primarily through basal
metabolism and physical activity Positive energy balance
Energy intake > energy expended
Results in weight gain
Negative energy balance
Energy intake < energy expended
Results in weight loss
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ENERGY BALANCE
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ESTIMATING KCAL
CONTENT IN FOOD Bomb calorimeter
Burns food inside a
chamber surrounded
by water Heat is given off as
food is burned
The increase in water
temperature indicates
the amount of energy
in the food
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MACRONUTRIENTS AND FAT
STORAGE Most fat is stored directly into adipose
tissue
Body has unlimited ability to store fat (as
fat)
Limited CHO can be stored as glycogen
Most CHO is used as a energy source
Excessive CHO will be converted into fat
(for storage)
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Macronutrients and Fat Storage
Protein is primarily used for tissue synthesis
Adults generally consume more protein than
needed for tissue synthesis
Some protein will be converted into fat (for
storage)
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MACRONUTRIENTS AND FAT
STORAGE Body prefers to use CHO as energy source
Only excess intake of CHO and protein will
be turned into fat
Fat will remain as fat for storage
Physical activity encourages the burning of
dietary fat
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ENERGY IN vs ENERGY OUT
Basal Metabolism
Dietary Intake Physical Activity
Thermic Effect of food
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BASAL METABOLISM
The minimum energy expended to keep a
resting, awake body alive
~60-70% of the total energy needs
Includes energy needed for maintaining a
heartbeat, respiration, body temperature
Amount of energy needed varies between
individuals
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INFLUENCES ON BASAL
METABOLISM Body surface area (weight, height)
Gender
Body temperature Thyroid hormone
Age
Kcal intake
Pregnancy
Use of caffeine and tobacco
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PHYSICAL ACTIVITY
Increases energy expenditure beyond BMR
Varies widely among individuals
More activity, more energy burned
Lack of activity is the major cause of
obesity
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THERMIC EFFECT OF FOOD
Energy used to digest, absorb, and
metabolize food nutrients
Sales tax of total energy consumed ~5-10% above the total energy consumed
TEF is higher for CHO and protein than fat
Less energy is used to transfer dietary fat
into adipose stores
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NONEXERCISE ACTIVITY
THERMOGENESIS Nonvoluntary physical activity triggered by
overeating
Fidgeting
Over eating increases sympathetic nervous
system activity
Resists weight gain
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MEASUREMENT OF
BODYS ENERGY NEEDS Direct calorimetry
Measures heat output from the body using an
insulated chamber
Expensive and complex
Indirect calorimetry
Measures the amount of oxygen a person uses
A relationship exists between the bodys use of
energy and oxygen
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HARRIS-BENEDICT EQUATION
FOR THE BODYS ENERGY
NEEDS
ESTIMATES RESTING ENERGY needs
Considers height, weight, age, and gender
For men:
66.5 + 13.8x(kg) + 5x(cm) - 6.8x(age in yr.)
For women:
655.1 + 9.6x(kg) + 1.8x(cm) - 4.7x(age in yr.)
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SAMPLE CALCULATIONS
Man: 21 yr., 510 (171 cm), 155# (70 kg)
66.5 + 13.8x(70kg) + 5x(171cm) - 6.8x(21)
= 1745 kcal/day
Woman: 21 yr., 510 (171 cm), 155# (70kg)
655.1 + 9.6x(70kg) + 1.8x(171cm) -
4.7x(21)= 1536 kcal/day
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ESTIMATED ENERGY
REQUIREMENT (EER) FOR MEN
EER =662 - (9.53 x AGE) + PA x (15.91 x
WEIGHT + 539.6 x HEIGHT )
FOR WOMEN
EER = 354(6.91 x AGE) + PA x (9.36 x
WEIGHT + 726 x HEIGHT)
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WHY DO YOU EAT?
Hunger
Physiological (internal) drive to eat
Controlled by internal body
Appetite
Psychological (external) drive to eat
Often in the absence of hunger
e.g., seeing/smelling fresh baked chocolate chip
cookies
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WHY WE EAT
Appetite is affected by a variety of external
forces
Combination of internal and external signalsdrive us to eat
Not a perfect system; desire to eat can be
overwhelming
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SATIETY REGULATOR
The hypothalamus
When feeding cells are stimulated, they signal
you to eat
When satiety cells are stimulated, they signal
you to stop eating
Sympathetic nervous system
When activity increases, it signals you to stop
eating
When activity decreases, it signals you to eat
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Influences of Satiety
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Influences of Satiety
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WHAT IS A HEALTHY BODY
WEIGHT? Based on how you feel, weight history, fat
distribution, family history of obesity-
related disease, current health status, andlifestyle
Current height/weight standards only
provide guides
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BODY MASS INDEX (BMI)
The preferred weight-for-height standard
Calculation:
Body wt (in kg) OR Body wt (in lbs) x 703.1
[Ht (in m)]2 [Ht (in inches)]2
Health risks increase when BMI is > 25
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ESTIMATION OF HEALTHY
WEIGHTFor men:106 pounds for the first 5 feet
add 6 pounds per each inch over five feet
A man who is 510 should weigh 166 lbs.
For women:
100 pounds for the first 5 feet
add 5 pounds per each inch over five feet
A women who is 510 should weigh 150 lbs.
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OBESITY
Excessive amount of body fat
Women with > 30-35% body fat
Men with > 25% body fat Increased risk for health problems
Are usually overweight
Measurements using calipers
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ESTIMATION OF BODY FAT
Underwater weighing(Fig. 13-5)
Most accurate
Fat is less dense than
lean tissue Fat floats
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ESTIMATION OF BODY FAT
Bioelectrical impedanceLow-energy current to the body that measures
the resistance of electrical flow
Fat is resistant to electrical flow; the more theresistance, the more body fat you have
X-ray photon absorptiometry
An X-ray body scan that allows for thedetermination of body fat
Infrared light
Assess the interaction of fat and protein in the
arm muscle
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BODY FAT DISTRIBUTION
Upper-body (android) obesity--Apple shape
Associated with more heart disease, HTN,
Type II Diabetes
Abdominal fat is released right into the liver
Fat affects livers ability to clear insulin and
lipoprotein
Encouraged by testosterone and excessive
alcohol intake
Defined as waist to hip ratio of >1.0 in men
and >0.8 in women
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BODY FAT DISTRIBUTION
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BODY FAT DISTRIBUTION
Lower-body (gynecoid) obesity--Pear shape
Encouraged by estrogen and progesterone
After menopause, upper-body obesity
appears
Less health risk than upper-body obesity
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OVERWEIGHT AND OBESITY
Underweight = BMI < 18.5
Healthy weight = BMI 18.5-24.9
Overweight = BMI 25-29.9
Obese = BMI 30-39.9
Severely obese = BMI >40
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JUVENILE-ONSET OBESITY
Develops in infancy or childhood
Increase in the number of adipose
cells
Adipose cells have long life span and
need to store fat Makes it difficult to loose the fat
(weight loss)
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ADULT-ONSET OBESITY
Develops in adulthood
Fewer (number of) adipose cells
These adipose cells are larger (stores excess
amount of fat)
If weight gain continues, the number of
adipose cells can increase
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CAUSES OF OBESITY
Nature debate
Identical twins raised apart have similar
weights Genetics account for ~40% of weight
differences
Genes affect metabolic rate, fuel use, brainchemistry
Thrifty metabolism gene allows for more fat
storage to protect against famine
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CAUSES OF OBESITY
Nurture debate
Environmental factors influence weight
Learned eating habits
Activity factor (or lack of)
Poverty and obesity
Female obesity is rooted in childhood
obesity
Male obesity appears after age 30
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NATURE AND NURTURE
Obesity is nurture allowing nature to
express itself
Location of fat is influenced by genetics
A child with no obese parents has a 10%
chance of becoming obese
A child with 1 obese parent has a 40%
chance
A child with 2 obese parents has a 80%
chance
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NATURE VS. NURTURE
Those at risk for obesity will face a lifelong
struggle with weight
Gene does not control destiny
Increased physical activity, moderate intake
can promote healthy weight
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WHY DIETS DONT WORK
Obesity is a chronic disease
Treatment requires long-term lifestyle changes
Dieters are misdirected
More concerned about weight loss than healthy
lifestyle
Unrealistic weight expectations
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WHY DIETS DONT WORK
Body defends itself against weight loss
Thyroid hormone concentrations (BMR)
drop during weight loss and make it moredifficult to lose weight
Activity of lipoprotein lipase increases
making it more efficient at taking up fat forstorage
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WHY DIETS DONT WORK
Weight cycling (yo-yo dieting)
Typically weight loss is not maintained
Weight lost consists of fat and lean tissue
Weight gained after weight loss is primarily
adipose tissue
Weight gained is usually more than weight
lost
Associated with upper body fat deposition
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WHY DIETS DONT WORKWeight gain in adulthood
Weight gain is common from ages 25-44
BMR decreases with age
Inactive lifestyle
Changes in body composition
Fluid is usually the first weight lost Loss in lean body tissue means lowering the
BMR
Very little fat is lost during weight loss
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LIFESTYLE VS. WEIGHT LOSS
Prevention of obesity is easier than curing
Balance energy in(take) with energy
out(put)
Focus on improving food habits
Focus on increase physical activities
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WHAT IT TAKES TO LOSE A
POUND Body fat contains 3500 kcal per pound
Fat storage (body fat plus supporting lean
tissues) contains 2700 kcal per pound
Must have an energy deficit of 2700-3500
kcal to lose a pound per week
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DO THE MATH
To lose one pound, you must create a deficit of 2700-3500
kcal
So to lose a pound in 1 week (7 days), try cutting back on
your kcal intake and increase physical activity so that youcreate a deficit of 400-500 kcal per day
- 500 kcal x 7 days = - 3500 kcal = 1 pound of weight loss
day week in 1 week
SOUND WEIGHT LOSS
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SOUND WEIGHT LOSS
PROGRAM
Slow & steady weight loss
Adapted to individuals habits and tastes
Contains enough kcal to minimize hunger andfatigue
Contains common foods
Fit into any social situation See a physician before starting
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CUTTING BACK
Control calorie intake by being aware of
kcal and fat content of foods
Fat Free does not mean Calories Free(or All You Can Eat)
Read food labels
Estimate kcal using the exchange system
Keep a food diary
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REGULAR PHYSICAL
ACTIVITY Fat use is enhanced with regular physical
activity
Increases energy expenditure
Duration and regularity are important
Make it a part of a daily routine
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BEHAVIOR MODIFICATION
Modify problem (eating) behaviors
Chain-breaking
Stimulus control
Cognitive restructuring
Contingency management
Self-monitoring
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CHAIN-BREAKING
Breaking the link between two behaviors
These links can lead to excessive intake
Snacking while watching T.V.
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STIMULUS CONTROL
Alternating the environment to minimize
the stimuli for eating
Puts you in charge of temptations
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COGNITIVE RESTRUCTURING
Changing your frame of mind regarding
eating
Replace eating due to stress with walking
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CONTINGENCY
MANAGEMENT Forming a plan of action in response to a
situation
Rehearse in advance appropriate responsesto pressure of eating at parties
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SELF-MONITORING
Tracking foods eaten and conditions
affecting eating
Helps you understand your eating habits
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WEIGHT MAINTENANCE Prevent relapse
Occasional lapse is fine, but take charge
immediately
Continue to practice newly learned behavior Requires motivation, movement, and
monitoring
Have social supportEncouragement from friends/ family/
professionals
DIETING CAN BE
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DIETING CAN BE
HAZARDOUS TO YOUR
HEALTH
Weight regained consists of a higher
percentage of body fat than before Less healthy than before dieting
Weight loss diet should not be
considered unless you are committed
and motivated
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Diet Drugs
Amphetamine (Phenteramine)
Prolongs the activity of epinephrine and
norepinephrine in the brainDecreases appetite
Not recommended for long term use
Sibutramine (Meridia)Enhances norepinephrine and serotonin activity
Decreases appetite(eat less)
Not recommended for people with HTN
GASTROPLASTY STOMACH
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GASTROPLASTY - STOMACH
STAPLING
Common surgical procedure for treatingsevere obesity
Reduces the stomach size (from 4 cups) to
half a shot glass size (1 oz) Overeating will result in rapid vomiting
Smaller stomach promotes satiety earlier
75% will lose ~50% of excess body weight
Costly
Dumping syndrome
GASTROPLASTY
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GASTROPLASTY
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UNDERWEIGHT IS ALSO A
PROBLEM 15-25% below healthy weight or BMI of
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TREATMENT FOR
UNDERWEIGHT Intake of energy-dense foods (energy input)
Encourage meals and snacks
Reduce activity (energy output)
To gain a pound you need a total excess
intake of 2700-3500 kcal